J. Degen

发表

Role of Olivary Electrical Coupling in Cerebellar Motor Learning

J. Deuchars, H. Monyer, C. I. Zeeuw, 2008, Neuron.

Structural and Functional Diversity of Connexin Genes in the Mouse and Human Genome

J. Degen, K. Willecke, Jürgen Eiberger, 2002, Biological chemistry.

Slow Conduction and Enhanced Anisotropy Increase the Propensity for Ventricular Tachyarrhythmias in Adult Mice With Induced Deletion of Connexin43

Tobias Opthof, Jacques M T de Bakker, Harold V M van Rijen, 2004, Circulation.

Expression of Connexin36 in Cone Pedicles and OFF-Cone Bipolar Cells of the Mouse Retina

Josef Ammermüller, Hannah Monyer, Andreas Feigenspan, 2004, The Journal of Neuroscience.

Connexin36 mediates gap junctional coupling of alpha‐ganglion cells in mouse retina

Timm Schubert, Hannah Monyer, R. Weiler, 2005, The Journal of comparative neurology.

Tagger—A Swiss army knife for multiomics to dissect cell type–specific mechanisms of gene expression in mice

V. Bansal, W. Jackson, J. Degen, 2019, PLoS biology.

Cardiomyocyte-restricted deletion of connexin43 during mouse development.

Michael D. Schneider, P. Doevendans, J. Degen, 2006, Journal of molecular and cellular cardiology.

Connexin32 can restore hearing in connexin26 deficient mice.

J. Degen, K. Willecke, T. Moser, 2011, European journal of cell biology.

Ablation of Neuronal Ceramide Synthase 1 in Mice Decreases Ganglioside Levels and Expression of Myelin-associated Glycoprotein in Oligodendrocytes*

J. Degen, K. Willecke, E. Dere, 2012, The Journal of Biological Chemistry.

Quality control of astrocyte‐directed Cre transgenic mice: The benefits of a direct link between loss of gene expression and reporter activation

J. Degen, K. Willecke, C. Steinhäuser, 2009, Glia.

The conditional connexin43G138R mouse mutant represents a new model of hereditary oculodentodigital dysplasia in humans.

J. Degen, K. Willecke, B. Fleischmann, 2008, Human molecular genetics.

Replacement by a lacZ reporter gene assigns mouse connexin36, 45 and 43 to distinct cell types in pancreatic islets.

J. Degen, K. Willecke, A. Charollais, 2004, Experimental cell research.

Adult Ceramide Synthase 2 (CERS2)-deficient Mice Exhibit Myelin Sheath Defects, Cerebellar Degeneration, and Hepatocarcinomas*

J. Degen, K. Willecke, D. Hartmann, 2009, The Journal of Biological Chemistry.

Deletion of Connexin45 in Mouse Retinal Neurons Disrupts the Rod/Cone Signaling Pathway between AII Amacrine and ON Cone Bipolar Cells and Leads to Impaired Visual Transmission

R. Weiler, J. Ammermüller, J. Degen, 2005, The Journal of Neuroscience.

Expression pattern of lacZ reporter gene representing connexin36 in transgenic mice

J. Degen, K. Willecke, R. S. van der Giessen, 2004, The Journal of comparative neurology.

Functional Expression of the New Gap Junction Gene Connexin47 Transcribed in Mouse Brain and Spinal Cord Neurons

J. Degen, K. Willecke, K. Schilling, 2001, The Journal of Neuroscience.

Functional Expression of the Murine Connexin 36 Gene Coding for a Neuron-Specific Gap Junctional Protein

B. Teubner, J. Degen, K. Willecke, 2000, The Journal of Membrane Biology.

Functional characterization of enzymes catalyzing ceramide phosphoethanolamine biosynthesis in mice[S]

J. Degen, K. Willecke, D. Vance, 2015, Journal of Lipid Research.

Connexin 47 (Cx47)-Deficient Mice with Enhanced Green Fluorescent Protein Reporter Gene Reveal Predominant Oligodendrocytic Expression of Cx47 and Display Vacuolized Myelin in the CNS

K. Wellershaus, J. Degen, K. Willecke, 2003, The Journal of Neuroscience.

Ceramide synthase 4 deficiency in mice causes lipid alterations in sebum and results in alopecia.

J. Degen, K. Willecke, P. Dörmann, 2014, The Biochemical journal.

Characterization of Panglial Gap Junction Networks in the Thalamus, Neocortex, and Hippocampus Reveals a Unique Population of Glial Cells.

D. Cope, V. Crunelli, J. Degen, 2015, Cerebral cortex.

Functional role of connexin43 gap junction channels in adult mouse heart assessed by inducible gene deletion.

J. Degen, K. Willecke, J. D. de Bakker, 2004, Journal of molecular and cellular cardiology.

Characterization of connexin30.3-deficient mice suggests a possible role of connexin30.3 in olfaction.

J. Degen, K. Willecke, E. Dere, 2007, European journal of cell biology.

The murine gap junction gene connexin36 is highly expressed in mouse retina and regulated during brain development

J. Degen, K. Willecke, G. Söhl, 1998, FEBS letters.

A new conditional mouse mutant reveals specific expression and functions of connexin36 in neurons and pancreatic beta-cells.

J. Deuchars, K. Wellershaus, J. Degen, 2008, Experimental cell research.

Efficient genome engineering by targeted homologous recombination in mouse embryos using transcription activator-like effector nucleases

J. Degen, M. Beyer, J. Schultze, 2014, Nature Communications.

The oligodendroglial precursor cell line Oli-neu represents a cell culture system to examine functional expression of the mouse gap junction gene connexin29 (Cx29)

J. Degen, B. Odermatt, G. Söhl, 2013, Front. Pharmacol..

Visual Transmission Deficits in Mice with Targeted Disruption of the Gap Junction Gene Connexin36

R. Weiler, A. Feigenspan, J. Ammermüller, 2001, The Journal of Neuroscience.

Connexin30.2 containing gap junction channels decelerate impulse propagation through the atrioventricular node.

Alexander Ghanem, Klaus Tiemann, Thorsten Lewalter, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Behavioral alterations and changes in Ca/calmodulin kinase II levels in the striatum of connexin36 deficient mice

J. P. Huston, J. Degen, K. Willecke, 2012, Behavioural Brain Research.

New insights into the expression and function of neural connexins with transgenic mouse mutants

J. Degen, K. Willecke, B. Odermatt, 2004, Brain Research Reviews.

Inactivation of Ceramide Synthase 6 in Mice Results in an Altered Sphingolipid Metabolism and Behavioral Abnormalities*

J. Degen, K. Willecke, E. Dere, 2013, The Journal of Biological Chemistry.

Peripheral lymphangiogenesis in mice depends on ectodermal connexin-26 (Gjb2)

J. Degen, K. Willecke, T. Bald, 2011, Journal of Cell Science.

Ceramide Synthase 5 Is Essential to Maintain C16:0-Ceramide Pools and Contributes to the Development of Diet-induced Obesity*

M. Hoch, J. Degen, P. Dörmann, 2016, The Journal of Biological Chemistry.

Connexin Genes in the Mouse and Human Genome

J. Degen, K. Willecke, Jürgen Eiberger, 2001, Cell communication & adhesion.

Pathologic and Phenotypic Alterations in a Mouse Expressing a Connexin47 Missense Mutation That Causes Pelizaeus-Merzbacher–Like Disease in Humans

J. Degen, K. Willecke, E. Dere, 2011, PLoS genetics.

- 1-Ceramide Synthase 5 Is Essential to Maintain C 16 : 0 Ceramide Pools and Contributes to the Development of Diet Induced Obesity

M. Hoch, J. Degen, P. Dörmann, 2016 .

Inactivation of ceramide synthase 2 catalytic activity in mice affects transcription of genes involved in lipid metabolism and cell division.

J. Degen, K. Willecke, D. Hartmann, 2018, Biochimica et biophysica acta. Molecular and cell biology of lipids.

Dual reporter approaches for identification of Cre efficacy and astrocyte heterogeneity

J. Degen, K. Willecke, J. Trotter, 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.