E. Théâtre

发表

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

David C. Wilson, Judy H. Cho, C. Gieger, 2012, Nature.

IBD risk loci are enriched in multigenic regulatory modules encompassing putative causative genes

David C. Wilson, Judy H. Cho, C. Gieger, 2018, Nature Communications.

Comparative performances of machine learning methods for classifying Crohn Disease patients using genome-wide genotyping data

Judy H. Cho, John P. Ferguson, G. Wainrib, 2019, Scientific Reports.

Inherited determinants of Crohn's disease and ulcerative colitis phenotypes: a genetic association study

Tariq Ahmad, Judy H Cho, Vibeke Andersen, 2016, The Lancet.

NF-κB-Independent Role of IKKα/IKKβ in Preventing RIPK1 Kinase-Dependent Apoptotic and Necroptotic Cell Death during TNF Signaling.

M. Bertrand, J. Bertin, P. Vandenabeele, 2015, Molecular cell.

Sensitivity of intestinal fibroblasts to TNF-related apoptosis-inducing ligand-mediated apoptosis in Crohn's disease

P. Delvenne, J. Belaiche, E. Louis, 2008, Scandinavian journal of gastroenterology.

Induction of nuclear factor-κB and its downstream genes by TNF-α and IL-1β has a pro-apoptotic role in pancreatic beta cells

T. Ørntoft, E. Théâtre, D. Eizirik, 2008, Diabetologia.

DUSP3 Phosphatase Deficiency or Inhibition Limit Platelet Activation and Arterial Thrombosis Running title: Musumeci et al.; DUSP3, a new player in arterial thrombosis

T. Mustelin, T. Chung, E. Théâtre, 2014 .

Analysis of Genes Associated With Monogenic Primary Immunodeficiency Identifies Rare Variants in XIAP in Patients With Crohn's Disease.

B. Charloteaux, M. Mni, D. Franchimont, 2018, Gastroenterology.

Biological Safety Evaluation of KiOmedine® CM-chitosan, an Innovative Non-animal Carboxymethyl-Chitosan Biomaterial Intended for Injectable Biomedical Applications

E. Théâtre, S. Gautier, J. Bentin, 2020 .

Dual-Specificity Phosphatase 3 Deficiency or Inhibition Limits Platelet Activation and Arterial Thrombosis

T. Mustelin, T. Chung, E. Théâtre, 2014, Circulation.

Fine-mapping inflammatory bowel disease loci to single variant resolution

Hailiang Huang, Kyle Kai-How Farh, Andre Franke, 2017, Nature.

High density mapping of the MHC identifies a shared role for HLA-DRB1*01:03 in inflammatory bowel diseases and heterozygous advantage in ulcerative colitis

Hailiang Huang, Stephan Ripke, Andre Franke, 2014, Nature Genetics.

Materials for : Host-microbe interactions shape genetic risk for inflammatory bowel disease

Judy H. Cho, C. Gieger, M. Daly, 2012 .

Comparative performances of machine learning methods for classifying Crohn Disease patients using genome-wide genotyping data

Judy H. Cho, John P. Ferguson, G. Wainrib, 2019, Scientific Reports.

Genome-Wide Copy Number Variation Scan Identifies Complement Component C4 as Novel Susceptibility Gene for Crohn's Disease

Y. Moreau, J. Vermeesch, C. Robberecht, 2016, Inflammatory bowel diseases.

An efficient algorithm to perform multiple testing in epistasis screening

Louis Wehenkel, Kristel Van Steen, François Van Lishout, 2013, BMC Bioinformatics.

CCDC88B is required for pathogenesis of inflammatory bowel disease

E. Louis, M. Georges, M. Kubo, 2017, Nature Communications.

DUSP 3 Phosphatase Deficiency or Inhibition Limit Platelet Activation and Arterial Thrombosis

T. Mustelin, T. Chung, E. Théâtre, 2014 .

Overexpression of CD39 in Mouse Airways Promotes Bacteria-Induced Inflammation

L. Bettendorff, V. Bours, E. Théâtre, 2012, The Journal of Immunology.

Epilepsy, hippocampal sclerosis and febrile seizures linked by common genetic variation around SCN1A

A. Ramasamy, I. Scheffer, H. Hakonarson, 2013, Brain : a journal of neurology.

Association mapping of inflammatory bowel disease loci to single variant resolution

Masa Umicevic Mirkov, Judy H. Cho, M. Daly, 2015, bioRxiv.

Background—A limitation of current antiplatelet therapies is their inability to separate thrombotic events from bleeding occurrences. A better understanding of the molecular mechanisms leading to platelet activation is important for the development of improved therapies. Recently, protein tyrosine p

T. Mustelin, T. Chung, E. Théâtre, 2015 .

The TNF/ADAM 17 system: implication of an ADAM 17 haplotype in the clinical response to infliximab in Crohn's disease

J. Belaiche, P. Rutgeerts, A. Gossum, 2006, Pharmacogenetics and genomics.

NF-k B-Independent Role o f IKK a / IKK b in Preventing RIPK 1 Kinase-Dependent Apoptotic and Necroptotic Cell Death during TNF Signaling Graphical Abstract Highlights

M. Bertrand, J. Bertin, P. Vandenabeele, 2015 .

A P2X ion channel-triggered NF-kappaB pathway enhances TNF-alpha-induced IL-8 expression in airway epithelial cells.

V. Bours, E. Théâtre, C. Oury, 2009, American journal of respiratory cell and molecular biology.

DUSP3 Genetic Deletion Confers M2-like Macrophage–Dependent Tolerance to Septic Shock

T. Mustelin, E. Théâtre, C. Libert, 2015, The Journal of Immunology.

CCDC88B is required for pathogenesis of inflammatory bowel disease

E. Louis, M. Georges, M. Kubo, 2017, Nature Communications.