Ching Shang

发表

A long non-coding RNA protects the heart from pathological hypertrophy

E. Ashley, Weihong Xu, D. Bernstein, 2014, Nature.

Pbx/Meis Deficiencies Demonstrate Multigenetic Origins of Congenital Heart Disease

K. Stankunas, N. Copeland, N. Jenkins, 2008, Circulation research.

A COMMUNITY OVERLAP STRATEGY REVEALS CENTRAL GENES AND NETWORKS IN HEART FAILURE AUTHORS

Kevin S. Smith, Kevin Scott Smith, Mingyao Li, 2016 .

Pbx1 functions in distinct regulatory networks to pattern the great arteries and cardiac outflow tract

K. Stankunas, M. Cleary, Ching Shang, 2008, Development.

Systems Genomics Identifies a Key Role for Hypocretin/Orexin Receptor-2 in Human Heart Failure.

Clint L. Miller, D. Absher, R. Myers, 2015, Journal of the American College of Cardiology.

Characterization of the mechanism of action of lanraplenib, a novel spleen tyrosine kinase inhibitor, in models of lupus nephritis

Ching Shang, A. Clarke, K. Currie, 2020, BMC Rheumatology.

Pathologic gene network rewiring implicates PPP1R3A as a central cardioprotective factor in pressure overload heart failure

Kevin S. Smith, H. Hakonarson, Mingyao Li, 2016, bioRxiv.

Pbx1 activates Fgf10 in the mesenchyme of developing lungs

Pei Han, Wei Li, Ching Shang, 2014, Genesis.

Title Pbx 1 activates Fgf 10 in the mesenchyme of developing lungs

Pei Han, Wei Li, Ching Shang, 2014 .

Epicardial calcineurin-NFAT signals through Smad2 to direct coronary smooth muscle cell and arterial wall development.

T. Quertermous, Pei Han, Wei Li, 2014, Cardiovascular research.

Silencing of MYH7 ameliorates disease phenotypes in human iPSC-Cardiomyocytes.

E. Ashley, M. Wheeler, B. Pruitt, 2020, Physiological genomics.

Dissociation of disease phenotype and allele silencing in hypertrophic cardiomyopathy

E. Ashley, M. Wheeler, B. Pruitt, 2019, bioRxiv.

Chromatin regulation by Brg1 underlies heart muscle development and disease

E. Ashley, Pei Han, Jin Yang, 2010, Nature.

Epigenetic response to environmental stress: Assembly of BRG1-G9a/GLP-DNMT3 repressive chromatin complex on Myh6 promoter in pathologically stressed hearts.

Dean Y. Li, D. Bernstein, Chiou-Hong Lin, 2016, Biochimica et biophysica acta.

Chromatin Remodeling in Heart Failure

Pei Han, Jin Yang, Ching Shang, 2016 .

Targeting LOXL2 for cardiac interstitial fibrosis and heart failure treatment

T. Quertermous, Peng-Sheng Chen, Jin Yang, 2016, Nature Communications.

Targeting LOXL 2 for cardiac interstitial fibrosis and heart failure treatment

T. Quertermous, Peng-Sheng Chen, Jin Yang, 2016 .

Pathologic gene network rewiring implicates PPP1R3A as a central regulator in pressure overload heart failure

Michael J. Gloudemans, Kevin S. Smith, H. Hakonarson, 2019, Nature Communications.

Apelin and APJ orchestrate complex tissue-specific control of cardiomyocyte hypertrophy and contractility in the hypertrophy-heart failure transition.

P. Tsao, E. Ashley, D. Bernstein, 2018, American journal of physiology. Heart and circulatory physiology.

Chromatin regulation by Brg1 underlies heart muscle development and disease

E. Ashley, Pei Han, Jin Yang, 2010, Nature.

Allele-Specific Silencing Ameliorates Restrictive Cardiomyopathy Due to a Human Myosin Regulatory Light Chain Mutation.

Nikhil A. Jain, E. Ashley, M. Wheeler, 2019, Circulation.

Allele-specific silencing ameliorates restrictive cardiomyopathy due to a human myosin regulatory light chain mutation

Nikhil A. Jain, E. Ashley, M. Wheeler, 2019, bioRxiv.

Endocardial Brg1 represses ADAMTS1 to maintain the microenvironment for myocardial morphogenesis.

K. Stankunas, Jiang I. Wu, Ching Shang, 2008, Developmental cell.

Brg1 governs distinct pathways to direct multiple aspects of mammalian neural crest cell development

K. Stankunas, D. Bernstein, T. Meyer, 2013, Proceedings of the National Academy of Sciences.

Pathological Ace2-to-Ace enzyme switch in the stressed heart is transcriptionally controlled by the endothelial Brg1–FoxM1 complex

Chiou-Hong Lin, T. Quertermous, Pei Han, 2016, Proceedings of the National Academy of Sciences.

Brg1 governs a positive feedback circuit in the hair follicle for tissue regeneration and repair.

K. Stankunas, M. Longaker, Pei Han, 2013, Developmental cell.

In Vivo Post–Cardiac Arrest Myocardial Dysfunction Is Supported by Ca2+/Calmodulin-Dependent Protein Kinase II–Mediated Calcium Long-Term Potentiation and Mitigated by Alda-1, an Agonist of Aldehyde Dehydrogenase Type 2

E. Ashley, P. Kohl, Z. Ali, 2016, Circulation.

In vivo Post-cardiac arrest myocardial dysfunction is supported by CaMKII-mediated calcium long-term potentiation and mitigated by Alda-1 , an Agonist of Aldehyde Dehydrogenase Type 2 Running Title : Post-cardiac arrest myocardial dysfunction

Daria, E. Ashley, P. Kohl, 2016 .