Jamie L. Marshall

发表

Sensitive spatial genome wide expression profiling at cellular resolution

Evan Z. Macosko, Fei Chen, Evan Murray, 2020, bioRxiv.

Sarcospan reduces dystrophic pathology: stabilization of the utrophin–glycoprotein complex

Jamie L. Marshall, Angela K. Peter, R. Crosbie, 2008, The Journal of cell biology.

High Throughput Screening for Compounds That Alter Muscle Cell Glycosylation Identifies New Role for N-Glycans in Regulating Sarcolemmal Protein Abundance and Laminin Binding

Jamie L. Marshall, S. Nelson, L. Wells, 2012, The Journal of Biological Chemistry.

Differentiation-related glycan epitopes identify discrete domains of the muscle glycocalyx.

Jamie L. Marshall, K. Moremen, L. Baum, 2016, Glycobiology.

Stabilization of the cardiac sarcolemma by sarcospan rescues DMD-associated cardiomyopathy.

Jamie L. Marshall, J. Chamberlain, A. Beedle, 2019, JCI insight.

Sarcospan Regulates Cardiac Isoproterenol Response and Prevents Duchenne Muscular Dystrophy–Associated Cardiomyopathy

Jamie L. Marshall, Michelle S. Parvatiyar, R. H. Crosbie-Watson, 2015, Journal of the American Heart Association.

Jagged 1 Rescues the Duchenne Muscular Dystrophy Phenotype

Jamie L. Marshall, K. Lindblad-Toh, E. Karlsson, 2015, Cell.

MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms.

Jamie L. Marshall, L. Kunkel, M. Gasperini, 2014, The Journal of clinical investigation.

The potential of sarcospan in adhesion complex replacement therapeutics for the treatment of muscular dystrophy

Jamie L. Marshall, L. Baum, B. J. McMorran, 2013, The FEBS journal.

Works Title Sarcospan : a small protein with large potential for Duchenne muscular dystrophy

Jamie L. Marshall, R. Crosbie-Watson, R. Crosbie‐Watson, 2013 .

Sarcospan: a small protein with large potential for Duchenne muscular dystrophy

Jamie L. Marshall, R. Crosbie-Watson, R. Crosbie‐Watson, 2013, Skeletal Muscle.

High levels of sarcospan are well tolerated and act as a sarcolemmal stabilizer to address skeletal muscle and pulmonary dysfunction in DMD

Jamie L. Marshall, Thien-Minh Nguyen, M. Spencer, 2016, Human molecular genetics.

Dystrophin and utrophin expression require sarcospan: loss of α7 integrin exacerbates a newly discovered muscle phenotype in sarcospan-null mice.

Jamie L. Marshall, D. Burkin, E. Chou, 2012, Human Molecular Genetics.

Sarcospan-dependent Akt activation is required for utrophin expression and muscle regeneration

Jamie L. Marshall, J. Holmberg, Angela K. Peter, 2012, The Journal of cell biology.

Loss of sarcospan exacerbates pathology in mdx mice, but does not affect utrophin amelioration of disease.

Jamie L. Marshall, E. Gibbs, R. Crosbie, 2021, Human molecular genetics.

Sarcospan integration into laminin-binding adhesion complexes that ameliorate muscular dystrophy requires utrophin and α7 integrin.

Jamie L. Marshall, D. Burkin, J. Holmberg, 2015, Human molecular genetics.

Reversal of neurobehavioral social deficits in dystrophic mice using inhibitors of phosphodiesterases PDE5A and PDE9A

Jamie L. Marshall, J. Spinazzola, J. Harms, 2016, Translational Psychiatry.

Stabilization of the cardiac sarcolemma by sarcospan rescues DMD-associated cardiomyopathy.

Aaron M. Beedle, Jamie L. Marshall, J. Chamberlain, 2019, JCI insight.

The potential of sarcospan in adhesion complex replacement therapeutics for the treatment of muscular dystrophy

Jamie L. Marshall, L. Baum, B. J. McMorran, 2013, The FEBS journal.

MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms.

Jamie L. Marshall, L. Kunkel, M. Gasperini, 2014, The Journal of clinical investigation.

Jagged 1 Rescues the Duchenne Muscular Dystrophy Phenotype

Jamie L. Marshall, K. Lindblad-Toh, E. Karlsson, 2015, Cell.