Paul G A Volders

Myokit: A simple interface to cardiac cellular electrophysiology.

Michael Clerx, Pieter Collins, Enno de Lange, 2016, Progress in biophysics and molecular biology.

Beat-to-Beat variability of repolarization determines proarrhythmic outcome in dogs susceptible to drug-induced torsades de pointes.

Paul G A Volders, Marc A Vos, Jørgen Matz, 2006, Journal of the American College of Cardiology.

Diastolic Spontaneous Calcium Release From the Sarcoplasmic Reticulum Increases Beat-to-Beat Variability of Repolarization in Canine Ventricular Myocytes After &bgr;-Adrenergic Stimulation

Najah Abi-Gerges, Paul G A Volders, Jordi Heijman, 2013, Circulation research.

Increased Short-Term Variability of Repolarization Predicts d-Sotalol–Induced Torsades de Pointes in Dogs

Milan Stengl, Paul G A Volders, P. Volders, 2004, Circulation.

Local control of β-adrenergic stimulation: Effects on ventricular myocyte electrophysiology and Ca(2+)-transient.

Yoram Rudy, Paul G A Volders, Jordi Heijman, 2011, Journal of molecular and cellular cardiology.

Electromechanical window negativity in genotyped long-QT syndrome patients: relation to arrhythmia risk.

Michael J Ackerman, Thor Edvardsen, Paul G A Volders, 2015, European heart journal.

In vivo mechanisms precipitating torsades de pointes in a canine model of drug-induced long-QT1 syndrome.

Paul G A Volders, P. Volders, Hua-rong Lu, 2007, Cardiovascular research.

I(Ks) restricts excessive beat-to-beat variability of repolarization during beta-adrenergic receptor stimulation.

Jean-Pierre Valentin, Najah Abi-Gerges, Paul G A Volders, 2010, Journal of molecular and cellular cardiology.