A. Rozkalne

发表

Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease

D. Holtzman, B. Hyman, M. Meyer-Luehmann, 2008, Nature.

Preservation of Neuronal Number Despite Age-Related Cortical Brain Atrophy in Elderly Subjects Without Alzheimer Disease

B. Hyman, M. Frosch, L. Lipsitz, 2008, Journal of neuropathology and experimental neurology.

Fam65b is important for formation of the HDAC6‐dysferlin protein complex during myogenic cell differentiation

A. Rozkalne, L. Kunkel, A. Balasubramanian, 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Mouse regenerating myofibers detected as false-positive donor myofibers with anti-human spectrin.

A. Rozkalne, E. Gussoni, J. Morgan, 2014, Human gene therapy.

Inhibition of Sirtuin 2 with Sulfobenzoic Acid Derivative AK1 is Non-Toxic and Potentially Neuroprotective in a Mouse Model of Frontotemporal Dementia

G. Carlson, Rose Pitstick, T. Spires-Jones, 2012, Front. Pharmacol..

Amyloid β Induces the Morphological Neurodegenerative Triad of Spine Loss, Dendritic Simplification, and Neuritic Dystrophies through Calcineurin Activation

Tara Spires-Jones, Brian J Bacskai, Kishore Kuchibhotla, 2010, The Journal of Neuroscience.

Explorer Calcineurin inhibition with systemic FK 506 treatment increases dendritic branching and dendritic spine density in healthy adult mouse brain

B. Hyman, T. Spires-Jones, A. Rozkalne, 2018 .

CD82 Is a Marker for Prospective Isolation of Human Muscle Satellite Cells and Is Linked to Muscular Dystrophies.

J. Spinazzola, A. Rozkalne, L. Kunkel, 2016, Cell stem cell.

Human fetal skeletal muscle contains a myogenic side population that expresses the melanoma cell-adhesion molecule.

A. Rozkalne, E. Gussoni, A. Lapan, 2012, Human molecular genetics.

POMK mutations disrupt muscle development leading to a spectrum of neuromuscular presentations.

C. Walsh, T. Yu, A. Rozkalne, 2014, Human molecular genetics.

Apolipoprotein E: Isoform Specific Differences in Tertiary Structure and Interaction with Amyloid-β in Human Alzheimer Brain

Phillip B. Jones, B. Hyman, T. Spires-Jones, 2011, PloS one.

G‐protein coupled receptor 56 promotes myoblast fusion through serum response factor‐ and nuclear factor of activated T‐cell‐mediated signalling but is not essential for muscle development in vivo

A. Rozkalne, E. Gussoni, A. Kopin, 2013, The FEBS journal.

Explorer Passive immunotherapy rapidly increases structural plasticity in a mouse model of Alzheimer disease

B. Hyman, M. Meyer-Luehmann, T. Spires-Jones, 2017 .

Passive immunotherapy rapidly increases structural plasticity in a mouse model of Alzheimer disease

B. Hyman, M. Meyer-Luehmann, T. Spires-Jones, 2009, Neurobiology of Disease.

Calcineurin inhibition with FK506 ameliorates dendritic spine density deficits in plaque-bearing Alzheimer model mice

B. Hyman, T. Spires-Jones, A. Rozkalne, 2011, Neurobiology of Disease.

Curcumin labels amyloid pathology in vivo, disrupts existing plaques, and partially restores distorted neurites in an Alzheimer mouse model

B. Hyman, M. Garcia-Alloza, A. Rozkalne, 2007, Journal of neurochemistry.

A single dose of passive immunotherapy has extended benefits on synapses and neurites in an Alzheimer's disease mouse model

B. Hyman, E. Stern, T. Spires-Jones, 2009, Brain Research.

Calcineurin inhibition with systemic FK506 treatment increases dendritic branching and dendritic spine density in healthy adult mouse brain

B. Hyman, T. Spires-Jones, A. Rozkalne, 2011, Neuroscience Letters.

Antagonistic Regulation of Actin Dynamics and Cell Motility by TRPC5 and TRPC6 Channels

A. Rozkalne, H. Pavenstädt, J. Schlondorff, 2010, Science Signaling.

Explorer Rapid appearance and local toxicity of amyloid-beta plaques in a mouse model of Alzheimer's disease

D. Holtzman, B. Hyman, M. Meyer-Luehmann, 2012 .