David P. Wilson

发表

Mathematics is applied by everyone except applied mathematicians

David P. Wilson, 2009, Appl. Math. Lett..

Experience with Software Quality / Usability Metrics

David P. Wilson, Judy Lau, David Wilson, 1998 .

Understanding the role of thermal fluctuations in DNA looping

Noel C. Perkins, Sachin Goyal, Jens-Christian Meiners, 2007, SPIE International Symposium on Fluctuations and Noise.

Ca2+ Activation of Smooth Muscle Contraction

David P. Wilson, C. Sutherland, M. Walsh, 2002, The Journal of Biological Chemistry.

Calcium desensitisation in late polymicrobial sepsis is associated with loss of vasopressor sensitivity in a murine model

David P. Wilson, J. Beltrame, R. Young, 2015, Intensive Care Medicine Experimental.

Integrin-linked kinase is responsible for Ca2+-independent myosin diphosphorylation and contraction of vascular smooth muscle.

David P. Wilson, C. Sutherland, M. Walsh, 2005, The Biochemical journal.

Phosphorylation of the protein phosphatase type 1 inhibitor protein CPI-17 by protein kinase C.

David P. Wilson, C. Sutherland, M. Walsh, 2007, Methods in molecular biology.

Integrin-linked kinase is responsible for Ca 2 +-independent myosin diphosphorylation and contraction of vascular smooth muscle

David P. Wilson, C. Sutherland, M. Walsh, 2005 .

Sex differences in vascular reactivity of coronary artery bypass graft conduits

David P. Wilson, J. Beltrame, Victor Lamin, 2019, Heart and Vessels.

Bradykinin receptor antagonists attenuate neointimal proliferation

David P. Wilson, L. Yau, J. Werner, 2001 .

Heteromultimeric TRPC6-TRPC7 Channels Contribute to Arginine Vasopressin-Induced Cation Current of A7r5 Vascular Smooth Muscle Cells

David P. Wilson, D. Welsh, Emma J. Walsh, 2006, Circulation research.

Phosphorylation of the myosin phosphatase target subunit by integrin-linked kinase.

David P. Wilson, T. Haystead, D. Hartshorne, 2002, The Biochemical journal.

Mechanisms Responsible for Serotonin Vascular Reactivity Sex Differences in the Internal Mammary Artery

David P. Wilson, J. Beltrame, Victor Lamin, 2018, Journal of the American Heart Association.

Cellular Physiology of Angiotensin II Receptors in Vascular Smooth Muscle Cells

David P. Wilson, L. Yau, P. Zahradka, 1998 .

EVIDENCE FOR THE INVOLVEMENT OF CALMODULIN THAT IS BOUND TO THE TRITON-INSOLUBLE FRACTION EVEN IN THE ABSENCE OF Ca 2 *

David P. Wilson, C. Sutherland, M. Walsh, 2002 .

Potential mechanisms of the acute coronary syndrome presentation in patients with the coronary slow flow phenomenon - insight from a plasma proteomic approach.

David P. Wilson, P. Hoffmann, J. Beltrame, 2012, International journal of cardiology.

Endothelial Denudation of Isolated Human Internal Mammary ArterySegments

David P. Wilson, J. Beltrame, Victor Lamin, 2015 .

Thromboxane A2-induced contraction of rat caudal arterial smooth muscle involves activation of Ca2+ entry and Ca2+ sensitization: Rho-associated kinase-mediated phosphorylation of MYPT1 at Thr-855, but not Thr-697.

David P. Wilson, C. Sutherland, M. Walsh, 2005, The Biochemical journal.

Characterization of tightly associated smooth muscle myosin-myosin light-chain kinase-calmodulin complexes.

David P. Wilson, F. Hong, P. Brewer, 2009, Journal of molecular biology.

Activation of MMP-2 in response to vascular injury is mediated by phosphatidylinositol 3-kinase-dependent expression of MT1-MMP.

Gregory E. J. Harding, David P. Wilson, P. Zahradka, 2004, American journal of physiology. Heart and circulatory physiology.

Activation of Smooth Muscle Myosin Light Chain Kinase by Calmodulin

David P. Wilson, Jonathan P. Davis, J. Johnson, 2002, The Journal of Biological Chemistry.

Identification of the linker histone H1 as a protein kinase Cε-binding protein in vascular smooth muscle

David P. Wilson, Mingcai Zhao, C. Sutherland, 2004 .

Inhibition of zipper-interacting protein kinase function in smooth muscle by a myosin light chain kinase pseudosubstrate peptide.

David P. Wilson, E. Ihara, M. Walsh, 2007, American journal of physiology. Cell physiology.

Structural and stoichiometric determinants of Ca2+ release-activated Ca2+ (CRAC) channel Ca2+-dependent inactivation.

David P. Wilson, G. Barritt, G. Rychkov, 2014, Biochimica et biophysica acta.

Glu¹⁰⁶ in the Orai1 pore contributes to fast Ca²⁺-dependent inactivation and pH dependence of Ca²⁺ release-activated Ca²⁺ (CRAC) current.

David P. Wilson, G. Rychkov, N. Scrimgeour, 2012, The Biochemical journal.

Heterogeneity of L- and T-Channels in the Vasculature: Rationale for the Efficacy of Combined L- and T-Blockade

David P. Wilson, D. Saint, J. Beltrame, 2009, Hypertension.

The role of RhoA and Rho-associated kinase in vascular smooth muscle contraction

David P. Wilson, M. Walsh, K. Swärd, 2003, Current hypertension reports.

Heteromultimeric TRPC 6-TRPC 7 Channels Contribute to Arginine Vasopressin-Induced Cation Current of A 7 r 5 Vascular Smooth Muscle Cells

David P. Wilson, D. Welsh, Emma J. Walsh, 2006 .

Attenuated platelet aggregation in patients with septic shock is independent from the activity state of myosin light chain phosphorylation or a reduction in Rho kinase-dependent inhibition of myosin light chain phosphatase

David P. Wilson, A. Deane, S. O'Connor, 2015, Intensive Care Medicine Experimental.

Identification of a novel interaction between the Ca(2+)-binding protein S100A11 and the Ca(2+)- and phospholipid-binding protein annexin A6.

David P. Wilson, N. Chang, R. Winkfein, 2007, American journal of physiology. Cell physiology.