P. Gubellini

发表

Dopaminergic control of synaptic plasticity in the dorsal striatum

P. Calabresi, B. Picconi, D. Centonze, 2001, The European journal of neuroscience.

Unilateral dopamine denervation blocks corticostriatal LTP.

P. Calabresi, B. Picconi, D. Centonze, 1999, Journal of neurophysiology.

Dopamine and cAMP-Regulated Phosphoprotein 32 kDa Controls Both Striatal Long-Term Depression and Long-Term Potentiation, Opposing Forms of Synaptic Plasticity

P. Greengard, P. Calabresi, B. Picconi, 2000, The Journal of Neuroscience.

Glutamate-Triggered Events Inducing Corticostriatal Long-Term Depression

P. Calabresi, D. Centonze, P. Gubellini, 1999, The Journal of Neuroscience.

Activation of M1-like muscarinic receptors is required for the induction of corticostriatal LTP

P. Calabresi, D. Centonze, P. Gubellini, 1999, Neuropharmacology.

Blockade of M2‐like muscarinic receptors enhances long‐term potentiation at corticostriatal synapses

P. Calabresi, D. Centonze, P. Gubellini, 1998 .

Selective involvement of mGlu1 receptors in corticostriatal LTD

P. Calabresi, D. Centonze, P. Gubellini, 2001, Neuropharmacology.

A Critical Role of the Nitric Oxide/cGMP Pathway in Corticostriatal Long-Term Depression

P. Calabresi, D. Centonze, P. Gubellini, 1999, The Journal of Neuroscience.

Distinct Roles of D1 and D5 Dopamine Receptors in Motor Activity and Striatal Synaptic Plasticity

P. Calabresi, D. Centonze, P. Gubellini, 2003, The Journal of Neuroscience.

Synaptic transmission in the striatum: from plasticity to neurodegeneration

G Bernardi, P. Calabresi, D. Centonze, 2000, Progress in Neurobiology.

Postnatal Tshz3 Deletion Drives Altered Corticostriatal Function and Autism Spectrum Disorder–like Behavior

P. Gubellini, B. Jacq, P. Salin, 2019, Biological Psychiatry.

Deep brain stimulation in neurological diseases and experimental models: From molecule to complex behavior

P. Gubellini, C. Baunez, P. Salin, 2009, Progress in Neurobiology.

Electrophysiological and behavioral evidence that modulation of metabotropic glutamate receptor 4 with a new agonist reverses experimental parkinsonism

P. Gubellini, M. Amalric, C. Beurrier, 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Targeted Tshz3 deletion in corticostriatal circuit components segregates core autistic behaviors

P. Gubellini, P. Salin, Dorian Chabbert, 2022, Translational Psychiatry.

Targeted Tshz3 deletion in corticostriatal circuit components segregates core autistic behaviors

P. Gubellini, P. Salin, Dorian Chabbert, 2022, Translational Psychiatry.

Synaptic scaling up in medium spiny neurons of aged BACHD mice: A slow-progression model of Huntington's disease

P. Gubellini, F. Petit, G. Bonvento, 2016, Neurobiology of Disease.

Ciliary Neurotrophic Factor Protects Striatal Neurons against Excitotoxicity by Enhancing Glial Glutamate Uptake

P. Gubellini, G. Bonvento, C. Beurrier, 2010, PloS one.

Ubiquitin-Mediated Stress Response in a Rat Model of Brain Transient Ischemia/Hypoxia

P. Gubellini, G. Bisso, S. Fortuna, 2004, Neurochemical Research.

Correction: Targeted Tshz3 deletion in corticostriatal circuit components segregates core autistic behaviors

P. Gubellini, P. Salin, Dorian Chabbert, 2022, Translational Psychiatry.

ã Federation of European Neuroscience Societies Activity- and age-dependent GABAergic synaptic plasticity in the developing rat hippocampus

Y. Ben-Ari, P. Gubellini, Y. Ben‐Ari, 2001 .

Endogenous Neurotrophins Are Required for the Induction of GABAergic Long-Term Potentiation in the Neonatal Rat Hippocampus

Y. Ben-Ari, P. Gubellini, J. Gaïarsa, 2005, The Journal of Neuroscience.

Metabotropic glutamate receptor subtype 4 selectively modulates both glutamate and GABA transmission in the striatum: implications for Parkinson’s disease treatment

P. Gubellini, A. Pisani, C. Melon, 2009, Journal of neurochemistry.

Synergy between l-DOPA and a novel positive allosteric modulator of metabotropic glutamate receptor 4: Implications for Parkinson's disease treatment and dyskinesia

P. Gubellini, L. Goff, C. Melon, 2013, Neuropharmacology.

Metabotropic glutamate receptors and striatal synaptic plasticity: implications for neurological diseases

Antonio Pisani, Paolo Calabresi, P. Calabresi, 2004, Progress in Neurobiology.

Dopamine, Acetylcholine and Nitric Oxide Systems Interact to Induce Corticostriatal Synaptic Plasticity

P. Calabresi, D. Centonze, P. Gubellini, 2003, Reviews in the neurosciences.

A Synaptic Mechanism Underlying the Behavioral Abnormalities Induced by Manganese Intoxication

Giuseppe Sancesario, Paolo Calabresi, P. Calabresi, 2001, Neurobiology of Disease.

Tissue plasminogen activator controls multiple forms of synaptic plasticity and memory

P. Calabresi, D. Centonze, P. Gubellini, 2000, The European journal of neuroscience.

Corticostriatal LTP requires combined mGluR1 and mGluR5 activation

P. Calabresi, D. Centonze, P. Gubellini, 2003, Neuropharmacology.

Metabotropic glutamate receptor 5 mediates the potentiation of N-methyl-D-aspartate responses in medium spiny striatal neurons

G Bernardi, P. Calabresi, B. Picconi, 2001, Neuroscience.

Inhibition of Mitochondrial Complex II Induces a Long-Term Potentiation of NMDA-Mediated Synaptic Excitation in the Striatum Requiring Endogenous Dopamine

P. Greengard, P. Calabresi, B. Picconi, 2001, The Journal of Neuroscience.

Acetylcholine-mediated modulation of striatal function

P. Calabresi, D. Centonze, P. Gubellini, 2000, Trends in Neurosciences.

Endogenous ACh enhances striatal NMDA‐responses via M1‐like muscarinic receptors and PKC activation

P. Calabresi, D. Centonze, P. Gubellini, 1998, The European journal of neuroscience.

Striatal spiny neurons and cholinergic interneurons express differential ionotropic glutamatergic responses and vulnerability: Implications for ischemia and Huntington's disease

P. Calabresi, D. Centonze, P. Gubellini, 1998, Annals of neurology.

TSHZ3 deletion causes an autism syndrome and defects in cortical projection neurons

P. Gubellini, B. Jacq, N. Šestan, 2016, Nature Genetics.

Downstream mechanisms triggered by mitochondrial dysfunction in the basal ganglia: from experimental models to neurodegenerative diseases.

P. Calabresi, B. Picconi, P. Gubellini, 2010, Biochimica et biophysica acta.

Effects of GPi and STN inactivation on physiological, motor, cognitive and motivational processes in animal models of Parkinson's disease.

Christelle Baunez, P. Gubellini, C. Baunez, 2010, Progress in brain research.

Molecular, Cellular and Electrophysiological Changes Triggered by High-Frequency Stimulation of the Subthalamic Nucleus in Animal Models of Parkinson’s Disease

P. Gubellini, P. Salin, 2009 .

Ciliary Neurotrophic Factor Activates Astrocytes, Redistributes Their Glutamate Transporters GLAST and GLT-1 to Raft Microdomains, and Improves Glutamate Handling In Vivo

Philippe Hantraye, Emmanuel Brouillet, Gilles Bonvento, 2006, The Journal of Neuroscience.

Experimental Parkinsonism Alters Endocannabinoid Degradation: Implications for Striatal Glutamatergic Transmission

P. Calabresi, B. Picconi, D. Centonze, 2002, The Journal of Neuroscience.

Targeted Tshz3 deletion in corticostriatal circuit components segregates core autistic behaviors

P. Gubellini, P. Salin, Dorian Chabbert, 2021, bioRxiv.

Postsynaptic autism spectrum disorder genes and synaptic dysfunction

P. Gubellini, P. Bonsi, L. Fasano, 2021, Neurobiology of Disease.

Genetic Models of Parkinson’s

P. Gubellini, P. Kachidian, 2020 .

Camk2a-Cre and Tshz3 Expression in Mouse Striatal Cholinergic Interneurons: Implications for Autism Spectrum Disorder

P. Gubellini, B. Habermann, Dorian Chabbert, 2021, Frontiers in Genetics.

Tissue plasminogen activator is required for corticostriatal long‐term potentiation

Antonio Pisani, Alessandro Martorana, Paolo Calabresi, 2002, The European journal of neuroscience.

Activation of dopamine D1‐like receptors excites LTS interneurons of the striatum

Enrico Bracci, Antonio Pisani, Paolo Calabresi, 2002, The European journal of neuroscience.

Impaired Excitatory Transmission in the Striatum of Rats Chronically Intoxicated with Manganese

P. Calabresi, D. Centonze, P. Gubellini, 2001, Experimental Neurology.

Neuronal vulnerability following inhibition of mitochondrial complex II: a possible ionic mechanism for Huntington's disease

P. Calabresi, B. Picconi, D. Centonze, 2004, Molecular and Cellular Neuroscience.

Progressive Parkinsonism by acute dysfunction of excitatory amino acid transporters in the rat substantia nigra

P. Gubellini, P. Salin, C. Melon, 2014, Neurobiology of Disease.

Role of glutamate transporters in corticostriatal synaptic transmission

P. Gubellini, G. Bonvento, L. Goff, 2009, Neuroscience.

Chronic high‐frequency stimulation of the subthalamic nucleus and L‐DOPA treatment in experimental parkinsonism: effects on motor behaviour and striatal glutamate transmission

P. Gubellini, P. Salin, A. Eusebio, 2006, The European journal of neuroscience.

Animal models of Parkinson's disease: An updated overview.

P. Gubellini, P. Kachidian, 2015, Revue neurologique.

Update in neurosciences Animal models of Parkinson ’ s disease : An updated overview Modèles animaux de la maladie de Parkinson : un panorama

P. Gubellini, P. Kachidian, 2015 .

Metabolic, synaptic and behavioral impact of 5‐week chronic deep brain stimulation in hemiparkinsonian rats

P. Gubellini, P. Salin, F. Durif, 2016, Journal of neurochemistry.

Construct Validity and Cross Validity of a Test Battery Modeling Autism Spectrum Disorder (ASD) in Mice

P. Gubellini, X. Caubit, P. Roubertoux, 2019, Behavior Genetics.

Construct Validity and Cross Validity of a Test Battery Modeling Autism Spectrum Disorder (ASD) in Mice

P. Gubellini, X. Caubit, P. Roubertoux, 2019, Behavior Genetics.

Metabotropic Glutamate Receptors and Parkinson’s Disease: Basic and Preclinical Neuroscience

P. Gubellini, Y. Smith, M. Amalric, 2017 .

Differential contribution of dopamine D2S and D2L receptors in the modulation of glutamate and GABA transmission in the striatum

P. Calabresi, D. Centonze, P. Gubellini, 2004, Neuroscience.

Induction of corticostriatal LTP by 3-nitropropionic acid requires the activation of mGluR1/PKC pathway

Paolo Calabresi, P. Calabresi, D. Centonze, 2004, Neuropharmacology.

Receptor Subtypes Involved in the Presynaptic and Postsynaptic Actions of Dopamine on Striatal Interneurons

Antonio Pisani, Alessandro Usiello, Paolo Calabresi, 2003, The Journal of Neuroscience.

Dopamine D2 Receptor-Mediated Inhibition of Dopaminergic Neurons in Mice Lacking D2L Receptors

Antonio Pisani, Alessandro Usiello, Paolo Calabresi, 2002, Neuropsychopharmacology.

Targeting striatal cholinergic interneurons in Parkinson’s disease: Focus on metabotropic glutamate receptors

P. Calabresi, D. Centonze, P. Gubellini, 2003, Neuropharmacology.

Editorial: Neuroprotection and Disease Modification in Parkinson’s Disease

P. Gubellini, S. Pérez-Lloret, F. Capani, 2021, Frontiers in Pharmacology.

High-Frequency Stimulation of the Subthalamic Nucleus Potentiates l-DOPA-Induced Neurochemical Changes in the Striatum in a Rat Model of Parkinson's Disease

P. Gubellini, P. Salin, L. Goff, 2007, The Journal of Neuroscience.

Deficits of glutamate transmission in the striatum of experimental hemiballism

P. Calabresi, B. Picconi, D. Centonze, 2006, Neuroscience.

Permissive role of interneurons in corticostriatal synaptic plasticity

P. Calabresi, D. Centonze, P. Gubellini, 1999, Brain Research Reviews.

Distinct effects of mGlu4 receptor positive allosteric modulators at corticostriatal vs. striatopallidal synapses may differentially contribute to their antiparkinsonian action

P. Gubellini, L. Goff, C. Melon, 2014, Neuropharmacology.

Inhibition of mitochondrial complex II alters striatal expression of genes involved in glutamatergic and dopaminergic signaling: possible implications for Huntington's disease

P. Calabresi, D. Centonze, P. Gubellini, 2004, Neurobiology of Disease.

Subthalamic nucleus lesion reverses motor abnormalities and striatal glutamatergic overactivity in experimental parkinsonism

P. Calabresi, B. Picconi, D. Centonze, 2005, Neuroscience.

An abnormal striatal synaptic plasticity may account for the selective neuronal vulnerability in Huntington's disease

P. Calabresi, B. Picconi, D. Centonze, 2001, Neurological Sciences.

Dopamine-Dependent Long-Term Potentiation Induced by 3-Nitropropionic Acid in Striatal Medium Spiny Neurons

P. Calabresi, D. Centonze, P. Gubellini, 2002 .

NR2B Subunit Exerts a Critical Role in Postischemic Synaptic Plasticity

P. Calabresi, B. Picconi, D. Centonze, 2006, Stroke.

Levodopa treatment reverses endocannabinoid system abnormalities in experimental parkinsonism

P. Calabresi, B. Picconi, D. Centonze, 2003, Journal of neurochemistry.

Blockade of M2-like muscarinic receptors enhances long-term potentiation at corticostriatal synapses.

G Bernardi, P. Calabresi, D. Centonze, 1998, The European journal of neuroscience.

Downstream mechanisms triggered by mitochondrial dysfunction in the basal ganglia: from experimental models to neurodegenerative diseases.

P. Calabresi, B. Picconi, P. Gubellini, 2010, Biochimica et biophysica acta.

Interhemispheric reactivity of the subthalamic nucleus sustains progressive dopamine neuron loss in asymmetrical parkinsonism

P. Gubellini, P. Salin, L. Goff, 2024, Neurobiology of Disease.