D. Fage

发表

Neurochemical characteristics of amisulpride, an atypical dopamine D2/D3 receptor antagonist with both presynaptic and limbic selectivity.

K. Chergui, F. Gonon, H. Schoemaker, 1997, The Journal of pharmacology and experimental therapeutics.

Neuronal versus endothelial origin of vasoactive acetylcholine in pial vessels

E. Mackenzie, E. Hamel, D. Fage, 1987, Brain Research.

The pharmacology of native N-methtl-D-aspartate receptor subtypes: Different receptors control the release of different striatal and spinal transmitters

C. Carter, D. Fage, M. Nankai, 1998, Progress in Neuro-Psychopharmacology and Biological Psychiatry.

Opposing effects of D-1 and D-2 receptor antagonists on acetylcholine levels in the rat striatum.

D. Fage, B. Scatton, 1986, European journal of pharmacology.

The effects ofN-methyl-d-aspartate and kainate lesions of the rat striatum on striatal ornithine decar☐ylase activity and polyamine levels

C. Carter, D. Fage, B. Scatton, 1991, Brain Research.

Local infusion of interleukin-6 attenuates the neurotoxic effects of NMDA on rat striatal cholinergic neurons

D. Fage, J. Bénavidès, X. Vigé, 1992, Neuroscience Letters.

Hypophysectomy fails to affect the supersensitivity of striatal dopamine target cells induced by prolonged haloperidol treatment.

D. Fage, B. Scatton, 1984, European journal of pharmacology.

Influence of lithium on biochemical manifestations of striatal dopamine target cell supersensitivity induced by prolonged haloperidol treatment.

D. Fage, A. Oblin, B. Scatton, 1983, European journal of pharmacology.

Release of spermidine from the rat cortex following permanent middle cerebral artery occlusion

C. Carter, D. Fage, B. Scatton, 1995, Fundamental & clinical pharmacology.

Implication of the polyamines in the neurotoxic effects of N-methyl-D-aspartate.

C. Carter, D. Fage, J. Bénavidès, 1992, Neurological research.

Neurotoxic effects of the intrastriatal injection of spermine and spermidine: lack of involvement of NMDA receptors

C. Carter, D. Fage, B. Scatton, 1992, Brain Research.

Peripheral type benzodiazepine binding sites are a sensitive indirect index of neuronal damage

C. Carter, D. Fage, J. Bénavidès, 1987, Brain Research.

Difluoromethyl ornithine protects against the neurotoxic effects of intrastriatally administered N-methyl-D-aspartate in vivo.

C. Carter, D. Fage, B. Scatton, 1991, European journal of pharmacology.

Time course of the changes in striatal acetylcholine levels induced by pergolide and haloperidol after lesion of the nigrostriatal dopaminergic pathways in the rat

C. Feuerstein, D. Fage, B. Scatton, 1984, Brain Research.

Effects of cyclo (Leu-Gly) on neurochemical indices of striatal dopaminergic supersensitivity induced by prolonged haloperidol treatment.

D. Fage, B. Scatton, C. Le Douarin, 1984, Life sciences.

Ornithine decarboxylase inhibition or NMDA receptor antagonism reduce cortical polyamine efflux associated with dialysis probe implantation

C. Carter, D. Fage, B. Scatton, 1993, Neuroscience Letters.

Selective Release of Spermine and Spermidine from the Rat Striatum by N‐Methyl‐d‐Aspartate Receptor Activation In Vivo

C. Carter, D. Fage, B. Scatton, 1992, Journal of neurochemistry.

Striatal NMDA receptor subtypes: the pharmacology of N-methyl-D-aspartate-evoked dopamine, gamma-aminobutyric acid, acetylcholine and spermidine release.

C. Carter, D. Fage, M. Nankai, 1995, European journal of pharmacology.

Excitatory Amino Acid Influence on Striatal Cholinergic Transmission

D. Fage, B. Scatton, 1986 .

Evidence for Differing Origins of the Serotonergic Innervation of Major Cerebral Arteries and Small Pial Vessels in the Rat

E. Mackenzie, G. Bonvento, D. Fage, 1991, Journal of neurochemistry.

2-Oxo-[14C]glutarate is taken up by glutamatergic nerve terminals in the rat striatum

M. Savasta, C. Carter, D. Fage, 1986, Neuroscience Letters.

Neurochemical studies on the existence, origin and characteristics of the serotonergic innervation of small pial vessels

D. Duverger, E. Mackenzie, D. Fage, 1985, Brain Research.

Effects of amisulpride, an atypical antipsychotic which blocks preferentially presynaptic dopamine autoreceptors, on integrated functional cerebral activity in the rat

D. Fage, B. Scatton, A. Cudennec, 1997, Brain Research.

Inhibition of synaptosomal veratridine-induced sodium influx by antidepressants and neuroleptics used in chronic pain

C. Carter, D. Fage, A. Deffois, 1996, Neuroscience Letters.

Improved Method for Determination of Acetylcholine, Choline, and Other Biogenic Amines in a Single Brain Tissue Sample Using High Performance Liquid Chromatography and Electrochemical Detection

D. Fage, A. Zekhnini, A. Beley, 1987 .

Evidence for native NMDA receptor subtype pharmacology as revealed by differential effects on the NMDA-evoked release of striatal neuromodulators: Eliprodil, ifenprodil and other native NMDA receptor subtype selective compounds

C. Carter, D. Fage, M. Nankai, 1996, Neurochemistry International.

NMDA Receptor Subtype Selectivity: Eliprodil, Polyamine Spider Toxins, Dextromethorphan, and Desipramine Selectively Block NMDA‐Evoked Striatal Acetylcholine but Not Spermidine Release

C. Carter, D. Fage, M. Nankai, 1995, Journal of neurochemistry.

Pharmacology of N-methyl-D-aspartate-evoked [3H]noradrenaline release in adult rat spinal cord.

C. Carter, D. Fage, M. Klarica, 1996, European journal of pharmacology.

Relative selectivity of 6,7-dihydroxy-2-dimethylaminotetralin, N-n-propyl-3-(3-hydroxyphenyl)piperidine, N-n-propylnorapomorphine and pergolide as agonists at striatal dopamine autoreceptors and postsynaptic dopamine receptors.

Y. Claustre, D. Fage, B. Scatton, 1985, The Journal of pharmacology and experimental therapeutics.

Small pial vessels, but not choroid plexus, exhibit specific biochemical correlates of functional cholinergic innervation

E. Mackenzie, E. Hamel, D. Fage, 1990, Brain Research.

The quantification of brain lesions with anω 3 site ligand: a critical analysis of animal models of cerebral ischaemia and neurodegeneration

D. Duverger, E. Mackenzie, D. Fage, 1990, Brain Research.

Evidence for, and nature of, the tonic inhibitory influence of habenulointerpeduncular pathways upon cerebral dopaminergic transmission in the rat

D. Fage, B. Scatton, T. Nishikawa, 1986, Brain Research.

Quantitative autoradiography of 3H-nomifensine binding sites in rat brain.

M. Dubocovich, D. Fage, B. Scatton, 1985, Life sciences.

Influence of GABA mimetics and lithium on biochemical manifestations of striatal dopamine target cell hypersensitivity.

D. Fage, A. Oblin, B. Scatton, 1985, Psychopharmacology. Supplementum.

Imaging of primary and remote ischaemic and excitotoxic brain lesions. An autoradiographic study of peripheral type benzodiazepine binding sites in the rat and cat

D. Duverger, E. Mackenzie, D. Fage, 1988, Brain Research.

Cortical ablation fails to influence striatal dopamine target cell supersensitivity induced by nigrostriatal denervation in the rat

C. Feuerstein, D. Fage, B. Scatton, 1987, Brain Research.

Recent findings in central dopaminergic and cholinergic neurotransmission of roman high- and low-avoidance (RHA/Verh and RLA/Verh) rats

Y. Claustre, D. Fage, B. Scatton, 1987, Behavioural Brain Research.

Decrease in both choline acetyltransferase activity and EEG patterns in the hippocampal formation of the rat following septal macroelectrode implantation

J. Delacour, D. Fage, B. Scatton, 1984, Brain Research.

Synergism between the NMDA receptor antagonistic effects of ifenprodil and the glycine antagonist, 7-chlorokynurenate, in vivo.

C. Carter, D. Fage, C. Voltz, 1994, European journal of pharmacology.

Monoamine abnormalities in the brain of scrapie-infected rats

D. Fage, B. Scatton, M. Bassant, 1984, Brain Research.