Radmila Terentyeva

发表

Abnormal intrastore calcium signaling in chronic heart failure.

Z. Kubalová, D. Terentyev, S. Viatchenko‐Karpinski, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Pyridostigmine improves cardiac function and rhythmicity through RyR2 stabilization and inhibition of STIM1‐mediated calcium entry in heart failure

D. Terentyev, Radmila Terentyeva, C. Carnes, 2021, Journal of cellular and molecular medicine.

miR-1 Overexpression Enhances Ca 2 (cid:1) Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56 (cid:2) and Causing CaMKII-Dependent Hyperphosphorylation of RyR2

T. Elton, D. Terentyev, Radmila Terentyeva, 2009 .

Redox modification of ryanodine receptors by mitochondria‐derived reactive oxygen species contributes to aberrant Ca2+ handling in ageing rabbit hearts

D. Terentyev, Radmila Terentyeva, G. Koren, 2013, The Journal of physiology.

Redox Modification of Ryanodine Receptors Contributes to Sarcoplasmic Reticulum Ca2+ Leak in Chronic Heart Failure

Dmitry Terentyev, Savita Khanna, Chandan K Sen, 2008, Circulation research.

SK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR

Tae Yun Kim, D. Terentyev, Radmila Terentyeva, 2017, Cardiovascular research.

Cellular Biology Shortened Ca Signaling Refractoriness Underlies Cellular Arrhythmogenesis in a Postinfarction Model of Sudden Cardiac Death

G. Billman, D. Terentyev, Radmila Terentyeva, 2012 .

Rectification on Cardiac Repolarization

Tae Yun Kim, P. Bronk, D. Terentyev, 2022 .

Interleukin-1β, Oxidative Stress, and Abnormal Calcium Handling Mediate Diabetic Arrhythmic Risk

D. Terentyev, Radmila Terentyeva, S. Dudley, 2021, JACC. Basic to translational science.

Increased RyR2 activity is exacerbated by calcium leak-induced mitochondrial ROS

D. Terentyev, Radmila Terentyeva, S. Györke, 2020, Basic Research in Cardiology.

Sarcoplasmic reticulum-mitochondria communication; implications for cardiac arrhythmia.

D. Terentyev, Radmila Terentyeva, R. Clements, 2021, Journal of molecular and cellular cardiology.

MicroRNA-1 and -133 Increase Arrhythmogenesis in Heart Failure by Dissociating Phosphatase Activity from RyR2 Complex

Dmitry Terentyev, Cynthia A. Carnes, Sandor Györke, 2011, PloS one.

Endurance exercise training normalizes repolarization and calcium-handling abnormalities, preventing ventricular fibrillation in a model of sudden cardiac death.

G. Billman, B. Liu, D. Terentyev, 2012, Journal of applied physiology.

miR-1 Overexpression Enhances Ca2+ Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2

Dmitry Terentyev, Sandor Györke, T. Elton, 2009, Circulation research.

Hyperphosphorylation of RyRs Underlies Triggered Activity in Transgenic Rabbit Model of LQT2 Syndrome

A. Karma, D. Terentyev, Radmila Terentyeva, 2014, Circulation research.

Tetrahydrobiopterin depletion and NOS2 uncoupling contribute to heart failure-induced alterations in atrial electrophysiology.

J. Zweier, T. Elton, D. Terentyev, 2011, Cardiovascular research.

Chronic heart failure and the substrate for atrial fibrillation.

T. Nakayama, D. Terentyev, Y. Nishijima, 2009, Cardiovascular research.

Dietary Omega-3 Fatty Acids Promote Arrhythmogenic Remodeling of Cellular Ca2+ Handling in a Postinfarction Model of Sudden Cardiac Death

G. Billman, D. Terentyev, Radmila Terentyeva, 2013, PloS one.

Hyperphosphorylation of RyR 2 and Causing CaMKII-Dependent α by Targeting PP 2 A Regulatory Subunit B 56 Release and Promotes Cardiac Arrhythmogenesis 2 + Overexpression Enhances

T. Elton, Radmila Terentyeva, D. Feldman, 2009 .

Modulation of SR Ca release by luminal Ca and calsequestrin in cardiac myocytes: effects of CASQ2 mutations linked to sudden cardiac death.

Dmitry Terentyev, Donald M Bers, Serge Viatchenko-Karpinski, 2008, Biophysical journal.

Abnormal Interactions of Calsequestrin With the Ryanodine Receptor Calcium Release Channel Complex Linked to Exercise-Induced Sudden Cardiac Death

S. Priori, C. Napolitano, N. Blom, 2006, Circulation research.

Triadin Overexpression Stimulates Excitation-Contraction Coupling and Increases Predisposition to Cellular Arrhythmia in Cardiac Myocytes

D. Terentyev, S. Viatchenko‐Karpinski, Radmila Terentyeva, 2005, Circulation research.

Modulation of cytosolic and intra‐sarcoplasmic reticulum calcium waves by calsequestrin in rat cardiac myocytes

Z. Kubalová, D. Terentyev, S. Viatchenko‐Karpinski, 2004, The Journal of physiology.

Abnormal Calcium Signaling and Sudden Cardiac Death Associated With Mutation of Calsequestrin

S. Priori, C. Napolitano, D. Terentyev, 2004, Circulation research.

Protein Phosphatases Decrease Sarcoplasmic Reticulum Calcium Content by Stimulating Calcium Release in Cardiac Myocytes

D. Terentyev, S. Viatchenko‐Karpinski, Radmila Terentyeva, 2003, The Journal of physiology.

Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.

D. Terentyev, Radmila Terentyeva, G. Koren, 2014, American journal of physiology. Heart and circulatory physiology.

Pharmacological Modulation of Mitochondrial Ca2+ Content Regulates Sarcoplasmic Reticulum Ca2+ Release via Oxidation of the Ryanodine Receptor by Mitochondria-Derived Reactive Oxygen Species

Tae Yun Kim, P. Bronk, D. Terentyev, 2018, Front. Physiol..

Ero1α-Dependent ERp44 Dissociation From RyR2 Contributes to Cardiac Arrhythmia

Tae Yun Kim, Jonathan P. Davis, Xun Ai, 2022, Circulation research.

Redox modification of ryanodine receptors underlies calcium alternans in a canine model of sudden cardiac death.

Dmitry Terentyev, Serge Viatchenko-Karpinski, Arun Sridhar, 2009, Cardiovascular research.

Sexual dimorphism in bidirectional SR-mitochondria crosstalk in ventricular cardiomyocytes

Beth S. Lee, D. Terentyev, Radmila Terentyeva, 2023, Basic Research in Cardiology.

MCU overexpression evokes disparate dose-dependent effects on mito-ROS and spontaneous Ca2+ release in hypertrophic rat cardiomyocytes.

D. Terentyev, Radmila Terentyeva, S. Györke, 2021, American journal of physiology. Heart and circulatory physiology.

Shortened Ca2+ Signaling Refractoriness Underlies Cellular Arrhythmogenesis in a Postinfarction Model of Sudden Cardiac Death

G. Billman, D. Terentyev, Radmila Terentyeva, 2012, Circulation research.

PKA phosphorylation underlies functional recruitment of sarcolemmal SK2 channels in ventricular myocytes from hypertrophic hearts

Tae Yun Kim, P. Bronk, D. Terentyev, 2019, The Journal of physiology.

Impact of ISK Voltage and Ca2+/Mg2+-Dependent Rectification on Cardiac Repolarization.

Tae Yun Kim, P. Bronk, D. Terentyev, 2020, Biophysical journal.

NaV1.6 dysregulation within myocardial T-tubules by D96V calmodulin enhances proarrhythmic sodium and calcium mishandling

Jonathan P. Davis, D. Terentyev, Radmila Terentyeva, 2023, The Journal of clinical investigation.

The role of spatial organization of Ca2+ release sites in the generation of arrhythmogenic diastolic Ca2+ release in myocytes from failing hearts

D. Terentyev, Radmila Terentyeva, C. Carnes, 2017, Basic Research in Cardiology.

Repolarization abnormalities and afterdepolarizations in a canine model of sudden cardiac death.

G. Billman, D. Terentyev, Y. Nishijima, 2008, American journal of physiology. Regulatory, integrative and comparative physiology.

fibrillationin animals susceptible to ventricular submaximal onset and heart rate recovery responses to Effect of endurance exercise training on heart rate

J. Seidman, G. Billman, C. Seidman, 2015 .

Arrhythmogenic adverse effects of cardiac glycosides are mediated by redox modification of ryanodine receptors

D. Terentyev, Radmila Terentyeva, C. Carnes, 2010, The Journal of physiology.

Transient Outward K+ Current (Ito) Underlies the Right Ventricular Initiation of Polymorphic Ventricular Tachycardia in a Transgenic Rabbit Model of Long-QT Syndrome Type 1

Tae Yun Kim, A. Karma, Z. Qu, 2018, Circulation. Arrhythmia and electrophysiology.

Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.

D. Terentyev, Radmila Terentyeva, G. Koren, 2014, American journal of physiology. Heart and circulatory physiology.

The relationship between arrhythmogenesis and impaired contractility in heart failure: role of altered ryanodine receptor function.

D. Terentyev, Y. Nishijima, Radmila Terentyeva, 2011, Cardiovascular research.

Hyperphosphorylation of RyRs Underlies Triggered Activity in Transgenic Rabbit Model of LQT2 Syndrome

A. Karma, D. Terentyev, Radmila Terentyeva, 2014, Circulation research.

Shortened Ca 2 (cid:1) Signaling Refractoriness Underlies Cellular Arrhythmogenesis in a Postinfarction Model of Sudden Cardiac Death

G. Billman, D. Terentyev, Radmila Terentyeva, 2012 .

Abstract 18186: Arrhythmogenic Adverse Effects of Cardiac Glycosides are Mediated by Redox Modification of Ryanodine Receptors

D. Terentyev, Radmila Terentyeva, C. Carnes, 2010 .

STIM1 ablation impairs exercise-induced physiologic cardiac hypertrophy and dysregulates autophagy in mouse hearts.

Jonathan P. Davis, D. Terentyev, Radmila Terentyeva, 2023, Journal of applied physiology.

Maternal exercise preserves offspring cardiovascular health via oxidative regulation of the ryanodine receptor

D. Terentyev, Radmila Terentyeva, D. Nassal, 2024, Molecular metabolism.