J. A. Black

发表

Differential expression of sodium channel genes in retinal ganglion cells.

S. Dib-Hajj, S. Waxman, J. Fjell, 1997, Brain research. Molecular brain research.

Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury

Stephen G Waxman, S. Waxman, J. A. Black, 2003, The Journal of comparative neurology.

Molecular Reconstruction of Nodes of Ranvier after Remyelination by Transplanted Olfactory Ensheathing Cells in the Demyelinated Spinal Cord

S. Waxman, J. A. Black, J. Kocsis, 2006, The Journal of Neuroscience.

Protection of the axonal cytoskeleton in anoxic optic nerve by decreased extracellular calcium

S. Waxman, B. Ransom, J. A. Black, 1993, Brain Research.

Freeze–fracture studies on unmyelinated axolemma of rat cervical sympathetic trunk : correlation with saxitoxin binding

S. Waxman, J. A. Black, J. Black, 1988, Proceedings of the Royal Society of London. Series B. Biological Sciences.

Macromolecular structure of the Schwann cell membrane Perinodal microvilli

S. Waxman, J. A. Black, J. Black, 1987, Journal of the Neurological Sciences.

Effects of delayed myelination by oligodendrocytes and Schwann cells on the macromolecular structure of axonal membrane in rat spinal cord

S. Waxman, J. A. Black, T. Sims, 1986, Journal of neurocytology.

Axo-glial relations in the retina-optic nerve junction of the adult rat: freeze-fracture observations on axon membrane structure

S. Waxman, J. A. Black, C. Hildebrand, 1985, Journal of neurocytology.

Plasticity of sodium channel expression in DRG neurons in the chronic constriction injury model of neuropathic pain

R. LaMotte, S. Dib-Hajj, S. Waxman, 1999, PAIN®.

Diverse functions and dynamic expression of neuronal sodium channels.

S. Dib-Hajj, S. Waxman, J. A. Black, 2002, Novartis Foundation symposium.

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

Masaki Tanaka, S. Waxman, J. A. Black, 2004, Pain.

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

S. Waxman, J. Newcombe, J. A. Black, 2010, Brain : a journal of neurology.

Rat optic nerve: Disruption of gliogenesis with 5-azacytidine during early postnatal development

S. Waxman, B. Ransom, J. A. Black, 1985, Brain Research.

Carbonic anhydrase activity develops postnatally in the rat optic nerve.

S. Waxman, B. Ransom, J. A. Black, 1987, Brain research.

GDNF and NGF reverse changes in repriming of TTX-sensitive Na(+) currents following axotomy of dorsal root ganglion neurons.

S. Dib-Hajj, S. Waxman, J. A. Black, 2002, Journal of neurophysiology.

Sodium channels and pain.

S. Dib-Hajj, S. Waxman, J. A. Black, 1999, Proceedings of the National Academy of Sciences of the United States of America.

Neuroma Removal for Neuropathic Pain: Efficacy and Predictive Value of Lidocaine Infusion

S. Waxman, J. A. Black, T. Jensen, 2010, The Clinical journal of pain.

Axonal Protection with Sodium Channel Blocking Agents in Models of Multiple Sclerosis

S. Waxman, J. A. Black, Kenneth J. Smith, 2013 .

A Novel Persistent Tetrodotoxin-Resistant Sodium Current In SNS-Null And Wild-Type Small Primary Sensory Neurons

S G Waxman, S. Dib-Hajj, S. Waxman, 1999, The Journal of Neuroscience.

Correlation of Nav1.8 and Nav1.9 sodium channel expression with neuropathic pain in human subjects with lingual nerve neuromas

S. Waxman, J. A. Black, F. Boissonade, 2013, Molecular pain.

Voltage-gated sodium channels: therapeutic targets for pain.

S. Dib-Hajj, S. Waxman, J. A. Black, 2009, Pain medicine.

Two tetrodotoxin‐resistant sodium channels in human dorsal root ganglion neurons

S. Dib-Hajj, S. Waxman, J. A. Black, 1999, FEBS letters.

Dynamics of sodium channel Nav1.5 expression in astrocytes in mouse models of multiple sclerosis

S. Waxman, J. A. Black, Shujun Liu, 2014, Neuroreport.

Upregulation of a silent sodium channel after peripheral, but not central, nerve injury in DRG neurons.

Y. H. Chen, S. Waxman, J. A. Black, 1999, Journal of neurophysiology.

Localization of the tetrodotoxin‐resistant sodium channel NaN in nociceptors

S. Dib-Hajj, S. Waxman, J. Fjell, 2000, Neuroreport.

Schwann cells modulate sodium channel expression in spinal sensory neurons in vitro

S. Dib-Hajj, S. Waxman, J. A. Black, 1997, Glia.

Down-regulation of transcripts for Na channel alpha-SNS in spinal sensory neurons following axotomy.

S. Dib-Hajj, S. Waxman, J. A. Black, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Differential role of GDNF and NGF in the maintenance of two TTX-resistant sodium channels in adult DRG neurons.

S. Dib-Hajj, S. Waxman, J. Fjell, 1999, Brain research. Molecular brain research.

Membrane specialization and axo-glial association in the rat retinal nerve fibre layer: freeze-fracture observations

S. Waxman, J. A. Black, C. Hildebrand, 1984, Journal of neurocytology.

Sodium channel Nav1.7 in vascular myocytes, endothelium, and innervating axons in human skin

S. Waxman, F. Rice, J. A. Black, 2015, Molecular pain.

Macromolecular Structure of Axonal Membrane in the Optic Nerve of the Jimpy Mouse

S. Waxman, R. Fields, J. A. Black, 1988, Journal of neuropathology and experimental neurology.

Phenytoin protects central axons in experimental autoimmune encephalomyelitis

S. Waxman, J. A. Black, J. Black, 2008, Journal of the Neurological Sciences.

Neuroprotection of axons with phenytoin in experimental allergic encephalomyelitis

A. Lo, S. Waxman, J. A. Black, 2002, Neuroreport.

Exacerbation of experimental autoimmune encephalomyelitis after withdrawal of phenytoin and carbamazepine

S. Waxman, J. A. Black, Shujun Liu, 2007, Annals of neurology.

Dynamic aspects of sodium channel expression in astrocytes.

S. Waxman, J. A. Black, H. Sontheimer, 1993, Advances in neurology.

Sodium channels in the cytoplasm of Schwann cells.

S. Waxman, J. A. Black, K. Angelides, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Sodium channels in the cytoplasm of Schwann cells.

J. M. Ritchie, S. Waxman, J. A. Black, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Freeze‐fracture heterogeneity of the axolemma of premyelinated fibers in the CNS

S. Waxman, J. A. Black, R. Foster, 1982, Neurology.

Immuno-Localization of Sodium Channels in Axon Membrane and Astrocytes and Schwann Cells in vivo and in vitro

A. Cornell-Bell, S. Waxman, J. A. Black, 1990 .

Sodium channels in astrocytes of rat optic nerve in situ: Immuno‐electron microscopic studies

S. Waxman, J. A. Black, K. Angelides, 1999, Glia.

Filipin-cholesterol binding in CNS axons prior to myelination: evidence for microheterogeneity in premyelinated axolemma

S. Waxman, R. Fields, J. A. Black, 1987, Brain Research.

Differences in intramembranous particle distribution in the paranodal axolemma are not associated with functional differences of dorsal and ventral roots

S. Waxman, R. Fields, J. A. Black, 1986, Neuroscience Letters.

In vivo NGF deprivation reduces SNS expression and TTX-R sodium currents in IB4-negative DRG neurons.

S. Waxman, J. Fjell, K. Fried, 1999, Journal of neurophysiology.

Distribution of sodium channels in chronically demyelinated spinal cord axons: immunoultrastructural localization and electrophysiological observations. Brain Research 544 (1991) 59-70.

S. Waxman, J. A. Black, J. Kocsis, 1997, Brain research.

Nav1.5 sodium channels in macrophages in multiple sclerosis lesions

S. Waxman, J. Newcombe, J. A. Black, 2013, Multiple sclerosis.

A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons.

A. M. Rush, S. Dib-Hajj, S. Waxman, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Ion channels in spinal cord astrocytes in vitro. I. Transient expression of high levels of Na+ and K+ channels.

S. Waxman, J. A. Black, H. Sontheimer, 1992, Journal of neurophysiology.

Na(+)-current expression in rat hippocampal astrocytes in vitro: alterations during development.

A. Cornell-Bell, S. Waxman, J. A. Black, 1991, Journal of neurophysiology.

Spinal cord astrocytes in vitro: Phenotypic diversity and sodium channel immunoreactivity

S. Waxman, J. A. Black, H. Sontheimer, 1993, Glia.

Freeze-fracture ultrastructure of rat C.N.S. and P.N.S. nonmyelinated axolemma

S. Waxman, J. A. Black, R. Foster, 1981, Journal of neurocytology.

Membrane structure of vesiculotubular complexes in developing axons in rat optic nerve: freeze—fracture evidence for sequential membrane assembly

S. Waxman, J. A. Black, J. Black, 1985, Proceedings of the Royal Society of London. Series B. Biological Sciences.

Membrane ultrastructure of developing axons in glial cell deficient rat spinal cord

S. Waxman, J. A. Black, T. Sims, 1985, Journal of neurocytology.

Freeze-fracture ultrastructure of developing and adult non-myelinated ganglion cell axolemma in the retinal nerve fibre layer

S. Waxman, J. A. Black, R. Foster, 1983, Journal of neurocytology.

Sodium channel expression in hypothalamic osmosensitive neurons in experimental diabetes

S. Waxman, J. A. Black, J. P. Klein, 2002, Neuroreport.

NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy.

S. Dib-Hajj, S. Waxman, J. A. Black, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Abnormal expression of SNS/PN3 sodium channel in cerebellar Purkinje cells following loss of myelin in the taiep rat.

S. Dib-Hajj, S. Waxman, I. Duncan, 1999, Neuroreport.

Differential up-regulation of sodium channel α- and β1-subunit mRNAs in cultured embryonic DRG neurons following exposure to NGF

S. Waxman, K. Zur, J. A. Black, 1995 .

Type II brain sodium channel expression in non-neuronal cells: embryonic rat osteoblasts.

S. Waxman, W. Catterall, J. A. Black, 1995, Brain research. Molecular brain research.

Isoform‐specific expression of sodium channels in astrocytes in vitro: Immunocytochemical observations

S. Waxman, W. Catterall, B. Ransom, 1995, Glia.

Type II sodium channels in spinal cord astrocytes in situ: Immunocytochemical observations

S. Waxman, W. Catterall, B. Ransom, 1994, Glia.

The voltage-dependent sodium channel in mammalian CNS and PNS: antibody characterization and immunocytochemical localization

S. Waxman, J. A. Black, K. Angelides, 1990, Brain Research.

Na+ channel beta 1 subunit mRNA: differential expression in rat spinal sensory neurons.

S. Waxman, J. A. Black, Y. Oh, 1995, Brain research. Molecular brain research.

Expression of sodium channel α- and β-subunits in the nervous system of themyelin-deficient rat

S. Waxman, J. A. Black, P. Felts, 1995, Journal of neurocytology.

Expression of skeletal muscle‐type voltage‐gated Na+ channel in rat and human prostate cancer cell lines

S. Dib-Hajj, S. Waxman, J. A. Black, 1998, FEBS letters.

Sodium channel expression in optic nerve astrocytes chronically deprived of axonal contact

S. Waxman, B. Ransom, J. A. Black, 1992, Glia.

Two types of Na +‐currents in cultured rat optic nerve astrocytes: Changes with time in culture and with age of culture derivation

S. Waxman, B. Ransom, J. A. Black, 1991, Journal of neuroscience research.

Perinodal astrocytic processes at nodes of ranvier in developing normal and glial cell deficient rat spinal cord

S. Waxman, J. A. Black, T. Sims, 1985, Brain Research.

Molecular structure of the axolemma of developing axons following altered gliogenesis in rat optic nerve.

S. Waxman, J. A. Black, J. Black, 1986, Developmental biology.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.

Stephen G Waxman, S. Waxman, J. A. Black, 2006, Brain : a journal of neurology.

Tapered withdrawal of phenytoin removes protective effect in EAE without inflammatory rebound and mortality

S. Waxman, J. A. Black, S. Liu, 2014, Journal of the Neurological Sciences.

Specialization of astrocytic membrane at glia limitans in rat optic nerve: Freeze-fracture observations

S. Waxman, J. A. Black, J. Black, 1985, Neuroscience Letters.

Specificity of cell-cell coupling in rat optic nerve astrocytes in vitro.

S. Waxman, J. A. Black, H. Sontheimer, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Sodium channel expression detected with antibody 7493 in A2B5+ and A2B5− astrocytes from rat optic nerve in vitro

S. Waxman, J. A. Black, K. Angelides, 1990, Glia.

The expression of sodium channels in astrocytes in situ and in vitro.

S. Waxman, J. A. Black, H. Sontheimer, 1992, Progress in brain research.

Macromolecular Structure of Axonal Membrane during Acute Experimental Allergic Encephalomyelitis in Rat and Guinea Pig Spinal Cord

S. Waxman, J. A. Black, M. Smith, 1987, Journal of neuropathology and experimental neurology.

Membrane‐Associated Sodium Channels and Cytoplasmic Precursors in Glial Cells

S. Waxman, J. A. Black, K. Angelides, 1991, Annals of the New York Academy of Sciences.

Ca2+ toxicity due to reverse Na+/Ca2+ exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav1.7 mutation.

S. Waxman, J. A. Black, G. Lauria, 2015, Journal of neurophysiology.

Ca 2 toxicity due to reverse Na / Ca 2 exchange contributes to degeneration of neurites of DRG neurons induced by a neuropathy-associated Nav 1 . 7 mutation

S. Waxman, J. A. Black, G. Lauria, 2015 .

Retinal involvement in multiple sclerosis

S. Waxman, J. A. Black, J. Black, 2007, Neurology.

Pharmacological modification of axon membrane molecules and cell transplantation as approaches to the restoration of conduction in demyelinated axons.

S. Waxman, J. A. Black, J. Kocsis, 1993, Research publications - Association for Research in Nervous and Mental Disease.

Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.

Stephen G Waxman, A. Lo, S. Waxman, 2004, Brain : a journal of neurology.

Sodium-calcium exchanger and multiple sodium channel isoforms in intra-epidermal nerve terminals

S. Waxman, J. A. Black, A. Gasser, 2010, Molecular pain.

Endogenous NMDA-receptor activation regulates glutamate release in cultured spinal neurons.

S. Waxman, J. A. Black, A. Robert, 1998, Journal of neurophysiology.

Contactin Associates with Sodium Channel Nav1.3 in Native Tissues and Increases Channel Density at the Cell Surface

A. M. Rush, S. Dib-Hajj, S. Waxman, 2004, The Journal of Neuroscience.

Direct Interaction with Contactin Targets Voltage-gated Sodium Channel Nav1.9/NaN to the Cell Membrane*

Z. Lian, S. Dib-Hajj, S. Waxman, 2001, The Journal of Biological Chemistry.

Sensory neuron-specific sodium channel SNS is abnormally expressed in the brains of mice with experimental allergic encephalomyelitis and humans with multiple sclerosis.

J Newcombe, S G Waxman, S. Dib-Hajj, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Primary motor neurons fail to up‐regulate voltage‐gated sodium channel Nav1.3/brain type III following axotomy resulting from spinal cord injury

S. Waxman, J. A. Black, B. C. Hains, 2002, Journal of neuroscience research.

Sodium channels in normal and pathological pain.

S. Dib-Hajj, S. Waxman, J. A. Black, 2010, Annual review of neuroscience.

Sodium channels, excitability of primary sensory neurons, and the molecular basis of pain

S. Dib-Hajj, S. Waxman, J. Fjell, 1999, Muscle & nerve.

NaV1.7: Stress-induced changes in immunoreactivity within magnocellular neurosecretory neurons of the supraoptic nucleus

S. Waxman, J. A. Black, J. Hoeijmakers, 2013, Molecular pain.

From genes to pain: Nav1.7 and human pain disorders

Sulayman D. Dib-Hajj, Stephen G. Waxman, S. Dib-Hajj, 2007, Trends in Neurosciences.