D. Ferguson

发表

Limitations of chromosome classification by multicolor karyotyping.

D. Gisselsson, J. Fletcher, C. Lee, 2001, American journal of human genetics.

Hematopoietic stem cells are acutely sensitive to Acd shelterin gene inactivation.

D. Ferguson, C. Keegan, I. Maillard, 2014, The Journal of clinical investigation.

rearrangements due to a hypomorphic RAG1 mutation Leaky severe combined immunodeficiency and aberrant DNA

F. Alt, D. Schatz, D. Ferguson, 2009 .

The MRE11 GAR motif regulates DNA double-strand break processing and ATR activation

S. Richard, Zhenbao Yu, D. Ferguson, 2011, Cell Research.

Checkpoint failure and chromosomal instability without lymphomagenesis in Mre11(ATLD1/ATLD1) mice.

F. Alt, P. Hunt, D. Ferguson, 2003, Molecular cell.

Reversible mislocalization of a disease-associated MRE11 splice variant product

D. Ferguson, J. Regal, Joshua A Regal, 2018, Scientific Reports.

The Polycomb group protein EZH2 impairs DNA repair in breast epithelial cells.

S. Merajver, S. Varambally, A. Chinnaiyan, 2005, Neoplasia.

Proapoptotic BID is required for myeloid homeostasis and tumor suppression.

F. Alt, S. Korsmeyer, J. Kutok, 2003, Genes & development.

A polyglutamine expansion disease protein sequesters PTIP to attenuate DNA repair and increase genomic instability.

D. Ferguson, G. Dressler, Zhigang Yu, 2012, Human molecular genetics.

Multiple roles for Mre11 at uncapped telomeres

D. Ferguson, Sandy Chang, Xiaolan Guo, 2009, Nature.

Functional Interaction of H2AX, NBS1, and p53 in ATM-Dependent DNA Damage Responses and Tumor Suppression

F. Alt, D. Ferguson, C. Bassing, 2005, Molecular and Cellular Biology.

Leaky SCID and aberrant DNA rearrangements due to a hypomorphic RAG1 mutation

F. Alt, D. Ferguson, Hwei-Ling Cheng, 2008 .

Structure of REC2, a recombinational repair gene of Ustilago maydis, and its function in homologous recombination between plasmid and chromosomal sequences

D. Ferguson, W. Holloman, B. Rubin, 1994, Molecular and cellular biology.

Pten dependence distinguishes hematopoietic stem cells from leukemia-initiating cells

S. Morrison, D. Ferguson, Ömer H. Yilmaz, 2006 .

Tpp1/Acd maintains genomic stability through a complex role in telomere protection

D. Ferguson, G. Hammer, J. Hutz, 2007, Chromosome Research.

Recombinational repair of gaps in DNA is asymmetric in Ustilago maydis and can be explained by a migrating D-loop model.

D. Ferguson, W. Holloman, David O. Ferguson, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Chfr and RNF8 synergistically regulate ATM activation

D. Ferguson, M. Ljungman, Lin-Yu Lu, 2011, Nature Structural &Molecular Biology.

Post-transplant lymphoproliferative disorder with ileal stricture masquerading as Crohn disease

D. Ferguson, D. Arber, L. Lamps, 2022, Human Pathology Reports.

Interaction between Ustilago maydis REC2 and RAD51 genes in DNA repair and mitotic recombination.

D. Ferguson, M. Rendi, M. Rice, 1997, Genetics.

Mre11 Nuclease Activity Has Essential Roles in DNA Repair and Genomic Stability Distinct from ATM Activation

D. Ferguson, Sandy Chang, Gerwin H. Westfield, 2008, Cell.

Rad50 depletion impacts upon ATR-dependent DNA damage responses.

D. Ferguson, H. Zhong, Mark Eckersdorff, 2005, Human molecular genetics.

Recruitment and activation of the ATM kinase in the absence of DNA damage sensors

D. Ferguson, A. Hartlerode, Yipin Wu, 2015, Nature Structural &Molecular Biology.

Mre11 regulates CtIP–dependent double strand break repair by interaction with CDK2

D. Ferguson, Jeffrey M Buis, Trina Stoneham, 2011, Nature Structural &Molecular Biology.

Rad54 and DNA Ligase IV cooperate to maintain mammalian chromatid stability.

F. Alt, D. Ferguson, K. Mills, 2004, Genes & development.

DNA ligase IV deficiency in mice leads to defective neurogenesis and embryonic lethality via the p53 pathway.

F. Alt, R. DePinho, N. Sharpless, 2000, Molecular cell.

Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development

F. Alt, R. DePinho, D. Ferguson, 2000, Nature.

A Deletion at the Mouse Xist Gene Exposes Trans-effects That Alter the Heterochromatin of the Inactive X Chromosome and the Replication Time and DNA Stability of Both X Chromosomes

A. Chess, M. Teitell, D. Ferguson, 2006, Genetics.

DNA double strand break repair and chromosomal translocation: Lessons from animal models

F. Alt, D. Ferguson, David O Ferguson, 2001, Oncogene.

The interplay between nonhomologous end-joining and cell cycle checkpoint factors in development, genomic stability, and tumorigenesis.

F. Alt, R. DePinho, N. Sharpless, 2000, Cold Spring Harbor symposia on quantitative biology.

Mammalian Rif1 contributes to replication stress survival and homology-directed repair

D. Ferguson, S. B. Buonomo, T. de Lange, 2009, The Journal of cell biology.

Oncogenic Myc translocations are independent of chromosomal location and orientation of the immunoglobulin heavy chain locus

D. Ferguson, W. Dubois, A. Kovalchuk, 2012, Proceedings of the National Academy of Sciences.

Mouse model of colonic adenoma-carcinoma progression based on somatic Apc inactivation.

Kathleen R. Cho, E. Fearon, D. Ferguson, 2007, Cancer research.

Pten dependence distinguishes haematopoietic stem cells from leukaemia-initiating cells

S. Morrison, D. Ferguson, Wei Guo, 2006, Nature.

Nonhomologous end-joining proteins are required for V(D)J recombination, normal growth, and neurogenesis.

F. Alt, D. Ferguson, J. Chaudhuri, 1999, Cold Spring Harbor symposia on quantitative biology.

The role of DNA breaks in genomic instability and tumorigenesis

F. Alt, D. Ferguson, K. Mills, 2003, Immunological reviews.

Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AX

F. Alt, Heikyung Suh, D. Livingston, 2002, Proceedings of the National Academy of Sciences of the United States of America.

MRE11 Promotes Tumorigenesis by Facilitating Resistance to Oncogene-Induced Replication Stress.

D. Ferguson, J. Sekiguchi, M. Dinkelmann, 2017, Cancer research.

Dysregulated TCL1 requires the germinal center and genome instability for mature B-cell transformation.

F. Alt, M. Teitell, D. Ferguson, 2006, Blood.

Histone H2AX A Dosage-Dependent Suppressor of Oncogenic Translocations and Tumors

F. Alt, Heikyung Suh, D. Ferguson, 2003, Cell.

Impaired nonhomologous end-joining provokes soft tissue sarcomas harboring chromosomal translocations, amplifications, and deletions.

L. Chin, F. Alt, R. DePinho, 2001, Molecular cell.

Disease-associated MRE11 mutants impact ATM/ATR DNA damage signaling by distinct mechanisms.

D. Ferguson, J. Regal, Joshua A Regal, 2013, Human molecular genetics.

Leaky severe combined immunodeficiency and aberrant DNA rearrangements due to a hypomorphic RAG1 mutation.

F. Alt, D. Schatz, D. Ferguson, 2009, Blood.

Murine models of IDH-wild-type glioblastoma exhibit spatial segregation of tumor initiation and manifestation during evolution

Jun Z. Li, B. Ross, D. Ferguson, 2020, Nature Communications.

Deficiencies of GM-CSF and Interferon γ Link Inflammation and Cancer

F. Alt, M. Mihm, G. Dranoff, 2003, The Journal of experimental medicine.

Genetic interactions between ATM and the nonhomologous end-joining factors in genomic stability and development

F. Alt, D. Ferguson, A. Nussenzweig, 2001, Proceedings of the National Academy of Sciences of the United States of America.

The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations.

F. Alt, R. DePinho, D. Ferguson, 2000, Proceedings of the National Academy of Sciences of the United States of America.

ATRX loss promotes tumor growth and impairs nonhomologous end joining DNA repair in glioma

D. Ferguson, Chris Jones, V. Gorbunova, 2016, Science Translational Medicine.

Artemis and p53 cooperate to suppress oncogenic N-myc amplification in progenitor B cells.

F. Alt, D. Ferguson, J. Manis, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Unrepaired DNA Breaks in p53-Deficient Cells Lead to Oncogenic Gene Amplification Subsequent to Translocations

F. Alt, D. Ferguson, C. Morton, 2002, Cell.

Defective embryonic neurogenesis in Ku-deficient but not DNA-dependent protein kinase catalytic subunit-deficient mice.

F. Alt, D. Ferguson, J. Chun, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Multiple functions of MRN in end-joining pathways during isotype class switching

D. Ferguson, Yipin Wu, J. Sekiguchi, 2009, Nature Structural &Molecular Biology.

Genetic p53 deficiency partially rescues the adrenocortical dysplasia phenotype at the expense of increased tumorigenesis.

R. Kuick, D. Ferguson, G. Hammer, 2009, Cancer cell.

DNA Double-Strand Break Repair: A Relentless Hunt Uncovers New Prey

D. Ferguson, J. Sekiguchi, 2006, Cell.

Defective DNA Repair and Increased Genomic Instability in Artemis-deficient Murine Cells

David Lombard, Frederick W. Alt, David G. Schatz, 2003, The Journal of experimental medicine.

MOF and H4 K16 Acetylation Play Important Roles in DNA Damage Repair by Modulating Recruitment of DNA Damage Repair Protein Mdc1

P. Pan, J. Min, Y. Dou, 2010, Molecular and Cellular Biology.

Mutation avoidance and DNA repair proficiency in Ustilago maydis are differentially lost with progressive truncation of the REC1 gene product

D. Ferguson, K. Onel, M. Thelen, 1995, Molecular and cellular biology.

The Essential DNA Damage Response Complex MRN Is Dispensable for the Survival and Function of Purkinje Neurons

D. Ferguson, Zhao-Qi Wang, C. Geis, 2022, Frontiers in Aging Neuroscience.