Mohamed Chahine

发表

A wireless system for combined heart optogenetics and electrocardiography recording

Benoit Gosselin, Leonard L. Gagnon, Gabriel Gagnon-Turcotte, 2017, 2017 IEEE International Symposium on Circuits and Systems (ISCAS).

Assessment Of Noise Impacts At Airports

Mutasem El-Fadel, Toufic Mezher, Mohamed Chahine, 2002 .

SCN5A Polymorphism Restores Trafficking of a Brugada Syndrome Mutation on a Separate Gene

Mohamed Chahine, Sandro Sorrentino, Steven Poelzing, 2006, Circulation.

A novel mutation in SCN5A, delQKP 1507-1509, causing long QT syndrome: role of Q1507 residue in sodium channel inactivation.

Mohamed Chahine, P. Guicheney, V. Fressart, 2003, Journal of molecular and cellular cardiology.

A distinct de novo expression of Nav1.5 sodium channels in human atrial fibroblasts differentiated into myofibroblasts

Mohamed Chahine, Olivier Thériault, N. Benamer, 2012, The Journal of physiology.

Pyrethroids Differentially Alter Voltage-Gated Sodium Channels from the Honeybee Central Olfactory Neurons

Mohamed Chahine, Y. Le Conte, M. Chahine, 2014, PloS one.

Role of auxiliary β1-, β2-, and β3-subunits and their interaction with Nav1.8 voltage-gated sodium channel

Mohamed Chahine, I. Kinghorn, K. Vijayaragavan, 2004 .

Biophysical characterisation of the persistent sodium current of the Nav1.6 neuronal sodium channel: a single-channel analysis

Mohamed Chahine, M. Chahine, P. Bois, 2010, Pflügers Archiv - European Journal of Physiology.

Gain-of-function mutation of Nav1.5 in atrial fibrillation enhances cellular excitability and lowers the threshold for action potential firing.

Robert Lemery, Mohamed Chahine, Hai Huang, 2009, Biochemical and biophysical research communications.

Cell membrane expression of cardiac sodium channel Na(v)1.5 is modulated by alpha-actinin-2 interaction.

Mohamed Chahine, Alan H Beggs, Hai Huang, 2010, Biochemistry.

Sodium channel mutations in paramyotonia congenita uncouple inactivation from activation

Ming Zhou, Mohamed Chahine, Richard Horn, 1994, Neuron.

Enzyme Domain Affects the Movement of the Voltage Sensor in Ascidian and Zebrafish Voltage-sensing Phosphatases*

Yasushi Okamura, Mohamed Chahine, Hirohide Iwasaki, 2008, Journal of Biological Chemistry.

Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.

Mohamed Chahine, Olivier Thériault, M. Boutjdir, 2017, The Canadian journal of cardiology.

Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C.

Mohamed Chahine, M. Boutjdir, K. Vijayaragavan, 2004, Journal of neurophysiology.

Biophysics, pathophysiology, and pharmacology of ion channel gating pores

Mohamed Chahine, Pascal Gosselin-Badaroudine, M. Chahine, 2014, Front. Pharmacol..

Expression and Intracellular Localization of an SCN5A Double Mutant R1232W/T1620M Implicated in Brugada Syndrome

Mohamed Chahine, M. Chahine, Said Acharfi, 2002, Circulation research.

New Insights into Cardiac and Brain Sodium Channels Modulation by Beta Blockers

Mohamed Chahine, M. Chahine, 2011, Front. Pharmacol..

Biophysical properties of human Na v1.7 splice variants and their regulation by protein kinase A.

Mohamed Chahine, J. Krupp, L. Dahllund, 2008, Journal of neurophysiology.

Gating pore currents, a new pathological mechanism underlying cardiac arrhythmias associated with dilated cardiomyopathy

Mohamed Chahine, Pascal Gosselin-Badaroudine, M. Chahine, 2015, Channels.