C. Van Lint

发表

Host-pathogen interactome mapping for HTLV-1 and -2 retroviruses

M. Vidal, Tomoko Hirozane-Kishikawa, D. Hill, 2012, Retrovirology.

Tuning of AKT-pathway by Nef and its blockade by protease inhibitors results in limited recovery in latently HIV infected T-cell line

R. König, J. Gatot, C. Van Lint, 2016, Scientific Reports.

An Interferon Regulatory Factor Binding Site in the U5 Region of the Bovine Leukemia Virus Long Terminal Repeat Stimulates Tax-Independent Gene Expression

V. Kiermer, A. Burny, C. Van Lint, 1998, Journal of Virology.

New pyridinone derivatives as potent HIV-1 nonnucleoside reverse transcriptase inhibitors.

C. Van Lint, F. Durant, Kiet Le Van, 2009, Journal of medicinal chemistry.

The Prdm13 histone methyltransferase encoding gene is a Ptf1a-Rbpj downstream target that suppresses glutamatergic and promotes GABAergic neuronal fate in the dorsal neural tube.

M. C. Jørgensen, P. Serup, C. Van Lint, 2014, Developmental biology.

HIV-1 transcription and latency: an update

Alessandro Marcello, C. Van Lint, A. Marcello, 2013, Retrovirology.

Synergistic Activation of Human Immunodeficiency Virus Type 1 Promoter Activity by NF-κB and Inhibitors of Deacetylases: Potential Perspectives for the Development of Therapeutic Strategies

Ben Berkhout, Vincent Bours, B. Berkhout, 2002, Journal of Virology.

Transcriptional Regulation of the Bovine Leukemia Virus Promoter by the Cyclic AMP-response Element Modulator τ Isoform*

A. Burny, C. Van Lint, Y. De Launoit, 2007, Journal of Biological Chemistry.

HMGA1 recruits CTIP2-repressed P-TEFb to the HIV-1 and cellular target promoters

C. Van Lint, Sebastian Eilebrecht, A. Benecke, 2014, Nucleic acids research.

CTIP2 is a negative regulator of P-TEFb

F. Dequiedt, C. Van Lint, X. Graña, 2013, Proceedings of the National Academy of Sciences.

Histone deacetylase inhibitor trichostatin A sustains sodium pervanadate-induced NF-kappaB activation by delaying IkappaBalpha mRNA resynthesis: comparison with tumor necrosis factor alpha.

C. Van Lint, G. Haegeman, J. Piette, 2007, The Journal of biological chemistry.

HIC1 controls cellular- and HIV-1- gene transcription via interactions with CTIP2 and HMGA1

C. Van Lint, F. Forouzanfar, Sebastian Eilebrecht, 2016, Scientific Reports.

Transcriptional regulation of the murine brca2 gene by CREB/ATF transcription factors.

C. Van Lint, D. Monté, Y. De Launoit, 2003, Biochemical and biophysical research communications.

Limited HIV-1 Reactivation in Resting CD4+ T cells from Aviremic Patients under Protease Inhibitors

J. Gatot, C. Van Lint, Amit Kumar, 2016, Scientific Reports.

Primate lentiviruses use at least three alternative strategies to suppress NF-κB-mediated immune activation

C. Van Lint, F. Kirchhoff, M. Peeters, 2017, PLoS pathogens.

Characterization of new RNA polymerase III and RNA polymerase II transcriptional promoters in the Bovine Leukemia Virus genome

A. Burny, C. Van Lint, C. Vanhulle, 2016, Scientific Reports.

Hyperactivated NF-κB and AP-1 Transcription Factors Promote Highly Accessible Chromatin and Constitutive Transcription across the Interleukin-6 Gene Promoter in Metastatic Breast Cancer Cells

C. Van Lint, G. Haegeman, D. Chalbos, 2009, Molecular and Cellular Biology.

On the way to find a cure: Purging latent HIV‐1 reservoirs

C. Van Lint, C. Schwartz, S. Bouchat, 2017, Biochemical pharmacology.

A Defect in Nucleosome Remodeling Prevents IL-12(p35) Gene Transcription in Neonatal Dendritic Cells

C. Van Lint, M. Goldman, M. Libin, 2004, The Journal of experimental medicine.

Jurkat-Derived (J-Lat, J1.1, and Jurkat E4) and CEM-Derived T Cell Lines (8E5 and ACH-2) as Models of Reversible Proviral Latency.

C. Van Lint, G. Poli, A. Rodari, 2022, Methods in molecular biology.

Novel role of UHRF1 in the epigenetic repression of the latent HIV-1

B. Berkhout, C. Van Lint, C. Rouzioux, 2022, EBioMedicine.

Preclinical shock strategies to reactivate latent HIV-1: an update

C. Van Lint, G. Darcis, Benoit Van Driessche, 2016, Current opinion in HIV and AIDS.

POSITIVE TRANSCRIPTION ELONGATION FACTOR B IS POORLY EXPRESSED AND IS SEQUESTERED IN RESTING CELLS

C. Van Lint, G. Darcis, Benoit Van Driessche, 2016 .

Synthesis, biological evaluation and molecular modeling studies of quinolonyl diketo acid derivatives: new structural insight into the HIV-1 integrase inhibition.

C. Van Lint, J. Wouters, J. Mouscadet, 2011, European journal of medicinal chemistry.

Structural and theoretical studies of [6-bromo-1-(4-fluorophenylmethyl)-4(1H)-quinolinon-3-yl)]-4-hydroxy-2-oxo-3-butenoïc acid as HIV-1 integrase inhibitor.

C. Van Lint, J. Wouters, J. Mouscadet, 2009, Bioorganic & medicinal chemistry letters.

Posttranscriptional Regulation of HIV-1 Gene Expression during Replication and Reactivation from Latency by Nuclear Matrix Protein MATR3

B. Berkhout, C. Van Lint, C. Rouzioux, 2018, mBio.

Inhibition of Histone Deacetylases Induces Bovine Leukemia Virus Expression In Vitro and In Vivo

C. Van Lint, R. Kettmann, L. Willems, 2002, Journal of Virology.

Deacetylase Inhibitors and the Viral Transactivator TaxBLV Synergistically Activate Bovine Leukemia Virus Gene Expression via a cAMP-responsive Element- and cAMP-responsive Element-binding Protein-dependent Mechanism*

A. Burny, C. Van Lint, Y. De Launoit, 2004, Journal of Biological Chemistry.

Nerve Growth Factor Receptor Signaling Induces Histone Acetyltransferase Domain-dependent Nuclear Translocation of p300/CREB-binding Protein-associated Factor and hGCN5 Acetyltransferases*

C. Van Lint, T. Karpova, James G. McNally, 2004, Journal of Biological Chemistry.

DNA cytosine methylation in the Bovine Leukemia Virus promoter is associated with latency in a Lymphoma-derived B-cell line : potential involvement of direct inhibition of CREB/CREM/ATF binding

A. Burny, C. Van Lint, R. Kettmann, 2010, Journal of Biological Chemistry.

Purification of SUMO-1 modified IκBα and complex formation with NF-κB.

V. Villeret, C. Van Lint, Y. De Launoit, 2011, Protein expression and purification.

Ferulic acid derivatives block coronaviruses HCoV-229E and SARS-CoV-2 replication in vitro

C. Van Lint, Mathilde Galais, S. Pasquereau, 2022, Scientific Reports.

Chromatin disruption in the promoter of human immunodeficiency virus type 1 during transcriptional activation.

C. Van Lint, E. Verdin, C. Lint, 1993, The EMBO journal.

Expression, purification, and structural prediction of the Ets transcription factor ERM.

C. Van Lint, J. Ruysschaert, Y. De Launoit, 2006, Biochimica et biophysica acta.

COUP-TF interacting protein 2 represses the initial phase of HIV-1 gene transcription in human microglial cells

C. Van Lint, D. Aunis, S. Chasserot-Golaz, 2005, Nucleic acids research.

Shocking HIV-1 with immunomodulatory latency reversing agents.

C. Van Lint, G. Darcis, Anna Kula-Pacurar, 2021, Seminars in immunology.

Current Status of Latency Reversing Agents Facing the Heterogeneity of HIV-1 Cellular and Tissue Reservoirs

C. Van Lint, A. Kula, A. Marcello, 2020, Frontiers in Microbiology.

Microglial Cells: The Main HIV-1 Reservoir in the Brain

C. Van Lint, A. Marcello, C. Schwartz, 2019, Front. Cell. Infect. Microbiol..

CpG Methylation Controls Reactivation of HIV from Latency

C. Van Lint, D. Olive, E. Verdin, 2009, PLoS pathogens.

CpG methylation controls reactivation of HIV from latency

C. Van Lint, D. Olive, E. Verdin, 2010, Retrovirology.

Suboptimal Enhancer Sequences Are Required for Efficient Bovine Leukemia Virus Propagation In Vivo: Implications for Viral Latency

A. Burny, C. Van Lint, R. Kettmann, 2001, Journal of Virology.

Depicting HIV-1 Transcriptional Mechanisms: A Summary of What We Know

C. Van Lint, A. Rodari, A. Dutilleul, 2020, Viruses.

Proceedings of the Frontiers of Retrovirology Conference 2016

R. König, D. Elleder, R. Sachidanandam, 2016, Retrovirology.

Transcription factor binding sites downstream of the human immunodeficiency virus type 1 transcription start site are important for virus infectivity

C. Van Lint, E. Verdin, S. Emiliani, 1997, Journal of virology.

Immune Hyperactivation of HIV-1-Infected T Cells Mediated by Tat and the CD28 Pathway

C. Van Lint, E. Verdin, M. Ott, 1997, Science.

The expression of a small fraction of cellular genes is changed in response to histone hyperacetylation.

C. Van Lint, E. Verdin, S. Emiliani, 1996, Gene expression.

Bovine Leukemia Virus SU Protein Interacts with Zinc, and Mutations within Two Interacting Regions Differently Affect Viral Fusion and Infectivity In Vivo

J. Gatot, A. Burny, C. Van Lint, 2002, Journal of Virology.

A transcriptional regulatory element is associated with a nuclease-hypersensitive site in the pol gene of human immunodeficiency virus type 1

A. Burny, C. Van Lint, E. Verdin, 1994, Journal of virology.

Overlapping CRE and E Box Motifs in the Enhancer Sequences of the Bovine Leukemia Virus 5′ Long Terminal Repeat Are Critical for Basal and Acetylation-Dependent Transcriptional Activity of the Viral Promoter: Implications for Viral Latency

A. Burny, C. Van Lint, R. Kettmann, 2004, Journal of Virology.

Differential Regulation of NF-κB-Mediated Proviral and Antiviral Host Gene Expression by Primate Lentiviral Nef and Vpu Proteins

C. Van Lint, F. Kirchhoff, B. Hahn, 2015, Cell reports.

The human T-cell leukemia virus type-1 tax oncoprotein dissociates NF-κB p65RelA-Stathmin complexes and causes catastrophic mitotic spindle damage and genomic instability.

C. Van Lint, L. Ratner, Katie Smith, 2019, Virology.

The Molecular Biology of HIV Latency.

C. Van Lint, D. Purcell, G. Khoury, 2018, Advances in experimental medicine and biology.

Characterization of a human RPD3 ortholog, HDAC3.

C. Van Lint, E. Verdin, W. Fischle, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Human Immunodeficiency Virus Type-1 Tat/Co-activator Acetyltransferase Interactions Inhibit p53Lys-320Acetylation and p53-responsive Transcription*

C. Van Lint, T. Karpova, James G. McNally, 2003, The Journal of Biological Chemistry.

Prdm12 specifies V1 interneurons through cross-repressive interactions with Dbx1 and Nkx6 genes in Xenopus

K. Lewis, C. Van Lint, A. Pierani, 2015, Development.

Diversity of acetylation targets and roles in transcriptional regulation: the human immunodeficiency virus type 1 promoter as a model system.

C. Van Lint, V. Quivy, 2002, Biochemical pharmacology.

Upstream Stimulatory Factors Binding to an E Box Motif in the R Region of the Bovine Leukemia Virus Long Terminal Repeat Stimulates Viral Gene Expression*

V. Kiermer, A. Burny, C. Van Lint, 2002, The Journal of Biological Chemistry.

Molecular Control of HIV and SIV Latency.

C. Van Lint, F. Kirchhoff, S. Bouchat, 2017, Current topics in microbiology and immunology.

HIV Latency: Should We Shock or Lock?

C. Van Lint, G. Darcis, Benoit Van Driessche, 2017, Trends in immunology.

B-oligomer of pertussis toxin inhibits HIV-1 LTR-driven transcription through suppression of NF-kappaB p65 subunit activity.

M. Bukrinsky, C. Van Lint, S. Iordanskiy, 2002, Virology.

Cytoplasmic IκBα Increases NF-κB-independent Transcription through Binding to Histone Deacetylase (HDAC) 1 and HDAC3*

C. Van Lint, J. Piette, M. Merville, 2003, Journal of Biological Chemistry.

An In-Depth Comparison of Latency-Reversing Agent Combinations in Various In Vitro and Ex Vivo HIV-1 Latency Models Identified Bryostatin-1+JQ1 and Ingenol-B+JQ1 to Potently Reactivate Viral Gene Expression

Arsène Burny, Anna Kula, J. Gatot, 2015, PLoS pathogens.

LSD1 cooperates with CTIP2 to promote HIV-1 transcriptional silencing

C. Van Lint, D. Aunis, C. Schwartz, 2011, Nucleic acids research.

Tax-Independent Gene Expression Long Terminal Repeat Stimulates the U 5 Region of the Bovine Leukemia Virus An Interferon Regulatory Factor Binding Site in

V. Kiermer, A. Burny, C. Van Lint, 1998 .

The intragenic enhancer of human immunodeficiency virus type 1 contains functional AP-1 binding sites

A. Burny, C. Van Lint, E. Verdin, 1991, Journal of virology.

Exogenous Nef Protein Activates NF-κB, AP-1, and c-Jun N-Terminal Kinase and Stimulates HIV Transcription in Promonocytic Cells

Bharat B. Aggarwal, C. Van Lint, S. Manna, 2003, The Journal of Biological Chemistry.

Regulation of Human Immunodeficiency Virus Type 1 Gene Expression by Clade-Specific Tat Proteins

M. Wainberg, A. Burny, C. Van Lint, 2005, Journal of Virology.

Histone methyltransferase inhibitors induce HIV-1 recovery in resting CD4+ T cells from HIV-1-infected HAART-treated patients

J. Gatot, C. Van Lint, C. Rouzioux, 2012, AIDS.

Transcription factor binding sites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivity

A. Burny, C. Van Lint, C. Vanhulle, 2006, Retrovirology.

Transcription factor binding aites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivity

A. Burny, C. Van Lint, C. Vanhulle, 2006, Retrovirology.

HIV Persistence and the Prospect of Long-Term Drug-Free Remissions for HIV-Infected Individuals

C. Van Lint, T. Chun, C. Rouzioux, 2010, Science.

Inhibition of HIV-1 gene transcription by KAP1 in myeloid lineage

C. Van Lint, B. Fahrenkrog, C. Schwartz, 2020, Scientific Reports.

Internal transcriptional regulatory elements in HIV-1 and other retroviruses.

C. Van Lint, E. Verdin, 1995, Cellular and molecular biology.

Mutations in the tat Gene Are Responsible for Human Immunodeficiency Virus Type 1 Postintegration Latency in the U1 Cell Line

C. Van Lint, E. Verdin, M. Ott, 1998, Journal of Virology.

Transcriptional activation and chromatin remodeling of the HIV‐1 promoter in response to histone acetylation.

C. Van Lint, E. Verdin, M. Ott, 1996, The EMBO journal.

Applications of CRISPR/Cas9 tools in deciphering the mechanisms of HIV-1 persistence.

C. Van Lint, G. Darcis, Amina Ait-Ammar, 2019, Current opinion in virology.

HIV‐1 regulation of latency in the monocyte‐macrophage lineage and in CD4+ T lymphocytes

C. Van Lint, D. Aunis, C. Schwartz, 2010, Journal of leukocyte biology.

NF‐κB‐dependent control of HIV‐1 transcription by the second coding exon of Tat in T cells

C. Van Lint, A. Varin, G. Herbein, 2008, Journal of leukocyte biology.

The Werner Syndrome Helicase Is a Cofactor for HIV-1 Long Terminal Repeat Transactivation and Retroviral Replication*

C. Van Lint, R. Monnat, D. Skiest, 2007, Journal of Biological Chemistry.

HIV‐1 Tat transcriptional activity is regulated by acetylation

A. Burny, C. Van Lint, K. Jeang, 1999, The EMBO journal.

Isoquinolinesulphonamide Derivatives Inhibit Transcriptional Elongation of Human Immunodeficiency Virus Type 1 RNA in a Promyelocytic Model of Latency

C. Van Lint, E. Verdin, O. Ho, 1999, Antiviral chemistry & chemotherapy.

Epigenetic Mechanisms of HIV-1 Persistence

C. Van Lint, R. Verdikt, O. Hernalsteens, 2021, Vaccines.

Epigenetic crosstalk in chronic infection with HIV-1

C. Van Lint, Amina Ait-Ammar, R. Verdikt, 2020, Seminars in Immunopathology.

Molecular control of HIV-1 postintegration latency: implications for the development of new therapeutic strategies

C. Van Lint, Laurence Colin, 2009, Retrovirology.

Anti-Leukemia Activity of MS-275 Histone Deacetylase Inhibitor Implicates 4-1BBL/4-1BB Immunomodulatory Functions

C. Van Lint, D. Olive, A. Restouin, 2009, PloS one.

Transcriptional behavior of the HIV-1 promoter in context of the BACH2 prominent proviral integration gene.

J. Hauber, C. Van Lint, T. Günther, 2020, Virus research.

Recruitment of chromatin‐modifying enzymes by CTIP2 promotes HIV‐1 transcriptional silencing

F. Dequiedt, C. Van Lint, D. Aunis, 2007, The EMBO journal.

Sequential treatment with 5‐aza‐2′‐deoxycytidine and deacetylase inhibitors reactivates HIV‐1

Anna Kula, J. Gatot, C. Van Lint, 2015, EMBO molecular medicine.

Synergistic Activation of HIV-1 Expression by Deacetylase Inhibitors and Prostratin: Implications for Treatment of Latent Infection

Arsène Burny, Dolores Vaira, J. Gatot, 2009, PloS one.

Role of the cellular factor CTCF in the regulation of bovine leukemia virus latency and three-dimensional chromatin organization

Peter H. L. Krijger, A. Burny, C. Van Lint, 2022, Nucleic Acids Research.

Gene activation and gene silencing: a subtle equilibrium.

A. Burny, C. Van Lint, C. Vanhulle, 2004, Cloning and stem cells.

CD32+CD4+ T Cells Are Highly Enriched for HIV DNA and Can Support Transcriptional Latency

B. Berkhout, C. Van Lint, N. Kootstra, 2020, Cell reports.

Reactivation capacity by latency-reversing agents ex vivo correlates with the size of the HIV-1 reservoir

Anna Kula, C. Van Lint, C. Rouzioux, 2017, AIDS.

A point mutation in the HIV-1 Tat responsive element is associated with postintegration latency.

C. Van Lint, J. Brady, E. Verdin, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Immunity drives TET1 regulation in cancer through NF-κB

C. Sotiriou, C. Van Lint, K. Willard-Gallo, 2018, Science Advances.

Synthetic Vpr Protein Activates Activator Protein-1, c-Jun N-terminal Kinase, and NF-κB and Stimulates HIV-1 Transcription in Promonocytic Cells and Primary Macrophages*

C. Van Lint, B. Aggarwal, B. Roques, 2005, Journal of Biological Chemistry.

Heterogeneous HIV-1 Reactivation Patterns of Disulfiram and Combined Disulfiram+Romidepsin Treatments

A. Burny, C. Van Lint, C. Rouzioux, 2019, Journal of acquired immune deficiency syndromes.

Chromatin-associated regulation of HIV-1 transcription: implications for the development of therapeutic strategies.

C. Van Lint, S. de Walque, V. Quivy, 2007, Sub-cellular biochemistry.

Administration of HDAC inhibitors to reactivate HIV-1 expression in latent cellular reservoirs: implications for the development of therapeutic strategies.

C. Van Lint, D. Demonté, V. Quivy, 2004, Biochemical pharmacology.

Implication of the cellular factor CTCF in the regulation of Bovine Leukemia Virus latency and tridimensional chromatin organization

Peter H. L. Krijger, C. Van Lint, W. de Laat, 2021, bioRxiv.

HIV-1 transcriptional silencing caused by TRIM22 inhibition of Sp1 binding to the viral promoter

A. Das, B. Berkhout, C. Van Lint, 2015, Retrovirology.

Expression of the Ets transcription factor Erm is regulated through a conventional PKC signaling pathway in the Molt4 lymphoblastic cell line

C. Van Lint, D. Monté, Y. De Launoit, 2005, FEBS letters.

Human T-cell leukemia virus type-1 Tax oncoprotein regulates G-protein signaling.

M. Boxus, F. Dequiedt, A. Burny, 2007, Blood.

HIV-1 Vpr mediates the depletion of the cellular repressor CTIP2 to counteract viral gene silencing

C. Van Lint, F. Forouzanfar, C. Schwartz, 2019, Scientific Reports.

Active Transcription of the Human FASL/CD95L/TNFSF6 Promoter Region in T Lymphocytes Involves Chromatin Remodeling

C. Van Lint, D. Olive, I. Hirsch, 2006, Journal of Biological Chemistry.

Reactivation of simian immunodeficiency virus reservoirs in the brain of virally suppressed macaques

R. Siliciano, C. Van Lint, J. Clements, 2016, AIDS.

OP 1.6 – 00138 Role of UHRF1 in HIV-1 transcriptional repression through epigenetic and non-epigenetic mechanisms

C. Van Lint, M. Bendoumou, A. Aït-Ammar, 2022, Journal of virus eradication.

HIV-1 chromatin, transcription, and the regulatory protein Tat.

C. Van Lint, E. Verdin, Laurence Colin, 2014, Methods in molecular biology.

Epigenetic control of HIV-1 post integration latency: implications for therapy

C. Van Lint, Amit Kumar, G. Herbein, 2015, Clinical Epigenetics.

Long-Term Spontaneous Control of HIV-1 Is Related to Low Frequency of Infected Cells and Inefficient Viral Reactivation

C. Van Lint, J. Martinez-Picado, J. Prado, 2015, Journal of Virology.

Epigenetic control of HIV-1 post integration latency: implications for therapy

C. Van Lint, Amit Kumar, G. Herbein, 2015, Clinical Epigenetics.

Mass spectrometry and CLIP-seq analysis reveal BCL11b interactions with RNA processing pathways

C. Van Lint, C. Schwartz, O. Rohr, 2020, bioRxiv.

Stop HUSHing on SIV/HIV

C. Van Lint, 2018, Nature Microbiology.

Repression of Human T-lymphotropic virus type 1 Long Terminal Repeat sense transcription by Sp1 recruitment to novel Sp1 binding sites

A. Burny, C. Van Lint, A. Kula, 2017, Scientific Reports.

Human IL-12(p35) gene activation involves selective remodeling of a single nucleosome within a region of the promoter containing critical Sp1-binding sites.

C. Van Lint, M. Goldman, S. Goriely, 2003, Blood.

A pervasive role of histone acetyltransferases and deacetylases in an NF-kappaB-signaling code.

A. Burny, C. Van Lint, M. Calao, 2008, Trends in biochemical sciences.

Regulation at multiple levels of NF-kappaB-mediated transactivation by protein acetylation.

C. Van Lint, Carine Van Lint, Vincent Quivy, 2004, Biochemical pharmacology.

Potentiation of Tumor Necrosis Factor-Induced NF-κB Activation by Deacetylase Inhibitors Is Associated with a Delayed Cytoplasmic Reappearance of IκBα

A. Burny, C. Van Lint, J. Piette, 2003, Molecular and Cellular Biology.

Distinct Mechanisms Trigger Apoptosis in Human Immunodeficiency Virus Type 1-Infected and in Uninfected Bystander T Lymphocytes

C. Van Lint, E. Verdin, G. Herbein, 1998, Journal of Virology.

Interferon-inducible TRIM22 contributes to maintenance of HIV-1 proviral latency in T cell lines.

A. Das, B. Berkhout, C. Van Lint, 2019, Virus research.

Stop HUSHing on SIV/HIV

C. Van Lint, 2018, Nature Microbiology.

Mechanisms Regulating Expression of the Tumor Necrosis Factor-related light Gene

C. Van Lint, D. Olive, R. Castellano, 2002, The Journal of Biological Chemistry.

The Current Status of Latency Reversing Agents for HIV-1 Remission.

C. Van Lint, G. Darcis, A. Rodari, 2021, Annual review of virology.

Solution structure of the N-terminal transactivation domain of ERM modified by SUMO-1.

V. Villeret, C. Van Lint, D. Monté, 2010, Biochemical and biophysical research communications.

Chromatin disruption in the promoter of Bovine Leukemia Virus during transcriptional activation

A. Burny, C. Van Lint, Laurence Colin, 2011, Nucleic acids research.

Role of chromatin in HIV-1 transcriptional regulation.

C. Van Lint, 2000, Advances in pharmacology.

A complex network of transcription factors and epigenetic regulators involved in bovine leukemia virus transcriptional regulation

C. Van Lint, Maxime Bellefroid, Estelle Plant, 2023, Retrovirology.

Potentiation of Tumor Necrosis Factor-Induced NF-κB Activation by Deacetylase Inhibitors Is Associated with a Delayed Cytoplasmic Reappearance of IκBα

A. Burny, C. Van Lint, J. Piette, 2003, Molecular and Cellular Biology.

HIV-1 Tat Interactions with p300 and PCAF Transcriptional Coactivators Inhibit Histone Acetylation and Neurotrophin Signaling through CREB*

C. Van Lint, D. Skiest, D. Burns, 2005, Journal of Biological Chemistry.

Molecular control of HIV - 1 postintegration latency: implications for therapeutic strategies

C. Van Lint, 2012, Retrovirology.

Editorial: The relevance of molecular mechanisms in HIV-1 latency and reactivation from latency

C. Van Lint, O. Rohr, Anna Kula-Pacurar, 2023, Frontiers in Cellular and Infection Microbiology.

Cytoplasmic IkappaBalpha increases NF-kappaB-independent transcription through binding to histone deacetylase (HDAC) 1 and HDAC3.

C. Van Lint, J. Piette, M. Merville, 2003, The Journal of biological chemistry.

Proceedings of the Frontiers of Retrovirology Conference 2016

Sebastian A. Wagner, R. König, D. Elleder, 2016, Retrovirology.

HIV-1 transcriptional silencing caused by TRIM22 inhibition of Sp1 binding to the viral promoter

A. Das, B. Berkhout, C. Van Lint, 2015, Retrovirology.

Identification of small molecule antivirals against HTLV-1 by targeting the hDLG1-Tax-1 protein-protein interaction

C. Van Lint, J. Twizere, F. Delvigne, 2023, bioRxiv.

Artificial Zinc Finger Fusions Targeting Sp1-binding Sites and the trans-Activator-responsive Element Potently Repress Transcription and Replication of HIV-1*

C. Van Lint, M. Hur, W. Seol, 2005, Journal of Biological Chemistry.

Inhibition of the NF-kappa B transcription factor increases Bax expression in cancer cell lines.

C. Van Lint, John Calvin Reed, M. Merville, 2001, Oncogene.

The AP-1 Binding Sites Located in the pol Gene Intragenic Regulatory Region of HIV-1 Are Important for Viral Replication

A. Bergamaschi, A. Burny, C. Van Lint, 2009, PLoS ONE.

Transcription factor binding aites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivity

A. Burny, C. Van Lint, C. Vanhulle, 2006, Retrovirology.

Transcription factor binding sites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivity

A. Burny, C. Van Lint, Y. De Launoit, 2005, Nucleic Acids Research.

HIV‐1 regulation of latency in the monocyte‐macrophage lineage and in CD4+ T lymphocytes

C. Van Lint, D. Aunis, C. Schwartz, 2010, Journal of leukocyte biology.

Regulation of Human Immunodeficiency Virus Type 1 Gene Expression by Clade-Specific Tat Proteins

Nathalie, M. Wainberg, A. Burny, 2005, Journal of Virology.

Prdm12 specifies V1 interneurons through cross-repressive interactions with Dbx1 and Nkx6 genes in Xenopus

K. Lewis, C. Van Lint, A. Pierani, 2015, Development.

Molecular mechanisms involved in HIV-1 transcriptional latency and reactivation: implications for the development of therapeutic strategies.

A. Burny, C. Van Lint, J. Piette, 2004, Bulletin et memoires de l'Academie royale de medecine de Belgique.

DNA Cytosine Methylation in the Bovine Leukemia Virus Promoter Is Associated with Latency in a Lymphoma-derived B-cell Line

A. Burny, C. Van Lint, R. Kettmann, 2010, The Journal of Biological Chemistry.

Transcription factor binding sites in the pol gene intragenic regulatory region of HIV-1 are important for virus infectivity

A. Burny, C. Van Lint, Y. De Launoit, 2005, Nucleic acids research.