S. Cialfi

发表

Characterization of Wnt/β-catenin signaling in rhabdomyosarcoma

S. Annavarapu, S. Cialfi, C. Dominici, 2013, Laboratory Investigation.

PLK1 targets NOTCH1 during DNA damage and mitotic progression

I. Screpanti, D. Bellavia, S. Checquolo, 2019, The Journal of Biological Chemistry.

Gefitinib in combination with oral topotecan and cyclophosphamide in relapsed neuroblastoma: Pharmacological rationale and clinical response

A. Jenkner, A. Donfrancesco, S. Cialfi, 2010, Pediatric blood & cancer.

Functional cooperation between ASK1 and p21Waf1/Cip1 in the balance of cell-cycle arrest, cell death and tumorigenesis of stressed keratinocytes

I. Screpanti, S. Cialfi, H. Ichijo, 2021, Cell death discovery.

Yeast-Based Screen to Identify Natural Compounds with a Potential Therapeutic Effect in Hailey-Hailey Disease

S. Cialfi, D. Uccelletti, Azzurra Zonfrilli, 2018, International journal of molecular sciences.

MicroRNAs as Modulators of the Immune Response in T-Cell Acute Lymphoblastic Leukemia

I. Screpanti, S. Cialfi, M. P. Felli, 2022, International journal of molecular sciences.

The deregulated expression of miR-125b in acute myeloid leukemia is dependent on the transcription factor C/EBPα

I. Screpanti, D. Bellavia, S. Checquolo, 2015, Leukemia.

Glutathione S-transferase ϴ-subunit as a phenotypic suppressor of pmr1Δ strain, the Kluyveromyces lactis model for Hailey-Hailey disease.

S. Cialfi, D. Uccelletti, G. Biolcati, 2016, Biochimica et biophysica acta.

KlHsl1 is a component of glycerol response pathways in the milk yeast Kluyveromyces lactis.

H. Heipieper, S. Cialfi, M. Wésolowski-Louvel, 2011, Microbiology.

KlHsl 1 is a component of glycerol response pathways in the milk yeast Kluyveromyces lactis

H. Heipieper, S. Cialfi, M. Wésolowski-Louvel, 2011 .

miR ‐ 125 b / NRF 2 / HO ‐ 1 axis is involved in protection against oxidative stress of cystic fibrosis : A pilot study

I. Screpanti, S. Cialfi, A. Pizzuti, 2021 .

Elevated levels of miR-145 correlate with SMAD3 down-regulation in cystic fibrosis patients.

S. Cialfi, C. Dominici, A. Pizzuti, 2013, Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society.

5FU/Oxaliplatin-Induced Jagged1 Cleavage Counteracts Apoptosis Induction in Colorectal Cancer: A Novel Mechanism of Intrinsic Drug Resistance

M. V. Giuli, I. Screpanti, D. Bellavia, 2022, Frontiers in Oncology.

The Translation Factor eIF6 Is a Notch-Dependent Regulator of Cell Migration and Invasion

S. Cialfi, P. Londei, D. Benelli, 2012, PloS one.

Cross talk among Notch3, pre-TCR, and Tal1 in T-cell development and leukemogenesis.

I. Screpanti, S. Cialfi, L. Frati, 2006, Blood.

Complex multipathways alterations and oxidative stress are associated with Hailey–Hailey disease

I. Screpanti, D. Taruscio, S. Cialfi, 2010, The British journal of dermatology.

Author's response to reviews Title:Deep Sequencing the microRNA profile in rhabdomyosarcoma reveals down-regulation of miR-378 family members. Authors:

S. Cialfi, H. McDowell, A. Pizzuti, 2014 .

Hypotonic, Acidic Oxidizing Solution Containing Hypochlorous Acid (HClO) as a Potential Treatment of Hailey-Hailey Disease

I. Screpanti, S. Cialfi, Azzurra Zonfrilli, 2019, Molecules.

Corrigendum: The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease

I. Screpanti, S. Cialfi, D. Uccelletti, 2017, Scientific reports.

The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease

I. Screpanti, S. Cialfi, D. Uccelletti, 2016, Scientific Reports.

Efficacy of the melanocortin analogue Nle4-D-Phe7-α-melanocyte-stimulating hormone in the treatment of patients with Hailey–Hailey disease

I. Screpanti, S. Cialfi, G. Biolcati, 2013, Clinical and experimental dermatology.

Oxidative stress activation of miR‐125b is part of the molecular switch for Hailey–Hailey disease manifestation

I. Screpanti, G. Melino, D. Taruscio, 2011, Experimental dermatology.

Synergistic Post-Transcriptional Regulation of the Cystic Fibrosis Transmembrane conductance Regulator (CFTR) by miR-101 and miR-494 Specific Binding

S. Cialfi, C. Dominici, A. Pizzuti, 2011, PloS one.

Glucocorticoid sensitivity of T-cell lymphoblastic leukemia/lymphoma is associated with glucocorticoid receptor-mediated inhibition of Notch1 expression

I. Screpanti, D. Bellavia, S. Checquolo, 2013, Leukemia.

Differential subcellular localization regulates c-Cbl E3 ligase activity upon Notch3 protein in T-cell leukemia

G. Ferrara, I. Screpanti, D. Bellavia, 2010, Oncogene.

Constitutively active Notch1 induces growth arrest of HPV-positive cervical cancer cells via separate signaling pathways.

I. Screpanti, S. Cialfi, L. Frati, 2005, Experimental cell research.

Corrigendum: The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease

I. Screpanti, S. Cialfi, D. Uccelletti, 2017, Scientific Reports.

Deep Sequencing the microRNA profile in rhabdomyosarcoma reveals down-regulation of miR-378 family members

S. Cialfi, C. Dominici, H. McDowell, 2014, BMC Cancer.

A sketch of known and novel MYCN-associated miRNA networks in neuroblastoma

S. Cialfi, C. Dominici, H. McDowell, 2017, Oncology reports.

The p53 codon 72 Pro/Pro genotype identifies poor-prognosis neuroblastoma patients: correlation with reduced apoptosis and enhanced senescence by the p53-72P isoform.

M. L. Belli, S. Cialfi, C. Dominici, 2012, Neoplasia.

The p 53 Codon 72 Pro / Pro Genotype Identifies Poor-Prognosis Neuroblastoma Patients : Correlation with Reduced Apoptosis and Enhanced Senescence by the p 53-72 P Isoform 1 , 2

M. L. Belli, S. Cialfi, C. Dominici, 2012 .

Novel TONSL variants cause SPONASTRIME dysplasia and associate with spontaneous chromosome breaks, defective cell proliferation and apoptosis.

D. Taruscio, S. Cialfi, S. Castellana, 2020, Human molecular genetics.

Intracellular NADPH Levels Affect the Oligomeric State of the Glucose 6-Phosphate Dehydrogenase

S. Cialfi, A. Tramonti, D. De Biase, 2012, Eukaryotic Cell.

The epigenetic factor BORIS/CTCFL regulates the NOTCH3 gene expression in cancer cells.

I. Screpanti, S. Chiaretti, S. Cialfi, 2014, Biochimica et biophysica acta.

DNA Damage Stress: Cui Prodest?

S. Cialfi, R. Palermo, Azzurra Zonfrilli, 2019, International journal of molecular sciences.

Loss of Notch1-dependent p21Waf1/Cip1 expression influences the Notch1 outcome in tumorigenesis

I. Screpanti, D. Bellavia, S. Checquolo, 2014, Cell cycle.

“In vitro toxicity studies of zinc oxide nano- and microrods on mammalian cells: A comparative analysis”

M. S. Sarto, S. Cialfi, D. Uccelletti, 2016 .

Notch3/Jagged1 Circuitry Reinforces Notch Signaling and Sustains T-ALL

L. Di Marcotullio, I. Screpanti, D. Bellavia, 2014, Neoplasia.

Deep Sequencing the microRNA profile in rhabdomyosarcoma reveals down-regulation of miR-378 family members

S. Cialfi, C. Dominici, H. McDowell, 2014, BMC Cancer.

Loss of ATP2C1 function promotes trafficking and degradation of NOTCH1: Implications for Hailey‐Hailey disease

I. Screpanti, D. Bellavia, S. Checquolo, 2023, Experimental dermatology.