I. Y. Haddad

发表

"Natural surfactant and hyperoxic lung injury in primates".

S. Matalon, I. Y. Haddad, 1994, Journal of applied physiology.

Exuberant Inflammation in Nicotinamide Adenine Dinucleotide Phosphate-Oxidase-Deficient Mice After Allogeneic Marrow Transplantation1

I. Y. Haddad, A. Panoskaltsis-Mortari, B. Blazar, 2002, The Journal of Immunology.

NITRIC OXIDE MODULATION OF GENE EXPRESSION

S. Matalon, I. Y. Haddad, Sha Zhu, 1998 .

History of Pulmonary Complications in hildren after Bone Marrow Transplantation

D. Cornfield, I. Y. Haddad, B. Blazar, 2004 .

Impact of a Standardized Treatment Guideline for Pediatric Iatrogenic Opioid Dependence: A Quality Improvement Initiative.

I. Y. Haddad, R. Gerkin, T. Youssef, 2016, The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG.

Hyperoxic lung injury increases HOCl-modified lung proteins and NA,K-adenosine triphosphatase nitrotyrosine content.

I. Y. Haddad, D. Ingbar, J. Heinecke, 1999, Chest.

High levels of peroxynitrite are generated in the lungs of irradiated mice given cyclophosphamide and allogeneic T cells. A potential mechanism of injury after marrow transplantation.

I. Y. Haddad, D. Ingbar, B. Blazar, 1999, American journal of respiratory cell and molecular biology.

Quantitation of nitrotyrosine levels in lung sections of patients and animals with acute lung injury.

S. Matalon, I. Y. Haddad, G. Pataki, 1994, The Journal of clinical investigation.

Mechanisms of Nitric Oxide Induced Injury to the Alveolar Epithelium

S. Matalon, I. Y. Haddad, Sha Zhu, 1998 .

Concurrent generation of nitric oxide and superoxide damages surfactant protein A.

S. Matalon, I. Y. Haddad, J. Beckman, 1994, The American journal of physiology.

Mechanisms of peroxynitrite-induced injury to pulmonary surfactants.

S. Matalon, I. Y. Haddad, H. Ischiropoulos, 1993, The American journal of physiology.

Nitration of surfactant protein A results in decreased ability to aggregate lipids.

S. Matalon, I. Y. Haddad, H. Ischiropoulos, 1996, The American journal of physiology.

Inhibition of alveolar type II cell ATP and surfactant synthesis by nitric oxide.

S. Matalon, I. Y. Haddad, J. Crow, 1996, The American journal of physiology.

Allogeneic Marrow Transplantation Phosphate-Oxidase-Deficient Mice After Adenine Dinucleotide Exuberant Inflammation in Nicotinamide

I. Y. Haddad, B. Blazar, Shuxia Yang, 2002 .

Effects of oxidant stress on inflammation and survival of iNOS knockout mice after marrow transplantation.

D. Cornfield, I. Y. Haddad, B. Blazar, 2001, American journal of physiology. Lung cellular and molecular physiology.

LUNG REJECTION Safety of Inhaled Nitric Oxide After Lung Transplantation

D. Cornfield, I. Y. Haddad, C. Milla, 2003 .

Safety of inhaled nitric oxide after lung transplantation.

D. Cornfield, I. Y. Haddad, C. Milla, 2003, The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation.

Cyclophosphamide prevents systemic keratinocyte growth factor-induced up-regulation of surfactant protein A after allogeneic transplant in mice.

D. Lacey, S. Matalon, I. Y. Haddad, 2000, American journal of respiratory and critical care medicine.

Inhibition of surfactant function by copper-zinc superoxide dismutase (CuZn-SOD).

S. Matalon, I. Y. Haddad, B. Nieves-Cruz, 1997, Journal of applied physiology.

Concurrent Generation of Nitric Oxide and Superoxide Damages the Pulmonary Surfactant System

S. Matalon, I. Y. Haddad, 1996 .

Inhaled nitric oxide injures the pulmonary surfactant system of lambs in vivo.

S. Matalon, I. Y. Haddad, V. Demarco, 1996, The American journal of physiology.

Myeloperoxidase deficiency enhances inflammation after allogeneic marrow transplantation.

D. Cornfield, I. Y. Haddad, S. Hazen, 2004, American journal of physiology. Lung cellular and molecular physiology.

Interactions of keratinocyte growth factor with a nitrating species after marrow transplantation in mice.

D. Lacey, D. Cornfield, I. Y. Haddad, 1999, American Journal of Physiology.

Interactions of keratinocyte growth factor with a nitrating species after marrow transplantation in mice.

D. Lacey, D. Cornfield, I. Y. Haddad, 1999, American journal of physiology. Lung cellular and molecular physiology.

Nitric oxide inhibits heterologous CFTR expression in polarized epithelial cells.

S. Matalon, I. Y. Haddad, T. Jilling, 1999, American journal of physiology. Lung cellular and molecular physiology.

Modulation of adenovirus-mediated gene transfer by nitric oxide.

S. Matalon, I. Y. Haddad, E. Sorscher, 1997, American journal of respiratory cell and molecular biology.

Human surfactant protein a suppresses T cell-dependent inflammation and attenuates the manifestations of idiopathic pneumonia syndrome in mice.

I. Y. Haddad, D. Ingbar, B. Blazar, 2001, American journal of respiratory cell and molecular biology.

A rose by any other name is yet a rose: acute respiratory failure in children.

D. Cornfield, I. Y. Haddad, 2003, American journal of respiratory and critical care medicine.

Simultaneous absence of surfactant proteins A and D increases lung inflammation and injury after allogeneic HSCT in mice.

I. Y. Haddad, B. Blazar, M. Steiner, 2009, American journal of physiology. Lung cellular and molecular physiology.

Surfactant protein A is a required mediator of keratinocyte growth factor after experimental marrow transplantation.

D. Lacey, I. Y. Haddad, B. Blazar, 2003, American journal of physiology. Lung cellular and molecular physiology.

Activated alveolar macrophage-derived nitric oxide predicts the development of lung damage after marrow transplantation in mice.

I. Y. Haddad, D. Ingbar, B. Blazar, 1999, Chest.

Structural and functional alterations of surfactant protein A by peroxynitrite.

S. Matalon, I. Y. Haddad, J. Beckman, 1994, Chest.

Pediatric Lung Transplantation

D. Cornfield, I. Y. Haddad, 2009 .

Myeloperoxidase deﬁciency enhances inﬂammation after allogeneic marrow transplantation

D. Cornfield, I. Y. Haddad, S. Hazen, 2004 .

Nitric oxide inhibits heterologous CFTR expression in polarized epithelial cells

S. Matalon, I. Y. Haddad, T. Jilling, 1999 .

Nitration of surfactant protein A (SP-A) tyrosine residues results in decreased mannose binding ability.

S. Matalon, I. Y. Haddad, S. Zhu, 1996, Archives of biochemistry and biophysics.

Stem cell transplantation and lung dysfunction

I. Y. Haddad, 2013, Current opinion in pediatrics.

KGF pretreatment decreases B7 and granzyme B expression and hastens repair in lungs of mice after allogeneic BMT.

D. Lacey, I. Y. Haddad, P. Bitterman, 2000, American journal of physiology. Lung cellular and molecular physiology.

ALUNG July 21/1

S. Matalon, I. Y. Haddad, T. Jilling, 1999 .

Voltage-gated K(+)-channel activity in ovine pulmonary vasculature is developmentally regulated.

D. Cornfield, I. Y. Haddad, E. Resnik, 2000, American journal of physiology. Lung cellular and molecular physiology.

Inflammatory cytokines and the development of pulmonary complications after allogeneic hematopoietic cell transplantation in patients with inherited metabolic storage disorders.

D. Cornfield, I. Y. Haddad, B. Blazar, 2006, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

Dependence of surfactant function on extracellular pH: mechanisms and modifications.

S. Matalon, I. Y. Haddad, B. Holm, 1994, Journal of applied physiology.

The Spectrum of Idiopathic Pneumonia Syndrome: Risk Factors, Diagnosis, and Treatment

I. Y. Haddad, Budi Wiryawan, 2011 .

exerciseof SP-A-deficient mice to hyperoxia or

I. Y. Haddad, A. Jobe, B. Yoder, 2015 .

Mechanisms and Physiological Sequelae of Reactive Species Injury to the Alveolar Epithelium

S. Matalon, I. Y. Haddad, Sha Zhu, 1999 .

An official American Thoracic Society research statement: noninfectious lung injury after hematopoietic stem cell transplantation: idiopathic pneumonia syndrome.

Angela Panoskaltsis-Mortari, Matthias Griese, I. Y. Haddad, 2011, American journal of respiratory and critical care medicine.

Natural history of pulmonary complications in children after bone marrow transplantation.

D. Cornfield, I. Y. Haddad, B. Blazar, 2005, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

Absence of host tumor necrosis factor receptor 1 attenuates manifestations of idiopathic pneumonia syndrome.

I. Y. Haddad, B. Blazar, C. Milla, 2005, American journal of physiology. Lung cellular and molecular physiology.

Intercellular adhesion molecule-I (ICAM-I, CD54) deficiency segregates the unique pathophysiological requirements for generating idiopathic pneumonia syndrome (IPS) versus graft-versus-host disease following allogeneic murine bone marrow transplantation.

I. Y. Haddad, B. Blazar, J. Hermanson, 2001, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

Surfactant Protein a Decreases Lung Injury and Mortality after Murine Marrow Transplantation

I. Y. Haddad, B. Blazar, C. Milla, 2022 .

Pneumonia and Empyema

D. Cornfield, I. Y. Haddad, 2008, The Respiratory Tract in Pediatric Critical Illness and Injury.

Surfactant protein A decreases lung injury and mortality after murine marrow transplantation.

I. Y. Haddad, B. Blazar, C. Milla, 2002, American journal of respiratory cell and molecular biology.