M. Morishima

发表

A pivotal role for endogenous TGF-β-activated kinase-1 in the LKB1/AMP-activated protein kinase energy-sensor pathway

Michael D. Schneider, A. Baldini, J. Dyck, 2006, Proceedings of the National Academy of Sciences.

Foxc2 influences alveolar epithelial cell differentiation during lung development

M. Morishima, S. Enerbäck, J. Tamaoki, 2017, Development, growth & differentiation.

Summary Tbx 1 has a dual role in the morphogenesis of the cardiac outflow tract

B. Bruneau, R. Schwartz, A. Baldini, 2004 .

Ultrastructural changes in the choriocapillaris of N-methyl-N-nitrosourea-induced retinal degeneration in C57BL/6 mice

M. Morishima, H. Fujieda, K. Shimizu, 2020, Medical Molecular Morphology.

TBX1 is required for inner ear morphogenesis.

F. Vitelli, A. Baldini, M. Morishima, 2003, Human molecular genetics.

Hypoplasia of cushion ridges in the proximal outflow tract elicits formation of a right ventricle‐to‐aortic route in retinoic acid‐induced complete transposition of the great arteries in the mouse: Scanning electron microscopic observations of corrosion cast models

M. Morishima, M. Nakazawa, Y. Nakajima, 1996, The Anatomical record.

Visceroatrial heterotaxy syndrome in the NOD mouse with special reference to atrial situs.

M. Morishima, M. Ando, A. Takao, 1991, Teratology.

Ripply3, a Tbx1 repressor, is required for development of the pharyngeal apparatus and its derivatives in mice

M. Morishima, R. Matsuoka, H. Yagi, 2011, Development.

Influence of genetic and maternal diabetes in the pathogenesis of visceroatrial heterotaxy in mice.

M. Morishima, M. Ando, H. Yasui, 1996, Teratology.

Developmental spectrum of cardiac outflow tract anomalies encompassing transposition of the great arteries and dextroposition of the aorta: Pathogenic effect of extrinsic retinoic acid in the mouse embryo

M. Morishima, M. Ando, M. Nakazawa, 1999, The Anatomical record.

Morphological observations on the pathogenetic process of transposition of the great arteries induced by retinoic acid in mice.

M. Morishima, H. Yasui, M. Nakazawa, 1995, Circulation.

Genetic factors are major determinants of phenotypic variability in a mouse model of the DiGeorge/del22q11 syndromes

M. Morishima, Tu V. Huynh, E. Lindsay, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Ece1 and Tbx1 define distinct pathways to aortic arch morphogenesis

A. Baldini, M. Morishima, M. Yanagisawa, 2003, Developmental dynamics : an official publication of the American Association of Anatomists.

Tbx1 haploinsufficiency identified by functional scanning of the DiGeorge syndrome region is the cause of aortic arch defects in mice.

P. Scambler, A. Bradley, F. Vitelli, 2001 .

A genetic link between Tbx1 and fibroblast growth factor signaling.

F. Vitelli, A. Baldini, M. Morishima, 2002, Development.

Tbx1 haploinsufficiency in the DiGeorge syndrome region causes aortic arch defects in mice

P. Scambler, A. Bradley, F. Vitelli, 2001, Nature.

Altered distribution of collagen type I and hyaluronic acid in the cardiac outflow tract of mouse embryos destined to develop transposition of the great arteries

M. Morishima, H. Yasui, M. Nakazawa, 2007, Heart and Vessels.

Distribution of fibronectin, type I collagen, type IV collagen, and laminin in the cardiac jelly of the mouse embryonic heart with retinoic acid‐induced complete transposition of the great arteries

M. Morishima, M. Nakazawa, Y. Nakajima, 1997, The Anatomical record.

Dominant lethality of the mouse skeletal mutation tail-short (Ts) is determined by the Ts allele from mating partners.

M. Morishima, T. Shiroishi, H. Yasui, 1998, Genomics.

Engineered chromosome-based genetic mapping establishes a 3.7 Mb critical genomic region for Down syndrome-associated heart defects in mice

P. Ye, M. Morishima, A. Pao, 2014, Human Genetics.

Genetic analysis of Down syndrome-associated heart defects in mice

Li Zhang, Antonio Baldini, N. Nowak, 2011, Human Genetics.

Duplication of the entire 22.9 Mb human chromosome 21 syntenic region on mouse chromosome 16 causes cardiovascular and gastrointestinal abnormalities.

N. Nowak, M. Morishima, J. Conroy, 2007, Human molecular genetics.

Tbx1 mutation causes multiple cardiovascular defects and disrupts neural crest and cranial nerve migratory pathways.

F. Vitelli, A. Baldini, M. Morishima, 2002, Human molecular genetics.

Vasohibin-2 modulates tumor onset in the gastrointestinal tract by normalizing tumor angiogenesis

M. Morishima, Yasuhiro Suzuki, Yasufumi Sato, 2014, Molecular Cancer.

A Deficiency in the Region Homologous to Human 17q21.33–q23.2 Causes Heart Defects in Mice

A. Bradley, A. Baldini, M. Morishima, 2006, Genetics.

Tbx1 expression in pharyngeal epithelia is necessary for pharyngeal arch artery development

Joshua W. Vincentz, F. Vitelli, A. Baldini, 2005, Development.

Tbx1 has a dual role in the morphogenesis of the cardiac outflow tract

B. Bruneau, R. Schwartz, A. Baldini, 2004, Development.

The role of Foxc2 gene in lung development

M. Morishima, T. Kume, J. Tamaoki, 2014 .

Pathological study of Japanese quail embryo with acid α-glucosidase deficiency during early development

M. Morishima, M. Nakazawa, T. Kikuchi, 1996, Acta Neuropathologica.

Visceroatrial heterotaxy syndrome induced by maternal hyperthermia in the rat

M. Morishima, M. Ando, H. Yasui, 1995, Cardiology in the Young.

Tbx1 and Digeorge Syndrome: A Genetic Link between Cardiovascular and Pharyngeal Development

A. Baldini, M. Morishima, Huansheng Xu, 2007 .

Cardiac outflow tract septation process in the mouse model of transposition of the great arteries.

M. Morishima, M. Ando, H. Yasui, 1997, Teratology.

Effects of Environmental Hyperthermia on Cardiovascular Function in the Rat Embryo

M. Morishima, M. Nakazawa, A. Takao, 1991, Pediatric Research.

Anomalous looping, atrioventricular cushion dysplasia, and unilateral ventricular hypoplasia in the mouse embryos with right isomerism induced by retinoic acid

M. Morishima, M. Nakazawa, E. Aikawa, 1998, The Anatomical record.

Situs variation and cardiovascular anomalies in the transgenic mouse insertional mutation, inv.

M. Morishima, M. Ishibashi, M. Ando, 1998, Teratology.

Inhibition of outflow cushion mesenchyme formation in retinoic acid-induced complete transposition of the great arteries.

M. Morishima, M. Nakazawa, Y. Nakajima, 1996, Cardiovascular research.

Hemodynamics and Ventricular Function in the Day-12 Rat Embryo: Basic Characteristics and the Responses to Cardiovascular Drugs

M. Morishima, M. Nakazawa, Fusae Kajio, 1995, Pediatric Research.