Z. Ba

发表

Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac output.

P. Wang, Z. Ba, I. Chaudry, 1991, Archives of surgery.

UP‐REGULATION OF A NOVEL POTENT VASODILATORY PEPTIDE ADRENOMEDULLIN DURING POLYMICROBIAL SEPSIS

P. Wang, Z. Ba, I. Chaudry, 1998, Shock.

Reduction in vascular responsiveness to adrenomedullin during sepsis.

P. Wang, Z. Ba, I. Chaudry, 1999, The Journal of surgical research.

Mechanism of adrenal insufficiency following trauma and severe hemorrhage: role of hepatic 11beta-hydroxysteroid dehydrogenase.

P. Wang, Z. Ba, I. Chaudry, 1999, Archives of surgery.

Role of estrogen receptor subtypes in estrogen-induced organ-specific vasorelaxation after trauma-hemorrhage.

Z. Ba, I. Chaudry, 2008, American journal of physiology. Heart and circulatory physiology.

17beta-Estradiol modulates vasoconstriction induced by endothelin-1 following trauma-hemorrhage.

Z. Ba, I. Chaudry, K. Bland, 2007, American journal of physiology. Heart and circulatory physiology.

ATP-MgCl2 restores the depressed hepatocellular function and hepatic blood flow following hemorrhage and resuscitation.

P. Wang, Z. Ba, I. Chaudry, 1991, The Journal of surgical research.

Adrenomedullin binding protein-1 modulates vascular responsiveness to adrenomedullin in late sepsis.

Z. Ba, I. Chaudry, Ping Wang, 2002, American Journal of Physiology. Regulatory Integrative and Comparative Physiology.

Adrenomedullin Binding Protein-1 Modulates Vascular Responsiveness to Adrenomedullin During the Late Stage of Sepsis

Z. Ba, I. Chaudry, Ping Wang, 2002 .

The Complex Pattern of Cytokines in Sepsis Association Between Prostaglandins, Cachectin, and Interleukins

Z. Ba, I. Chaudry, A. Ayala, 1991, Annals of surgery.

Alterations in tissue oxygen consumption and extraction after trauma and hemorrhagic shock

Z. Ba, I. Chaudry, K. Bland, 2000, Critical care medicine.

SYSTEMATIC ANALYSIS OF THE SALUTARY EFFECT OF ESTROGEN ON CARDIAC PERFORMANCE AFTER TRAUMA-HEMORRHAGE

Z. Ba, I. Chaudry, J. Hsu, 2008, Shock.

Gender differences in small intestinal endothelial function: inhibitory role of androgens.

Z. Ba, I. Chaudry, K. Bland, 2004, American journal of physiology. Gastrointestinal and liver physiology.

Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxation.

Z. Ba, I. Chaudry, P. Wang, 1994, The American journal of physiology.

Endothelial cell dysfunction occurs after hemorrhage in nonheparinized but not in preheparinized models.

Z. Ba, I. Chaudry, P. Wang, 1993, The Journal of surgical research.

Differential alterations in plasma IL-6 and TNF levels after trauma and hemorrhage.

Z. Ba, I. Chaudry, P. Wang, 1991, The American journal of physiology.

Alcohol ingestion before burn injury decreases splanchnic blood flow and oxygen delivery.

Z. Ba, I. Chaudry, K. Bland, 2005, American journal of physiology. Heart and circulatory physiology.

Gender differences in small intestinal perfusion following trauma hemorrhage: the role of endothelin-1.

Z. Ba, I. Chaudry, K. Bland, 2005, American journal of physiology. Gastrointestinal and liver physiology.

Continuous resuscitation after hemorrhage and acute fluid replacement improves cardiovascular responses.

Z. Ba, I. Chaudry, K. Bland, 2001, Surgery.

Testosterone receptor blockade after trauma and hemorrhage attenuates depressed adrenal function.

Z. Ba, I. Chaudry, K. Bland, 2000, American journal of physiology. Regulatory, integrative and comparative physiology.

Is gut the "motor" for producing hepatocellular dysfunction after trauma and hemorrhagic shock?

Z. Ba, I. Chaudry, K. Bland, 1998, The Journal of surgical research.

17??-ESTRADIOL MODULATES VASOCONSTRICTION INDUCED BY ENDOTHELIN-1 FOLLOWING TRAUMAHEMORRHAGE

Z. Ba, I. Chaudry, K. Bland, 2006 .

Flutamide induces relaxation in large and small blood vessels.

Z. Ba, I. Chaudry, K. Bland, 2002, Archives of surgery.

Attenuation of vascular endothelial dysfunction by testosterone receptor blockade after trauma and hemorrhagic shock.

Z. Ba, I. Chaudry, K. Bland, 2001, Archives of surgery.

Diltiazem restores cardiac output and improves renal function after hemorrhagic shock and crystalloid resuscitation.

Z. Ba, I. Chaudry, D. Meldrum, 1992, The American journal of physiology.

Hepatocellular dysfunction after severe hypotension in the absence of blood loss is associated with the increased IL-6 and PGE2.

Z. Ba, I. Chaudry, K. Bland, 1998, The Journal of surgical research.

Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsis.

Z. Ba, I. Chaudry, P. Wang, 1995, The American journal of physiology.

Measurement of hepatic blood flow after severe hemorrhage: lack of restoration despite adequate resuscitation.

Z. Ba, I. Chaudry, J. Burkhardt, 1992, The American journal of physiology.

Trauma-hemorrhage and resuscitation in the mouse: effects on cardiac output and organ blood flow.

Z. Ba, I. Chaudry, J. Burkhardt, 1993, The American journal of physiology.

Adequate crystalloid resuscitation restores but fails to maintain the active hepatocellular function following hemorrhagic shock.

Z. Ba, I. Chaudry, J. Hauptman, 1990, The Journal of trauma.

Upregulation of Kupffer cell beta-adrenoceptors and cAMP levels during the late stage of sepsis.

Z. Ba, I. Chaudry, P. Wang, 1998, Biochimica et biophysica acta.

Nitric oxide. To block or enhance its production during sepsis?

Z. Ba, I. Chaudry, P. Wang, 1994, Archives of surgery.

Does early infusion of red blood cells after trauma and hemorrhage improve organ functions?

Z. Ba, I. Chaudry, K. Bland, 2000, Critical care medicine.

Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6.

Z. Ba, I. Chaudry, P. Wang, 1997, Archives of Surgery.

Endothelial cell dysfunction occurs very early following trauma-hemorrhage and persists despite fluid resuscitation.

Z. Ba, I. Chaudry, P. Wang, 1993, The American journal of physiology.

Is prostacyclin responsible for producing the hyperdynamic response during early sepsis?

Z. Ba, I. Chaudry, K. Bland, 2000, Critical care medicine.

Tumor necrosis factor-alpha administration increases Kupffer cell cyclic adenosine monophosphate levels.

Z. Ba, I. Chaudry, P. Wang, 1995, Shock.

Preheparinization improves organ function after hemorrhage and resuscitation.

Z. Ba, I. Chaudry, G. Singh, 1990, The American journal of physiology.

Pentoxifylline restores cardiac output and tissue perfusion after trauma-hemorrhage and decreases susceptibility to sepsis.

Z. Ba, I. Chaudry, M. Zhou, 1993, Surgery.

Mechanism of the beneficial effects of pentoxifylline on hepatocellular function after trauma hemorrhage and resuscitation.

Z. Ba, I. Chaudry, A. Ayala, 1992, Surgery.

Liver endothelial cell dysfunction occurs early following hemorrhagic shock and persists despite crystalloid resuscitation.

Z. Ba, I. Chaudry, A. Biondo, 1996, The Journal of surgical research.

Pentoxifylline attenuates the depressed endothelial cell function and vascular muscle contractility following trauma and hemorrhagic shock.

Z. Ba, I. Chaudry, P. Wang, 1994, The Journal of trauma.

Diltiazem administration after crystalloid resuscitation restores active hepatocellular function and hepatic blood flow after severe hemorrhagic shock.

Z. Ba, I. Chaudry, R. Dean, 1991, Surgery.

ROLE OF KUPFFER CELLS IN INTERLEUKIN‐6 RELEASE FOLLOWING TRAUMA‐HEMORRHAGE AND RESUSCITATION

Z. Ba, Ping Wang, A. Ayala, 1994, Shock.

Salutary effects of ATP-MgCl2 on the depressed endothelium-dependent relaxation during hyperdynamic sepsis.

Z. Ba, I. Chaudry, K. Bland, 1999, Critical care medicine.

Measurement of circulating blood volume in vivo after trauma-hemorrhage and hemodilution.

Z. Ba, I. Chaudry, A. Ayala, 1994, The American journal of physiology.

Severe hypoxemia in the absence of blood loss depresses hepatocellular function and up-regulates IL-6 and PGE2.

Z. Ba, I. Chaudry, P. Wang, 1997, Biochimica et biophysica acta.

endothelial cell function hemorrhage and resuscitation: role of vascular Gender dimorphic tissue perfusion response after acute

Z. Ba, I. Chaudry, K. Bland, 2016 .

Gender dimorphic tissue perfusion response after acute hemorrhage and resuscitation: role of vascular endothelial cell function.

Z. Ba, I. Chaudry, K. Bland, 2003, American journal of physiology. Heart and circulatory physiology.

Pentoxifylline maintains vascular endothelial cell function during hyperdynamic and hypodynamic sepsis.

Z. Ba, I. Chaudry, T. J. Wood, 1996, Surgery.

SUSTAINED ELEVATION IN CIRCULATING CATECHOLAMINE LEVELS DURING POLYMICROBIAL SEPSIS

Z. Ba, I. Chaudry, S. Reich, 1995, Shock.

233; ATP‐MgCl2 RESTORES THE DEPRESSED ENDOTHELIAL CELL FUNCTION AFTER HEMORRHAGIC SHOCK, and RESUSCITATION

Z. Ba, I. Chaudry, P. Wang, 1994 .

Tumor necrosis factor-alpha produces hepatocellular dysfunction despite normal cardiac output and hepatic microcirculation.

Z. Ba, I. Chaudry, A. Ayala, 1993, The American journal of physiology.

The small intestine plays an important role in upregulating CGRP during sepsis.

Z. Ba, I. Chaudry, M. Zhou, 2001, American journal of physiology. Regulatory, integrative and comparative physiology.

The pivotal role of adrenomedullin in producing hyperdynamic circulation during the early stage of sepsis.

Z. Ba, I. Chaudry, K. Bland, 1998, Archives of surgery.

Pentoxifylline maintains hepatocellular function and improves cardiac performance during early sepsis.

Z. Ba, I. Chaudry, P. Wang, 1997, The Journal of trauma.

HEPATOCELLULAR DYSFUNCTION OCCURS EARLIER THAN THE ONSET OF HYPERDYNAMIC CIRCULATION DURING SEPSIS

Z. Ba, I. Chaudry, Ping Wang, 1995, Shock.

ADMINISTRATION OF A MATRIX METALLOPROTEINASE INHIBITOR AFTER HEMORRHAGE IMPROVES CARDIOVASCULAR AND HEPATOCELLULAR FUNCTION

Z. Ba, I. Chaudry, Ping Wang, 1996, Shock.

Differential alterations in microvascular perfusion in various organs during early and late sepsis.

Z. Ba, I. Chaudry, P. Wang, 1992, The American journal of physiology.

Increase in hepatic blood flow during early sepsis is due to increased portal blood flow.

Z. Ba, I. Chaudry, P. Wang, 1991, The American journal of physiology.

p38 MAPK-dependent eNOS upregulation is critical for 17beta-estradiol-mediated cardioprotection following trauma-hemorrhage.

Z. Ba, I. Chaudry, K. Bland, 2008, American journal of physiology. Heart and circulatory physiology.

Alterations in circulating blood volume during polymicrobial sepsis.

Z. Ba, I. Chaudry, S. Tait, 1993, Circulatory shock.

Hepatocellular dysfunction persists during early sepsis despite increased volume of crystalloid resuscitation.

Z. Ba, I. Chaudry, A. Ayala, 1991, The Journal of trauma.

ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitation.

Z. Ba, I. Chaudry, P. Wang, 1995, The American journal of physiology.

ATP-MgCl2 administration normalizes macrophage cAMP and beta-adrenergic receptors after hemorrhage and resuscitation.

Z. Ba, I. Chaudry, S. Tait, 1994, The American journal of physiology.

ATP-MgCl2 restores the depressed cardiac output following trauma and severe hemorrhage even in the absence of blood resuscitation.

Z. Ba, I. Chaudry, P. Wang, 1992, Circulatory shock.

Mechanism of the beneficial effects of ATP-MgCl2 following trauma-hemorrhage and resuscitation: downregulation of inflammatory cytokine (TNF, IL-6) release.

Z. Ba, I. Chaudry, A. Ayala, 1992, The Journal of surgical research.

ATP-MgCl2 restores renal microcirculation following trauma and severe hemorrhage.

Z. Ba, I. Chaudry, A. Ayala, 1992, Canadian journal of physiology and pharmacology.

Inhibition of the biologic activity of tumor necrosis factor maintains vascular endothelial cell function during hyperdynamic sepsis.

Z. Ba, I. Chaudry, T. J. Wood, 1996, The Journal of trauma.

Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitation.

Z. Ba, I. Chaudry, P. Wang, 1992, The American journal of physiology.

Effects of nonanticoagulant heparin on cardiovascular and hepatocellular function after hemorrhagic shock.

Z. Ba, I. Chaudry, K. Holme, 1996, The American journal of physiology.

Liver endothelial cell function is depressed only during hypodynamic sepsis.

Z. Ba, I. Chaudry, P. Wang, 1997, The Journal of surgical research.

Downregulation of hepatic β-adrenergic receptors after trauma and hemorrhagic shock

Z. Ba, I. Chaudry, S. Tait, 1996 .

Testosterone receptor blockade after trauma and hemorrhage attenuates depressed adrenal function.

Z. Ba, I. Chaudry, K. Bland, 2000, American journal of physiology. Regulatory, integrative and comparative physiology.

The pivotal role of adrenomedullin in producing hyperdynamic circulation during the early stage of sepsis.

Z. Ba, I. Chaudry, K. Bland, 1998, Archives of surgery.