Ruth E. Thomas

发表

The PINK1/Parkin pathway regulates mitochondrial morphology

Angela C. Poole, Ruth E. Thomas, Laurie A Andrews, 2008, Proceedings of the National Academy of Sciences.

Autophagy accounts for approximately one-third of mitochondrial protein turnover and is protein selective

Gennifer E. Merrihew, James W. MacDonald, M. MacCoss, 2019, Autophagy.

Glucocerebrosidase deficiency promotes protein aggregation through dysregulation of extracellular vesicles

Gennifer E. Merrihew, M. MacCoss, Ruth E. Thomas, 2018, PLoS genetics.

Slowed protein turnover in aging Drosophila reflects a shift in cellular priorities.

Gennifer E. Merrihew, M. MacCoss, Ruth E. Thomas, 2021, The journals of gerontology. Series A, Biological sciences and medical sciences.

The Mitochondrial Fusion-Promoting Factor Mitofusin Is a Substrate of the PINK1/Parkin Pathway

Angela C. Poole, Ruth E. Thomas, L. Pallanck, 2010, PloS one.

Melanocortin-5 receptor deficiency reduces a pheromonal signal for aggression in male mice.

M. Novotny, Ruth E. Thomas, R. Cone, 2004, Chemical senses.

Melanocortin-5 receptor deficiency promotes defensive behavior in male mice

Ruth E. Thomas, R. Cone, C. Morgan, 2004, Hormones and Behavior.

Exocrine Gland Dysfunction in MC5-R-Deficient Mice: Evidence for Coordinated Regulation of Exocrine Gland Function by Melanocortin Peptides

Ximena Opitz-Araya, M. Low, Ruth E. Thomas, 1997, Cell.

Loss of the Drosophilam-AAA mitochondrial protease paraplegin results in mitochondrial dysfunction, shortened lifespan, and neuronal and muscular degeneration

Ruth E. Thomas, L. Pallanck, Gautam Pareek, 2018, Cell Death & Disease.

Glucocerebrosidase Deficiency in Drosophila Results in α-Synuclein-Independent Protein Aggregation and Neurodegeneration

T. Montine, Ruth E. Thomas, L. Pallanck, 2016, PLoS genetics.

The PINK1–Parkin pathway promotes both mitophagy and selective respiratory chain turnover in vivo

Gennifer E. Merrihew, M. MacCoss, Ruth E. Thomas, 2013, Proceedings of the National Academy of Sciences.

PINK1-Parkin Pathway Activity Is Regulated by Degradation of PINK1 in the Mitochondrial Matrix

Ruth E. Thomas, Laurie A Andrews, L. Pallanck, 2014, PLoS genetics.

VCP Is Essential for Mitochondrial Quality Control by PINK1/Parkin and this Function Is Impaired by VCP Mutations

Ruth E. Thomas, L. Pallanck, Rebecca B. Smith, 2013, Neuron.

Lon protease inactivation in Drosophila causes unfolded protein stress and inhibition of mitochondrial translation

Ruth E. Thomas, L. Pallanck, D. Morris, 2018, Cell Death Discovery.

VCP Is Essential for Mitochondrial Quality Control by PINK1/Parkin and this Function Is Impaired by VCP Mutations

Ruth E. Thomas, L. Pallanck, Rebecca B. Smith, 2013, Neuron.

Glucocerebrosidase reduces the spread of protein aggregation in a Drosophila melanogaster model of neurodegeneration by regulating proteins trafficked by extracellular vesicles.

Ruth E. Thomas, L. Pallanck, D. Raftery, 2021, PLoS genetics.

Lon protease inactivation in Drosophila causes unfolded protein stress and inhibition of mitochondrial translation

Ruth E. Thomas, L. Pallanck, D. Morris, 2018, Cell Death Discovery.

Loss of the Drosophilam-AAA mitochondrial protease paraplegin results in mitochondrial dysfunction, shortened lifespan, and neuronal and muscular degeneration

Ruth E. Thomas, L. Pallanck, Gautam Pareek, 2018, Cell Death & Disease.

Glucocerebrosidase reduces the spread of protein aggregation in a Drosophila melanogaster model of neurodegeneration by regulating proteins trafficked by extracellular vesicles

Ruth E. Thomas, L. Pallanck, K. Jewett, 2020, bioRxiv.