H. Huang

发表

KLF6 Gene and early melanoma development in a collagen I-rich extracellular environment.

S. Friedman, W. Cavenee, S. Huh, 2010, Journal of the National Cancer Institute.

The potential of TRAIL for cancer chemotherapy

W. Cavenee, M. Nagane, H. Huang, 2001, Apoptosis.

CD95‐mediated Apoptosis of Human Glioma Cells: Modulation by Epidermal Growth Factor Receptor Activity

M. Weller, W. Cavenee, H. Huang, 2002, Brain pathology.

Functional analysis of wild-type and malignant glioma derived CDKN2Aβ alleles: Evidence for an RB-independent growth suppressive pathway

W. Cavenee, W. Arap, D. Sidransky, 1997, Oncogene.

Advances in Brief Absence of PTEN / MMAC 1 GermLine Mutations in Sporadic Bannayan-Riley-Ruvalcaba Syndrome 1

Marilyn, Dharam, F. Furnari, 2006 .

Novel monoclonal antibody specific for the de2‐7 epidermal growth factor receptor (EGFR) that also recognizes the EGFR expressed in cells containing amplification of the EGFR gene

A. Scott, W. Cavenee, A. Burgess, 2002, International journal of cancer.

Genistein‐induced neuronal differentiation is associated with activation of extracellular signal‐regulated kinases and upregulation of p21 and N‐cadherin

S. Hung, H. Huang, Hao-jen Huang, 2005, Journal of cellular biochemistry.

A novel seven transmembrane receptor induced during the early steps of astrocyte differentiation identified by differential expression

W. Cavenee, F. Furnari, H. Huang, 2002, Journal of neurochemistry.

in Vivo and in Vitro Ligand with Tumor Necrosis Factor-related Apoptosis-inducing Agents in Human Gliomas Causes Synergistic Cytotoxicity Increased Death Receptor 5 Expression by Chemotherapeutic

W. Cavenee, M. Nagane, H. Huang, 2000 .

Advances in Brief Increased Death Receptor 5 Expression by Chemotherapeutic Agents in Human Gliomas Causes Synergistic Cytotoxicity with Tumor Necrosis Factor-related Apoptosis-inducing Ligand in Vitro and in Vivo

W. Cavenee, M. Nagane, H. Huang, 2000 .

Analysis of the P300/CBP-Associated Factor (PCAF) Gene in Astrocytic Tumors

W. Cavenee, H. Huang, H. Nishimori, 2004, Journal of Neuro-Oncology.

Anatomic Sites Activities in Promoting Tumor Angiogenesis at Different Vascular Endothelial Growth Factor Isoforms Display Distinct Updated

W. Cavenee, M. Nagane, H. Huang, 2001 .

Vascular endothelial growth factor isoforms display distinct activities in promoting tumor angiogenesis at different anatomic sites.

W. Cavenee, M. Nagane, H. Huang, 2001, Cancer research.

Bioinformatic identification of IGF 1 as a hub gene in hepatocellular carcinoma ( HCC ) and in-vitro analysis of the chemosensitizing effect of miR-379 via suppressing the IGF 1 / IGF 1 R signaling pathway

H. Huang, J. Yang, LI Y.-H., 2016 .

Loss of Heterozygosity (LOH)

W. Cavenee, H. Huang, G. Robertson, 2002 .

Causes of drug resistance and novel therapeutic opportunities for the treatment of glioblastoma.

W. Cavenee, M. Nagane, H. Huang, 1999, Drug resistance updates : reviews and commentaries in antimicrobial and anticancer chemotherapy.

Mutant epidermal growth factor receptor signaling down-regulates p27 through activation of the phosphatidylinositol 3-kinase/Akt pathway in glioblastomas.

Motoo Nagane, Yoshitaka Narita, W. Cavenee, 2002, Cancer research.

The Enhanced Tumorigenic Activity of a Mutant Epidermal Growth Factor Receptor Common in Human Cancers Is Mediated by Threshold Levels of Constitutive Tyrosine Phosphorylation and Unattenuated Signaling*

H. Wiley, W. Cavenee, M. Nagane, 1997, The Journal of Biological Chemistry.

Replacement of the pl 6 / CDKN 2 Gene Suppresses Human Glioma Cell Growth 1

W. Cavenee, F. Furnari, W. Arap, 2006 .

PIK3CA mutations in plasma circulating tumor DNA predict survival and treatment outcomes in patients with advanced cancers

F. Meric-Bernstam, H. Huang, F. Janku, 2021, ESMO open.

Synthesis and Neoangiogenesis ( Flt-1 ) / Vascular Endothelial Growth Factor Receptor-1 Astrocytomas Is Associated with Fms-like Tyrosine Kinase-1 Expression of the Ets-1 Transcription Factor in Human Updated

O. Wiestler, W. Cavenee, H. Huang, 1999 .

Point mutations can inactivate in vitro and in vivo activities of p16INK4a/CDKN2A in human glioma

W. Cavenee, W. Arap, H. Huang, 1997, Oncogene.

Release of an inhibitor of angiogenesis upon induction of wild type p53 expression in glioblastoma cells

Erwin G. Van Meir, W. Cavenee, H. Huang, 1994, Nature Genetics.

Advances in Brief The Phosphoinositol Phosphatase Activity of PTEN Mediates a Serum-sensitive Gl Growth Arrest in Glioma Cells

F. Furnari, H. Huang, Webster, 2006 .

Enhanced Tumorigenic Behavior of Glioblastoma Cells Expressing a Truncated Epidermal Growth Factor Receptor Is Mediated through the Ras-Shc-Grb2 Pathway*

J R Feramisco, S A Prigent, W. Cavenee, 1996, The Journal of Biological Chemistry.

Expression and distribution of vascular endothelial growth factor protein in human brain tumors

A. von Deimling, O. Wiestler, W. Cavenee, 1997, Acta Neuropathologica.

Expression of vascular endothelial growth factor in human brain tumors

W. Cavenee, H. Huang, R. Nishikawa, 1998, Acta Neuropathologica.

The Protein Tyrosine Phosphatase TCPTP Suppresses the Tumorigenicity of Glioblastoma Cells Expressing a Mutant Epidermal Growth Factor Receptor*

W. Cavenee, F. Furnari, H. Huang, 2001, The Journal of Biological Chemistry.

A mutant epidermal growth factor receptor common in human glioma confers enhanced tumorigenicity.

W. Cavenee, H. Huang, X. Ji, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Silencing of p16/CDKN2 expression in human gliomas by methylation and chromatin condensation.

M. Berger, J. Costello, W. Cavenee, 1996, Cancer research.

Expression of the Ets-1 transcription factor in human astrocytomas is associated with Fms-like tyrosine kinase-1 (Flt-1)/vascular endothelial growth factor receptor-1 synthesis and neoangiogenesis.

O. Wiestler, W. Cavenee, H. Huang, 1999, Cancer research.

Drug resistance of human glioblastoma cells conferred by a tumor-specific mutant epidermal growth factor receptor through modulation of Bcl-XL and caspase-3-like proteases.

W. Cavenee, M. Nagane, H. Huang, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Monoclonal antibody 806 inhibits the growth of tumor xenografts expressing either the de2-7 or amplified epidermal growth factor receptor (EGFR) but not wild-type EGFR.

A. Scott, W. Cavenee, A. Burgess, 2001, Cancer research.

Growth suppression of glioma cells by PTEN requires a functional phosphatase catalytic domain.

W. Cavenee, F. Furnari, H. Huang, 1997, Proceedings of the National Academy of Sciences of the United States of America.

A common mutant epidermal growth factor receptor confers enhanced tumorigenicity on human glioblastoma cells by increasing proliferation and reducing apoptosis.

O. Bogler, W. Cavenee, M. Nagane, 1996, Cancer research.

Induction of vascular endothelial growth factor expression in endothelial cells by platelet-derived growth factor through the activation of phosphatidylinositol 3-kinase.

W. Cavenee, H. Huang, D. Wang, 1999, Cancer research.

Suppression of glioblastoma angiogenicity and tumorigenicity by inhibition of endogenous expression of vascular endothelial growth factor.

W. Cavenee, M. Nagane, S. Cheng, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Intracerebral tumor-associated hemorrhage caused by overexpression of the vascular endothelial growth factor isoforms VEGF121 and VEGF165 but not VEGF189.

W. Cavenee, M. Nagane, S. Cheng, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Tyrphostin AG 1478 preferentially inhibits human glioma cells expressing truncated rather than wild-type epidermal growth factor receptors.

W. Cavenee, H. Huang, H. Huang, 1996, Cancer research.

The phosphoinositol phosphatase activity of PTEN mediates a serum-sensitive G1 growth arrest in glioma cells.

W. Cavenee, F. Furnari, H. Huang, 1998, Cancer research.

A peptide derived from the non-receptor-binding region of urokinase plasminogen activator inhibits glioblastoma growth and angiogenesis in vivo in combination with cisplatin.

A. Gown, M. Skelly, W. Cavenee, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Growth suppression of intracranial xenografted glioblastomas overexpressing mutant epidermal growth factor receptors by systemic administration of monoclonal antibody (mAb) 806, a novel monoclonal antibody directed to the receptor.

A. Scott, W. Cavenee, H. Huang, 2001, Cancer research.

Increased death receptor 5 expression by chemotherapeutic agents in human gliomas causes synergistic cytotoxicity with tumor necrosis factor-related apoptosis-inducing ligand in vitro and in vivo.

W. Cavenee, M. Nagane, H. Huang, 2000, Cancer research.

SETA: a novel SH3 domain-containing adapter molecule associated with malignancy in astrocytes.

W. Cavenee, F. Furnari, H. Huang, 2000, Neuro-oncology.

The chromosome 10 monosomy common in human melanomas results from loss of two separate tumor suppressor loci.

W. Cavenee, M. Nagane, H. Huang, 1999, Cancer research.

Malignant transformation of p53-deficient astrocytes is modulated by environmental cues in vitro.

W. Cavenee, M. Nagane, H. Huang, 1999, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Loss of wild-type p53 bestows a growth advantage on primary cortical astrocytes and facilitates their in vitro transformation.

W. Cavenee, H. Huang, H. Huang, 1995, Cancer research.