M. Vadas

发表

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

G. Goodall, P. A. Gregory, A. Bert, 2008, Nature Cell Biology.

Assessing IRES activity in the HIF-1α and other cellular 5′ UTRs

G. Goodall, A. Bert, M. Vadas, 2006 .

Cloning, Characterization, and Chromosomal Location of a Novel Human K+-Cl− Cotransporter*

J. Gamble, M. Vadas, R. D'Andrea, 1999, The Journal of Biological Chemistry.

YAP and the RhoC regulator ARHGAP18, are required to mediate flow-dependent endothelial cell alignment

J. Gamble, M. Vadas, Jia Li, 2020, Cell Communication and Signaling.

Expression profiling reveals functionally important genes and coordinately regulated signaling pathway genes during in vitro angiogenesis.

J. Gamble, G. Goodall, A. Tsykin, 2005, Physiological genomics.

Angiopoietin-1 Is an Antipermeability and Anti-Inflammatory Agent In Vitro and Targets Cell Junctions

J. Gamble, M. Vadas, G. Yancopoulos, 2000, Circulation research.

Transforming growth factor-beta inhibits E-selectin expression on human endothelial cells.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1993, Journal of immunology.

Recombinant human interleukin 5 is a selective activator of human eosinophil function

J. Gamble, M. Vadas, I. G. Young, 1988, The Journal of experimental medicine.

Chronic increases in sphingosine kinase-1 activity induce a pro-inflammatory, pro-angiogenic phenotype in endothelial cells

J. Gamble, M. Vadas, Malcolm D. Smith, 2009, Cellular & Molecular Biology Letters.

Phenoxodiol, an experimental anticancer drug, shows potent antiangiogenic properties in addition to its antitumour effects

J. Gamble, M. Vadas, J. Drew, 2006, International journal of cancer.

Assessing IRES activity in the HIF-1alpha and other cellular 5' UTRs.

G. Goodall, A. Bert, M. Vadas, 2006, RNA.

changes: inhibition by angiopoietin-1 in thrombin-induced endothelial permeability ζ Role of protein kinase C

M. Vadas, J. Drew, C. Hahn, 2013 .

Role of protein kinase Czeta in thrombin-induced endothelial permeability changes: inhibition by angiopoietin-1.

J. Gamble, M. Vadas, J. Drew, 2004, Blood.

Caveolae control the anti-inflammatory phenotype of senescent endothelial cells

J. Gamble, M. Vadas, R. Parton, 2014, Aging cell.

Synergism between membrane gangliosides and Arg-Gly-Asp-directed glycoprotein receptors in attachment to matrix proteins by melanoma cells

J. Werkmeister, M. Vadas, G. Burns, 1988, The Journal of cell biology.

Endothelial adhesiveness for blood neutrophils is inhibited by transforming growth factor-beta.

J. Gamble, M. Vadas, 1988, Science.

Novel Cytokine-independent Induction of Endothelial Adhesion Molecules Regulated by Platelet/Endothelial Cell Adhesion Molecule (CD31)

J. Gamble, M. Vadas, S. Albelda, 1997, The Journal of cell biology.

Transforming growth factor-beta 1 inhibits the production of IL-8 and the transmigration of neutrophils through activated endothelium.

William B. Smith, J. Gamble, M. Vadas, 1996, Journal of immunology.

Recombinant human granulocyte-macrophage colony-stimulating factor stimulates in vitro mature human neutrophil and eosinophil function, surface receptor expression, and survival.

J. Gamble, M. Vadas, C. Begley, 1986, The Journal of clinical investigation.

Dissociation of TNF‐alpha cytotoxic and proinflammatory activities by p55 receptor‐ and p75 receptor‐selective TNF‐alpha mutants.

William B. Smith, P. Vandenabeele, J. Gamble, 1994, The EMBO journal.

Loss of sphingosine kinase 1 predisposes to the onset of diabetes via promoting pancreatic β‐cell death in diet‐induced obese mice

M. Vadas, Y. Qi, Jinbiao Chen, 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Down‐modulation of receptors for granulocyte colony‐stimulating factor on human neutrophils by granulocyte‐activating agents

M. Vadas, N. Nicola, A. Lopez, 1986, Journal of cellular physiology.

Localization of the G-CSF gene on chromosome 17 proximal to the breakpoint in the t(15;17) in acute promyelocytic leukemia.

M. F. Shannon, M. Vadas, O. Garson, 1987 .

Granulocyte-macrophage colony-stimulating factor is a stimulant of platelet-activating factor and superoxide anion generation by human neutrophils.

M. Vadas, A. Stewart, M. DeNichilo, 1991, The Journal of biological chemistry.

Activation of granulocyte cytotoxic function by purified mouse colony-stimulating factors.

M. Vadas, A. Burgess, W. Sewell, 1983, Journal of immunology.

Nucleotide sequence of the mouse VEGF 3'UTR and quantitative analysis of sites of polyadenylation.

G. Goodall, M. Vadas, W. Risau, 2001, Biochimica et biophysica acta.

Interleukin-8 induces neutrophil transendothelial migration.

William B. Smith, J. Gamble, M. Vadas, 1991, Immunology.

H-2 gene complex restricts transfer of delayed-type hypersensitivity in mice.

J. Gamble, M. Vadas, J. Miller, 1975, Proceedings of the National Academy of Sciences of the United States of America.

Stress-induced premature senescence mediated by a novel gene, SENEX, results in an anti-inflammatory phenotype in endothelial cells.

J. Gamble, M. Vadas, R. Stocker, 2010, Blood.

Signals for activation of the GM-CSF promoter and enhancer in T cells.

M. F. Shannon, M. Vadas, P. Cockerill, 1997, Critical reviews in immunology.

High Density Lipoproteins (HDL) Interrupt the Sphingosine Kinase Signaling Pathway

J. Gamble, M. Vadas, P. Barter, 1999, The Journal of Biological Chemistry.

Interleukin-3/erythropoietin fusion proteins: in vitro effects on hematopoietic cells.

M. Vadas, P. Quesenberry, M. McMahon, 1993, Experimental hematology.

Basal and angiopoietin-1-mediated endothelial permeability is regulated by sphingosine kinase-1.

J. Gamble, M. Vadas, R. Proia, 2008, Blood.

Identification of circulating sphingosine kinase-related metabolites for prediction of type 2 diabetes

Weiqi Wang, M. Vadas, Xin Gao, 2021, Journal of translational medicine.

Tumor necrosis factor-alpha induces adhesion molecule expression through the sphingosine kinase pathway.

J. Gamble, A. Bert, M. Vadas, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Targeting miR-27a/VE-cadherin interactions rescues cerebral cavernous malformations in mice

J. Gamble, M. Vadas, Jia Li, 2020, PLoS biology.

ARHGAP18 Protects Against Thoracic Aortic Aneurysm Formation by Mitigating the Synthetic and Proinflammatory Smooth Muscle Cell Phenotype

J. Gamble, M. Vadas, R. Jeremy, 2017, Circulation research.

The VE-Cadherin/β-catenin signalling axis regulates immune cell infiltration into tumours.

J. Gamble, M. Vadas, Jia Li, 2020, Cancer letters.

ARHGAP18: A Flow‐Responsive Gene That Regulates Endothelial Cell Alignment and Protects Against Atherosclerosis

J. Gamble, M. Vadas, M. Schwartz, 2019, Journal of the American Heart Association.

Phosphorylation of cold shock domain/Y‐box proteins by ERK2 and GSK3β and repression of the human VEGF promoter

G. Goodall, A. Bert, M. Vadas, 2005, FEBS letters.

Insights into defibrotide as therapy for veno‐occlusive disease

J. Gamble, M. Vadas, 2009, Hepatology.

The effects of recombinant tumour necrosis factor (cachectin) on metabolism in isolated rat adipocyte, hepatocyte and muscle preparations.

J. Gamble, M. Vadas, A. Rofe, 1987, The Biochemical journal.

Deletion of sphingosine kinase 1 ameliorates hepatic steatosis in diet-induced obese mice: Role of PPARγ.

J. Gamble, M. Vadas, Xin Gao, 2016, Biochimica et biophysica acta.

JAM-C Induces Endothelial Cell Permeability Through Its Association and Regulation of &bgr;3 Integrins

J. Gamble, M. Vadas, M. Aurrand-Lions, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Interleukin-5, interleukin-3, and granulocyte-macrophage colony-stimulating factor cross-compete for binding to cell surface receptors on human eosinophils.

M. Vadas, M. Elliott, D. Gillis, 1991, The Journal of biological chemistry.

Purified colony-stimulating factors enhance the survival of human neutrophils and eosinophils in vitro: a rapid and sensitive microassay for colony-stimulating factors.

C. Begley, M. Vadas, N. Nicola, 1986, Blood.

Liver-specific deletion of miR-181ab1 reduces liver tumour progression via upregulation of CBX7

E. El-Omar, J. Gamble, M. Vadas, 2022, Cellular and Molecular Life Sciences.

Interferon-y Upregulates Interleukin-3 ( IL-3 ) Receptor Expression in Human Endothelial Cells and Synergizes With IL-3 in Stimulating Major Histocompatibility Complex Class I 1 Expression and Cytokine Production

William, M. Vadas, M. Dottore, 2002 .

Monoclonal antibody 7G3 recognizes the N-terminal domain of the human interleukin-3 (IL-3) receptor alpha-chain and functions as a specific IL-3 receptor antagonist.

J. Gamble, G. Goodall, M. Vadas, 1996, Blood.

Neutrophils activated by granulocyte-macrophage colony-stimulating factor express receptors for interleukin-3 which mediate class II expression.

M. Vadas, E. Korpelainen, C. Hawrylowicz, 1995, Blood.

Interferon-gamma upregulates interleukin-3 (IL-3) receptor expression in human endothelial cells and synergizes with IL-3 in stimulating major histocompatibility complex class II expression and cytokine production.

J. Gamble, M. Vadas, M. Dottore, 1995, Blood.

Human Interleukin‐3 inhibits the binding of granulocyte‐macrophage colony‐stimulating factor and interleukin‐5 to basophils and strongly enhances their functional activity

A. B. Lyons, M. Vadas, L. To, 1990, Journal of cellular physiology.

Specific binding of human interleukin-3 and granulocyte-macrophage colony-stimulating factor to human basophils.

A. B. Lyons, M. Vadas, L. To, 1990, The Journal of allergy and clinical immunology.

Recombinant human interleukin-3 and granulocyte-macrophage colony-stimulating factor show common biological effects and binding characteristics on human monocytes.

M. Vadas, L. To, M. Elliott, 1989, Blood.

A human interleukin 3 analog with increased biological and binding activities.

M. F. Shannon, S. Barry, M. Vadas, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Phosphorylation-dependent translocation of sphingosine kinase to the plasma membrane drives its oncogenic signalling

M. Vadas, B. Wattenberg, S. Pitson, 2005, The Journal of experimental medicine.

Characterization of a spontaneously transformed human endothelial cell line.

J. Gamble, M. Vadas, G. Cockerill, 1994, Laboratory investigation; a journal of technical methods and pathology.

A new assay for the measurement of the attachment of neutrophils and other cell types to endothelial cells.

J. Gamble, M. Vadas, 1988, Journal of immunological methods.

Recombinant human tumor necrosis factor-alpha. Regulation of N-formylmethionylleucylphenylalanine receptor affinity and function on human neutrophils.

M. Vadas, W. Marasco, A. Lopez, 1988, The Journal of clinical investigation.

The role of sphingosine kinase 1 in cancer: oncogene or non-oncogene addiction?

J. Gamble, M. Vadas, G. McCaughan, 2008, Biochimica et biophysica acta.

leads to ligand independence and tumorigenicity granulocyte-macrophage colony-stimulating factor, and IL-5 that A mutation of the common receptor subunit for interleukin-3 (IL-3),

M. Vadas, R. D'Andrea, J. Rayner, 2011 .

A mutation of the common receptor subunit for interleukin-3 (IL-3), granulocyte-macrophage colony-stimulating factor, and IL-5 that leads to ligand independence and tumorigenicity.

M. Vadas, R. D'Andrea, J. Rayner, 1994, Blood.

A model for the interaction of the GM-CSF, IL-3 and IL-5 receptors with their ligands.

G. Goodall, M. Vadas, C. Bagley, 1993, Growth factors.

Requirement for nuclear factor (NF)-kappa B p65 and NF-interleukin-6 binding elements in the tumor necrosis factor response region of the granulocyte colony-stimulating factor promoter.

M. F. Shannon, M. Vadas, S. Gerondakis, 1994, Blood.

Sphingosine kinase-1 enhances endothelial cell survival through a PECAM-1-dependent activation of PI-3K/Akt and regulation of Bcl-2 family members.

J. Gamble, M. Vadas, C. Hahn, 2005, Blood.

A cytokine receptor gene cluster in the X-Y pseudoautosomal region?

M. Vadas, R. D'Andrea, G. Sutherland, 1993, Blood.

Regulation of hepatic insulin signaling and glucose homeostasis by sphingosine kinase 2

J. Gamble, M. Vadas, J. Burchfield, 2020, Proceedings of the National Academy of Sciences.

Stimulation of proliferation, differentiation, and function of human cells by primate interleukin 3.

M. F. Shannon, J. Gamble, M. Vadas, 1987, Proceedings of the National Academy of Sciences of the United States of America.

GM-CSF, IL-3 and IL-5: cross-competition on human haemopoietic cells.

M. Vadas, M. Elliott, M. Elliott, 1992, Immunology today.

Interactions between human eosinophils and schistosomula of schistosoma mansoni. II. The mechanism of irreversible eosinophil adherence

M. Vadas, B. Sherry, J. David, 1979, The Journal of experimental medicine.

Enhanced helminthotoxic capacity of eosinophils from patients with eosinophilia.

M. Vadas, J. David, E. Carvalho, 1980, The New England journal of medicine.

Murine eosinophil differentiation factor. An eosinophil-specific colony- stimulating factor with activity for human cells

M. Vadas, C. Begley, D. Warren, 1986, The Journal of experimental medicine.

Eosinophil activation by colony-stimulating factor in man: metabolic effects and analysis by flow cytometry.

M. Vadas, G. Varigos, D. Metcalf, 1983, Blood.

Enhancement of human blood eosinophil cytotoxicity by semi-purified eosinophil colony-stimulating factor(s)

M. Vadas, J. David, N. Nicola, 1982, The Journal of experimental medicine.

CHAPTER 16 – Immunological Influences in Cardiovascular Disease

M. Vadas, 1985 .

A new method for the purification of human eosinophils and neutrophils, and a comparison of the ability of these cells to damage schistosomula of Schistosoma mansoni.

M. Vadas, J. David, T. A. Siongok, 1979, Journal of immunology.

Antibody-dependent eosinophil-mediated damage to schistosomula of Schistosoma mansoni: lack of requirement for oxidative metabolism.

M. Vadas, J. David, M. Robbins, 1981, Journal of immunology.

Beyond liver fibrosis: Hepatic stellate cell senescence links obesity to liver cancer by way of the microbiome

J. Gamble, M. Vadas, G. McCaughan, 2014, Hepatology.

A novel tumor necrosis factor-responsive transcription factor which recognizes a regulatory element in hemopoietic growth factor genes

M. F. Shannon, M. Vadas, M. Lenardo, 1990, Molecular and cellular biology.

Hemopoietic colony-stimulating factors.

M. Vadas, N. Nicola, Nicos A. Nicola, 1984, Immunology today.

Low fluid shear stress conditions contribute to activation of cerebral cavernous malformation signalling pathways.

J. Gamble, M. Vadas, Jia Li, 2019, Biochimica et biophysica acta. Molecular basis of disease.

Restoring endocrine response in breast cancer cells by inhibition of the sphingosine kinase-1 signaling pathway.

M. Vadas, Lijun Wang, P. Xia, 2009, Endocrinology.

Transendothelial Migration Cleaved Annexin A1 in Neutrophil A Proinflammatory Role for Proteolytically

J. Gamble, M. Vadas, Y. Khew-Goodall, 2010 .

cell differentiation marker An autoantibody reactive with nuclei of polymorphonuclear neutrophils: a

M. Vadas, S. Whittingham, G. Morstyn, 2011 .

Specific human granulocyte-macrophage colony-stimulating factor antagonists.

M. F. Shannon, M. Vadas, M. Dottore, 1994, Proceedings of the National Academy of Sciences of the United States of America.

The receptor for interleukin 3 is selectively induced in human endothelial cells by tumor necrosis factor alpha and potentiates interleukin 8 secretion and neutrophil transmigration.

J. Gamble, G. Goodall, M. Vadas, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Differential binding of IL-3 and GM-CSF to human monocytes.

M. Vadas, J. Moss, M. Dottore, 1992, Growth factors.

Reciprocal inhibition of binding between interleukin 3 and granulocyte-macrophage colony-stimulating factor to human eosinophils.

M. Vadas, S. Clark, D. Gillis, 1989, Proceedings of the National Academy of Sciences of the United States of America.

A comparison of eosinophil‐activating factor (EAF) with other monokines and lymphokines

M. Vadas, A. Butterworth, J. Taverne, 1986, European journal of immunology.

Metcalf colony-stimulating factors neutrophils and eosinophils in vitro : a rapid and sensitive microassay for Purified colony-stimulating factors enhance the survival of human

C. Begley, M. Vadas, N. Nicola, 2005 .

Transendothelial migration of neutrophils involves integrin-associated protein (CD47).

J. Gamble, M. Vadas, E. Brown, 1995, Proceedings of the National Academy of Sciences of the United States of America.

The detection and initial characterization of colony-stimulating factors in synovial fluid.

M. Vadas, C. Begley, D. Metcalf, 1988, Clinical and experimental immunology.

Biologic properties in vitro of a recombinant human granulocyte-macrophage colony-stimulating factor.

Clark, M. Vadas, C. Begley, 1986, Blood.

Nedocromil sodium and cromolyn (sodium cromoglycate) selectively inhibit antibody-dependent granulocyte-mediated cytotoxicity.

J. Gamble, M. Vadas, T. H. Rand, 1988, International archives of allergy and applied immunology.

Regulation of the adhesion of neutrophils to endothelium.

J. Gamble, M. Vadas, 1990, Biochemical pharmacology.

Newer aspects of regulation of human granulocyte function.

M. Vadas, 1984, Australian and New Zealand journal of medicine.

New frontiers for IL-5.

M. Vadas, C. Bagley, Angel Lopez, 1997, The Journal of allergy and clinical immunology.

Sphingosine kinase-1 associates with integrin {alpha}V{beta}3 to mediate endothelial cell survival.

J. Gamble, M. Vadas, C. Hahn, 2009, The American journal of pathology.

Correlation between the stimulation of human neutrophil function by monoclonal antibody and by colony-stimulating factor.

M. Vadas, N. Nicola, A. Lopez, 1985, Blood.

Eosinophil-mediated killing of schistosomula of Schistosoma mansoni: oxidative requirement for enhancement by eosinophil colony stimulating factor (CSF-alpha) and supernatants with eosinophil cytotoxicity enhancing activity (E-CEA).

M. Vadas, J. David, H. Lenzi, 1984, Cellular immunology.

Maturation In Vivo of Schistosoma mansoni Schistosomula After Culture In Vitro with Granulocytes and Antibody

M. Vadas, J. David, A. Dessein, 1983, Infection and immunity.

Studies on the Mechanisms of Immunity in Human Schistosomiasis

M. Vadas, R. Sturrock, A. Dessein, 1982, Immunological reviews.

Interactions between human eosinophils and schistosomula of Schistosoma mansoni. I. Stable and irreversible antibody-dependent adherence.

M. Vadas, B. Sherry, J. David, 1980, Journal of immunology.

Reconstitution of T Cell-Specific Transcription Directed by Composite NFAT/Oct Elements1

A. Bert, M. Vadas, P. Cockerill, 2000, The Journal of Immunology.

A T cell-specific enhancer in the interleukin-3 locus is activated cooperatively by Oct and NFAT elements within a DNase I-hypersensitive site.

A. Bert, M. Vadas, P. Cockerill, 1997, Immunity.

Parasite immunity and the major histocompatibility complex

M. Vadas, 2005, Immunogenetics.

Evidence for the control of Eosinophilia by the major histocompatibility complex in mice

M. Vadas, W. Sewell, 2004, Immunogenetics.

Synthesis and expression of the gene encoding human interleukin-3.

M. F. Shannon, M. Vadas, A. Lopez, 1989, Gene.

Carbohydrate-specific monoclonal antibodies bind to human granulocytes and stimulate antibody-dependent cellular cytotoxicity.

M. Vadas, C. Civin, V. Ginsburg, 1989, Archives of biochemistry and biophysics.

The IIb-IIIa glycoprotein complex that mediates platelet aggregation is directly implicated in leukocyte adhesion

C. Begley, J. Werkmeister, M. Vadas, 1986, Cell.

An autoantibody reactive with nuclei of polymorphonuclear neutrophils: a cell differentiation marker

M. Vadas, John W. Wilson, S. Whittingham, 1981 .

An autoantibody reactive with nuclei of polymorphonuclear neutrophils: a cell differentiation marker.

M. Vadas, S. Whittingham, J. Wilson, 1981, Blood.

SEPARATION OF HUMAN MONOCYTES AND GUINEA PIG MACROPHAGES BY DENSITY GRADIENTS OF METRIZAMIDE

M. Vadas, B. Sherry, J. David, 1981 .

CHAPTER 20 – Immunological Studies on Schistosomes Cultured in Vitro

M. Vadas, A. Butterworth, 1979 .

Synovial fluid neutrophils of patients with rheumatoid arthritis have membrane antigen changes that reflect activation.

M. Vadas, P. Emery, G. Burns, 1988, Annals of the rheumatic diseases.

Localization of the G-CSF gene on chromosome 17 proximal to the breakpoint in the t(15;17) in acute promyelocytic leukemia.

M. F. Shannon, M. Vadas, O. Garson, 1987, Blood.

The Major Histocompatibility Complex: Influence on Immune Reactivity and T‐Lymphocyte Activation

M. Vadas, J. Miller, M. Vadas, 1977, Scandinavian journal of immunology.

Sphingosine kinase transmits estrogen signaling in human breast cancer cells.

M. Vadas, Lijun Wang, S. Pitson, 2003, Molecular endocrinology.

Studies on antigen-induced arthritis in mice. III. Cell and serum transfer experiments.

I. Mackay, M. Vadas, G. Mitchell, 1977, Journal of immunology.

Residue 21 of human granulocyte‐macrophage colony‐stimulating factor is critical for biological activity and for high but not low affinity binding.

M. F. Shannon, M. Vadas, M. Dottore, 1992, The EMBO journal.

Endothelial cell adhesiveness for human T lymphocytes is inhibited by transforming growth factor-beta 1.

J. Gamble, M. Vadas, 1991, Journal of immunology.

P-selectin interacts with a beta 2-integrin to enhance phagocytosis.

M. Vadas, M. Berndt, D. Cooper, 1994, Journal of immunology.

Role of major histocompatibility complex gene products in delayed-type hypersensitivity.

J. Gamble, M. Vadas, J. Miller, 1976, Proceedings of the National Academy of Sciences of the United States of America.

Recombinant interleukin 4 stimulates human immunodeficiency virus production by infected monocytes and macrophages.

M. Vadas, T. Sorrell, A. Cunningham, 1992, The Journal of general virology.

The effects of markedly raised intracellular sphingosine kinase-1 activity in endothelial cells

J. Gamble, M. Vadas, V. Limaye, 2009, Cellular & Molecular Biology Letters.

Regulation of human eosinophil production and function by interleukin-5.

M. F. Shannon, M. Vadas, A. Lopez, 1992, Immunology series.

Sphingosine Kinase-1 Associates with Integrin (cid:1) V (cid:2) 3 to Mediate Endothelial Cell Survival

J. Gamble, M. Vadas, C. Hahn, 2009 .

Cytokines and allergy.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1994, The Journal of allergy and clinical immunology.

Estrogen transactivates EGFR via the sphingosine 1-phosphate receptor Edg-3: the role of sphingosine kinase-1

Andrew S. Holmes, A. Ullrich, M. Vadas, 2006, The Journal of cell biology.

Nuclear proteins interacting with the promoter region of the human granulocyte/macrophage colony-stimulating factor gene.

M. F. Shannon, J. Gamble, M. Vadas, 1988, Proceedings of the National Academy of Sciences of the United States of America.

A radioisotopic method to measure delayed type hypersensitivity in the mouse. I. Studies in sensitized and normal mice.

J. Gamble, M. Vadas, J. Miller, 1975, International archives of allergy and applied immunology.

Studies on the mechanism of the enhancement of delayed-type hypersensitivity by pertussigen.

M. Vadas, W. Sewell, R. Scollay, 1984, Journal of immunology.

Role of cell surface receptors in the regulation of intracellular killing of bacteria by murine peritoneal exudate neutrophils

M. Vadas, P. McDonald, P. Hart, 1986, Infection and immunity.

[Antigen-induced arthritis in mice: genetic and immunologic studies].

M. Vadas, J. Miller, G. Mitchell, 1978, Verhandlungen der Deutschen Gesellschaft fur Rheumatologie.

Pertussigen enhances antigen-driven interferon-γ production by sensitized lymphoid cells

M. Vadas, W. Sewell, J. McKimm-Breschkin, 1986 .

T-cell functional regions of the human IL-3 proximal promoter.

M. F. Shannon, M. Vadas, G. Ryan, 1994, Molecular reproduction and development.

Novel mechanisms of the transendothelial migration of leukocytes.

M. Vadas, B. Engelhardt, B. Imhof, 2001, Trends in immunology.

Human tumournecrosis factor-alpha (TNF-a)directly stimulates arachidonic acidrelease inhumanneutrophils

M. Vadas, A. W. Murray, Y. Atkinson, 1990 .

Mononuclear cell-mediated enhancement of granulocyte function in man.

M. Vadas, N. Nicola, G. Johnson, 1984, Journal of immunology.

Human interleukin 4 regulates the phenotype of lymphocytes generated during mixed lymphocyte culture and inhibits the IL-2-induced development of LAK function in normal and leukaemic cells.

M. Vadas, G. Burns, C. Juttner, 1989, Leukemia research.

Activation of antibody-dependent cell-mediated cytotoxicity of human neutrophils and eosinophils by separate colony-stimulating factors.

M. Vadas, N. Nicola, D. Metcalf, 1983, Journal of immunology.

Two contiguous residues in human interleukin-3, Asp21 and Glu22, selectively interact with the alpha- and beta-chains of its receptor and participate in function.

M. F. Shannon, G. Goodall, S. Barry, 1994, The Journal of biological chemistry.

Adhesion protein GMP140 inhibits superoxide anion release by human neutrophils.

J. Gamble, M. Vadas, C. S. Wong, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Human myeloid growth factors

M. Vadas, Y. Atkinson, M. Shannon, 1987, Immunology and cell biology.

Identification of a human granulocyte functional antigen (GFA-2) involved in antibody-dependent cell-mediated cytotoxicity and phagocytosis.

M. Vadas, A. Butterworth, A. Lopez, 1985, Journal of immunology.

Selective enhancement of the expression of granulocyte functional antigens 1 and 2 on human neutrophils.

M. Vadas, D. Williamson, A. Lopez, 1985, Proceedings of the National Academy of Sciences of the United States of America.

The clonal proliferation in vitro of enriched populations of human promyelocytes and myelocytes.

C. Begley, M. Vadas, D. Metcalf, 1985, Blood.

Regulation of human granulocyte function by products derived from murine tumors.

M. Vadas, I. Clark‐Lewis, 1985, Experimental hematology.

Stimulation of human granulocyte function by monoclonal antibody WEM-G1.

M. Vadas, A. Lopez, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Inhibition of human monocyte adhesion by interleukin-4.

J. Gamble, M. Vadas, M. Elliott, 1991, Blood.

Role of colony-stimulating factors in leucocyte responses to inflammation and infection.

J. Gamble, M. Vadas, A. Lopez, 1991, Current opinion in immunology.

Enhancement of the intensity, persistence, and passive transfer of delayed-type hypersensitivity lesions by pertussigen in mice

M. Vadas, W. Sewell, J. Munoz, 1983, The Journal of experimental medicine.

Localization of the human G-CSF gene to the region of a breakpoint in the translocation typical of acute promyelocytic leukemia

M. F. Shannon, M. Vadas, G. Sutherland, 1988, Human Genetics.

Sphingosine kinase interacts with TRAF2 and dissects tumor necrosis factor-alpha signaling.

J. Gamble, M. Vadas, R. D'Andrea, 2002, The Journal of biological chemistry.

Regulation of human monocyte adherence by granulocyte-macrophage colony-stimulating factor.

J. Gamble, M. Vadas, M. Elliott, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Mechanism of the interaction mediating killing of Schistosoma mansoni by human eosinophils.

M. Vadas, J. David, A. Butterworth, 1980, The American journal of tropical medicine and hygiene.

Peptide insertions in domain 4 of hbeta(c), the shared signalling receptor subunit for GM-CSF, IL3 and IL5, induce ligand-independent activation.

S. Barry, M. Vadas, R. D'Andrea, 2001, Cytokine.

Recruitment and Activation of SHP-1 Protein-tyrosine Phosphatase by Human Platelet Endothelial Cell Adhesion Molecule-1 (PECAM-1)

D. Jackson, J. Gamble, M. Vadas, 1998, The Journal of Biological Chemistry.

Roles of the N and C terminal domains of the interleukin-3 receptor alpha chain in receptor function.

G. Goodall, S. Barry, M. Vadas, 1997, Blood.

Functional studies on purified eosinophils and neutrophils from patients with Schistosoma mansoni infections.

M. Vadas, J. David, R. Sturrock, 1980, Clinical and experimental immunology.

Genetic control of eosinophilia in mice: gene(s) expressed in bone marrow-derived cells control high responsiveness.

M. Vadas, 1982, Journal of immunology.

In vitro enhancement of the helminthotoxic capacity of human blood eosinophils.

M. Vadas, J. David, A. Dessein, 1981, The Australian journal of experimental biology and medical science.

Cyclophosphamide pretreatment induces eosinophilia to nonparasite antigens.

M. Vadas, 1981, Journal of immunology.

Regulation of Eosinophil Function by P-Selectin

J. Gamble, M. Vadas, Angel F. Lopez, 2019, Eosinophils in Allergy and Inflammation.

Identification of residues in the first and fourth helices of human granulocyte-macrophage colony-stimulating factor involved in biologic activity and in binding to the alpha- and beta-chains of its receptor.

M. F. Shannon, M. Vadas, M. Dottore, 1994, Blood.

Stimulation of neutrophils by tumor necrosis factor.

J. Gamble, M. Vadas, S. Klebanoff, 1986, Journal of immunology.

Acquisition of murine major histocompatibility complex gene products by schistosomula of Schistosoma mansoni

B. F. Hall, A. Sher, M. Vadas, 1978, The Journal of experimental medicine.

The cryptome: a subset of the proteome, comprising cryptic peptides with distinct bioactivities.

M. Vadas, L. Ilag, A. Smith, 2006, Drug discovery today.

Short course of systemic corticosteroids in sinonasal polyposis: A double‐blind, randomized, placebo‐controlled trial with evaluation of outcome measures

M. Vadas, P. Hissaria, D. Gillis, 2006, The Journal of allergy and clinical immunology.

Short course of systemic corticosteroids in sinonasal polyposis: A double‐blind, randomized, placebo‐controlled trial with evaluation of outcome measures

M. Vadas, P. Hissaria, D. Gillis, 2008, The Journal of allergy and clinical immunology.

Activation of Sphingosine Kinase by Tumor Necrosis Factor-α Inhibits Apoptosis in Human Endothelial Cells*

J. Gamble, M. Vadas, Lijun Wang, 1999, The Journal of Biological Chemistry.

Ly and Ia antigen phenotypes of T cells involved in delayed-type hypersensitivity and in suppression

J. Gamble, M. Vadas, J. Miller, 1976, The Journal of experimental medicine.

Stimulation of the adherence of neutrophils to umbilical vein endothelium by human recombinant tumor necrosis factor.

J. Gamble, M. Vadas, J. Harlan, 1985, Proceedings of the National Academy of Sciences of the United States of America.

Sphingosine Kinase Interacts with TRAF2 and Dissects Tumor Necrosis Factor-α Signaling*

J. Gamble, M. Vadas, Lijun Wang, 2002, The Journal of Biological Chemistry.

Phospholipid composition of reconstituted high density lipoproteins influences their ability to inhibit endothelial cell adhesion molecule expression.

J. Gamble, M. Vadas, P. Barter, 2000, Journal of lipid research.

Factors influencing the ability of HDL to inhibit expression of vascular cell adhesion molecule-1 in endothelial cells.

J. Gamble, M. Vadas, P. Barter, 1998, Arteriosclerosis, thrombosis, and vascular biology.

High-density lipoproteins inhibit cytokine-induced expression of endothelial cell adhesion molecules.

J. Gamble, M. Vadas, P. Barter, 1995, Arteriosclerosis, thrombosis, and vascular biology.

Regulation of granulocyte-macrophage colony-stimulating factor and E- selectin expression in endothelial cells by cyclosporin A and the T- cell transcription factor NFAT

J. Gamble, A. Bert, M. Vadas, 1995 .

Hypoxic regulation of vascular endothelial growth factor mRNA stability requires the cooperation of multiple RNA elements.

G. Goodall, M. Vadas, W. Risau, 1999, Molecular biology of the cell.

Regulation of vascular leak and recovery from ischemic injury by general and VE-cadherin-restricted miRNA antagonists of miR-27.

J. Kench, J. Gamble, N. Tran, 2013, Blood.

miR‐181a mediates TGF‐β‐induced hepatocyte EMT and is dysregulated in cirrhosis and hepatocellular cancer

J. Gamble, M. Vadas, G. McCaughan, 2015, Liver international : official journal of the International Association for the Study of the Liver.

A Proinflammatory Role for Proteolytically Cleaved Annexin A1 in Neutrophil Transendothelial Migration

Samantha L. Williams, J. Gamble, M. Vadas, 2010, The Journal of Immunology.

Characterization of GMP-140 (P-selectin) as a circulating plasma protein

J. Gamble, M. Vadas, J. Gorman, 1992, The Journal of experimental medicine.

cell transcription factor NFAT T- E- selectin expression in endothelial cells by cyclosporin A and the Regulation of granulocyte-macrophage colony-stimulating factor and

J. Gamble, A. Bert, M. Vadas, 2011 .

Regulation of granulocyte-macrophage colony-stimulating factor and E-selectin expression in endothelial cells by cyclosporin A and the T-cell transcription factor NFAT.

J. Gamble, A. Bert, M. Vadas, 1995, Blood.

Angiogenesis: models and modulators.

J. Gamble, M. Vadas, G. Cockerill, 1995, International review of cytology.

ARHGAP18: an endogenous inhibitor of angiogenesis, limiting tip formation and stabilizing junctions

J. Gamble, M. Vadas, T. Becker, 2014, Small GTPases.

Cytokines increase human hemopoietic cell adhesiveness by activation of very late antigen (VLA)-4 and VLA-5 integrins

M. Vadas, D. Leavesley, J. Lévesque, 1995, The Journal of experimental medicine.

IL‐3 receptor expression, regulation and function in cells of the Vasculature.

J. Gamble, M. Vadas, E. Korpelainen, 1996, Immunology and cell biology.

Age-associated stresses induce an anti-inflammatory senescent phenotype in endothelial cells

J. Gamble, M. Vadas, Gábor Hutás, 2013, Aging.

The aging endothelium

J. Gamble, M. Vadas, K. Ting, 2021, Vascular biology.

Transmigration of Leukocytes

M. Vadas, Y. Khew-Goodall, B. Stein, 2001 .

The vascular endothelial growth factor mRNA contains an internal ribosome entry site

G. Goodall, M. Vadas, W. Risau, 1998, FEBS letters.

Active subnetwork recovery with a mechanism-dependent scoring function; with application to angiogenesis and organogenesis studies

Tibério S. Caetano, Michael A. Charleston, Ilana Lichtenstein, 2013, BMC Bioinformatics.

Lack of effect of serum amyloid A (SAA) on the ability of high-density lipoproteins to inhibit endothelial cell adhesion molecule expression.

J. Gamble, M. Vadas, P. Barter, 2001, Atherosclerosis.

Sphingosine kinase 1 is a critical component of the copper-dependent FGF1 export pathway.

M. Vadas, R. Soldi, A. Mandinova, 2007, Experimental cell research.

Dysregulated hematopoiesis and a progressive neurological disorder induced by expression of an activated form of the human common beta chain in transgenic mice.

M. Vadas, T. Gonda, J. Finnie, 1998, The Journal of clinical investigation.

Interleukin 4 is at 5q31 and interleukin 6 is at 7p15

M. F. Shannon, M. Vadas, G. Sutherland, 1988, Human Genetics.

Activation of sphingosine kinase 1 by ERK1/2‐mediated phosphorylation

M. Vadas, B. Wattenberg, S. Pitson, 2003, The EMBO journal.

High-Density Lipoproteins Neutralize C-Reactive Protein Proinflammatory Activity

J. Gamble, M. Vadas, P. Barter, 2004, Circulation.

A novel mechanism of repression of the vascular endothelial growth factor promoter, by single strand DNA binding cold shock domain (Y-box) proteins in normoxic fibroblasts.

Gregory J Goodall, Peter Diamond, G. Goodall, 2002, Nucleic acids research.

Immune evasion by Schistosoma mansoni: loss of susceptibility to antibody or complement-dependent eosinophil attack by schistosomula cultured in medium free of macromolecules

M. Vadas, J. Samuelson, B. Sherry, 1981, Parasitology.

Selective inhibition of the induction phase of delayed type hypersensitivity in mice by niridazole.

M. Vadas, C. Bernard, 1981, Clinical immunology and immunopathology.

Cytolytic T lymphocytes recognize alloantigens on schistosomula of Schistosoma mansoni, but fail to induce damage.

A. Sher, M. Vadas, E. Martz, 1979, Journal of immunology.

Eosinophilia and acquisition of resistance to Nematospiroides dubius in mice sensitized with adult worms.

M. Vadas, J. Hurley, 1983, The Australian journal of experimental biology and medical science.

TGF-beta and endothelial cells inhibit VCAM-1 expression on human vascular smooth muscle cells.

J. Gamble, M. Vadas, L. Noack, 1995, Arteriosclerosis, thrombosis, and vascular biology.

Histocompatibility linked immune responsiveness and restrictions imposed on sensitized lymphocytes

J. Gamble, M. Vadas, J. Miller, 1977, The Journal of experimental medicine.

Prevention of activated neutrophil adhesion to endothelium by soluble adhesion protein GMP140.

J. Gamble, M. Vadas, M. Skinner, 1990, Science.

The transforming growth factor family and the endothelium

J. Gamble, M. Vadas, P. Xia, 2001 .

B1 integrin activation inhibits in vitro tube formation: effects on cell migration, vacuole coalescence and lumen formation.

J. Gamble, M. Vadas, J. Furze, 1999, Endothelium : journal of endothelial cell research.

Regulation of in vitro capillary tube formation by anti-integrin antibodies

J. Gamble, M. Vadas, G. Russ, 1993, The Journal of cell biology.

The Poly-cistronic miR-23-27-24 Complexes Target Endothelial Cell Junctions: Differential Functional and Molecular Effects of miR-23a and miR-23b

J. Gamble, M. Vadas, W. Ritchie, 2016, Molecular therapy. Nucleic acids.

The Poly-cistronic miR-23-27-24 Complexes Target Endothelial Cell Junctions: Differential Functional and Molecular Effects of miR-23a and miR-23b.

J. Gamble, M. Vadas, W. Ritchie, 2016, Molecular therapy. Nucleic acids.

promyelocytes and myelocytes The clonal proliferation in vitro of enriched populations of human

C. Begley, M. Vadas, Angel F. Lopez, 2011 .

Cold shock domain proteins repress transcription from the GM-CSF promoter.

M. F. Shannon, M. Vadas, L. Coles, 1996, Nucleic acids research.

The Human IL-3 Locus Is Regulated Cooperatively by Two NFAT-Dependent Enhancers That Have Distinct Tissue-Specific Activities1

M. Vadas, P. Cockerill, A. Hawwari, 2002, The Journal of Immunology.

Activities Enhancers That Have Distinct Tissue-Specific Cooperatively by Two NFAT-Dependent The Human IL-3 Locus Is Regulated

M. Vadas, P. Cockerill, A. Hawwari, 2002 .

An Ordered Array of Cold Shock Domain Repressor Elements across Tumor Necrosis Factor-responsive Elements of the Granulocyte-Macrophage Colony-stimulating Factor Promoter*

M. F. Shannon, M. Vadas, L. Coles, 2000, The Journal of Biological Chemistry.

The human granulocyte-macrophage colony-stimulating factor gene is autonomously regulated in vivo by an inducible tissue-specific enhancer.

A. Bert, M. Vadas, P. Cockerill, 1999, Proceedings of the National Academy of Sciences of the United States of America.

The granulocyte-macrophage colony-stimulating factor/interleukin 3 locus is regulated by an inducible cyclosporin A-sensitive enhancer.

M. F. Shannon, A. Bert, M. Vadas, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Recombinant human interleukin-3 stimulation of hematopoiesis in humans: loss of responsiveness with differentiation in the neutrophilic myeloid series.

M. Vadas, L. To, M. Elliott, 1988, Blood.

M. F. Shannon, M. Vadas, M. Dottore, 1994, Blood.

A multi-protein complex containing cold shock domain (Y-box) and polypyrimidine tract binding proteins forms on the vascular endothelial growth factor mRNA. Potential role in mRNA stabilization.

G. Goodall, A. Bert, M. Vadas, 2004, European journal of biochemistry.

The Protein Tyrosine Phosphatase Pez Is a Major Phosphatase of Adherens Junctions and Dephosphorylates β-Catenin

J. Gamble, M. Vadas, Y. Khew-Goodall, 2003 .

The solution structure of the cytokine-binding domain of the common beta-chain of the receptors for granulocyte-macrophage colony-stimulating factor, interleukin-3 and interleukin-5.

M. Vadas, G. Booker, T. Mulhern, 2000, Journal of molecular biology.

transcriptional and posttranscriptional level Human interleukin-3 mRNA accumulation is controlled at both the

M. F. Shannon, M. Vadas, Angel F. Lopez, 2011 .

Chronic expression of P-selectin on endothelial cells stimulated by the T-cell cytokine, interleukin-3.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1996, Blood.

Human interleukin-3 mRNA accumulation is controlled at both the transcriptional and posttranscriptional level.

M. F. Shannon, M. Vadas, A. Lopez, 1991, Blood.

A splice-isoform of vesicle-associated membrane protein-1 (VAMP-1) contains a mitochondrial targeting signal.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1998, Molecular biology of the cell.

Extracellular truncations of h beta c, the common signaling subunit for interleukin-3 (IL-3), granulocyte-macrophage colony-stimulating factor (GM-CSF), and IL-5, lead to ligand-independent activation.

G. Goodall, S. Barry, M. Vadas, 1996, Blood.

Endothelial adhesion molecules in atherogenesis. A concerto or a solo?

J. Gamble, M. Vadas, 1996, Circulation research.

Roles of the N and C Terminal Domains of the Interleukin-3 Receptor α Chain in Receptor Function

S. Barry, M. Vadas, Angel F. Lopez, 1997 .

Human Granulocyte-Macrophage Colony-Stimulating Factor Enhancer Function Is Associated with Cooperative Interactions between AP-1 and NFATp/c

M. F. Shannon, A. Bert, M. Vadas, 1994 .

Lumen formation during angiogenesis in vitro involves phagocytic activity, formation and secretion of vacuoles, cell death, and capillary tube remodelling by different populations of endothelial cells

J. Gamble, M. Vadas, Geoffrey T. Meyer, 1997, The Anatomical record.

Regulation by the H-2 gene complex of delayed type hypersensitivity

J. Gamble, M. Vadas, A. Whitelaw, 1977, Immunogenetics.

Genetic and T-lymphocyte influences on eosinophilia

M. Vadas, W. Sewell, 1984 .

Cold Shock Domain Factors Activate the Granulocyte-Macrophage Colony-stimulating Factor Promoter in Stimulated Jurkat T Cells*

M. Vadas, M. Shannon, L. Coles, 2001, The Journal of Biological Chemistry.

Eukaryotic Elongation Factor 1A Interacts with Sphingosine Kinase and Directly Enhances Its Catalytic Activity*

M. Vadas, S. Pitson, P. Moretti, 2008, Journal of Biological Chemistry.

TGF-β and Endothelial Cells Inhibit VCAM-1 Expression on Human Vascular Smooth Muscle Cells

J. Gamble, M. Vadas, L. Noack, 1995 .

The Tumour Vasculature as a Target to Modulate Leucocyte Trafficking

J. Gamble, M. Vadas, Y. Qi, 2021, Cancers.

PPARγ Agonists Ameliorate Endothelial Cell Activation via Inhibition of Diacylglycerol–Protein Kinase C Signaling Pathway: Role of Diacylglycerol Kinase

J. Gamble, M. Vadas, V. Chatterjee, 2004, Circulation research.

Structure-Function Studies of Lymphokines by Total Chemical Synthesis

L. Hood, M. Vadas, S. Kent, 1987 .

The Calmodulin-binding Site of Sphingosine Kinase and Its Role in Agonist-dependent Translocation of Sphingosine Kinase 1 to the Plasma Membrane*

M. Vadas, C. Bagley, S. Pitson, 2006, Journal of Biological Chemistry.

The Nucleotide-binding Site of Human Sphingosine Kinase 1*

M. Vadas, C. Bagley, B. Wattenberg, 2002, The Journal of Biological Chemistry.

Human sphingosine kinase: purification, molecular cloning and characterization of the native and recombinant enzymes.

J. Gamble, M. Vadas, L. Vandeleur, 2000, The Biochemical journal.

IL-3 and granulocyte-macrophage colony-stimulating factor stimulate two distinct phases of adhesion in human monocytes.

J. Gamble, M. Vadas, M. Elliott, 1990, Journal of immunology.

Transcriptional regulation of mouse granulocyte-macrophage colony-stimulating factor/IL-3 locus.

Cameron S. Osborne, M. Vadas, C. Osborne, 1995, Journal of immunology.

Ability of reconstituted high density lipoproteins to inhibit cytokine-induced expression of vascular cell adhesion molecule-1 in human umbilical vein endothelial cells.

J. Gamble, M. Vadas, P. Barter, 1999, Journal of lipid research.

A point mutant of human sphingosine kinase 1 with increased catalytic activity

M. Vadas, B. Wattenberg, P. Moretti, 2001, FEBS letters.

Activation of the Sphingosine Kinase–Signaling Pathway by High Glucose Mediates the Proinflammatory Phenotype of Endothelial Cells

Andrew S. Holmes, J. Gamble, M. Vadas, 2005, Circulation research.

Time sequence of the inhibition of endothelial adhesion molecule expression by reconstituted high density lipoproteins.

J. Gamble, M. Vadas, P. Barter, 2001, Atherosclerosis.

Immune reconstitution following peripheral blood stem cell autografting.

M. Vadas, L. To, C. Juttner, 1987, Bone Marrow Transplantation.

Sphingosine kinase regulates the rate of endothelial progenitor cell differentiation.

J. Gamble, M. Vadas, R. Proia, 2009, Blood.

Expression of a Catalytically Inactive Sphingosine Kinase Mutant Blocks Agonist-induced Sphingosine Kinase Activation

J. Gamble, M. Vadas, B. Wattenberg, 2000, The Journal of Biological Chemistry.

Regulation of haematopoiesis by growth factors – emerging insights and therapies

M. Vadas, Daniel Thomas, Angel Lopez, 2004, Expert opinion on biological therapy.

Primitive human hematopoietic progenitors adhere to P-selectin (CD62P).

A. Zannettino, M. Vadas, E. Butcher, 1995, Blood.

A vascular cell-restricted RhoGAP, p73RhoGAP, is a key regulator of angiogenesis.

J. Gamble, G. Goodall, M. Vadas, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Sinusoidal endothelium is essential for liver regeneration

J. Gamble, M. Vadas, G. McCaughan, 2011, Hepatology.

MicroRNA-199 a / b-3 p : A New Star in the Liver

J. Gamble, M. Vadas, G. McCaughan, 2011 .

Mechanisms of eosinophil mediated helminthotoxicity.

M. Vadas, K. Austen, J. David, 1980 .

Lysosomes in immunity to schistosomes and other helminths.

A. Sher, M. Vadas, J. David, 1979, Frontiers of biology.

Letter to the Editor: 1H and 15N chemical shift assignments for domain 4 of the common β-chain of the IL-3, IL-5 and GM-CSF receptors

M. Vadas, G. Booker, T. Mulhern, 1999 .

A Discontinuous Eight-Amino Acid Epitope in Human Interleukin-3 Binds the α-Chain of Its Receptor*

M. Vadas, C. Bagley, B. Cambareri, 1996, The Journal of Biological Chemistry.

Stat6 activation is essential for interleukin-4 induction of P-selectin transcription in human umbilical vein endothelial cells.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1999, Arteriosclerosis, thrombosis, and vascular biology.

Anti-Inflammatory Properties of High Density Lipoproteins

J. Gamble, M. Vadas, P. Barter, 2002 .

Differential Regulation of the Stability of Cytokine mRNAs in Lipopolysaccharide-activated Blood Monocytes in Response to Interleukin-10*

Gregory J. Goodall, Mathew A. Vadas, G. Goodall, 1996, The Journal of Biological Chemistry.

Regulation of adhesion and adhesion molecules in endothelium by transforming growth factor-beta.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1993, Current topics in microbiology and immunology.

Studies on antigen-induced arthritis in mice. II. Immunologic correlates of arthritis susceptibility in mice.

I. Mackay, M. Vadas, J. Miller, 1977, Journal of immunology.

Interleukin 4 and human immunodeficiency virus stimulate LFA-1-ICAM-1-mediated aggregation of monocytes and subsequent giant cell formation.

M. Vadas, A. Cunningham, F. Kazazi, 1994, The Journal of general virology.

Translocation of protein tyrosine phosphatase Pez/PTPD2/PTP36 to the nucleus is associated with induction of cell proliferation.

J. Gamble, M. Vadas, Y. Khew-Goodall, 2000, Journal of cell science.

Tnf modulation of endothelial and neutrophil adhesion

J. Gamble, M. Vadas, William B Smith, 1992 .

The vascular endothelium : structure and function

M. Vadas, V. Limaye, 2022 .

Chemotactic desensitization of neutrophils demonstrates interleukin-8 (IL-8)-dependent and IL-8-independent mechanisms of transmigration through cytokine-activated endothelium.

J. Gamble, M. Vadas, W. B. Smith, 1993, Immunology.

New therapeutic targets in vascular biology

M. Vadas, B. Imhof, 2003, Thrombosis and Haemostasis.

Therapeutic regulation of VE-cadherin with a novel oligonucleotide drug for diabetic eye complications using retinopathy mouse models

J. Gamble, M. Vadas, M. Gillies, 2018, Diabetologia.

Deletion of sphingosine kinase 1 inhibits liver tumorigenesis in diethylnitrosamine-treated mice

J. Gamble, M. Vadas, D. Kaczorowski, 2018, Oncotarget.

TGF beta down-regulates TLiSA1 expression and inhibits the differentiation of precursor lymphocytes into CTL and LAK cells.

M. Vadas, G. Burns, J. L. Scott, 1989, Immunology.

The protein tyrosine phosphatase Pez is a major phosphatase of adherens junctions and dephosphorylates beta-catenin.

J. Gamble, M. Vadas, Y. Khew-Goodall, 2003, Molecular biology of the cell.

The RhoGAP protein ARHGAP18/SENEX localizes to microtubules and regulates their stability in endothelial cells

J. Gamble, M. Vadas, M. Kavallaris, 2017, Molecular biology of the cell.

The CCT/TRiC chaperonin is required for maturation of sphingosine kinase 1.

M. Vadas, B. Wattenberg, S. Pitson, 2009, The international journal of biochemistry & cell biology.

An oncogenic role of sphingosine kinase

J. Gamble, M. Vadas, B. Wattenberg, 2000, Current Biology.

Antigen-initiated B lymphocyte differentiation. IX. Characterization of memory AFC progenitors by buoyant density and sedimentation velocity separation.

R. Schlegel, M. Vadas, M. Howard, 1977, Journal of immunology.

Differences between virgin and memory IgM-antibody-forming-cell precursor B-cells, and correlations with the heterogeneity present in B-cell populations from unimmunized mice.

M. Vadas, J. Schrader, 1976, Cellular immunology.

YAP and the RhoC regulator ARHGAP18, are required to mediate flow-dependent endothelial cell alignment

J. Gamble, M. Vadas, Jia Li, 2020, Cell communication and signaling : CCS.

Comparison of the ability of eosinophils and neutrophils, and of eosinophils from patients with S. mansoni infection and normal individuals, to mediate in vitro damage to schistosomula of S. mansoni.

M. Vadas, J. David, A. Butterworth, 1979, Advances in experimental medicine and biology.

Human tumour necrosis factor-alpha (TNF-alpha) directly stimulates arachidonic acid release in human neutrophils.

M. Vadas, A. W. Murray, A. Lopez, 1990, Immunology.

Three essential promoter elements mediate tumour necrosis factor and interleukin-1 activation of the granulocyte-colony stimulating factor gene.

M. F. Shannon, G. Goodall, M. Vadas, 1992, Growth factors.

Regulation de l'expression du gene gm-csf

M. Vadas, M. Shannon, 1988 .

Dissociation of TNF- cytotoxic and proinflammatory activities by p55 and p75 receptor-selective TNF- mutants

William B. Smith, P. Vandenabeele, J. Gamble, 1994 .

A radioisotopic method to measure delayed type hypersensitivity in the mouse. II. Cell transfer studies.

J. Gamble, M. Vadas, A. Whitelaw, 1975, International archives of allergy and applied immunology.

Restriction by H-2 gene complex of the transfer of delayed type hypersensitivity in mice.

M. Vadas, J. Miller, 1976, Advances in Experimental Medicine and Biology.

H-2 LINKED Ir GENE REGULATION OF DELAYED-TYPE HYPERSENSITIVITY IN MICE

J. Gamble, M. Vadas, A. Whitelaw, 1976 .

REGULATION OF LEUCOCYTE‐ENDOTHELIAL INTERACTIONS OF SPECIAL RELEVANCE TO ATHEROGENESIS

J. Gamble, M. Vadas, P. Barter, 1997, Clinical and experimental pharmacology & physiology.

An assay for sphingosine kinase activity using biotinylated sphingosine and streptavidin-coated membranes.

M. Vadas, C. Derian, S. Pitson, 2004, Analytical biochemistry.

Endothelial cell senescence-understanding ageing and disease

J. Gamble, M. Vadas, Yanjun Qi, 2023, Current Opinion in Physiology.

Antigen activation of T lymphocytes: influence of major histocompatibility complex.

M. Vadas, J. Miller, 1977, Cold Spring Harbor symposia on quantitative biology.

Establishment of a Human Umbilical Vein Endothelial Cell Line with Normal Functional Capabilities

M. Vadas, C. Halbert, P. Kaur, 1994 .

restricted miRNA antagonists of miR-27 - general and VE-cadherin Regulation of vascular leak and recovery from ischemic injury by

J. Kench, N. Tran, G. Goodall, 2013 .

Targeting Vascular Endothelial-Cadherin in Tumor-Associated Blood Vessels Promotes T-cell-Mediated Immunotherapy.

J. Gamble, M. Vadas, E. Dejana, 2017, Cancer research.

A granulocyte-colony-stimulating factor gene promoter element responsive to inflammatory mediators is functionally distinct from an identical sequence in the granulocyte-macrophage colony-stimulating factor gene.

M. F. Shannon, M. Vadas, E. Kuczek, 1991, Journal of immunology.

Recombinant human granulocyte-macrophage colony-stimulating factor (rH GM-CSF) regulates f Met-Leu-Phe receptors on human neutrophils.

M. Vadas, W. Marasco, G. Burns, 1988, Immunology.

The role of granulocyte-macrophage and granulocyte colony-stimulating factors in neutrophil transendothelial migration: comparison with interleukin-8.

J. Gamble, M. Vadas, W. B. Smith, 1994, Experimental hematology.

Potentiation of delayed-type hypersensitivity by pertussigen or cyclophosphamide with release of different lymphokines.

I. Mackay, M. Vadas, W. Sewell, 1987, Immunology.

Structure-function studies of human granulocyte-macrophage colony-stimulating factor. Identification of residues required for activity.

M. Vadas, S. Kent, L. To, 1988, Journal of immunology.

Depression of immunity to Naegleria fowleri in mice by selective depletion of neutrophils with a monoclonal antibody

M. Vadas, A. Ferrante, A. Lopez, 1988, Infection and immunity.

Human granulocyte-macrophage colony-stimulating factor enhancer function is associated with cooperative interactions between AP-1 and NFATp/c

A. Bert, M. Vadas, P. Cockerill, 1995, Molecular and cellular biology.

A sequence-specific single-strand DNA binding protein that contacts repressor sequences in the human GM-CSF promoter.

M. Vadas, L. Coles, F. Occhiodoro, 1994, Nucleic acids research.

Fcreceptors on mouse neutrophils andeosinophils: antigenic characteristics, isotype specificity andrelative cell membrane density measured byflow cytometry

M. Vadas, F. Battye, Angel Lopez, 1984 .

Delayed hypersensitivity induced by anti-T-cell-line antisera is enhanced by pertussigen and not restricted by histocompatibility genes.

J. Gamble, M. Vadas, W. Thomas, 1983, Proceedings of the National Academy of Sciences of the United States of America.

Multiple projection of the visual field to the medial portion of the dorsal lateral geniculate nucleus and the adjacent nuclei of the thalamus of the cat

M. Vadas, M. Vadas, P. O. Bishop, 1969, The Journal of comparative neurology.

Transforming growth factor-beta inhibits E-selectin expression on human endothelial cells.

J. Gamble, M. Vadas, Y. Khew-Goodall, 1993, The Journal of Immunology.

JAM-C Induces Endothelial Cell Permeability Through Its Association and Regulation of &bgr;3 Integrins

J. Gamble, M. Vadas, M. Aurrand-Lions, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Activation of Endothelial Extracellular Signal-Regulated Kinase Is Essential for Neutrophil Transmigration: Potential Involvement of a Soluble Neutrophil Factor in Endothelial Activation 1

J. Gamble, M. Vadas, Y. Khew-Goodall, 2003, The Journal of Immunology.

Fc receptors on mouse neutrophils and eosinophils: antigenic characteristics, isotype specificity and relative cell membrane density measured by flow cytometry.

M. Vadas, A. Lopez, F. Battye, 1985, Immunology.

Deletion of sphingosine kinase 1 ameliorates hepatic steatosis in diet-induced obese mice: Role of PPARγ.

J. Gamble, M. Vadas, Xin Gao, 2016, Biochimica et biophysica acta.

Cytokines increase human hemopoietic cell adhesiveness by activation of very late antigen (VLA)-4 and VLA-5 integrins

M. Vadas, Jean-Pierre Lévesque, David I. Leavesley, 1995, The Journal of experimental medicine.

Heterogeneity of recombinant granulocyte-macrophage colony-stimulating factor-mediated enhancement of neutrophil adherence to endothelium.

J. Gamble, M. Vadas, T. H. Rand, 1990, Experimental hematology.

Major histocompatibility complex gene products on macrophages influence T cell activation.

J. Gamble, M. Vadas, P. Mottram, 1978, Blood cells.

Major histocompatibility complex products restrict the adherence of cytolytic T lymphocytes to minor histocompatibility antigens or to trinitrophenyl determinants on schistosomula of Schistosoma mansoni.

A. Sher, M. Vadas, S. Burakoff, 1979, Proceedings of the National Academy of Sciences of the United States of America.

Regulation of granulocyte function by colony stimulating factors.

M. Vadas, A. F. López, 1984, Lymphokine research.