Z. Su

发表

PEG-3, a nontransforming cancer progression gene, is a positive regulator of cancer aggressiveness and angiogenesis.

Z. Su, N. Goldstein, H. Jiang, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Antisense inhibition of the PTI-1 oncogene reverses cancer phenotypes.

Z. Su, N. Goldstein, P. Fisher, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Astrocyte elevated gene-1 contributes to the pathogenesis of neuroblastoma

Z. Su, P. Fisher, Seok-Geun Lee, 2009, Oncogene.

Src kinase activation is mandatory for MDA-9/syntenin-mediated activation of Nuclear Factor-κB

Z. Su, P. Fisher, S. Das, 2010, Oncogene.

Activation of Ras/Raf protects cells from melanoma differentiation-associated gene-5-induced apoptosis

Z. Su, P. Fisher, L. Lin, 2006, Cell Death and Differentiation.

Mda-7, a novel melanoma differentiation associated gene with promise for cancer gene therapy.

Z. Su, N. Goldstein, C. S. Young, 2000, Advances in experimental medicine and biology.

Early Events in Methyl Methanesulfonate Enhancement of Adenovirus Transformation of Cloned Rat Embryo Fibroblast Cells

Z. Su, P. Fisher, Z. Su, 1989, Molecular carcinogenesis.

Wild-type adenovirus type 5 transforming genes function as transdominant suppressors of oncogenesis in mutant adenovirus type 5 transformed rat embryo fibroblast cells.

Z. Su, H. Jiang, P. Fisher, 1993, Cancer research.

Subtraction hybridization identifies a novel melanoma differentiation associated gene, mda-7, modulated during human melanoma differentiation, growth and progression.

Z. Su, N. Goldstein, H. Jiang, 1995, Oncogene.

Apoptosis mediates the selective toxicity of caffeic acid phenethyl ester (CAPE) toward oncogene-transformed rat embryo fibroblast cells.

Z. Su, N. Goldstein, P. Fisher, 1995, Anticancer research.

The melanoma differentiation associated gene mda-7 suppresses cancer cell growth.

Z. Su, N. Goldstein, H. Jiang, 1996, Proceedings of the National Academy of Sciences of the United States of America.

The melanoma differentiation-associated gene mda-6, which encodes the cyclin-dependent kinase inhibitor p21, is differentially expressed during growth, differentiation and progression in human melanoma cells.

Z. Su, H. Jiang, P. Fisher, 1995, Oncogene.

Astrocyte elevated gene-1 activates cell survival pathways through PI3K-Akt signaling

Z. Su, P. Fisher, Seok-Geun Lee, 2008, Oncogene.

Translational infidelity and human cancer: role of the PTI-1 oncogene.

Z. Su, N. Goldstein, P. Fisher, 1999, The international journal of biochemistry & cell biology.

Inhibition of AP-1 by SARI negatively regulates transformation progression mediated by CCN1

Z. Su, P. Fisher, Seok-Geun Lee, 2010, Oncogene.

Targeting inhibition of K-ras enhances Ad.mda-7-induced growth suppression and apoptosis in mutant K-ras colorectal cancer cells

Z. Su, P. Fisher, John Calvin Reed, 2007, Oncogene.

Evidence of a functional role for the cyclin-dependent kinase inhibitor p21(WAF1/CIP1/MDA6) in the reciprocal regulation of PKC activator-induced apoptosis and differentation in human myelomonocytic leukemia cells.

Z. Su, P. Fisher, S. Grant, 1998, Experimental cell research.

Identification of the human prostatic carcinoma oncogene PTI-1 by rapid expression cloning and differential RNA display.

Z. Su, P. Fisher, C. Olsson, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Induction of differentiation in human promyelocytic HL-60 leukemia cells activates p21, WAF1/CIP1, expression in the absence of p53.

Z. Su, H. Jiang, P. Fisher, 1994, Oncogene.

Ionizing radiation enhances therapeutic activity of mda-7/IL-24: overcoming radiation- and mda-7/IL-24-resistance in prostate cancer cells overexpressing the antiapoptotic proteins bcl-xL or bcl-2

Z. Su, John Calvin Reed, P. Dent, 2006, Oncogene.

The cancer growth suppressor gene mda-7 selectively induces apoptosis in human breast cancer cells and inhibits tumor growth in nude mice.

Z. Su, N. Goldstein, C. S. Young, 1998, Proceedings of the National Academy of Sciences of the United States of America.

A combinatorial approach for selectively inducing programmed cell death in human pancreatic cancer cells

Z. Su, N. Goldstein, P. Fisher, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Growth suppression and toxicity induced by caffeic acid phenethyl ester (CAPE) in type 5 adenovirus-transformed rat embryo cells correlate directly with transformation progression.

Z. Su, P. Fisher, Z. Su, 1994, Cancer research.

Growth inhibition and modulation of antigenic phenotype in human melanoma and glioblastoma multiforme cells by caffeic acid phenethyl ester (CAPE)

Z. Su, P. Fisher, S. Zucker, 1992, Cellular and molecular biology.

PEA3 sites within the progression elevated gene-3 (PEG-3) promoter and mitogen-activated protein kinase contribute to differential PEG-3 expression in Ha-ras and v-raf oncogene transformed rat embryo cells.

Z. Su, P. Dent, Y. Shi, 2001, Nucleic acids research.

Reciprocal subtraction differential RNA display: an efficient and rapid procedure for isolating differentially expressed gene sequences.

Z. Su, P. Fisher, D. Kang, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Analysis of viral and cellular gene expression during progression and suppression of the transformed phenotype in type 5 adenovirus-transformed rat embryo cells.

Z. Su, G. Duigou, P. Fisher, 1991, Oncogene.

Subtraction hybridization identifies a transformation progression-associated gene PEG-3 with sequence homology to a growth arrest and DNA damage-inducible gene.

Z. Su, P. Fisher, Y. Shi, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Enhanced delivery of mda-7/IL-24 using a serotype chimeric adenovirus (Ad.5/3) improves therapeutic efficacy in low CAR prostate cancer cells

Z. Su, P. Fisher, P. Dent, 2010, Cancer Gene Therapy.

A cancer terminator virus eradicates both primary and distant human melanomas

Z. Su, P. Fisher, P. Dent, 2008, Cancer Gene Therapy.

Defining the critical gene expression changes associated with expression and suppression of the tumorigenic and metastatic phenotype in Ha-ras-transformed cloned rat embryo fibroblast cells.

Z. Su, P. Fisher, S. Zimmer, 1993, Oncogene.

Induction of transformation progression in type 5 adenovirus-transformed rat embryo cells by a cloned protein kinase C beta 1 gene and reversal of progression by 5-azacytidine.

Z. Su, P. Fisher, R. Shen, 1994, Oncogene.

Cloning of differentially expressed genes in an HIV-1 resistant T cell clone by rapid subtraction hybridization, RaSH.

Z. Su, H. Jiang, P. Fisher, 2001, Gene.

Transcriptional switching model for the regulation of tumorigenesis and metastasis by the ha-ras oncogene - transcriptional changes in the ha-ras tumor-suppressor gene lysyl oxidase.

Z. Su, P. Fisher, S. Zimmer, 1995, International journal of oncology.

Caffeic acid phenethyl ester profoundly modifies protein synthesis profile in type 5 adenovirus-transformed cloned rat embryo fibroblast cells.

Z. Su, P. Fisher, D. Grunberger, 1997, International journal of oncology.

Chemoprevention gene therapy (CGT): novel combinatorial approach for preventing and treating pancreatic cancer.

Z. Su, P. Fisher, R. Dash, 2013, Current molecular medicine.

Fludarabine phosphate selectively inhibits growth and modifies the antigenic phenotype of human glioblastoma-multiforme cells expressing a multidrug resistance phenotype.

Z. Su, H. Jiang, P. Fisher, 1992, International journal of oncology.