C. Westphal

发表

The Neuroendocrine Protein 7B2 Is Required for Peptide Hormone Processing In Vivo and Provides a Novel Mechanism for Pituitary Cushing’s Disease

P. Leder, C. Westphal, L. Muller, 1999, Cell.

Loss of atm radiosensitizes multiple p53 null tissues.

P. Leder, C. Westphal, M. Kastan, 1998, Cancer research.

Genetic interactions between atm and p53 influence cellular proliferation and irradiation-induced cell cycle checkpoints.

P. Leder, C. Westphal, D. Fisher, 1997, Cancer research.

Cell-cycle signaling: Atm displays its many talents

C. Westphal, Christoph H. Westphal, 1997, Current Biology.

atm and p53 cooperate in apoptosis and suppression of tumorigenesis, but not in resistance to acute radiation toxicity

P. Leder, C. Westphal, D. Fisher, 1997, Nature Genetics.

Transposon-generated ‘knock-out’ and ‘knock-in’ gene-targeting constructs for use in mice

P. Leder, C. Westphal, 1997, Current Biology.

The mammalian mid-pachytene checkpoint: meiotic arrest in spermatocytes with a mutation in Atm alone or in combination with a Trp53 (p53) or Cdkn1a (p21/cip1) mutation

C. Westphal, D. D. de Rooij, T. Ashley, 2004, Cytogenetic and Genome Research.

Ataxia Telangiectasia Mutated Expression and Activation in the Testis1

C. Westphal, H. Kal, D. D. de Rooij, 2004, Biology of reproduction.

ATM Expression and Activation in the Testis 1

C. Westphal, H. Kal, T. Ashley, 2003 .

Erratum: Atm inactivation results in aberrant telomere clustering during meiotic prophase (Molecular and Cellular Biology (1999) 19:7 (5096-5105))

H. Scherthan, T. Pandita, C. Westphal, 1999 .

Atm-dependent interactions of a mammalian Chk1 homolog with meiotic chromosomes

C. Westphal, A. Carr, M. Hoekstra, 1997, Current Biology.

Atm Inactivation Results in Aberrant Telomere Clustering during Meiotic Prophase

Harry Scherthan, H. Scherthan, T. Pandita, 1999, Molecular and Cellular Biology.