J A Frangos

发表

Shear sensitivity in animal cell culture.

K J Gooch, J A Frangos, J. Frangos, 1993, Current opinion in biotechnology.

Femoral vein ligation increases bone mass in the hindlimb suspended rat.

J A Frangos, J. Frangos, A P Bergula, 1999, Bone.

Fluid shear stress increases membrane fluidity in endothelial cells: a study with DCVJ fluorescence.

J A Frangos, J. Frangos, M. Haidekker, 2000, American journal of physiology. Heart and circulatory physiology.

Modulation of GTPase activity of G proteins by fluid shear stress and phospholipid composition.

J A Frangos, J. Frangos, J. Nolan, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Analysis of temporal shear stress gradients during the onset phase of flow over a backward-facing step.

J A Frangos, J. Frangos, M. Haidekker, 2001, Journal of biomechanical engineering.

Effect of flow on prostaglandin E2 and inositol trisphosphate levels in osteoblasts.

J A Frangos, J. Frangos, K. M. Reich, 1991, The American journal of physiology.

Role of G proteins in shear stress-mediated nitric oxide production by endothelial cells.

J A Frangos, H. Jo, M. Kuchan, 1994, The American journal of physiology.

Pressure gradients and transport in the murine femur upon hindlimb suspension.

J A Frangos, J. Frangos, H. Stevens, 2006, Bone.

Review: Bone tissue engineering: The role of interstitial fluid flow

J A Frangos, J. Frangos, M V Hillsley, 1994, Biotechnology and bioengineering.

Shear stress increases endothelial platelet-derived growth factor mRNA levels.

J A Frangos, J. Frangos, H. Hsieh, 1991, The American journal of physiology.

Steady shear and step changes in shear stimulate endothelium via independent mechanisms--superposition of transient and sustained nitric oxide production.

J A Frangos, J. Frangos, T. Y. Huang, 1996, Biochemical and biophysical research communications.

Exogenous, basal, and flow‐induced nitric oxide production and endothelial cell proliferation

K J Gooch, J A Frangos, J. Frangos, 1997, Journal of cellular physiology.

Steady and Transient Fluid Shear Stress Stimulate NO Release in Osteoblasts Through Distinct Biochemical Pathways

J A Frangos, J. Frangos, T. McAllister, 1999, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Relative blood damage in the three phases of a prosthetic heart valve flow cycle.

G Rosenberg, D B Geselowitz, J A Frangos, 1993, ASAIO journal.

A new mock circulatory loop and its application to the study of chemical additive and aortic pressure effects on hemolysis in the Penn State electric ventricular assist device.

D B Geselowitz, J A Frangos, D. Geselowitz, 1994, Artificial organs.

Shear stress induced stimulation of mammalian cell metabolism

L V McIntire, J A Frangos, L. McIntire, 1988, Biotechnology and bioengineering.

Shear stress increases hydraulic conductivity of cultured endothelial monolayers.

J A Frangos, J. Tarbell, J. Frangos, 1995, The American journal of physiology.

Flow effects on prostacyclin production by cultured human endothelial cells.

L V McIntire, J A Frangos, L. McIntire, 1985, Science.

Temporal gradient in shear but not steady shear stress induces PDGF-A and MCP-1 expression in endothelial cells: role of NO, NF kappa B, and egr-1.

J A Frangos, J. Frangos, X. Bao, 1999, Arteriosclerosis, thrombosis, and vascular biology.

New fluorescent probes for the measurement of cell membrane viscosity.

J A Frangos, J. Frangos, T. Ling, 2001, Chemistry & biology.

Fluid flow stimulates rapid and continuous release of nitric oxide in osteoblasts.

J A Frangos, J. Frangos, T. McAllister, 1996, The American journal of physiology.

Activation of G proteins mediates flow-induced prostaglandin E2 production in osteoblasts.

J A Frangos, S Gudi, K M Reich, 1997, Endocrinology.