M. de la Luz Sierra

发表

Identification of a Family of Sorting Nexin Molecules and Characterization of Their Association with Receptors

D. Haft, C. Haft, M. de la Luz Sierra, 1998, Molecular and Cellular Biology.

D. Accili, M. Quon, M. de la Luz Sierra, 1992, Endocrine reviews.

Succinate dehydrogenase (SDH) D subunit (SDHD) inactivation in a growth-hormone-producing pituitary tumor: a new association for SDH?

P. Rustin, N. Patronas, M. de la Luz Sierra, 2012, The Journal of clinical endocrinology and metabolism.

Substitution of isoleucine for methionine at position 1153 in the beta-subunit of the human insulin receptor. A mutation that impairs receptor tyrosine kinase activity, receptor endocytosis, and insulin action.

M. Quon, M. de la Luz Sierra, S. Taylor, 1992, The Journal of biological chemistry.

G-CSF down-regulation of CXCR4 expression identified as a mechanism for mobilization of myeloid cells.

M. de la Luz Sierra, A. V. Gulino, C. Williams, 2006, Blood.

KCNJ5 mutations in the National Institutes of Health cohort of patients with primary hyperaldosteronism: an infrequent genetic cause of Conn's syndrome.

D. Hoffman, M. de la Luz Sierra, C. Stratakis, 2012, Endocrine-related cancer.

Identification of carboxypeptidase N as an enzyme responsible for C-terminal cleavage of stromal cell-derived factor-1alpha in the circulation.

Fuquan Yang, M. de la Luz Sierra, M. Narazaki, 2005, Blood.

Lipoprotein particles in patients with pediatric Cushing disease and possible cardiovascular risks

M. de la Luz Sierra, C. Stratakis, A. Remaley, 2019, Pediatric Research.

Differential processing of stromal-derived factor-1alpha and stromal-derived factor-1beta explains functional diversity.

Fuquan Yang, M. de la Luz Sierra, M. Narazaki, 2004, Blood.

Analysis of the juxtamembrane dileucine motif in the insulin receptor.

C. Haft, M. de la Luz Sierra, S. Taylor, 1998, Endocrinology.

Differences in adiposity in Cushing syndrome caused by PRKAR1A mutations: clues for the role of cyclic AMP signaling in obesity and diagnostic implications.

M. de la Luz Sierra, C. Stratakis, M. Lodish, 2014, The Journal of clinical endocrinology and metabolism.

The Transcription Factor Gfi1 Regulates G-csf Signaling and Neutrophil Development through the Ras Activator Rasgrp1 the Transcription Factor Growth Factor In- Dependence 1 (gfi1) and the Growth Fac- Tor Granulocyte Colony-stimulating Factor (g-csf) Are Individually Essential for Neu- Trophil Differ

D. Lowy, D. Maric, M. de la Luz Sierra, 2022 .

Transcription factor Gfi-1 induced by G-CSF is a negative regulator of CXCR4 in myeloid cells.

Peter J McCormick, Giovanna Tosato, M. de la Luz Sierra, 2007, Blood.

EphB2 and EphB4 receptors forward signaling promotes SDF-1-induced endothelial cell chemotaxis and branching remodeling.

Peter J McCormick, Giovanna Tosato, M. de la Luz Sierra, 2006, Blood.

Substitution of glutamic acid for alanine 1135 in the putative "catalytic loop" of the tyrosine kinase domain of the human insulin receptor. A mutation that impairs proteolytic processing into subunits and inhibits receptor tyrosine kinase activity.

M. Quon, M. de la Luz Sierra, L. Ottini, 1993, The Journal of biological chemistry.

A mutation in the tyrosine kinase domain of the insulin receptor associated with insulin resistance in an obese woman.

M. de la Luz Sierra, Simeon I. Taylor, L. Ottini, 1991, The Journal of clinical endocrinology and metabolism.

Selective expression of stromal-derived factor-1 in the capillary vascular endothelium plays a role in Kaposi sarcoma pathogenesis.

S. Pittaluga, M. de la Luz Sierra, R. Yarchoan, 2003, Blood.

SDH Subunit Mutation Status in Saliva: Genetic Testing in Patients with Pheochromocytoma

F. Faucz, M. de la Luz Sierra, K. Pacak, 2016, Hormone and Metabolic Research.

Bilateral Adrenal Hyperplasia as a Possible Mechanism for Hyperandrogenism in Women With Polycystic Ovary Syndrome.

E. Turkbey, A. Dobs, M. de la Luz Sierra, 2016, The Journal of clinical endocrinology and metabolism.

Human orthologs of yeast vacuolar protein sorting proteins Vps26, 29, and 35: assembly into multimeric complexes.

C. Haft, M. de la Luz Sierra, V. Barr, 2000, Molecular biology of the cell.

Familial pituitary apoplexy as the only presentation of a novel AIP mutation.

G. Chrousos, N. Patronas, M. de la Luz Sierra, 2013, Endocrine-related cancer.

Novel anti-inflammatory properties of the angiogenesis inhibitor vasostatin.

M. de la Luz Sierra, J. Schröder, E. Christophers, 2007, The Journal of investigative dermatology.

Not All Glucocorticoid-Induced Obesity is the Same: Differences in Adiposity Among Various Diagnostic Groups of Cushing Syndrome

M. de la Luz Sierra, C. Stratakis, M. Lodish, 2014, Hormone and Metabolic Research.

The transcription factor Gfi1 regulates G-CSF signaling and neutrophil development through the Ras activator RasGRP1.

D. Lowy, D. Maric, M. de la Luz Sierra, 2010, Blood.

Bilateral Adrenal Hyperplasia as a Possible Mechanism for Hyperandrogenism in Women With Polycystic Ovary Syndrome.

E. Turkbey, A. Dobs, M. de la Luz Sierra, 2016, Journal of Clinical Endocrinology and Metabolism.