P. Simpson‐Haidaris

发表

Integrin α v β 3-Mediated Endocytosis of Immobilized Fibrinogen by A549 Lung Alveolar Epithelial Cells

T. Odrljin, C. Haidaris, N. B. Lerner, 2001 .

Intracellular Localization of a Trypanosoma cruzi kDNA Minicircle Transcript Using RNA: RNA In Situ Hybridization

C. Haidaris, P. Simpson‐Haidaris, R. Thertulien, 1994, The Journal of eukaryotic microbiology.

The Role of Fibrin(ogen) in Transendothelial Cell Migration During Breast Cancer Metastasis

A. Sahni, P. Simpson‐Haidaris, B. Rybarczyk, 2011 .

Functional analysis of interleukin 6 response elements (IL-6REs) on the human gamma-fibrinogen promoter: binding of hepatic Stat3 correlates negatively with transactivation potential of type II IL-6REs.

P. Simpson‐Haidaris, H. Duan, P. Simpson-Haidaris, 2003, The Journal of biological chemistry.

Integrin alphavbeta3-mediated endocytosis of immobilized fibrinogen by A549 lung alveolar epithelial cells.

T. Odrljin, C. Haidaris, N. B. Lerner, 2001, American journal of respiratory cell and molecular biology.

Fibrinogen binding potentiates FGF‐2 but not VEGF induced expression of u‐PA, u‐PAR, and PAI‐1 in endothelial cells

A. Sahni, C. Francis, P. Simpson‐Haidaris, 2004, Journal of thrombosis and haemostasis : JTH.

Modification of Streptococcus mutans Cnm by PgfS Contributes to Adhesion, Endothelial Cell Invasion, and Virulence

P. Simpson‐Haidaris, J. Abranches, J. H. Miller, 2014, Journal of bacteriology.

Author's reply to Weijers et al. “Fibrin β15–42 domain is cryptic in intact fibrinogen: comment on the study by A. Sahni et al.”

A. Sahni, P. Simpson‐Haidaris, 2010, International Journal of Cancer.

Heterologous expression of Streptococcus mutans Cnm in Lactococcus lactis promotes intracellular invasion, adhesion to human cardiac tissues and virulence

P. Simpson‐Haidaris, J. Abranches, Peter A. Knight, 2017, Virulence.

Parasitoid wasp venom elevates sorbitol and alters expression of metabolic genes in human kidney cells

J. Werren, P. Simpson‐Haidaris, A. Siebert, 2018, bioRxiv.

Plasma fibrinogen γ′ chain content in the thrombotic microangiopathy syndrome

P. Simpson‐Haidaris, R. Bertina, T. Raife, 2007 .

Peroxisome Proliferator-Activated Receptor γ Ligands Enhance Human B Cell Antibody Production and Differentiation1

P. Simpson‐Haidaris, C. Baglole, T. Murant, 2009, The Journal of Immunology.

Antibody Production and Differentiation ) Ligands Enhance Human B Cell g (PPAR g Peroxisome Proliferator-Activated Receptor

P. Simpson‐Haidaris, C. Baglole, T. Murant, 2009 .

Post-transcriptional regulation of endothelial cell plasminogen activator inhibitor-1 expression during R. rickettsii infection.

P. Simpson‐Haidaris, V. Marder, D. Silverman, 2000, Microbial pathogenesis.

Integrin a v b 3-Mediated Endocytosis of Immobilized Fibrinogen by A 549 Lung Alveolar Epithelial Cells

T. Odrljin, C. Haidaris, N. B. Lerner, 2000 .

Mechanisms of cytopenia in human immunodeficiency virus infection.

P. Simpson‐Haidaris, J. Liesveld, C. Abboud, 1994, Blood reviews.

Thrombin cleavage enhances exposure of a heparin binding domain in the N-terminus of the fibrin beta chain.

T. Odrljin, P. Simpson‐Haidaris, S. Lawrence, 1996, Blood.

Fibrinogen synthesized by cancer cells augments the proliferative effect of fibroblast growth factor‐2 (FGF‐2)

A. Sahni, C. Francis, P. Simpson‐Haidaris, 2007, Journal of thrombosis and haemostasis : JTH.

The Collagen-Binding Protein Cnm Is Required for Streptococcus mutans Adherence to and Intracellular Invasion of Human Coronary Artery Endothelial Cells

P. Simpson‐Haidaris, R. Burne, J. Abranches, 2011, Infection and Immunity.

Cloning and characterization of a conserved region of human and rhesus macaque Pneumocystis carinii gpA.

C. Haidaris, P. Simpson‐Haidaris, T. Wright, 1995, Gene.

Conserved sequence homology of cysteine-rich regions in genes encoding glycoprotein A in Pneumocystis carinii derived from different host species

C. Haidaris, P. Simpson‐Haidaris, T. Wright, 1994, Infection and immunity.

Molecular characterization of KEX1, a kexin-like protease in mouse Pneumocystis carinii.

C. Haidaris, P. Simpson‐Haidaris, G. Weinberg, 2000, Gene.

The Carboxyl Terminus of Pneumocystis cariniiGlycoprotein A Encodes a Functional Glycosylphosphatidylinositol Signal Sequence*

Constantine G. Haidaris, C. Haidaris, P. Simpson‐Haidaris, 1998, The Journal of Biological Chemistry.

Molecular characterization of mouse Pneumocystis carinii surface glycoprotein A.

C. Haidaris, P. Simpson‐Haidaris, F. Gigliotti, 1998, DNA research : an international journal for rapid publication of reports on genes and genomes.

Induction of Fibrinogen Expression in the Lung Epithelium during Pneumocystis carinii Pneumonia

P. Simpson‐Haidaris, T. Wright, F. Gigliotti, 1998, Infection and Immunity.

Epitope Mapping of a Protective Monoclonal Antibody against Pneumocystis carinii with Shared Reactivity to Streptococcus pneumoniae Surface Antigen PspA

C. Haidaris, P. Simpson‐Haidaris, J. Wells, 2004, Infection and Immunity.

Antigenic properties of recombinant glycosylated and nonglycosylatedPneumocystis carinii glycoprotein A polypeptides expressed in baculovirus-infected insect cells

C. Haidaris, P. Simpson‐Haidaris, F. Gigliotti, 1998, Molecular biotechnology.

The incorporation of fibrinogen into extracellular matrix is dependent on active assembly of a fibronectin matrix.

T. Odrljin, P. Simpson‐Haidaris, D. Hocking, 2002, Journal of cell science.

Anticancer Role of PPARγ Agonists in Hematological Malignancies Found in the Vasculature, Marrow, and Eyes

S. Feldon, P. Simpson‐Haidaris, R. Phipps, 2010, PPAR research.

Innate Immune Response and Inflammation: Roles in Pathogenesis and Protection (Rickettsiaceae)

P. Simpson‐Haidaris, S. Sahni, E. Rydkina, 2012 .

Matrix-fibrinogen enhances wound closure by increasing both cell proliferation and migration.

P. Simpson‐Haidaris, B. Rybarczyk, S. O. Lawrence, 2003, Blood.

Invasion of human coronary artery endothelial cells by Streptococcus mutans OMZ175.

P. Simpson‐Haidaris, R. Burne, J. Abranches, 2009, Oral microbiology and immunology.

Cnm is a major virulence factor of invasive Streptococcus mutans and part of a conserved three-gene locus.

J. Miller, P. Simpson‐Haidaris, J. Abranches, 2014, Molecular oral microbiology.