Cecilia S. L. Lai

发表

Identification of FOXP2 truncation as a novel cause of developmental speech and language deficits.

N. Sykes, A. Monaco, F. Vargha-Khadem, 2005, American journal of human genetics.

FOXP2 expression during brain development coincides with adult sites of pathology in a severe speech and language disorder.

A. Monaco, S. Fisher, Cecilia S. L. Lai, 2003, Brain : a journal of neurology.

A forkhead-domain gene is mutated in a severe speech and language disorder

A. Monaco, F. Vargha-Khadem, S. Fisher, 2001, Nature.

Molecular evolution of FOXP2, a gene involved in speech and language

A. Monaco, S. Pääbo, W. Enard, 2002, Nature.

Deciphering the genetic basis of speech and language disorders.

A. Monaco, S. Fisher, Cecilia S. L. Lai, 2003, Annual review of neuroscience.

The chromosome 6p22 haplotype associated with dyslexia reduces the expression of KIAA0319, a novel gene involved in neuronal migration.

Richard Wade-Martins, Yu Wang, C. Francks, 2006, Human molecular genetics.

FOXP2 is not a major susceptibility gene for autism or specific language impairment.

G Baird, A. Bailey, G. Baird, 2002, American journal of human genetics.

A novel transcription factor is mutated in a severe speech and language disorder

A. Monaco, F. Vargha-Khadem, S. Fisher, 2001 .

The SPCH1 region on human 7q31: genomic characterization of the critical interval and localization of translocations associated with speech and language disorder.

A. Monaco, E. Green, F. Vargha-Khadem, 2000, American journal of human genetics.