M. Boutjdir

M. Boutjdir, N. El-Sherif, H. Himel, 2008, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.

Mutations in the Voltage Sensors of Domains I and II of Nav1.5 that are Associated with Arrhythmias and Dilated Cardiomyopathy Generate Gating Pore Currents

M. Boutjdir, M. Chahine, A. Moreau, 2015, Front. Pharmacol..

Interleukin-6 inhibition of hERG underlies risk for acquired long QT in cardiac and systemic inflammation

M. Boutjdir, P. Lazzerini, P. Capecchi, 2018, PloS one.

Novel function of α1D L-type calcium channel in the atria.

M. Boutjdir, A. Aromolaran, Y. Qu, 2017, Biochemical and biophysical research communications.

Modulation of Nav 1 . 7 and Nav 1 . 8 Peripheral Nerve Sodium Channels by Protein Kinase A and Protein Kinase C

M. Boutjdir, K. Vijayaragavan, M. Chahine, 2004 .

Evidence for functional role of εPKC isozyme in the regulation of cardiac Ca2+ channels

M. Boutjdir, D. Mochly‐Rosen, K. Hu, 2000 .

PKC isozyme selective regulation of cloned human cardiac delayed slow rectifier K current.

M. Boutjdir, D. Mochly‐Rosen, G. Xiao, 2003, Biochemical and biophysical research communications.

Impaired Ca2+ homeostasis is associated with atrial fibrillation in the alpha1D L-type Ca2+ channel KO mouse.

M. Boutjdir, S. Mancarella, N. El-Sherif, 2008, American journal of physiology. Heart and circulatory physiology.

Gene Expression of SERCA2a and L- and T-type Ca Channels during Human Heart Development

M. Boutjdir, Y. Qu, 2001, Pediatric Research.

Systemic Inflammation Rapidly Induces Reversible Atrial Electrical Remodeling: The Role of Interleukin‐6–Mediated Changes in Connexin Expression

M. Boutjdir, I. Bertolozzi, G. Cevenini, 2019, Journal of the American Heart Association.

Autoantibody:Autoantigen Competitor Decoys: Application to Cardiac Phenotypes

M. Boutjdir, T. Cardozo, Lila Cardozo, 2022, Frontiers in Immunology.

Reexpression of T-type Ca2+ channel gene and current in post-infarction remodeled rat left ventricle.

M. Boutjdir, L. Deng, B. Huang, 2000, Cardiovascular research.

Autoimmune and inflammatory K+ channelopathies in cardiac arrhythmias: Clinical evidence and molecular mechanisms.

M. Boutjdir, P. Lazzerini, P. Capecchi, 2019, Heart rhythm.

Systemic inflammation as a novel QT-prolonging risk factor in patients with torsades de pointes

M. Boutjdir, I. Bertolozzi, P. Lazzerini, 2017, Heart.

Cardiac 5-HT4 Serotoninergic Receptors, 52 kD SSA/Ro and Autoimmune-Associated Congenital Heart Block

M. Boutjdir, J. Buyon, M. Miranda-Carús, 2002 .

Congenital Long QT syndrome and torsade de pointes

M. Boutjdir, N. El-Sherif, G. Turitto, 2017, Annals of noninvasive electrocardiology : the official journal of the International Society for Holter and Noninvasive Electrocardiology, Inc.

Editorial: Cardioimmunology: Inflammation and Immunity in Cardiovascular Disease

M. Boutjdir, R. Hamilton, P. Lazzerini, 2019, Front. Cardiovasc. Med..

Unravelling Atrioventricular Block Risk in Inflammatory Diseases: Systemic Inflammation Acutely Delays Atrioventricular Conduction via a Cytokine‐Mediated Inhibition of Connexin43 Expression

M. Boutjdir, I. Bertolozzi, P. Lazzerini, 2021, Journal of the American Heart Association.

Silencing of Cav1.2 gene in neonatal cardiomyocytes by lentiviral delivered shRNA.

M. Boutjdir, R. Wadgaonkar, S. Mancarella, 2009, Biochemical and biophysical research communications.

Electrophysiologic Effects of Cocaine on Subendocardial Purkinje Fibers Surviving 1 Day of Myocardial Infarction

M. Boutjdir, N. El-Sherif, M. Assadi, 1995, Journal of cardiovascular electrophysiology.

Induced pluripotent stem-cell-derived cardiomyocytes: cardiac applications, opportunities, and challenges.

M. Boutjdir, M. Chahine, A. Moreau, 2017, Canadian journal of physiology and pharmacology.

TRPC channels, an overarching Ca(2+) paradigm in the developing heart.

M. Boutjdir, Y. Qu, 2011, Cardiovascular research.

Effects of Physical Parameters of Fulguration on Electrophysiological and Anatomical Properties of Canine Myocardium

M. Boutjdir, L. Guize, P. Bruneval, 1986, Pacing and clinical electrophysiology : PACE.

Effects of glyburide on ischemia-induced changes in extracellular potassium and local myocardial activation: a potential new approach to the management of ischemia-induced malignant ventricular arrhythmias.

M. Boutjdir, N. El-Sherif, M. Restivo, 1990, American heart journal.

α1- and β-adrenergic interactions on L-type calcium current in cardiac myocytes

M. Boutjdir, N. El-Sherif, M. Restivo, 1992, Pflügers Archiv.

A review of the cardiovascular and anti-atherogenic effects of ghrelin.

M. Boutjdir, D. Mikhailidis, E. Isenovic, 2013, Current pharmaceutical design.

Interleukin-6 Elevation Is a Key Pathogenic Factor Underlying COVID-19-Associated Heart Rate-Corrected QT Interval Prolongation

M. Boutjdir, G. Cevenini, M. Capecchi, 2022, Frontiers in Cardiovascular Medicine.

Autoimmune Calcium Channelopathies and Cardiac Electrical Abnormalities

M. Boutjdir, P. Lazzerini, P. Capecchi, 2019, Front. Cardiovasc. Med..

α1-Adrenergic Activation Inhibits β-Adrenergic–Stimulated Unitary Ca2+ Currents in Cardiac Ventricular Myocytes

M. Boutjdir, N. El-Sherif, Long Chen, 1996 .

Reduction of ischemia-induced electrophysiologic abnormalities by glucose-insulin infusion.

M. Boutjdir, G. Butrous, S. Bekheit, 1993, Journal of the American College of Cardiology.

Cardioimmunology of arrhythmias: the role of autoimmune and inflammatory cardiac channelopathies

M. Boutjdir, P. Lazzerini, P. Capecchi, 2018, Nature Reviews Immunology.

Regulation of Cardiac Voltage-Gated Sodium Channel by Kinases: Roles of Protein Kinases A and C.

M. Boutjdir, A. Aromolaran, M. Chahine, 2018, Handbook of experimental pharmacology.

Commentary: Systemic effects of IL-17 in inflammatory arthritis

M. Boutjdir, P. Lazzerini, P. Capecchi, 2019, Front. Cardiovasc. Med..

Rescue and Worsening of Congenital Heart Block‐Associated Electrocardiographic Abnormalities in Two Transgenic Mice

M. Boutjdir, S. Mancarella, Y. Qu, 2011, Journal of cardiovascular electrophysiology.

Autoantibodies from mothers of children with congenital heart block downregulate cardiac L-type Ca channels.

M. Boutjdir, Y. Qu, G. Xiao, 2001, Journal of molecular and cellular cardiology.

Early Afterdepolarizations and Arrhythmogenesis

M. Boutjdir, W. Craelius, N. El-Sherif, 1990 .

Relationship between the Soluble F11 Receptor and Annexin A5 in African Americans Patients with Type-2 Diabetes Mellitus

M. Boutjdir, M. Salifu, L. Salciccioli, 2022, Biomedicines.

Perinatal and Postnatal Expression of Cav1.3 α1D Ca2+ Channel in the Rat Heart

M. Boutjdir, Y. Yue, Y. Qu, 2011, Pediatric Research.

Gene expression of Na+/Ca2+ exchanger during development in human heart.

M. Boutjdir, N. El-Sherif, Y. Qu, 2000, Cardiovascular research.

Evidence for functional role of epsilonPKC isozyme in the regulation of cardiac Ca(2+) channels.

M. Boutjdir, D. Mochly‐Rosen, K. Hu, 2000, American journal of physiology. Heart and circulatory physiology.

Electrocardiographic abnormalities in a murine model injected with IgG from mothers of children with congenital heart block.

M. Boutjdir, J. Buyon, N. El-Sherif, 1999, Circulation.

Induction of antibodies reactive with SSA/Ro-SSB/La and development of congenital heart block in a murine model.

M. Boutjdir, J. Buyon, M. Miranda-Carús, 1998, Journal of immunology.

Arrhythmogenicity of IgG and anti-52-kD SSA/Ro affinity-purified antibodies from mothers of children with congenital heart block.

M. Boutjdir, J. Buyon, N. El-Sherif, 1997, Circulation research.

Inflammatory cytokines and cardiac arrhythmias: the lesson from COVID-19

M. Boutjdir, P. Lazzerini, P. Capecchi, 2022, Nature Reviews Immunology.

Plasma PCSK9 predicts microvascular function but not arterial stiffness in African-Americans with well controlled type 2 diabetes

M. Boutjdir, M. Salifu, L. Salciccioli, 2020, Journal of Translational Science.

IL-6 (Interleukin 6) Blockade and Heart Rate Corrected QT Interval Prolongation in COVID-19

M. Boutjdir, P. Lazzerini, F. Laghi-Pasini, 2020, Circulation. Arrhythmia and Electrophysiology.

mRNA and Protein Expression of SSA/Ro and SSB/La in Human Fetal Cardiac Myocytes Cultured Using a Novel Application of the Langendorff Procedure

M. Boutjdir, J. Buyon, E. Chan, 1999, Pediatric Research.

Serum and Immunoglobulin G from the Mother of a Child with Congenital Heart Block Induce Conduction Abnormalities and Inhibit L-Type Calcium Channels in a Rat Heart Model

M. Boutjdir, J. Buyon, N. El-Sherif, 1998, Pediatric Research.

Congenital Heart Block in a Murine Model SSA / Ro-SSB / La and Development of Induction of Antibodies Reactive with Buyon

M. Boutjdir, M. Miranda-Carús, E. Chan, 1998 .

Enhanced Delivery of Ligand-Conjugated Antisense Oligonucleotides (C16-HA-ASO) Targeting DMPK Transcripts for the Treatment of Myotonic Dystrophy Type 1.

M. Boutjdir, J. Puymirat, F. Rigo, 2022, Human gene therapy.

iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities

M. Boutjdir, J. Puymirat, I. Deschênes, 2021, Scientific Reports.

Pathophysiology of autoimmune-associated congenital heart block

M. Boutjdir, Y. Qu, 2012 .

Sodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutation

M. Boutjdir, A. Krahn, G. Klein, 2013, Front. Pharmacol..

Wenckebach Periods in Sinoatrial Block: Experimental and Clinical Evidence

M. Boutjdir, L. Guize, T. Lavergne, 1991, Pacing and clinical electrophysiology : PACE.

Electrophysiologic Effects of Quinidine and Hydroquinidine on Rabbit Atrium: A Comparative Study

M. Boutjdir, L. Guize, T. Lavergne, 1989 .

Pharmacological evaluation of early afterdepolarisations induced by sea anemone toxin (ATXII) in dog heart.

M. Boutjdir, N. El-Sherif, 1991, Cardiovascular research.

All members in the sphingomyelin synthase gene family have ceramide phosphoethanolamine synthase activity[S]

M. Boutjdir, Jibin Dong, Ting-bo Ding, 2015, Journal of Lipid Research.

Calreticulin negatively regulates the surface expression of Cav1.3 L-type calcium channel.

M. Boutjdir, Y. Qu, E. Karnabi, 2009, Biochemical and biophysical research communications.

Transient Hypogonadism Is Associated With Heart Rate–Corrected QT Prolongation and Torsades de Pointes Risk During Active Systemic Inflammation in Men

M. Boutjdir, I. Bertolozzi, G. Cevenini, 2021, Journal of the American Heart Association.

Ketanserin inhibits depolarization-activated outward potassium current in rat ventricular myocytes.

M. Boutjdir, N. El-Sherif, Z. H. Zhang, 1994, Circulation research.

Sodium Channels by Protein Kinase A and Protein

M. Boutjdir, A. Guia, N. Beaudet, 2015 .

Role of spatial dispersion of repolarization in reentry around a functional core versus reentry around a fixed anatomical core

M. Boutjdir, N. El-Sherif, H. Himel, 2019, Annals of noninvasive electrocardiology : the official journal of the International Society for Holter and Noninvasive Electrocardiology, Inc.

Evidence of Na Current Contribution to the Transient Outward Current in Cardiac Ventricular Myocytes

M. Boutjdir, N. El-Sherif, N. Stergiopoulos, 1996, Journal of cardiovascular pharmacology and therapeutics.

Unitary Current Analysis of L‐type Ca2+ Channels in Human Fetal Ventricular Myocytes

M. Boutjdir, N. El-Sherif, Long Chen, 1999, Journal of cardiovascular electrophysiology.

L-type Ca channel KO mouse

M. Boutjdir, S. Mancarella, N. El-Sherif, 2008 .

Rolipram, a PDE4 Inhibitor, Enhances the Inotropic Effect of Rat Heart by Activating SERCA2a

M. Boutjdir, Long Chen, Ming Xie, 2019, Front. Pharmacol..

Effects of caffeine and ryanodine on delayed afterdepolarizations and sustained rhythmic activity in 1-day-old myocardial infarction in the dog.

M. Boutjdir, N. El-Sherif, W. Gough, 1990, Circulation.

Addressing challenges faced by underrepresented biomedical investigators and efforts to address them: An NHLBI-PRIDE perspective.

M. Boutjdir, Ana F. Diallo, Forgive Avorgbedor, 2022, Journal of the National Medical Association.

Training Underrepresented Early-Career Faculty in Cardiovascular Health Research during COVID-19: Structural Inequities and Health Disparity.

M. Boutjdir, Ana F. Diallo, S. R. Ramos, 2021, Ethnicity & disease.

Marked QTc Prolongation and Torsades de pointes in Patients with Chronic Inflammatory Arthritis

M. Boutjdir, I. Bertolozzi, P. Lazzerini, 2016, Front. Cardiovasc. Med..

Recent Progress and Challenges in the Development of Antisense Therapies for Myotonic Dystrophy Type 1

M. Boutjdir, J. Puymirat, M. Chahine, 2022, International journal of molecular sciences.

Functional Basis of Sinus Bradycardia in Congenital Heart Block

M. Boutjdir, Y. Yue, Y. Qu, 2004, Circulation research.

Community-Engaged Intervention Mapping for Cardiovascular Disease Prevention in Black and Latinx Sexual Minority Men With HIV in New York City: Protocol for a Web-Based Mixed Methods Study

M. Boutjdir, M. Camacho-Rivera, S. R. Ramos, 2022, JMIR research protocols.

Alpha 1-adrenoceptor regulation of delayed afterdepolarizations and triggered activity in subendocardial Purkinje fibers surviving 1 day of myocardial infarction.

M. Boutjdir, N. El-Sherif, 1991, Journal of molecular and cellular cardiology.

Induction of autoimmune response to the extracellular loop of the HERG channel pore induces QTc prolongation in guinea‐pigs

M. Boutjdir, E. Sobie, R. Wieczorek, 2016, The Journal of physiology.

The dual-specificity kinases, TOPK and DYRK1A, are critical for oocyte maturation induced by wild-type--but not by oncogenic--ras-p21 protein.

M. Pincus, S. Pestka, M. Boutjdir, 2007, Frontiers in bioscience : a journal and virtual library.

Two dual specificity kinases are preferentially induced by wild-type rather than by oncogenic RAS-P21 in Xenopus oocytes.

M. Pincus, S. Pestka, M. Boutjdir, 2006, Frontiers in bioscience : a journal and virtual library.

Isolated atrioventricular block of unknown origin in the adult and autoimmunity: diagnostic and therapeutic considerations exemplified by 3 anti-Ro/SSA–associated cases

M. Boutjdir, P. Lazzerini, P. Capecchi, 2015, HeartRhythm case reports.

COVID-19, Arrhythmic Risk and Inflammation: Mind the Gap!

M. Boutjdir, P. Lazzerini, P. Capecchi, 2020, Circulation.

Cardioimmunology of arrhythmias: the role of autoimmune and inflammatory cardiac channelopathies

M. Boutjdir, P. Lazzerini, P. Capecchi, 2018, Nature Reviews Immunology.

Fir(e)ing the Rhythm

M. Boutjdir, P. Lazzerini, P. Capecchi, 2023, JACC. Basic to translational science.

Contribution of cytokine-mediated prolongation of QTc interval to the multi-hit theory of Torsade de Pointes.

M. Boutjdir, P. Lazzerini, P. Capecchi, 2023, Biochemical and biophysical research communications.

Arrhythmogenic mechanisms of interleukin-6 combination with hydroxychloroquine and azithromycin in inflammatory diseases

M. Boutjdir, P. Lazzerini, P. Capecchi, 2022, Scientific Reports.

Anti-Ro/SSA Antibodies and the Autoimmune Long-QT Syndrome

M. Boutjdir, P. Lazzerini, P. Capecchi, 2021, Frontiers in Medicine.

Inflammation as a Risk Factor in Cardiotoxicity: An Important Consideration for Screening During Drug Development

M. Boutjdir, E. Sobie, C. Campana, 2021, Frontiers in Pharmacology.

Cardiac Arrest Risk During Acute Infections

M. Boutjdir, I. Bertolozzi, G. Cevenini, 2020, Circulation. Arrhythmia and electrophysiology.

Emerging Arrhythmic Risk of Autoimmune and Inflammatory Cardiac Channelopathies

M. Boutjdir, P. Lazzerini, P. Capecchi, 2018, Journal of the American Heart Association.

Arrhythmogenicity of Anti-Ro/SSA Antibodies in Patients With Torsades de Pointes

M. Boutjdir, I. Bertolozzi, P. Lazzerini, 2016, Circulation. Arrhythmia and electrophysiology.

Pathogenesis of the Novel Autoimmune-Associated Long-QT Syndrome

C. January, M. Boutjdir, E. Sobie, 2015, Circulation.

Inhomogeneity of Cellular Refractoriness in Human Atrium: Factor of Arrhythmia?

M. Boutjdir, L. Guize, T. Lavergne, 1986, Pacing and clinical electrophysiology : PACE.

Cardiac Ion Channel Regulation in Obesity and the Metabolic Syndrome: Relevance to Long QT Syndrome and Atrial Fibrillation

M. Boutjdir, A. Aromolaran, 2017, Front. Physiol..

Modulation of Nav1.7 and Nav1.8 peripheral nerve sodium channels by protein kinase A and protein kinase C.

Mohamed Chahine, M. Boutjdir, K. Vijayaragavan, 2004, Journal of neurophysiology.

Cardiac 5-HT(4) serotoninergic receptors, 52kD SSA/Ro and autoimmune-associated congenital heart block.

M. Boutjdir, J. Buyon, M. Miranda-Carús, 2002, Journal of autoimmunity.

Mentored Training to Increase Diversity among Faculty in the Biomedical Sciences: The NHLBI Summer Institute Programs to Increase Diversity (SIPID) and the Programs to Increase Diversity among Individuals Engaged in Health-related Research (PRIDE).

G. Jean-Louis, T. Rice, D. Jeffe, 2017, Ethnicity & disease.

Cardiolipotoxicity, Inflammation, and Arrhythmias: Role for Interleukin-6 Molecular Mechanisms

M. Boutjdir, A. Aromolaran, Alessandra Alí, 2019, Front. Physiol..

The Role of Inflammation and Autoimmunity in Long QT Syndrome

M. Boutjdir, P. Lazzerini, P. Capecchi, 2019, Cardiac Repolarization.

RNase protection assay for quantifying gene expression levels.

M. Boutjdir, Y. Qu, 2007, Methods in molecular biology.

Regulation of cardiac excitability by protein kinase C isozymes.

M. Boutjdir, D. Mochly‐Rosen, J. Ferreira, 2012, Frontiers in bioscience.

Sudden Cardiac Death in Ischemic Heart Disease: Pathophysiology and Risk Stratification.

M. Boutjdir, N. El-Sherif, G. Turitto, 2017, Cardiac electrophysiology clinics.

Increased sarcoplasmic/endoplasmic reticulum calcium ATPase 2a activity underlies the mechanism of the positive inotropic effect of ivabradine

M. Boutjdir, Long Chen, Ming Xie, 2020 .

Protein kinase C activation inhibits Cav1.3 calcium channel at NH2-terminal serine 81 phosphorylation site.

M. Boutjdir, Y. Qu, M. Chahine, 2006, American journal of physiology. Heart and circulatory physiology.

C2 Region–Derived Peptides of β-Protein Kinase C Regulate Cardiac Ca2+ Channels

M. Boutjdir, D. Mochly‐Rosen, N. El-Sherif, 1997 .

Diminished basal phosphorylation level of phospholamban in the postinfarction remodeled rat ventricle: role of beta-adrenergic pathway, G(i) protein, phosphodiesterase, and phosphatases.

M. Boutjdir, N. El-Sherif, S. Wang, 1999, Circulation research.

Intestine-specific MTP and global ACAT2 deficiency lowers acute cholesterol absorption with chylomicrons and HDLs

M. Boutjdir, L. Rudel, J. Iqbal, 2014, Journal of Lipid Research.

Voltage/Calcium Uncoupling Underlies Sustained Torsade de Pointes Ventricular Tachyarrhythmia in an Experimental Model of Long QT Syndrome

M. Boutjdir, N. El-Sherif, H. Himel, 2021, Frontiers in Physiology.

Acquired Long QT Syndrome and Electrophysiology of Torsade de Pointes

M. Boutjdir, N. El-Sherif, G. Turitto, 2019, Arrhythmia & electrophysiology review.

Mibefradil, a T‐Type Calcium Channel Blocker, and Abnormal Rhythm in Subacute Myocardial Infarction

M. Boutjdir, 1999, Journal of cardiovascular electrophysiology.

Early Afterdepolarization Formation in Cardiac Myocyte:

M. Boutjdir, N. El-Sherif, M. Restivo, 1994, Journal of cardiovascular electrophysiology.

Racial Disparities in Ion Channelopathies and Inherited Cardiovascular Diseases Associated With Sudden Cardiac Death

M. Boutjdir, M. Chahine, J. Fontaine, 2022, Journal of the American Heart Association.

A Perspective on Promoting Diversity in the Biomedical Research Workforce: The National Heart, Lung, and Blood Institute's PRIDE Program.

G. Jean-Louis, T. Rice, D. Jeffe, 2016, Ethnicity & disease.

Role of pharmacotherapy in cardiac ion channelopathies.

M. Boutjdir, N. El-Sherif, 2015, Pharmacology & therapeutics.

Novel Molecular Mechanism Involving &agr;1D (Cav1.3) L-Type Calcium Channel in Autoimmune-Associated Sinus Bradycardia

M. Boutjdir, Y. Yue, Y. Qu, 2005, Circulation.

Molecular and ionic basis of congenital complete heart block.

M. Boutjdir, 2000, Trends in cardiovascular medicine.

Evidence for functional role of epsilonPKC isozyme in the regulation of cardiac Na(+) channels.

M. Boutjdir, D. Mochly‐Rosen, Y. Qu, 2001, American journal of physiology. Cell physiology.

High-fat diet-dependent modulation of the delayed rectifier K(+) current in adult guinea pig atrial myocytes.

M. Boutjdir, H. Colecraft, A. Aromolaran, 2016, Biochemical and biophysical research communications.

The kinetics of spontaneous calcium oscillations and arrhythmogenesis in the in vivo heart during ischemia/reperfusion.

Gil Bub, M. Boutjdir, G. Bub, 2006, Heart rhythm.

Pathophysiology of Cav1.3 L-type calcium channels in the heart

M. Boutjdir, Y. Qu, M. Chahine, 2023, Frontiers in Physiology.

Autoimmune cardiac channelopathies: the heart of the matter

M. Boutjdir, P. Lazzerini, P. Capecchi, 2017, Nature Reviews Cardiology.

Intestine specific MTP deficiency with global ACAT 2 gene ablation lowers acute cholesterol absorption with chylomicrons and high density lipoproteins

M. Boutjdir, L. Rudel, J. Iqbal, 2014 .

β- and α-adrenergic cross-signaling for L-type Ca current is impaired in transgenic mice with constitutive activation of εPKC

M. Boutjdir, Y. Yue, Y. Qu, 2004 .

Activation of εPKC reduces reperfusion arrhythmias and improves recovery from ischemia: optical mapping of activation patterns in the isolated guinea-pig heart.

M. Boutjdir, D. Mochly‐Rosen, N. El-Sherif, 2012, Biochemical and biophysical research communications.

Cellular and ionic basis of arrhythmias in postinfarction remodeled ventricular myocardium.

D Qin, M. Boutjdir, E. Caref, 1996, Circulation research.

Anti-Ro/SSA-antibodies and heart rhythm disturbances in the general population: the 'dark side of the immune'.

M. Boutjdir, P. Lazzerini, P. Capecchi, 2022, European heart journal.

C2 region-derived peptides of beta-protein kinase C regulate cardiac Ca2+ channels.

M. Boutjdir, D. Mochly‐Rosen, N. El-Sherif, 1997, Circulation research.

Arrhythmogenic cardiomyopathy as a myogenic disease: highlights from cardiomyocytes derived from human induced pluripotent stem cells

M. Boutjdir, P. Chevalier, A. Moreau, 2023, Frontiers in Physiology.

Generation of four myotonic dystrophy type 1 patient iPSC lines (CBRCULi002-A, CBRCULi003-A, CBRCULi004-A, CBRCULi005-A) and a control (CBRCULi001-A) derived from lymphoblastoids cell lines.

M. Boutjdir, J. Puymirat, M. Chahine, 2023, Stem cell research.

Optical mapping of activation patterns in an animal model of congenital heart block.

M. Boutjdir, M. Restivo, D. Kozhevnikov, 2001, American journal of physiology. Heart and circulatory physiology.

Proton Pump Inhibitors Directly Block hERG-Potassium Channel and Independently Increase the Risk of QTc Prolongation in a Large Cohort of US Veterans

M. Boutjdir, F. Fusi, I. Bertolozzi, 2021, Circulation. Arrhythmia and electrophysiology.

Potassium Channel Block and Novel Autoimmune-Associated Long QT Syndrome.

M. Boutjdir, P. Lazzerini, P. Capecchi, 2016, Cardiac electrophysiology clinics.

Alpha 1-adrenergic activation inhibits beta-adrenergic-stimulated unitary Ca2+ currents in cardiac ventricular myocytes.

M. Boutjdir, N. El-Sherif, L. Chen, 1996, Circulation research.

Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.

Mohamed Chahine, Olivier Thériault, M. Boutjdir, 2017, The Canadian journal of cardiology.

Functional interactions of Raf and MEK with Jun-N-terminal kinase (JNK) result in a positive feedback loop on the oncogenic Ras signaling pathway.

M. Pincus, S. Pestka, M. Boutjdir, 2005, Biochemistry.

Research Education and Mentoring Program in Cardiovascular Diseases for Under-Represented Junior Faculty From NHLBI SIPID/PRIDE.

T. Rice, D. Jeffe, J. Jobe, 2019, Journal of the American College of Cardiology.

Anti-Ro/SSA Antibodies Blocking Calcium Channels as a Potentially Reversible Cause of Atrioventricular Block in Adults.

S. F. Carbone, M. Boutjdir, I. Bertolozzi, 2023, JACC. Clinical electrophysiology.

Letter by Lazzerini et al Regarding Article, "Autoantibody Signature in Cardiac Arrest".

M. Boutjdir, P. Lazzerini, P. Capecchi, 2020, Circulation.

Electrophysiological Substrates for Gender Difference in the Incidence of Torsades de Pointes Arrhythmias

M. Boutjdir, N. El-Sherif, M. Restivo, 2019, Cardiac Mapping.

Electrophysiological Basis of ECG Characteristics of Torsades de Pointes in Long QT Syndrome

M. Boutjdir, N. El-Sherif, G. Turitto, 2014 .

of Torsades de Pointes in Long QT Syndrome

M. Boutjdir, N. El-Sherif, G. Turitto, 2014 .

Phosphorylation of the Consensus Sites of Protein Kinase A on α1D L-type Calcium Channel*

M. Boutjdir, R. Wadgaonkar, Y. Qu, 2009, Journal of Biological Chemistry.

M. Boutjdir, P. Capecchi, M. Acampa, 2023, JACC: Basic to Translational Science.

Novel Re-expression of L-type Calcium Channel Cav1.3 in Left Ventricles of Failing Human Heart.

M. Boutjdir, Y. Qu, M. Chahine, 2020, Heart rhythm.

A Novel Peptide/Antibody-Based Antiarrhythmic Approach to Long QT Syndrome and Beyond.

M. Boutjdir, P. Lazzerini, 2020, Journal of the American College of Cardiology.

Modulation of Nav 1 . 7 and Nav 1 . 8 Peripheral Nerve Sodium Channels by Protein Kinase A and Protein Kinase C

M. Boutjdir, K. Vijayaragavan, M. Chahine, 2004 .

Peripheral Nerve Sodium Channels by Protein Kinase A and Protein Kinase C

M. Boutjdir, K. Vijayaragavan, M. Chahine, 2003 .

Optical Mapping of Cardiomyocytes in Monolayer Derived from Induced Pluripotent Stem Cells

M. Boutjdir, M. Chahine, Mohammed Djemai, 2023, Cells.

Induced pluripotent stem-cell-derived cardiomyocytes: cardiac applications, opportunities, and challenges.

M. Boutjdir, M. Chahine, A. Moreau, 2017, Canadian journal of physiology and pharmacology.

Protein kinase C activation inhibits α1D L-type Ca channel: A single-channel analysis

M. Boutjdir, S. Mancarella, Y. Qu, 2008, Pflügers Archiv - European Journal of Physiology.

Ethnic and racial differences in Asian populations with ion channelopathies associated with sudden cardiac death

M. Boutjdir, Y. Qu, M. Chahine, 2023, Frontiers in cardiovascular medicine.

Protective role of protein kinase C epsilon activation in ischemia-reperfusion arrhythmia.

M. Boutjdir, Y. Yue, Y. Qu, 2006, Biochemical and biophysical research communications.

Biophysical, Molecular, and Pharmacological Characterization of Voltage-Dependent Sodium Channels From Induced Pluripotent Stem Cell-Derived Cardiomyocytes.

M. Boutjdir, B. Burger, M. Chahine, 2017, The Canadian journal of cardiology.

Contrasting effects of ischemia on the kinetics of membrane voltage and intracellular calcium transient underlie electrical alternans.

M. Boutjdir, G. Bub, N. El-Sherif, 2005, American journal of physiology. Heart and circulatory physiology.

Cardiac involvement in patient-specific induced pluripotent stem cells of myotonic dystrophy type 1: unveiling the impact of voltage-gated sodium channels

M. Boutjdir, Mohammed Djemai, Marion Pierre, 2023, Frontiers in physiology.

Increased sarcoplasmic/endoplasmic reticulum calcium ATPase 2a activity underlies the mechanism of the positive inotropic effect of ivabradine

M. Boutjdir, Long Chen, Ming Xie, 2020, Experimental physiology.

Expression of skeletal muscle Na(V)1.4 Na channel isoform in canine cardiac Purkinje myocytes.

M. Boutjdir, Y. Qu, M. Chahine, 2007, Biochemical and biophysical research communications.

Induction of autoimmune response to the extracellular loop of the HERG channel pore induces QTc prolongation in guinea‐pigs

M. Boutjdir, E. Sobie, R. Wieczorek, 2016, The Journal of physiology.

Localization and modulation of α1D (Cav1.3) L-type Ca channel by protein kinase A

M. Boutjdir, N. El-Sherif, Y. Qu, 2005 .

G. Jean-Louis, T. Rice, D. Jeffe, 2017, Ethnicity & disease.

Potassium Channel Block and Novel Autoimmune-Associated Long QT Syndrome.

M. Boutjdir, P. Lazzerini, P. Capecchi, 2016, Cardiac electrophysiology clinics.

Comment on "absence of an association between anti-Ro antibodies and prolonged QTc interval in systemic sclerosis: a multicenter study of 689 patients".

M. Boutjdir, P. Lazzerini, P. Capecchi, 2015, Seminars in arthritis and rheumatism.