Frederick Y. Wu

发表

Exposure Dose Control Techniques For Excimer Laser Lithography

David H. Tracy, Frederick Y. Wu, 1988, Advanced Lithography.

Cell Cycle Arrest by Kaposi's Sarcoma-Associated Herpesvirus Replication-Associated Protein Is Mediated at both the Transcriptional and Posttranslational Levels by Binding to CCAAT/Enhancer-Binding Protein α and p21CIP-1

Frederick Y. Wu, M. Fujimuro, M. Lane, 2003, Journal of Virology.

Cell Cycle Arrest by Kaposi’s Sarcoma-Associated Herpesvirus Replication-Associated Protein Is Mediated at both the Transcriptional and Posttranslational Levels by Binding to CCAAT/Enhancer-Binding Protein and p21

Frederick Y. Wu, M. Fujimuro, M. Lane, 2003 .

CCAAT/Enhancer-Binding Protein- (cid:2) Is Induced during the Early Stages of Kaposi’s Sarcoma-Associated Herpesvirus (KSHV) Lytic Cycle Reactivation and Together with the KSHV Replication and Transcription Activator (RTA) Cooperatively Stimulates the Viral RTA, MTA, and PAN Promoters

Frederick Y. Wu, G. Hayward, S. Wang, 2003 .

Origin-Independent Assembly of Kaposi's Sarcoma-Associated Herpesvirus DNA Replication Compartments in Transient Cotransfection Assays and Association with the ORF-K8 Protein and Cellular PML

Won-Jong Jang, Frederick Y. Wu, Jinsong Xiao, 2001, Journal of Virology.

Convergence of p53 and Transforming Growth Factor β (TGFβ) Signaling on Activating Expression of the Tumor Suppressor Gene maspin in Mammary Epithelial Cells*

Frederick Y. Wu, C. Arteaga, D. Friedman, 2007, Journal of Biological Chemistry.

Transforming growth factor {beta} (TGF-{beta})-Smad target gene protein tyrosine phosphatase receptor type kappa is required for TGF-{beta} function.

Frederick Y. Wu, C. Arteaga, I. Shin, 2005, Molecular and cellular biology.

Refractory lympho-epithelial carcinoma of the nasopharynx: a case report illustrating a protracted clinical course

Frederick Y. Wu, A. Cmelak, E. Yang, 2009, Head & neck oncology.

HER2 kinase domain mutation results in constitutive phosphorylation and activation of HER2 and EGFR and resistance to EGFR tyrosine kinase inhibitors.

Carlos L Arteaga, Frederick Y. Wu, A. Gazdar, 2006, Cancer cell.

Reduction of cytosolic p27(Kip1) inhibits cancer cell motility, survival, and tumorigenicity.

Frederick Y. Wu, J. Baselga, F. Rojo, 2006, Cancer research.

Phosphorylation of p27Kip1 at Thr-157 Interferes with Its Association with Importin α during G1 and Prevents Nuclear Re-entry*

Frederick Y. Wu, C. Arteaga, I. Shin, 2005, Journal of Biological Chemistry.

Regulation of Expression of the Epstein-Barr Virus BamHI-A Rightward Transcripts

Frederick Y. Wu, Jian Huang, L. Hutt-Fletcher, 2005, Journal of Virology.

CCAAT/Enhancer Binding Protein α Interacts with ZTA and Mediates ZTA-Induced p21CIP-1 Accumulation and G1 Cell Cycle Arrest during the Epstein-Barr Virus Lytic Cycle

Frederick Y. Wu, M. Fujimuro, G. Hayward, 2003, Journal of Virology.

Role of CCAAT/Enhancer-Binding Protein Alpha (C/EBPα) in Activation of the Kaposi's Sarcoma-Associated Herpesvirus (KSHV) Lytic-Cycle Replication-Associated Protein (RAP) Promoter in Cooperation with the KSHV Replication and Transcription Activator (RTA) and RAP

Frederick Y. Wu, M. Fujimuro, G. Hayward, 2003, Journal of Virology.

The Khanka Block, NE China, and its significance for the evolution of the Central Asian Orogenic Belt and continental accretion

Frederick Y. Wu, S. Wilde, Guochun Zhao, 2010 .

Lytic replication-associated protein (RAP) encoded by Kaposi sarcoma-associated herpesvirus causes p21CIP-1-mediated G1 cell cycle arrest through CCAAT/enhancer-binding protein-α

Frederick Y. Wu, M. Fujimuro, M. Lane, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Phospholipase Cγ1 is a physiological guanine nucleotide exchange factor for the nuclear GTPase PIKE

Frederick Y. Wu, S. Snyder, P. Suh, 2002, Nature.

Early Activation of the Kaposi's Sarcoma-Associated Herpesvirus RTA, RAP, and MTA Promoters by the Tetradecanoyl Phorbol Acetate-Induced AP1 Pathway

Frederick Y. Wu, G. Hayward, Q. Zheng, 2004, Journal of Virology.

A novel viral mechanism for dysregulation of β-catenin in Kaposi's sarcoma–associated herpesvirus latency

Frederick Y. Wu, M. Fujimuro, G. Hayward, 2003, Nature Medicine.

Spindle Cell Conversion by Kaposi's Sarcoma-Associated Herpesvirus: Formation of Colonies and Plaques with Mixed Lytic and Latent Gene Expression in Infected Primary Dermal Microvascular Endothelial Cell Cultures

Frederick Y. Wu, G. Hayward, R. Ambinder, 2001, Journal of Virology.

PIKE A Nuclear GTPase that Enhances PI3Kinase Activity and Is Regulated by Protein 4.1N

Seth Blackshaw, Frederick Y. Wu, S. Snyder, 2000, Cell.

Interaction with the Epstein-Barr Virus Helicase Targets Zta to DNA Replication Compartments

Frederick Y. Wu, G. Liao, S. Hayward, 2001, Journal of Virology.

Contribution of C/EBP Proteins to Epstein-Barr Virus Lytic Gene Expression and Replication in Epithelial Cells

Frederick Y. Wu, G. Hayward, Jian Huang, 2006, Journal of Virology.

Cell cycle arrest by Kaposi's sarcoma-associated herpesvirus replication-associated protein is mediated at both the transcriptional and posttranslational levels by binding to CCAAT/enhancer-binding protein alpha and p21(CIP-1).

Frederick Y. Wu, M. Fujimuro, M. Lane, 2003, Journal of virology.

HER2/Neu (ErbB2) signaling to Rac1-Pak1 is temporally and spatially modulated by transforming growth factor beta.

Frederick Y. Wu, C. Arteaga, I. Shin, 2006, Cancer research.

HER 2 / Neu ( ErbB 2 ) Signaling to Rac 1-Pak 1 Is Temporally and Spatially Modulated by Transforming Growth Factor B Shizhen

Frederick Y. Wu, C. Arteaga, I. Shin, 2006 .

Transforming Growth Factor (cid:2) (TGF- (cid:2) )-Smad Target Gene Protein Tyrosine Phosphatase Receptor Type Kappa Is Required for TGF- (cid:2) Function†

Frederick Y. Wu, C. Arteaga, I. Shin, 2022 .

Key geochronology of Mesozoic deformation in the Eastern Block of the North China Craton and its con

Frederick Y. Wu, S. Li, Zhong Han, 2004 .

Identification of Human Male Germ Cell-associated Kinase, a Kinase Transcriptionally Activated by Androgen in Prostate Cancer Cells*

Frederick Y. Wu, H. Kung, D. Robinson, 2002, The Journal of Biological Chemistry.