M. Bosma

发表

NaV1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms

J. Linton, W. Catterall, F. Yu, 2012, Proceedings of the National Academy of Sciences.

Intracellular responses to gonadotropin-releasing hormone in a clonal cell line of the gonadotrope lineage.

B. Hille, M. Bosma, J. Windle, 1991, Molecular endocrinology.

Ion channel development, spontaneous activity, and activity-dependent development in nerve and muscle cells.

William J Moody, W. Moody, M. Bosma, 2005, Physiological reviews.

A nonselective cation channel activated by membrane deformation in oocytes of the ascidianBoltenia villosa

W. Moody, M. Bosma, 1989, The Journal of Membrane Biology.

Signaling mechanisms during the response of pituitary gonadotropes to GnRH.

B. Hille, M. Bosma, F. W. Tse, 1995, Recent progress in hormone research.

PKA-dependent regulation of mKv1.1, a mouse Shaker-like potassium channel gene, when stably expressed in CHO cells

M. Bosma, B. Tempel, M. Allen, 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Early persistent activation of sperm K+ channels by the egg peptide speract.

M. Bosma, A. Darszon, D. Babcock, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Hyperpolarization of resting membrane potential causes retraction of spontaneous transients during mouse embryonic circuit development

M. Bosma, Amanda J. Tose, Hirofumi Watari, 2013, The Journal of physiology.

Modulation of M current in frog sympathetic ganglion cells.

B. Hille, M. Bosma, M. D. Leibowitz, 1990, Society of General Physiologists series.

Differential expression of membrane conductances underlies spontaneous event initiation by rostral midline neurons in the embryonic mouse hindbrain

M. Bosma, N. Abedini, Audrey M Moruzzi, 2009, The Journal of physiology.

Spontaneous, synchronous electrical activity in neonatal mouse cortical neurones

William J Moody, W. Moody, Rebekah J. Corlew, 2004, The Journal of physiology.

Development of Synchronized Activity of Cranial Motor Neurons in the Segmented Embryonic Mouse Hindbrain

J. J. Wright, M. Bosma, J. Gust, 2003, The Journal of physiology.

Midline serotonergic neurones contribute to widespread synchronized activity in embryonic mouse hindbrain

M. Bosma, A. K. McCabe, P. N. Hunt, 2005, The Journal of physiology.

The Type 1 Inositol 1,4,5-Trisphosphate Receptor Gene Is Altered in the opisthotonos Mouse

M. Bosma, B. Tempel, M. Regan, 1997, The Journal of Neuroscience.

Timing and mechanism of a window of spontaneous activity in embryonic mouse hindbrain development

M. Bosma, 2010, Annals of the New York Academy of Sciences.

Electrophysiological properties of a cell line of the gonadotrope lineage.

B. Hille, M. Bosma, Martha M. Bosma, 1992, Endocrinology.

Hormone-induced loss of surface membrane during maturation of starfish oocytes: differential effects on potassium and calcium channels.

W. Moody, M. Bosma, 1985, Developmental biology.

Primary role of the serotonergic midline system in synchronized spontaneous activity during development of the embryonic mouse hindbrain.

M. Bosma, A. K. McCabe, J. Gust, 2006, Journal of neurobiology.

Spontaneous activity in the developing mouse midbrain driven by an external pacemaker

M. Bosma, J. L. Kirkman, Wendy Rockhill, 2009, Developmental neurobiology.

Looping circuit: a novel mechanism for prolonged spontaneous [Ca2+]i increases in developing embryonic mouse brainstem

M. Bosma, Hirofumi Watari, Amanda J. Tose, 2014, The Journal of physiology.

Identification of the delayed rectifier potassium channel, Kv1.6, in cultured astrocytes

M. Bosma, B. Tempel, Sharon L. Smart, 1997, Glia.

From neuron to muscle to movement: a complete biomechanical model of Hydra contractile behaviors

Rafael Yuste, Adrienne L. Fairhall, Michael Regnier, 2020, bioRxiv.

A complete biomechanical model of Hydra contractile behaviors, from neural drive to muscle to movement.

R. Yuste, A. Fairhall, T. Daniel, 2023, Proceedings of the National Academy of Sciences of the United States of America.

Anion channels with multiple conductance levels in a mouse B lymphocyte cell line.

M. Bosma, 1989, The Journal of physiology.

Protein kinase C is not necessary for peptide-induced suppression of M current or for desensitization of the peptide receptors.

B. Hille, M. Bosma, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Retinoic acid inhibits Ca2+ currents and cell proliferation in a B‐lymphocyte cell line

M. Bosma, N. Sidell, 1988, Journal of cellular physiology.

Ion channel properties and episodic activity in isolated immortalized gonadotropin-releasing hormone (GnRH) neurons

M. Bosma, Martha M. Bosma, 1993, The Journal of Membrane Biology.

Properties and mechanisms of spontaneous activity in the embryonic chick hindbrain

M. Bosma, C. Easton, Sean M. Hughes, 2009, Developmental neurobiology.

NaV1.1 channels are critical for intercellular communication in the suprachiasmatic nucleus and for normal circadian rhythms

J. Linton, W. Catterall, J. Hurley, 2012, Proceedings of the National Academy of Sciences.

Marcroscopic and single-channel studies of two Ca2+ channel types in oocytes of the ascidianCiona intestinalis

W. Moody, M. Bosma, 1990, The Journal of Membrane Biology.

Cells Currents in Retinal Glial (Müller) Developmental Regulation of Calcium

W. Moody, A. Reichenbach, M. Bosma, 2015 .