K. Dry

发表

A gene (RPGR) with homology to the RCC1 guanine nucleotide exchange factor is mutated in X–linked retinitis pigmentosa (RP3)

A. Ciccodicola, M. D'urso, T. Meitinger, 1996, Nature Genetics.

Genetic analysis of a kindred with X-linked mental handicap and retinitis pigmentosa.

G. Spowart, K. Dry, A. Carothers, 1994, American journal of human genetics.

Catecholamine release from bovine adrenal chromaffin cells during anoxia or metabolic inhibition.

K. Dry, J. Phillips, A. M. Dart, 1991, Circulation research.

Analysis of three deletion breakpoints in Xp21.1 and the further localization of RP3.

K. Dry, A. Wright, S. Boyle, 1996, Genomics.

A recombination outside the BB deletion refines the location of the X linked retinitis pigmentosa locus RP3.

P. Sieving, J. Ott, M. Devoto, 1996, American journal of human genetics.

Linkage analysis‐in a family with complete type congenital stationary night blindness with and without myopia

K. Dry, A. Wright, M. Aldred, 1993, Clinical genetics.

Linkage analysis in X-linked congenital stationary night blindness.

K. Dry, M. Jay, P. Teague, 1992, Genomics.

Identification of a novel gene, ETX1, from Xp21.1, a candidate gene for X-linked retinitis pigmentosa (RP3)

K. Dry, A. Lennon, M. Aldred, 1995 .

ATM-dependent phosphorylation of heterogeneous nuclear ribonucleoprotein K promotes p53 transcriptional activation in response to DNA damage

K. Dry, S. Jackson, A. Moumen, 2013, Cell cycle.

Genetic Analysis ofaKindred withX-linked MentalHandicap and Retinitis Pigmentosa

A. Fielder, G. Spowart, K. Dry, 1994 .

Identification of a 5′ splice site mutation in the RPGR gene in a family with X‐linked retinitis pigmentosa (RP3)

K. Dry, A. Lennon, A. Wright, 1999, Human mutation.

Catecholamine release from isolated chromaffin cells under conditions of anoxia or metabolic inhibition

K. Dry, 1990 .