H. Ginsburg

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Excess hemoglobin digestion and the osmotic stability of Plasmodium falciparum-infected red blood cells.

T. Tiffert, V. Lew, H. Ginsburg, 2003, Blood.

Intraerythrocytic Plasmodium falciparum utilizes only a fraction of the amino acids derived from the digestion of host cell cytosol for the biosynthesis of its proteins.

H. Ginsburg, M. Krugliak, Jianmin Zhang, 2002, Molecular and biochemical parasitology.

Membrane transport proteins of the malaria parasite

K. Kirk, H. Ginsburg, Rowena E. Martin, 2009, Molecular microbiology.

Inhibition of glutathione-dependent degradation of heme by chloroquine and amodiaquine as a possible basis for their antimalarial mode of action.

H. Ginsburg, M. Krugliak, Jianmin Zhang, 1998, Biochemical pharmacology.

Caveat emptor: limitations of the automated reconstruction of metabolic pathways in Plasmodium.

H. Ginsburg, 2009, Trends in parasitology.

Digestion of the host erythrocyte by malaria parasites is the primary target for quinoline-containing antimalarials.

H. Ginsburg, M. Krugliak, S. Zarchin, 1986, Biochemical pharmacology.

Metabolic interconnection between the human malarial parasite Plasmodium falciparum and its host erythrocyte. Regulation of ATP levels by means of an adenylate translocator and adenylate kinase.

H. Ginsburg, J. Kanaani, 1989, The Journal of biological chemistry.

Progress in in silico functional genomics: the malaria Metabolic Pathways database.

H. Ginsburg, 2006, Trends in parasitology.

Reduced microbicidal and anti‐tumour activities of human monocytes after ingestion of Plasmodium falciparum‐infected red blood cells

H. Ginsburg, P. Fiori, P. Rappelli, 1993, Parasite immunology.

Abundant proton pumping in Plasmodium, but why?

H. Ginsburg, 2002 .

Abundant proton pumping in Plasmodium falciparum, but why?

H. Ginsburg, 2002, Trends in parasitology.

Can biochemical properties serve as selective pressure for gene selection during inter-species and endosymbiotic lateral gene transfer?

Reinhart Heinrich, H. Ginsburg, O. Ebenhöh, 2006, Systems biology.

Data mining of the transcriptome of Plasmodium falciparum: the pentose phosphate pathway and ancillary processes

Hagai Ginsburg, Zbynek Bozdech, Zbynek Bozdech, 2005, Malaria Journal.

Is the development of falciparum malaria in the human host limited by the availability of uninfected erythrocytes?

M. Hoshen, H. Ginsburg, 2002, Malaria Journal.

Zero-trans and infinite-cis uptake of galactose in human erythrocytes.

H. Ginsburg, W. Stein, 1975, Biochimica et biophysica acta.

The transport of chloroquine across human erythrocyte membranes is mediated by a simple symmetric carrier.

H. Ginsburg, A. Yayon, 1982, Biochimica et biophysica acta.

Chronotherapy of malaria: an approach to malaria chemotherapy.

H. Ginsburg, G. Cambiè, I. Landau, 1991, Parasitology today.

Fractionation of mouse malarious blood according to parasite developmental stage, using a Percoll-sorbitol gradient.

H. Ginsburg, D. Mazier, I. Landau, 1987, Annales de parasitologie humaine et comparee.

Plasmodium falciparum metabolic pathways (MPMP) project upgraded with a database of subcellular locations of gene products.

Leann Tilley, Hagai Ginsburg, H. Ginsburg, 2011, Trends in parasitology.

Elevation of erythrocyte zinc- and free protoporphyrins with metastatic spread in cancer patients.

H. Ginsburg, Z. Fuks, T. Peretz, 1986, European journal of cancer & clinical oncology.

Stage sensitivity of Plasmodium vinckei petteri to quinine, mefloquine, and pyrimethamine.

H. Ginsburg, V. Caillard, I. Landau, 1995, The Journal of parasitology.

Chloroquine inhibits the degradation of endocytic vesicles in human malaria parasites.

H. Ginsburg, A. Yayon, 1983, Cell biology international reports.

Susceptibility of human malaria parasites to chloroquine is pH dependent.

H. Ginsburg, A. Yayon, Z. Cabantchik, 1985, Proceedings of the National Academy of Sciences of the United States of America.

Enhancement of the antimalarial effect of chloroquine on drug-resistant parasite strains--a critical examination of the reversal of multidrug resistance.

H. Ginsburg, 1991, Experimental parasitology.

Kinetic modelling of chloroquine uptake by malaria-infected erythrocytes. Assessment of the factors that may determine drug resistance.

H. Ginsburg, W. Stein, 1991, Biochemical pharmacology.

On the mode of action of phlorizin as an antimalarial agent in in vitro cultures of Plasmodium falciparum.

H. Ginsburg, W. Breuer, Z. Cabantchik, 1987, Biochemical pharmacology.

How many functional transport pathways does Plasmodium falciparum induce in the membrane of its host erythrocyte?

H. Ginsburg, W. Stein, 2005, Trends in parasitology.

Impairment of Macrophage Functions after Ingestion of Plasmodium s Erythrocytes or Isolated Malarial Pigment

H. Ginsburg, P. Arese, E. Schwarzer, 2003 .

Antimalarial properties of soy-bean fat emulsions.

H. Ginsburg, M. Krugliak, E. Deharo, 1995, International journal for parasitology.

Antiplasmodial Properties of Acyl-Lysyl Oligomers in Culture and Animal Models of Malaria

H. Vial, H. Ginsburg, M. Krugliak, 2011, Antimicrobial Agents and Chemotherapy.

Diffusion within egg lecithin bilayers resembles that within soft polymers

H. Ginsburg, W. R. Lieb, W. Stein, 1978, The Journal of general physiology.

Impairment of Macrophage Functions after Ingestion

H. Ginsburg, P. Arese, G. Giribaldi, 1992 .

Pharmacokinetic-pharmacodynamic modelling of the anti-malarial activity of mefloquine

M. Hoshen, H. Ginsburg, W. Stein, 2001, Parasitology.

Mathematical modelling of the chemotherapy of Plasmodium falciparum malaria with artesunate: postulation of ‘dormancy’, a partial cytostatic effect of the drug, and its implication for treatment regimens

M. Hoshen, H. Ginsburg, W. Stein, 2000, Parasitology.

A non-radiolabelled ferriprotoporphyrin IX biomineralisation inhibition test for the high throughput screening of antimalarial compounds.

H. Ginsburg, A. Giménez, M. Sauvain, 2002, Experimental parasitology.

Antimalarial Activities of Dermaseptin S4 Derivatives

H. Ginsburg, M. Krugliak, A. Mor, 2000, Antimicrobial Agents and Chemotherapy.

Oriented reconstitution of red cell membrane proteins and assessment of their transmembrane disposition by immunoquenching of fluorescence.

H. Ginsburg, Z. Cabantchik, A. Darmon, 1985, Biochimica et biophysica acta.

Direct Interaction of Dermaseptin S4 Aminoheptanoyl Derivative with Intraerythrocytic Malaria Parasite Leading to Increased Specific Antiparasitic Activity in Culture*

H. Ginsburg, A. Mor, L. Gaidukov, 2002, The Journal of Biological Chemistry.

Experimental conditions for testing the inhibitory activity of chloroquine on the formation of beta-hematin.

H. Ginsburg, M. Sauvain, V. Muñoz, 2000, Experimental parasitology.

In Vitro Antiplasmodium Effects of Dermaseptin S4 Derivatives

H. Ginsburg, A. Mor, L. Gaidukov, 2002, Antimicrobial Agents and Chemotherapy.

Mathematical modelling of malaria chemotherapy: combining artesunate and mefloquine

M. Hoshen, H. Ginsburg, W. Stein, 2002, Parasitology.

Selective toxicity to malaria parasites by non-intercalating DNA-binding ligands.

H. Ginsburg, M. Krugliak, D. Williamson, 1993, Molecular and biochemical parasitology.

Cellular Uptake of Chloroquine Is Dependent on Binding to Ferriprotoporphyrin IX and Is Independent of NHE Activity in Plasmodium falciparum

H. Ginsburg, S. Ward, M. Mungthin, 1999, The Journal of cell biology.

On the use of single-cell fluorescence imaging for the measurement of intracellular ion concentrations.

H. Ginsburg, 2000, Parasitology today.

The mysteries of hemoglobin degradation in Plasmodium falciparum-infected erythrocytes.

H. Ginsburg, 2003, Trends in parasitology.

Anion transport inhibitors as suppressors of Plasmodium falciparum growth in in vitro cultures.

H. Ginsburg, M. Krugliak, Z. Cabantchik, 1983, Molecular pharmacology.

A search for natural bioactive compounds in Bolivia through a multidisciplinary approach. Part IV. Is a new haem polymerisation inhibition test pertinent for the detection of antimalarial natural products?

H. Ginsburg, M. Sauvain, V. Muñoz, 2000, Journal of ethnopharmacology.

Erythrophagocytosis in malaria: Host defence or menace to the macrophage?

H. Ginsburg, P. Arese, F. Turrini, 1991, Parasitology today.

Synchronization ofPlasmodium yoelii nigeriensis andP. y. killicki infection in the mouse by means of Percoll-glucose gradient stage fractionation: Determination of the duration of the schizogonic cycle

H. Ginsburg, P. Gautret, E. Deharo, 2004, Parasitology Research.

The susceptibility of the malarial parasite Plasmodium falciparum to quinoline-containing drugs is correlated to the lipid composition of the infected erythrocyte membranes.

H. Ginsburg, G. Shalmiev, 1993, Biochemical pharmacology.

Heme Degradation in the Presence of Glutathione

H. Ginsburg, H. Atamna, 1995, The Journal of Biological Chemistry.

Transglutaminase in Plasmodium parasites: activity and putative role in oocysts and blood stages.

H. Ginsburg, M. Krugliak, A. Warburg, 2001, Molecular and biochemical parasitology.

Analysis of additivity and synergism in the anti-plasmodial effect of purified compounds from plant extracts

H. Ginsburg, E. Deharo, 2011, Malaria Journal.

A call for using natural compounds in the development of new antimalarial treatments – an introduction

H. Ginsburg, E. Deharo, 2011, Malaria Journal.

Ethnopharmacology and malaria: new hypothetical leads or old efficient antimalarials?

H. Ginsburg, P. Rasoanaivo, B. Graz, 2008, International journal for parasitology.

Potent antimalarial activity of clotrimazole in in vitro cultures of Plasmodium falciparum.

T. Tiffert, H. Ginsburg, M. Krugliak, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Origin of reactive oxygen species in erythrocytes infected with Plasmodium falciparum.

H. Ginsburg, H. Atamna, Hagai Ginsburg, 1993, Molecular and biochemical parasitology.

New permeability pathways induced by the malarial parasite in the membrane of its host erythrocyte: Potential routes for targeting of drugs into infected cells

H. Ginsburg, W. Stein, 1987, Bioscience reports.

Current concepts and new ideas on the mechanism of action of quinoline-containing antimalarials.

H. Ginsburg, T. Geary, 1987, Biochemical pharmacology.

Characterization of permeation pathways appearing in the host membrane of Plasmodium falciparum infected red blood cells.

H. Ginsburg, M. Krugliak, Z. Cabantchik, 1985, Molecular and biochemical parasitology.

Fusion of liposomes with planar lipid bilayers.

H. Ginsburg, M. Razin, 1980, Biochimica et biophysica acta.

Correlation of erythrocyte and plasma levels of zinc, copper, and iron with evidence of metastatic spread in cancer patients

H. Ginsburg, Z. Fuks, A. Sulkes, 1985, Cancer.

Oxidative stress in malaria parasite-infected erythrocytes: host-parasite interactions.

K. Becker, H. Ginsburg, L. Tilley, 2004, International journal for parasitology.

Effects of cinnamic acid derivatives on in vitro growth of Plasmodium falciparum and on the permeability of the membrane of malaria-infected erythrocytes

H. Ginsburg, J. Kanaani, 1992, Antimicrobial Agents and Chemotherapy.

New permeability pathways induced in membranes of Plasmodium falciparum infected erythrocytes.

H. Ginsburg, M. Krugliak, O. Eidelman, 1983, Molecular and biochemical parasitology.

Facilitated transport of di- and trinitrophenolate ions across lipid membranes by valinomycin and nonactin.

H. Ginsburg, G. Stark, 1976, Biochimica et biophysica acta.

Radial Water and Solute Flows in Roots of Zea mays IV. ELECTRICAL POTENTIAL PROFILES ACROSS THE ROOT

H. Ginsburg, B. Ginzburg, 1974 .

Radial Water and Solute Flows in Roots of Zea mays: I. WATER FLOW

H. Ginsburg, B. Ginzburg, 1970 .

Effects of mitochondrial protein synthesis inhibitors on the incorporation of isoleucine into Plasmodium falciparum in vitro.

J. Blum, H. Ginsburg, A. Yayon, 1984, The Journal of protozoology.

Antimalarial effects of C18 fatty acids on Plasmodium falciparum in culture and on Plasmodium vinckei petteri and Plasmodium yoelii nigeriensis in vivo.

H. Ginsburg, M. Krugliak, C. Moretti, 1995, Experimental parasitology.

Interactions of hemin, antimalarial drugs and hemin-antimalarial complexes with phospholipid monolayers.

H. Ginsburg, R. Demel, Rudy A. Demel, 1984, Chemistry and physics of lipids.

An integrated model of chloroquine action.

H. Ginsburg, S. Ward, P. Bray, 1999, Parasitology today.

Inhibition by anti-malarial drugs of haemoglobin denaturation and iron release in acidified red blood cell lysates – a possible mechanism of their anti-malarial effect?

H. Ginsburg, M. Krugliak, T. Gabay, 1994, Parasitology.

Quinoline-containing antimalarials--mode of action, drug resistance and its reversal. An update with unresolved puzzles.

H. Ginsburg, M. Krugliak, Hagai Ginsburg, 1992, Biochemical pharmacology.

Kinetics of inhibition of glutathione-mediated degradation of ferriprotoporphyrin IX by antimalarial drugs.

H. Ginsburg, M. Krugliak, O. Famin, 1999, Biochemical pharmacology.

The status of zinc in malaria (Plasmodium falciparum) infected human red blood cells: stage dependent accumulation, compartmentation and effect of dipicolinate.

H. Ginsburg, M. Krugliak, R. Gorodetsky, 1986, Biochimica et biophysica acta.

Antimalarial drugs inhibit the phagocytosis of erythrocytes infected with Plasmodium falciparum.

H. Ginsburg, M. Krugliak, F. Turrini, 1996, Transactions of the Royal Society of Tropical Medicine and Hygiene.

Resistance of glucose-6-phosphate dehydrogenase deficiency to malaria: effects of fava bean hydroxypyrimidine glucosides on Plasmodium falciparum growth in culture and on the phagocytosis of infected cells

H. Ginsburg, M. Krugliak, J. Kanaani, 1996, Parasitology.

Mode of antimalarial effect of methylene blue and some of its analogues on Plasmodium falciparum in culture and their inhibition of P. vinckei petteri and P. yoelii nigeriensis in vivo.

H. Ginsburg, M. Krugliak, H. Atamna, 1996, Biochemical pharmacology.

Antiplasmodial Activity of Lauryl-Lysine Oligomers

H. Ginsburg, M. Krugliak, I. Radzishevsky, 2007, Antimicrobial Agents and Chemotherapy.

Structural and Functional Properties of the Anion Transport System Isolated from Human Erythrocyte Membranes

H. Ginsburg, Z. Cabantchik, J. Wolosin, 1977 .

invasion by Plasmodium falciparum The hydration state of human red blood cells and their susceptibility to

T. Tiffert, V. Lew, H. Ginsburg, 2013 .

The hydration state of human red blood cells and their susceptibility to invasion by Plasmodium falciparum.

T. Tiffert, V. Lew, H. Ginsburg, 2005, Blood.

The evolution of the new permeability pathways in Plasmodium falciparum--infected erythrocytes--a kinetic analysis.

H. Ginsburg, M. Krugliak, 2006, Experimental parasitology.

Galactose transport in human erythrocytes. The transport mechanism is resolved into two simple asymmetric antiparallel carriers.

H. Ginsburg, 1978, Biochimica et biophysica acta.

Functional characterization of anion transport system isolated from human erythrocyte membranes.

H. Ginsburg, J. Wolosin, Z. I. Cabantchik, 1977, The Journal of biological chemistry.

Effects of red blood cell potassium and hypertonicity on the growth ofPlasmodium falciparum in culture

H. Ginsburg, M. Krugliak, S. Friedman, 2004, Zeitschrift für Parasitenkunde.

Uptake of L-tryptophan by erythrocytes infected with malaria parasites (Plasmodium falciparum).

H. Ginsburg, M. Krugliak, 1983, Biochimica et biophysica acta.

The biochemistry of Plasmodium falciparum

H. Ginsburg, 2016 .

Inhibition of P. Falciparum Growth in vitro by Specific Inhibitors of Red Blood Cell Anion Transport

H. Ginsburg, M. Krugliak, Z. Cabantchik, 1981 .

Permselectivity changes in malaria (Plasmodium falciparum) infected human red blood cell membranes

H. Ginsburg, Z. Cabantchik, S. Kutner, 1983, Journal of cellular physiology.

Transport pathways in the malaria-infected erythrocyte. Their characterization and their use as potential targets for chemotherapy.

H. Ginsburg, 1994, Biochemical pharmacology.

Kinetic characterization of Na+/H+ antiport of Plasmodium falciparum membrane

H. Ginsburg, F. Turrini, D. Ghigo, 1993, Journal of cellular physiology.

How and Why Does the Malarial Parasite Permeabilize Its Host Cell Membrane

H. Ginsburg, 1988 .

Effects of mitochondrial inhibitors on intraerythrocytic Plasmodium falciparum in in vitro cultures.

H. Ginsburg, J. Jensen, T. Geary, 1986, The Journal of protozoology.

Selectivity properties of pores induced in host erythrocyte membrane by Plasmodium falciparum. Effect of parasite maturation.

H. Ginsburg, Z. Cabantchik, S. Kutner, 1986, Biochimica et biophysica acta.

Alterations in membrane permeability of malaria-infected human erythrocytes are related to the growth stage of the parasite.

H. Ginsburg, Z. Cabantchik, S. Kutner, 1982, Biochimica et biophysica acta.

The mitochondrion of Plasmodium falciparum visualized by rhodamine 123 fluorescence.

H. Ginsburg, J. Jensen, T. Geary, 1985, The Journal of protozoology.

The permeability properties of the parasite cell membrane.

H. Ginsburg, 1999, Novartis Foundation symposium.

How Plasmodium secures nutrients: new targets for drugs?

H. Ginsburg, 1994, Parasitology today.

Transport of uridine in human red blood cells. Demonstration of a simple carrier-mediated process

H. Ginsburg, Z. Cabantchik, Z. I. Cabantchik, 1977, The Journal of general physiology.

Hemoglobin denaturation and iron release in acidified red blood cell lysate--a possible source of iron for intraerythrocytic malaria parasites.

H. Ginsburg, T. Gabay, Hagai Ginsburg, 1993, Experimental parasitology.

Recognition signals for phagocytic removal of favic, malaria-infected and sickled erythrocytes.

H. Ginsburg, H. Lutz, P. Arese, 1991, Advances in experimental medicine and biology.

Single or multiple localization of ADP/ATP transporter in human malarial Plasmodium falciparum.

H. Ginsburg, I. Hatin, G. Jaureguiberry, 1992, Biochemical pharmacology.

Modulation of cerebral malaria by fasudil and other immune-modifying compounds.

H. Ginsburg, J. Golenser, N. Hunt, 2010, Experimental parasitology.

Artemisinin resistance in the malaria parasite, Plasmodium falciparum, originates from its initial transcriptional response

Zbynek Bozdech, P. Newton, H. Ginsburg, 2022, Communications Biology.

The mechanism of artemisinin resistance of Plasmodium falciparum malaria parasites originates in their initial transcriptional response

Zbynek Bozdech, P. Newton, H. Ginsburg, 2021, bioRxiv.

Role of internal domains of glycophorin in Plasmodium falciparum invasion of human erythrocytes

H. Ginsburg, W. Breuer, Z. Cabantchik, 1983, Infection and immunity.

An assay of malaria parasite invasion into human erythrocytes. The effects of chemical and enzymatic modification of erythrocyte membrane components.

H. Ginsburg, W. Breuer, Z. Cabantchik, 1983, Biochimica et biophysica acta.

The malarial pigment in rat infected erythrocytes and its interaction with chloroquine. A Mössbauer effect study.

H. Ginsburg, A. Yayon, E. Bauminger, 1984, Journal of Biological Chemistry.

Uptake of [3H]chloroquine by drug-sensitive and -resistant strains of the human malaria parasite Plasmodium falciparum.

H. Ginsburg, J. Jensen, T. Geary, 1986, Biochemical pharmacology.

Mathematical modelling of the within-host dynamics of Plasmodium falciparum

R Heinrich, Reinhart Heinrich, M. Hoshen, 2000, Parasitology.

Should chloroquine be laid to rest?

H. Ginsburg, 2005, Acta tropica.

Transfer of Functional Components into Plasma Membrane of Living Cells: A New Tool in Membrane Research

H. Ginsburg, D. Volsky, A. Loyter, 1980 .

Potentiation of the antimalarial action of chloroquine in rodent malaria by drugs known to reduce cellular glutathione levels.

H. Ginsburg, J. Golenser, M. Krugliak, 2003, Biochemical pharmacology.

Evidence for active water transport in a corn root preparation

H. Ginsburg, B. Ginzburg, 1971, The Journal of Membrane Biology.

Impairment of macrophage functions after ingestion of Plasmodium falciparum-infected erythrocytes or isolated malarial pigment [published erratum appears in J Exp Med 1993 Mar 1;177(3):following 873]

H. Ginsburg, P. Arese, E. Schwarzer, 1992, The Journal of experimental medicine.

Phagocytosis of Plasmodium falciparum-infected human red blood cells by human monocytes: involvement of immune and nonimmune determinants and dependence on parasite developmental stage.

H. Ginsburg, P. Arese, F. Bussolino, 1992, Blood.

Antioxidant defense in Plasmodium falciparum – data mining of the transcriptome

Zbynek Bozdech, H. Ginsburg, Hagai Ginsburg, 2004, Malaria Journal.

The transcriptome of Plasmodium vivax reveals divergence and diversity of transcriptional regulation in malaria parasites

Hagai Ginsburg, Bruce Russell, Francois Nosten, 2008, Proceedings of the National Academy of Sciences.

Kinetic modelling of the response of Plasmodium falciparum to chloroquine and its experimental testing in vitro. Implications for mechanism of action of and resistance to the drug.

H. Ginsburg, J. Jensen, T. Geary, 1990, Biochemical pharmacology.

Identification of the acidic compartment of Plasmodium falciparum‐infected human erythrocytes as the target of the antimalarial drug chloroquine.

H. Ginsburg, A. Yayon, Z. Cabantchik, 1984, The EMBO journal.

Chloroquine - some open questions on its antimalarial mode of action and resistance.

H. Ginsburg, M. Krugliak, 1999, Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy.

Effect of calcium antagonists on malaria susceptibility to chloroquine.

H. Ginsburg, 1988, Parasitology today.

Modelling the chloroquine chemotherapy of falciparum malaria: the value of spacing a split dose

M. Hoshen, H. Ginsburg, W. Stein, 1998, Parasitology.

Glutathione is involved in the antimalarial action of chloroquine and its modulation affects drug sensitivity of human and murine species of Plasmodium

H. Ginsburg, J. Golenser, 2003, Redox report : communications in free radical research.

The malaria parasite supplies glutathione to its host cell--investigation of glutathione transport and metabolism in human erythrocytes infected with Plasmodium falciparum.

H. Ginsburg, H. Atamna, Hagai Ginsburg, 1997, European journal of biochemistry.

Hexose-monophosphate shunt activity in intact Plasmodium falciparum-infected erythrocytes and in free parasites.

H. Ginsburg, H. Atamna, G. Pascarmona, 1994, Molecular and biochemical parasitology.

The redox status of malaria-infected erythrocytes: an overview with an emphasis on unresolved problems.

H. Ginsburg, H. Atamna, Ginsburg H And, 1994, Parasite.

The fate of ferriprotorphyrin IX in malaria infected erythrocytes in conjunction with the mode of action of antimalarial drugs.

H. Ginsburg, M. Krugliak, Jianmin Zhang, 1999, Molecular and biochemical parasitology.

Inhibition of malaria parasite invasion of human erythrocytes by a lymphocyte membrane polypeptide

H. Ginsburg, Y. Milner, R. Benzaquen-Geffin, 1987, Infection and immunity.

Excess haemoglobin digestion by malaria parasites: a strategy to prevent premature host cell lysis.

T. Tiffert, V. Lew, H. Ginsburg, 2004, Blood cells, molecules & diseases.

Antimalarial drug development and new targets.

H. Ginsburg, L. Tilley, W. Sirawaraporn, 2000, Parasitology today.

Na+/H+ antiporter, chloroquine uptake and drug resistance: inconsistencies in a newly proposed model.

H. Ginsburg, S. Ward, P. Bray, 1999, Parasitology today.

Antimalarial drugs: is the lysosomotropic hypothesis still valid?

H. Ginsburg, 1990, Parasitology today.

Erythrocyte stages of Plasmodium falciparum exhibit a high nitric oxide synthase (NOS) activity and release an NOS-inducing soluble factor

H. Ginsburg, F. Bussolino, P. Gautret, 1995, The Journal of experimental medicine.

Differential effects of 4-aminoquinoline-containing antimalarial drugs on hemoglobin digestion in Plasmodium falciparum-infected erythrocytes.

H. Ginsburg, O. Famin, 2002, Biochemical pharmacology.

Optimisation of flow cytometric measurement of parasitaemia in plasmodium-infected mice.

H. Ginsburg, J. Golenser, H Ginsburg, 2000, International journal for parasitology.

Model for iso-osmotic water flow in plant roots.

H. Ginsburg, 1971, Journal of theoretical biology.

Electrophysiological studies of malaria parasite-infected erythrocytes: current status.

P. Verloo, K. Kirk, K. Saliba, 2007, International journal for parasitology.

Analysis of plant root electropotentials.

H. Ginsburg, 1972, Journal of theoretical biology.

Effects of temperature on the transport of galactose in human erythrocytes.

H. Ginsburg, S. Yeroushalmy, 1978, The Journal of physiology.

Mössbauer studies of malaria

H. Ginsburg, A. Yayon, S. Ofer, 1983 .

RECONSTITUTION OF THE ERYTHROCYTE ANION TRANSPORT SYSTEM: IN VITRO AND IN VIVO APPROACHES *

H. Ginsburg, D. Volsky, A. Loyter, 1980, Annals of the New York Academy of Sciences.

Pharmacogenomic analyses of targeting the AT-rich malaria parasite genome with AT-specific alkylating drugs.

H. Ginsburg, M. Krugliak, J. Woynarowski, 2007, Molecular and biochemical parasitology.

Effects of quinoline-containing antimalarials on the erythrocyte membrane and their significance to drug action on Plasmodium falciparum.

H. Ginsburg, M. Krugliak, 1988, Biochemical pharmacology.

The effect of ferriprotoporphyrin IX and chloroquine on phospholipid monolayers and the possible implications to antimalarial activity.

H. Ginsburg, R. Demel, 1983, Biochimica et biophysica acta.

Biophysical analysis of novel transport pathways induced in red blood cell membranes

H. Ginsburg, W. Stein, 2005, The Journal of Membrane Biology.

The Homeostasis of Plasmodium falciparum-Infected Red Blood Cells

Clemens F. Kaminski, Hagai Ginsburg, Virgilio L. Lew, 2009, PLoS Comput. Biol..

Effects of chloroquine on the feeding mechanism of the intraerythrocytic human malarial parasite Plasmodium falciparum.

H. Ginsburg, A. Yayon, R. Timberg, 1984, The Journal of protozoology.

Absence of α‐Ketoglutarate Dehydrogenase Activity and Presence of CO2‐Fixing Activity in Plasmodium falciparum Grown In Vitro in Human Erythrocytes1

J. Blum, H. Ginsburg, 1984 .

The Plasmodium digestive vacuole and drug choice.

H. Ginsburg, 1996, Parasitology today.

Alkalinization of the food vacuole of malaria parasites by quinoline drugs and alkylamines is not correlated with their antimalarial activity.

H. Ginsburg, M. Krugliak, E. Nissani, 1989, Biochemical pharmacology.

The treatment of Plasmodium falciparum-infected erythrocytes with chloroquine leads to accumulation of ferriprotoporphyrin IX bound to particular parasite proteins and to the inhibition of the parasite's 6-phosphogluconate dehydrogenase.

H. Ginsburg, O. Famin, Hagai Ginsburg, 2003, Parasite.

Orientation of transmembrane polypeptides as revealed by antibody quenching of fluorescence.

H. Ginsburg, Z. Cabantchik, A. Darmon, 1984, Biochimica et biophysica acta.

Calcium transport and compartment analysis of free and exchangeable calcium in Plasmodium falciparum-infected red blood cells.

H. Ginsburg, R. Kramer, 1991, The Journal of protozoology.

Compartment analysis of ATP in malaria-infected erythrocytes.

H. Ginsburg, J. Kanaani, 1988, Biochemistry international.

Chronotherapy of malaria: identification of drug-sensitive stage of parasite and timing of drug delivery for improved therapy.

H. Ginsburg, G. Cambiè, V. Caillard, 1991, Annales de parasitologie humaine et comparee.

Experimental modifications of the circadian rythm of Plasmodium vinckei petteri following cryopreservation; probable resistance of the merozoïte to thawing.

H. Ginsburg, I. Landau, A. Chabaud, 1988, Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie.

QUININE, MEFLOQUINE, AND PYRIMETHAMINE

H. Ginsburg, V. Caillard, I. Landau, 1995 .

Chronotherapy of malaria: improved efficacy of timed chloroquine treatment of patients with Plasmodium falciparum infections.

H. Ginsburg, P. Ringwald, P. Rabarison, 1992, Transactions of the Royal Society of Tropical Medicine and Hygiene.

The pharmacokinetics of chloroquine in healthy and Plasmodium chabaudi-infected mice: implications for chronotherapy.

H. Ginsburg, C. Gaudebout, G. Cambiè, 1994, Parasite.

Arylpiperazines displaying preferential potency against chloroquine-resistant strains of the malaria parasite Plasmodium falciparum.

Hagai Ginsburg, Kelly Chibale, Miriam Krugliak, 2005, Biochemical pharmacology.

Killing of intraerythrocytic Plasmodium falciparum by lysosomotropic amino acid esters

H. Ginsburg, M. Krugliak, Jianmin Zhang, 2003, Parasitology Research.

The New Permeability Pathways Induced by the Malaria Parasite in the Membrane of the Infected Erythrocyte: Comparison of Results Using Different Experimental Techniques

H. Ginsburg, W. Stein, 2004, The Journal of Membrane Biology.

Covalent modification of the permeability pathways induced in the human erythrocyte membrane by the malarial parasite Plasmodium falciparum

H. Ginsburg, Z. Cabantchik, W. Breuer, 1987, Journal of cellular physiology.

Effects of uranyl ions on lipid bilayer membranes

H. Ginsburg, J. Wolosin, 1979 .

Transport and trafficking in the malaria-infected erythrocyte.

K. Kirk, H. Ginsburg, L. Tilley, 1999, Parasitology today.

Evidence from electron paramagnetic resonance for function-related conformation changes in the anion-transport protein of human erythrocytes.

H. Ginsburg, C. Grisham, S. E. O'Connor, 1981, European journal of biochemistry.

Effects of combinations of quinoline-containing antimalarials on Plasmodium falciparum in culture.

H. Ginsburg, J. Jensen, T. Geary, 1986, Annals of tropical medicine and parasitology.

The permeation of organic acids through lecithin bilayers. Resemblance to diffusion in polymers.

H. Ginsburg, J. Wolosin, 1975, Biochimica et biophysica acta.

The modulatory effect of membrane viscosity on structural and functional properties of the anion exchange protein of human erythrocytes.

H. Ginsburg, Z. Cabantchik, D. Baruch, 1986, Membrane biochemistry.

Do azoles and chloroquine share similar mechanisms of antimalarial action?

T. Tiffert, V. Lew, H. Ginsburg, 2002, Trends in parasitology.

falciparum-infected red blood cells Excess hemoglobin digestion and the osmotic stability of Plasmodium

T. Tiffert, V. Lew, H. Ginsburg, 2013 .

Response to: Allen and Kirk: Cell volume control in the Plasmodium-infected erythrocyte

T. Tiffert, V. Lew, H. Ginsburg, 2004 .

Hydrophobic interactions in Plasmodium falciparum invasion into human erythrocytes.

H. Ginsburg, W. Breuer, Z. Cabantchik, 1984, Molecular and biochemical parasitology.

Malaria Parasite Metabolic Pathways (MPMP) Upgraded with Targeted Chemical Compounds.

Alyaa M. Abdel-Haleem, H. Ginsburg, 2016, Trends in parasitology.

Studies on the antimalarial mode of action of quinoline-containing drugs: time-dependence and irreversibility of drug action, and interactions with compounds that alter the function of the parasite's food vacuole.

H. Ginsburg, M. Krugliak, 1991, Life sciences.

Effect of Ferriprotoporphyrin IX and Non-heme Iron on the Ca2+ Pump of Intact Human Red Cells

T. Tiffert, H. Ginsburg, D. Perdomo, 2000, The Journal of Membrane Biology.

RoleofInternal Domains ofGlycophorin inPlasmodium falciparum Invasion ofHumanErythrocytes

H. Ginsburg, I. Cabantchik, W. Breuer, 1983 .

Transport pathways in the malaria-infected erythrocyte: characterization and their use as potential targets for chemotherapy.

H. Ginsburg, 1994, Memorias do Instituto Oswaldo Cruz.

Redox metabolism in malaria: from genes, through biochemistry and pathology, to drugs

H. Ginsburg, 2003, Redox report : communications in free radical research.

Effects of lysosomotropic detergents on the human malarial parasite Plasmodium falciparum in in vitro culture.

H. Ginsburg, M. Krugliak, R. Firestone, 1989, Biochemical pharmacology.

Gentamicin and amikacin repress the growth of Plasmodium falciparum in culture, probably by inhibiting a parasite acid phospholipase.

H. Ginsburg, M. Krugliak, Z. Waldman, 1987, Life sciences.

Metabolism: Malaria parasite stands out

H. Ginsburg, 2010, Nature.

Effect of cholesterol-rich diet on the susceptibility of rodent malarial parasites to chloroquine chemotherapy.

H. Ginsburg, I. Landau, D. Baccam, 1988, Life sciences.

Protonophoric effects of antimalarial drugs and alkylamines in Escherichia coli membranes.

H. Ginsburg, E. Nissani, 1989, Biochimica et biophysica acta.

Functional incorporation of the human erythrocyte chloride exchange system into plasma membranes of Friend erythroleukemic cells by Sendai virus-induced cell fusion.

H. Ginsburg, D. Volsky, A. Loyter, 1980, Experimental cell research.

Inhibition of malaria parasite invasion of human erythrocytes by a lymphocyte membrane polypeptide

H. Ginsburg, Y. Milner, R. Benzaquen-Geffin, 1987, Infection and immunity.

Some effects of trinitrocresolate and valinomycin on Na and K transport across thin lipid bilayer membranes: A steady-state analysis with simultaneous tracer and electrical measurements

D. Tosteson, H. Ginsburg, M. Tosteson, 1978, The Journal of Membrane Biology.

Plasmodium falciparum: elimination of fungal and bacterial contamination from in vitro culture.

H. Ginsburg, A. Yayon, S. Friedman, 1984, Annals of tropical medicine and parasitology.

Zero-trans and equilibrium-exchange efflux and infinite-trans uptake of galactose by human erythrocytes.

H. Ginsburg, D. Ram, 1975, Biochimica et biophysica acta.

Radial Water and Solute Flow in Roots of Zea maysIII. EFFECT OF TEMPERATURE ON THO AND ION TRANSPORT

H. Ginsburg, B. Ginzburg, 1971 .

Direct fluorometric determination of erythrocyte free and zinc protoporphyrins in health and disease.

Z Fuks, H. Ginsburg, Z. Fuks, 1985, Clinical biochemistry.

Membrane transport proteins of the malaria parasite

K. Kirk, H. Ginsburg, Rowena E. Martin, 2009, Molecular microbiology.

Heme Degradation in the Presence of Glutathione A PROPOSED MECHANISM TO ACCOUNT FOR THE HIGH LEVELS OF NON-HEME IRON FOUND IN THE MEMBRANES OF HEMOGLOBINOPATHIC RED BLOOD CELLS*

H. Ginsburg, H. Atamna, 1995 .

Some reflections concerning host erythrocyte-malarial parasite interrelationships.

H. Ginsburg, 1990, Blood cells.