Richard M. Page

发表

Richard M. Page, 1999 .

Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease

Richard M. Page, H. Kretzschmar, G. Mitteregger, 2010, Nature Neuroscience.

Intraneuronal Aβ, non-amyloid aggregates and neurodegeneration in a Drosophila model of Alzheimer’s disease

Richard M. Page, D. Gubb, D. Lomas, 2005, Neuroscience.

Constitutive α- and β-secretase cleavages of the amyloid precursor protein are partially coupled in neurons, but not in frequently used cell lines

Richard M. Page, E. Kremmer, P. Dempsey, 2013, Neurobiology of Disease.

Chronic γ-secretase inhibition reduces amyloid plaque-associated instability of pre- and postsynaptic structures

Richard M. Page, T Bonhoeffer, T. Bonhoeffer, 2013, Molecular Psychiatry.

Fenton chemistry and oxidative stress mediate the toxicity of the β-amyloid peptide in a Drosophila model of Alzheimer’s disease

Robert Hider, Huw Lewis, Damian C Crowther, 2009, The European journal of neuroscience.

Attenuated Aβ42 Responses to Low Potency γ-Secretase Modulators Can Be Overcome for Many Pathogenic Presenilin Mutants by Second-generation Compounds*

Richard M. Page, C. Haass, K. Baumann, 2011, The Journal of Biological Chemistry.

β-Amyloid Precursor Protein Mutants Respond to γ-Secretase Modulators*♦

Richard M. Page, C. Haass, R. Page, 2010, The Journal of Biological Chemistry.

Generation of Aβ38 and Aβ42 Is Independently and Differentially Affected by Familial Alzheimer Disease-associated Presenilin Mutations and γ-Secretase Modulation*

Richard M. Page, A. Flohr, C. Haass, 2008, Journal of Biological Chemistry.