C. Lendon

发表

A common biological mechanism in cancer and Alzheimer's disease?

C. Lendon, C. Roe, M. Behrens, 2009, Current Alzheimer research.

The angiotensin 1-converting enzyme insertion (I)/deletion (D) polymorphism does not influence the extent of amyloid or tau pathology in patients with sporadic Alzheimer's disease

D. Mann, J. Lambert, T. Iwatsubo, 2002, Neuroscience Letters.

A polymorphism within intron 11 of the tau gene is not increased in frequency in patients with sporadic Alzheimer's disease, nor does it influence the extent of tau pathology in the brain

E. Green, J. Lambert, T. Iwatsubo, 2002, Neuroscience Letters.

The E280A presenilin 1 Alzheimer mutation produces increased Aβ42 deposition and severe cerebellar pathology

Kenneth S. Kosik, Francisco Lopera, Takaomi C. Saido, 1996, Nature Medicine.

Clinical features of early-onset Alzheimer disease in a large kindred with an E280A presenilin-1 mutation.

A. Ruiz-Linares, A. Goate, J. Norton, 1997, JAMA.

Human leucocyte antigen-A2 increases risk of Alzheimer's disease but does not affect age of onset in a Scottish population

D. Clair, N. Craddock, C. Lendon, 2000, Neuroscience Letters.

Clinical Features of Early-Onset Alzheimer Disease in a Large Kindred With an E280A Presenilin-1 Mutation

A. Ruiz-Linares, A. Goate, K. Kosik, 1997 .

Association study of Notch 4 polymorphisms with Alzheimer’s disease

D. Mann, J. Lambert, P. Amouyel, 2004, Journal of Neurology, Neurosurgery & Psychiatry.

A polymorphic variation in the interleukin 1A gene increases brain microglial cell activity in Alzheimer’s disease

E. Green, D. Mann, J. Lambert, 2004, Journal of Neurology, Neurosurgery & Psychiatry.

The allelic modulation of apolipoprotein E expression by oestrogen: potential relevance for Alzheimer’s disease

J. Lambert, C. Lendon, N. Coyle, 2004, Journal of Medical Genetics.

Tau load is associated with apolipoprotein E genotype and the amount of amyloid β protein, Aβ40, in sporadic and familial Alzheimer's disease

T. Iwatsubo, C. Lendon, S. Pickering-Brown, 2003, Neuropathology and applied neurobiology.

Association of 3′-UTR polymorphisms of the oxidised LDL receptor 1 (OLR1) gene with Alzheimer’s disease

S. DeKosky, F. Pasquier, D. Mann, 2003, Journal of medical genetics.

Pronounced impact of Th1/E47cs mutation compared with -491 AT mutation on neural APOE gene expression and risk of developing Alzheimer's disease.

J. Hardy, A. Delacourte, D. Galasko, 1998, Human molecular genetics.

Is there a relation between APOE expression and brain amyloid load in Alzheimer’s disease?

D. Mann, J. Lambert, P. Amouyel, 2005, Journal of Neurology, Neurosurgery & Psychiatry.

Association study of three polymorphisms of TGF-β1 gene with Alzheimer's disease

D. Mann, J. Lambert, P. Amouyel, 2002, Journal of neurology, neurosurgery, and psychiatry.

Association between apolipoprotein E e4 allele and arteriosclerosis, cerebral amyloid angiopathy, and cerebral white matter damage in Alzheimer’s disease

D. Mann, C. Lendon, S. Pickering-Brown, 2004, Journal of Neurology, Neurosurgery & Psychiatry.

A polymorphism in the angiotensin 1-converting enzyme gene is associated with damage to cerebral cortical white matter in Alzheimer's disease

D. Mann, J. Lambert, C. Lendon, 2004, Neuroscience Letters.

α-T-catenin is expressed in human brain and interacts with the Wnt signaling pathway but is not responsible for linkage to chromosome 10 in Alzheimer’s disease

M. Owen, J. Morris, P. Holmans, 2007, NeuroMolecular Medicine.

Parkinson ’ s Disease α-Catenin and Wnt Signaling Ox-Phose Genes and Alzheimer ’ s Disease NO and Tardive Dyskinesia Homocysteine and Oxyradical-Induced Cognitive Impairment

M. Owen, J. Morris, P. Holmans, 2004 .

Alpha-T-catenin is expressed in human brain and interacts with the Wnt signaling pathway but is not responsible for linkage to chromosome 10 in Alzheimer's disease.

M. Owen, P. Holmans, A. Myers, 2004, Neuromolecular medicine.