A. Grover

发表

GAB2 Alleles Modify Alzheimer's Risk in APOE ɛ4 Carriers

Winnie S. Liang, G. Alexander, D. Stephan, 2007, Neuron.

GAB2 alleles modify Alzheimer's risk in APOE epsilon4 carriers.

Winnie S. Liang, G. Alexander, D. Stephan, 2007, Neuron.

Association of missense and 5′-splice-site mutations in tau with the inherited dementia FTDP-17

Ronald C. Petersen, Frank Owen, Stanley Fahn, 1998, Nature.

Alzheimer's disease is associated with reduced expression of energy metabolism genes in posterior cingulate neurons

Winnie S. Liang, Tracy L. Niedzielko, J. Morris, 2008, Proceedings of the National Academy of Sciences.

Epigenetic changes in Alzheimer's disease: Decrements in DNA methylation

Joseph Rogers, Paul D. Coleman, Andrew Grover, 2010, Neurobiology of Aging.

Evidence for an association between KIBRA and late-onset Alzheimer's disease

Richard J. Caselli, Gene E. Alexander, Daniel Bandy, 2010, Neurobiology of Aging.

A novel tau mutation in exon 9 (1260V) causes a four-repeat tauopathy

A. Grover, M. Hutton, U. Passant, 2003, Experimental Neurology.

Hyperphosphorylation-induced self assembly of murine tau: a comparison with human tau

A. Grover, I. Grundke‐Iqbal, K. Iqbal, 2005, Journal of Neural Transmission.

Whole genome association analysis shows that ACE is a risk factor for Alzheimer's disease and fails to replicate most candidates from Meta-analysis.

D. Stephan, S. Melquist, A. Myers, 2010, International journal of molecular epidemiology and genetics.

CHIP and Hsp70 regulate tau ubiquitination, degradation and aggregation

John Hardy, Dennis W. Dickson, Andrew Grover, 2004 .

Effects on splicing and protein function of three mutations in codon N296 of tau in vitro

A. Grover, M. Hutton, S. Yen, 2002, Neuroscience Letters.

Microglial responses to amyloid β peptide opsonization and indomethacin treatment

J. Rogers, D. Mastroeni, A. Grover, 2005, Journal of Neuroinflammation.

Alzheimer disease PS-1 exon 9 deletion defined

A. Paetau, S. Lincoln, J. Hardy, 1999, Nature Medicine.

Peripheral clearance of amyloid β peptide by complement C3-dependent adherence to erythrocytes

Rena Li, G. Ahern, J. Rogers, 2006, Neurobiology of Aging.

Altered neuronal gene expression in brain regions differentially affected by Alzheimer's disease: a reference data set.

Eric M Reiman, Richard J Caselli, Joseph Rogers, 2008, Physiological genomics.

The epigenetics of Alzheimer's disease — additional considerations

J. Rogers, D. Mastroeni, A. Grover, 2011, Neurobiology of Aging.

Neuroinflammation in Alzheimer's disease and Parkinson's disease: are microglia pathogenic in either disorder?

J. Rogers, D. Mastroeni, A. Grover, 2007, International review of neurobiology.

Epigenetic mechanisms in Alzheimer's disease

J. Rogers, D. Mastroeni, A. Grover, 2011, Neurobiology of Aging.

Leptin Decreases Energy Expenditure Despite Increased Thyroid Hormone in Patients with Lipodystrophy.

A. Grover, Kong Y. Chen, Rebecca J. Brown, 2021, The Journal of clinical endocrinology and metabolism.

Gene expression correlates of neurofibrillary tangles in Alzheimer's disease

Kevin M. Brown, Roger Higdon, Joseph Rogers, 2006, Neurobiology of Aging.

5′ Splice Site Mutations in tau Associated with the Inherited Dementia FTDP-17 Affect a Stem-Loop Structure That Regulates Alternative Splicing of Exon 10*

A. Grover, M. Hutton, H. Houlden, 1999, The Journal of Biological Chemistry.

Epigenetic Differences in Cortical Neurons from a Pair of Monozygotic Twins Discordant for Alzheimer's Disease

J. Rogers, D. Mastroeni, A. Grover, 2009, PloS one.

Gene expression profiles in anatomically and functionally distinct regions of the normal aged human brain.

Eric M Reiman, Richard J Caselli, Joseph Rogers, 2007, Physiological genomics.

Subventricular zone neural progenitors from rapid brain autopsies of elderly subjects with and without neurodegenerative disease

E. Hol, J. Rogers, D. Mastroeni, 2009, The Journal of comparative neurology.

High-content siRNA screening of the kinome identifies kinases involved in Alzheimer's disease-related tau hyperphosphorylation

C. Beaudry, D. Stephan, E. Reiman, 2010, BMC Genomics.

Consistent decrease in global DNA methylation and hydroxymethylation in the hippocampus of Alzheimer's disease patients

P. Hof, H. Steinbusch, D. Mastroeni, 2013, Neurobiology of Aging.

Reduced RAN Expression and Disrupted Transport between Cytoplasm and Nucleus; A Key Event in Alzheimer’s Disease Pathophysiology

Winnie S. Liang, J. Rogers, W. Liang, 2013, PloS one.

Aberrant intracellular localization of H3k4me3 demonstrates an early epigenetic phenomenon in Alzheimer's disease

D. Mastroeni, A. Grover, P. Coleman, 2015, Neurobiology of Aging.

Microglial responses to dopamine in a cell culture model of Parkinson's disease

J. Rogers, D. Mastroeni, A. Grover, 2009, Neurobiology of Aging.

The L266V tau mutation is associated with frontotemporal dementia and Pick-like 3R and 4R tauopathy

M. Mesulam, D. Dickson, E. Bigio, 2003, Acta Neuropathologica.

Frequency of tau mutations in three series of non‐Alzheimer's degenerative dementia

R. Petersen, D. Neary, B. Boeve, 1999, Annals of neurology.

Reprogramming of tau alternative splicing by spliceosome-mediated RNA trans-splicing: Implications for tauopathies

M. Garcia-Blanco, A. Grover, S. Mansfield, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Neuronal gene expression in non-demented individuals with intermediate Alzheimer's Disease neuropathology

Winnie S. Liang, J. Morris, D. Stephan, 2010, Neurobiology of Aging.

Evidence for independent mechanisms and a multiple-hit model of tau assembly.

A. Grover, M. Hutton, S. Yen, 2006, Biochemical and biophysical research communications.

brain human functionally distinct regions of the normal aged Gene expression profiles in anatomically and

J. Morris, E. Reiman, R. Caselli, 2015 .

Evidence for an association between KIBRA and late-onset Alzheimer's disease

Jason J. Corneveaux, Winnie S. Liang, G. Alexander, 2010, Neurobiology of Aging.

Altered neuronal gene expression in brain regions differentially affected by Alzheimer's disease: a reference data set.

Winnie S. Liang, D. Stephan, E. Reiman, 2008, Physiological genomics.

Microglial responses to amyloid β peptide opsonization and indomethacin treatment

D. Mastroeni, A. Grover, R. Strohmeyer, 2005, Journal of Neuroinflammation.