N. Standen

发表

Potassium activation in Helix aspersa neurones under voltage clamp: a component mediated by calcium influx.

R. Meech, N. Standen, 1975, The Journal of physiology.

Calcium channels, potassium channels, and voltage dependence of arterial smooth muscle tone.

J B Patlak, J. Patlak, N. Standen, 1990, The American journal of physiology.

Modulation of hERG potassium currents in HEK‐293 cells by protein kinase C. Evidence for direct phosphorylation of pore forming subunits

N. Standen, I. Torrecilla, J. Mitcheson, 2007, The Journal of physiology.

The G. L. Brown Lecture. Potassium channels, metabolism and muscle

N. Standen, N. Standen, 1992, Experimental physiology.

The Role of the Membrane Potential of Endothelial and Smooth Muscle Cells in the Regulation of Coronary Blood Flow

N. Standen, M. Nelson, J. Daut, 1994, Journal of cardiovascular electrophysiology.

Regulation of single calcium channels in cerebral arteries by voltage, serotonin, and dihydropyridines.

N. Standen, M. Nelson, J. Worley, 1991, The American journal of physiology.

Principal role of adenylyl cyclase 6 in K+ channel regulation and vasodilator signalling in vascular smooth muscle cells

N. Standen, N. Davies, M. Perry, 2011, Cardiovascular research.

Calcium currents elicited by voltage steps and steady voltages in myocytes isolated from the rat basilar artery.

N. Standen, P. Langton, 1993, The Journal of physiology.

Comparative Pharmacology of Guinea Pig Cardiac Myocyte and Cloned hERG (IKr) Channel

Chris Pollard, N. Standen, J. Mitcheson, 2004, Journal of cardiovascular electrophysiology.

Kir6.2 limits Ca(2+) overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress.

N. Standen, R. D. Rainbow, D. Lodwick, 2013, American journal of physiology. Heart and circulatory physiology.

Phenylephrine preconditioning involves modulation of cardiac sarcolemmal K(ATP) current by PKC delta, AMPK and p38 MAPK.

N. Standen, M. Dickens, G. Rodrigo, 2011, Journal of molecular and cellular cardiology.

Mitochondrial dysfunction as the cause of the failure to precondition the diabetic human myocardium.

N. Standen, M. Galiñanes, B. Matata, 2006, Cardiovascular research.

Diazoxide causes early activation of cardiac sarcolemmal KATP channels during metabolic inhibition by an indirect mechanism.

N. Standen, N. Davies, G. Rodrigo, 2004, Cardiovascular research.

Protection from the effects of metabolic inhibition and reperfusion in contracting isolated ventricular myocytes via protein kinase C activation.

N. Standen, D. Hudman, 2004, Journal of molecular and cellular cardiology.

Dinitrophenol pretreatment of rat ventricular myocytes protects against damage by metabolic inhibition and reperfusion.

N. Standen, C. Lawrence, G. Rodrigo, 2002, Journal of molecular and cellular cardiology.

Evidence for mitochondrial K ATP channels as effectors of human myocardial preconditioning.

N. Standen, M. Galiñanes, S. Ghosh, 2000, Cardiovascular research.

Block of calcium-activated potassium channels in mammalian arterial myocytes by tetraethylammonium ions.

N. Standen, M. Nelson, P. Langton, 1991, The American journal of physiology.

KATP channels in vascular smooth muscle.

N. Standen, J. Quayle, 1994, Cardiovascular research.

K+ channel modulation in arterial smooth muscle.

N. Standen, J. Quayle, 1998, Acta physiologica Scandinavica.

The sulfonylurea glimepiride does not abolish myocardial protection afforded by either ischemic preconditioning or diazoxide

N. Standen, M. Mocanu, D. Yellon, 2000 .

Effect of metabolic inhibition on glimepiride block of native and cloned cardiac sarcolemmal KATP channels

N. Standen, N. Davies, C. Lawrence, 2002, British journal of pharmacology.

Failure to precondition pathological human myocardium.

N. Standen, M. Galiñanes, S. Ghosh, 2001, Journal of the American College of Cardiology.

The effect of gliclazide and glibenclamide on preconditioning of the human myocardium.

N. Standen, M. Galiñanes, M. Loubani, 2005, European journal of pharmacology.

Dual role of nNOS in ischemic injury and preconditioning

N. Standen, M. Galiñanes, A. Barua, 2010, BMC Physiology.

Evidence against the association of the sulphonylurea receptor with endogenous Kir family members other than KATP in coronary vascular smooth muscle

N. Standen, J. Quayle, G. Wellman, 1996, Pflügers Archiv.

Endothelin-I and angiotensin II inhibit arterial voltage-gated K+ channels through different protein kinase C isoenzymes.

N. Standen, N. Davies, R. D. Rainbow, 2009, Cardiovascular research.

Visualizing the temporal effects of vasoconstrictors on PKC translocation and Ca2+ signaling in single resistance arterial smooth muscle cells.

N. Standen, N. Davies, R. Challiss, 2008, American journal of physiology. Cell physiology.

Glucose reduces endothelin inhibition of voltage‐gated potassium channels in rat arterial smooth muscle cells

N. Standen, R. Rainbow, M. E. Hardy, 2006, The Journal of physiology.

Potassium channel activation and relaxation by nicorandil in rat small mesenteric arteries

N. Standen, C. Davie, M. Kubo, 1998, British journal of pharmacology.

Activation of ATP‐dependent K+ channels by hypoxia in smooth muscle cells isolated from the pig coronary artery.

N. Standen, C. Dart, 1995, The Journal of physiology.

Ryanodine receptors regulate arterial diameter and wall [Ca2+] in cerebral arteries of rat via Ca2+‐dependent K+ channels

N. Standen, M. Nelson, H. Knot, 1998, The Journal of physiology.

Caveolae Localize Protein Kinase A Signaling to Arterial ATP-Sensitive Potassium Channels

N. Standen, L. J. Sampson, C. Dart, 2004, Circulation research.

Angiotensin II-activated protein kinase C targets caveolae to inhibit aortic ATP-sensitive potassium channels.

N. Standen, L. J. Sampson, C. Dart, 2007, Cardiovascular research.

Role of potassium channels in the vascular response to endogenous and pharmacological vasodilators.

N. Standen, M. Nelson, J. Brayden, 1991, Blood vessels.

External cadmium and internal calcium block of single calcium channels in smooth muscle cells from rabbit mesenteric artery.

N. Standen, M. Nelson, J. Worley, 1989, Biophysical journal.

Enhancement of the vasorelaxant potency of nicorandil by metabolic inhibition and adenosine in the pig coronary artery.

N. Standen, C. Davie, 1998, Cardiovascular research.

Adenosine Activates Potassium Channels in Porcine Coronary Arterial Myocytes

N. Standen, C. Dart, 1994 .

Properties of the ATP-sensitive K+ current activated by levcromakalim in isolated pulmonary arterial myocytes

N. Standen, P. Langton, L. Clapp, 1994, The Journal of Membrane Biology.

Membrane Potential and Calcium Channels in Arterial Smooth Muscle Cells

N. Standen, M. Nelson, J. Brayden, 1989 .

Role of mitochondrial re-energization and Ca2+ influx in reperfusion injury of metabolically inhibited cardiac myocytes.

N. Standen, G. Rodrigo, 2005, Cardiovascular research.

The origin of calcium overload in rat cardiac myocytes following metabolic inhibition with 2,4-dinitrophenol.

N. Standen, C. Lawrence, R. D. Rainbow, 2002, Journal of molecular and cellular cardiology.

G protein-coupled receptor kinase 2 and arrestin2 regulate arterial smooth muscle P2Y-purinoceptor signalling

N. Standen, G. E. Morris, R. Challiss, 2010, Cardiovascular research.

Endothelin signalling in arterial smooth muscle is tightly regulated by G protein-coupled receptor kinase 2

N. Standen, G. E. Morris, R. Challiss, 2009, Cardiovascular research.

Effect of Adenosine and Intracellular GTP on KATP Channels of Mammalian Skeletal Muscle

N. Standen, N. Davies, A. Comtois, 1996, The Journal of Membrane Biology.

ATP-sensitive potassium channels.

N. Standen, G. Rodrigo, 2005, Current pharmaceutical design.

SUR2A C‐terminal fragments reduce KATP currents and ischaemic tolerance of rat cardiac myocytes

N. Standen, N. Davies, R. D. Rainbow, 2004, The Journal of physiology.

Mechanisms that regulate [Ca2+]i following depolarization in rat systemic arterial smooth muscle cells

N. Standen, N. Davies, T. Kamishima, 2000, The Journal of physiology.

INWARD RECTIFICATION IN SKELETAL MUSCLE FIBRES

F. Ashcroft, N. Standen, C. Leech, 1981 .

Cannabinoid CB1 receptors fail to cause relaxation, but couple via Gi/Go to the inhibition of adenylyl cyclase in carotid artery smooth muscle

N. Standen, R. Challiss, J. Boyle, 1999, British journal of pharmacology.

Modulation of the nitric oxide metabolism overcomes the unresponsiveness of the diabetic human myocardium to protection against ischemic injury.

N. Standen, M. Galiñanes, A. Barua, 2011, The Journal of surgical research.

Arrestins 2 and 3 differentially regulate ETA and P2Y2 receptor-mediated cell signaling and migration in arterial smooth muscle.

N. Standen, G. E. Morris, R. Challiss, 2012, American journal of physiology. Cell physiology.

Cardioprotection by preconditioning: KATP channels, metabolism, or both?

N. Standen, N. B. Standen, 2002, The Journal of physiology.

Reduced effectiveness of HMR 1098 in blocking cardiac sarcolemmal K(ATP) channels during metabolic stress.

N. Standen, N. Davies, R. D. Rainbow, 2005, Journal of molecular and cellular cardiology.

Evidence for mitochondrial K channels as effectors of human

N. Standen, S. Ghosh, M. Galiñanes, 2000 .

Properties of BKCa Channels Formed by Bicistronic Expression of hSloα and β1–4 Subunits in HEK293 Cells

N. Standen, N. Davies, J. Lippiat, 2003, The Journal of Membrane Biology.

G protein-coupled receptor kinase 2 and arrestin 2 regulate arterial smooth muscle P 2 Y-purinoceptor signalling

N. Standen, G. E. Morris, R. Challiss, 2010 .

Voltage-dependent ATP-sensitive potassium channels of skeletal muscle membrane

N. Standen, A. Spruce, P. Stanfield, 1985, Nature.

Arterial dilations in response to calcitonin gene-related peptide involve activation of K+ channels

N. Standen, M. Nelson, J. Hescheler, 1990, Nature.

Hyperpolarizing vasodilators activate ATP-sensitive K+ channels in arterial smooth muscle.

N. Standen, M. Nelson, N. Davies, 1989, Science.

Studies of the unitary properties of adenosine‐5'‐triphosphate‐regulated potassium channels of frog skeletal muscle.

N. Standen, A. Spruce, P. Stanfield, 1987, The Journal of physiology.

Angiotensin II inhibits and alters kinetics of voltage-gated K(+) channels of rat arterial smooth muscle.

N. Standen, N. Davies, Y. Hayabuchi, 2001, American journal of physiology. Heart and circulatory physiology.

Angiotensin II inhibits rat arterial KATP channels by inhibiting steady‐state protein kinase A activity and activating protein kinase Ce

N. Standen, N. Davies, Y. Hayabuchi, 2001, The Journal of physiology.

Angiotensin II Inhibition of ATP‐Sensitive K+ Currents in Rat Arterial Smooth Muscle Cells Through Protein Kinase C

N. Standen, J. Quayle, M. Kubo, 1997, The Journal of physiology.

Noradrenaline contracts arteries by activating voltage-dependent calcium channels

N. Standen, M. Nelson, J. Brayden, 1988, Nature.

Postnatal development of inotropic responses to nerve stimulation and tyramine in rat atria

N. Standen, 1977, Pflügers Archiv.

Effects of the BKCa channel activator, NS1619, on rat cerebral artery smooth muscle

N. Standen, P. Langton, J. Boyle, 1996, British journal of pharmacology.

ATP‐sensitive K+ channel activation by calcitonin gene‐related peptide and protein kinase A in pig coronary arterial smooth muscle

N. Standen, J. Quayle, G. Wellman, 1998, The Journal of physiology.

ATP-sensitive and inwardly rectifying potassium channels in smooth muscle.

N. Standen, M. Nelson, J. Quayle, 1997, Physiological reviews.

Rubidium block and rubidium permeability of the inward rectifier of frog skeletal muscle fibres.

N. Standen, P. Stanfield, 1980, The Journal of physiology.

Potassium depletion and sodium block of potassium currents under hyperpolarization in frog sartorius muscle.

N. Standen, P. Stanfield, 1979, The Journal of physiology.

A potential‐ and time‐dependent blockade of inward rectification in frog skeletal muscle fibres by barium and strontium ions.

N. Standen, P. Stanfield, 1978, The Journal of physiology.

Ca channel inactivation by intracellular Ca injection into Helix neurones

N. Standen, 1981, Nature.

The effect of intracellular pH on ATP‐dependent potassium channels of frog skeletal muscle.

N. Standen, N. Davies, P. Stanfield, 1992, The Journal of physiology.

The effects of injection of calcium ions and calcium chelators on calcium channel inactivation in Helix neurones.

N. Standen, T. Plant, T. A. Ward, 1983, The Journal of physiology.

CALCIUM INWARD CURRENT IN AN IDENTIFIED SNAIL

N. Standen, 2005 .

Calcium and sodium ions as charge carriers in the action potential of an identified snail neurone.

N. Standen, 1975, The Journal of physiology.

Properties of a calcium channel in snail neurones

N. Standen, 1974, Nature.

Glimepiride, a Novel Sulfonylurea, Does Not Abolish Myocardial Protection Afforded by Either Ischemic Preconditioning or Diazoxide

N. Standen, M. Mocanu, D. Yellon, 2001, Circulation.

A rate theory model for Mg2+ block of ATP‐dependent potassium channels of rat skeletal muscle.

N. Standen, N. Davies, P. Stanfield, 1996, The Journal of physiology.

Properties of BK(Ca) channels formed by bicistronic expression of hSloalpha and beta1-4 subunits in HEK293 cells.

N. Standen, N. Davies, J. Lippiat, 2003, The Journal of membrane biology.

Tetraethylammonium block of Slowpoke calcium-activated potassium channels expressed in Xenopus oocytes: Evidence for tetrameric channel formation

R. North, J. Adelman, N. Standen, 1994, Pflügers Archiv.

Separation and analysis of macroscopic currents

N. Standen, N. Davies, P. Langton, 1999 .

The Biology of K ATP Channels

N. Standen, 1995 .

The flickery block of ATP-dependent potassium channels of skeletal muscle by internal 4-aminopyridine

N. Standen, N. Davies, A. I. Pettit, 1991, Pflügers Archiv.

Calcium current inactivation in identified neurones of Helix aspersa.

N. Standen, T. Plant, 1981, The Journal of physiology.

ATP-dependent potassium channels of muscle cells: Their properties, regulation, and possible functions

N. W. Davies, N. B. Standen, P. R. Stanfield, 1991, Journal of bioenergetics and biomembranes.

Evidence for involvement of A‐kinase anchoring protein in activation of rat arterial KATP channels by protein kinase A

N. Standen, C. Dart, Y. Hayabuchi, 2001, The Journal of physiology.

Direct block of native and cloned (Kir2.1) inward rectifier K+ channels by chloroethylclonidine

N. Standen, C. Dart, R. Barrett-Jolley, 1999, British journal of pharmacology.

The postnatal development of adrenoceptor responses in isolated papillary muscles from rat

N. Standen, E. Mackenzie, 2004, Pflügers Archiv.

Potassium channels: Structure, classification, function and therapeutic potential

N. Standen, 1990 .

Block of cloned BKCa channels (rSlo)expressed in HEK 293 cells by N-methyl d-glucamine

N. Standen, N. Davies, J. Lippiat, 1998, Pflügers Archiv.

A residue in the intracellular vestibule of the pore is critical for gating and permeation in Ca2+‐activated K+ (BKCa) channels

N. Standen, J. Lippiat, N. Davies, 2000, The Journal of physiology.

A binding-site model for calcium channel inactivation that depends on calcium entry

N. Standen, P. Stanfield, 1982, Proceedings of the Royal Society of London. Series B. Biological Sciences.

Increase in the vasorelaxant potency of K(ATP) channel opening drugs by adenosine A(1) and A(2) receptors in the pig coronary artery.

N. Standen, C. Davie, D. Everitt, 1999, European journal of pharmacology.

17 Chapter 2 Voltage clamp techniques

J. Halliwell, N. Standen, T. Plant, 1999 .

Voltage clamp techniques

J. Halliwell, N. Standen, 1999 .

Voltage‐clamp studies of the calcium inward current in an identified snail neurone: comparison with the sodium inward current.

N. Standen, 1975, The Journal of physiology.

Inward rectification in skeletal muscle: A blocking particle model

N. Standen, P. Stanfield, 1978, Pflügers Archiv.

Preconditioning the human myocardium by simulated ischemia: studies on the early and delayed protection.

N. Standen, M. Galiñanes, S. Ghosh, 2000, Cardiovascular research.

The voltage‐dependent block of ATP‐sensitive potassium channels of frog skeletal muscle by caesium and barium ions.

N. Standen, J. Quayle, P. Stanfield, 1988, The Journal of physiology.

The effect of intracellular anions on ATP‐dependent potassium channels of rat skeletal muscle.

N. Standen, N. Davies, P. Stanfield, 1994, The Journal of physiology.

Adenosine‐activated potassium current in smooth muscle cells isolated from the pig coronary artery.

N. Standen, C. Dart, N B Standen, 1993, The Journal of physiology.

Properties of single potassium channels in vesicles formed from the sarcolemma of frog skeletal muscle.

N. Standen, P. Stanfield, T. A. Ward, 1985, The Journal of physiology.

Multiple blocking mechanisms of ATP‐sensitive potassium channels of frog skeletal muscle by tetraethylammonium ions.

N. Standen, A. Spruce, N. Davies, 1989, The Journal of physiology.

The KATP channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidation

N. Standen, B. Billups, C. Lawrence, 2001, British journal of pharmacology.

Functional and electrophysiological effects of a novel imidazoline‐based KATP channel blocker, IMID‐4F

N. Standen, M. Kubo, G. Mcpherson, 1999, British journal of pharmacology.

Activation of ATP-dependent K+ currents in intact skeletal muscle fibres by reduced intracellular pH

N. Standen, N. Davies, P. Stanfield, 1992, Proceedings of the Royal Society of London. Series B: Biological Sciences.

The intrinsic gating of inward rectifier K+ channels expressed from the murine IRK1 gene depends on voltage, K+ and Mg2+.

W. J. Brammar, N. Standen, N. Davies, 1994, The Journal of physiology.

Arrestins 2 and 3 differentially regulate ETA and P2Y2 receptor-mediated cell signaling and migration in arterial smooth muscle.

N. Standen, R. Challiss, C. Nelson, 2012, American journal of physiology. Cell physiology.

The action of external tetraethylammonium ions on unitary delayed rectifier potassium channels of frog skeletal muscle.

N. Standen, A. Spruce, P. Stanfield, 1987, The Journal of physiology.

Principal role of adenylyl cyclase 6 in K+ channel regulation and vasodilator signalling in vascular smooth muscle cells

N. Standen, N. Davies, M. Perry, 2011, Cardiovascular research.

Kir6.2 limits Ca(2+) overload and mitochondrial oscillations of ventricular myocytes in response to metabolic stress.

N. Standen, R. D. Rainbow, D. Lodwick, 2013, American journal of physiology. Heart and circulatory physiology.

K+ channels in cardiovascular medicine : from basic science to clinical practice

N. Standen, D. Escande, 1993 .

Calcium-mediated potassium activation in Helix neurones.

R. Meech, N. Standen, 1974, The Journal of physiology.

A new preparation for recording single-channel currents from skeletal muscle

N. Standen, P. Stanfield, T. A. Ward, 1984, Proceedings of the Royal Society of London. Series B. Biological Sciences.

Potassium channels: Structure, classification, function and therapeutic potential

N. Standen, 1990 .

Rubidium ions and the gating of delayed rectifier potassium channels of frog skeletal muscle.

N. Standen, A. Spruce, P. Stanfield, 1989, The Journal of physiology.