A. Ben-nun

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Spontaneous remission of autoimmune encephalomyelitis is inhibited by splenectomy, thymectomy or ageing

I. Cohen, Y. Ron, A. Ben-nun, 1980, Nature.

The rapid isolation of clonable antigen‐specific T lymphocyte lines capable of mediating autoimmune encephalomyelitis

I. Cohen, A. Ben-nun, H. Wekerle, 1981, European journal of immunology.

Experimental autoimmune encephalomyelitis (EAE) mediated by T cell lines: process of selection of lines and characterization of the cells.

I. Cohen, A. Ben-nun, 1982, Journal of immunology.

Vaccination against autoimmune disease with lines of autoimmune T lymphocytes.

I. Cohen, A. Ben-nun, J. Holoshitz, 1983, Immunology today.

Lines of T lymphocytes induce or vaccinate against autoimmune arthritis.

I. Cohen, A. Ben-nun, Y. Naparstek, 1983, Science.

Bacterial agents protect against autoimmune disease. I. Mice pre-exposed to Bordetella pertussis or Mycobacterium tuberculosis are highly refractory to induction of experimental autoimmune encephalomyelitis.

A. Ben-nun, D. Lehmann, 1992, Journal of autoimmunity.

Sclerosis-Associated HLA-DRB1*1501 and Predicted Epitope(s) for Multiple Encephalitogenic Epitope(s) for SJL/J Mice Encompasses the Immunodominant Major 36 - Basic Protein Region MOBP15 The Myelin-Associated Oligodendrocytic

M. Eisenstein, H. Lassmann, A. Ben-nun, 2004 .

A novel antigen-toxin chimeric protein: myelin basic protein-pseudomonas exotoxin (MBP-PE 40) for treatment of experimental autoimmune encephalomyelitis.

A. Ben-nun, E. Béraud, D. Soffer, 1999, Immunology letters.

Effect of the bm12 class II mutation on proliferative and cytokine responses of encephalitogenic T cells in myelin oligodendrocyte glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis.

A. Ben-nun, N. Kerlero de Rosbo, H. Gur, 1999, Journal of autoimmunity.

Prevention of experimental autoimmune encephalomyelitis and induction of tolerance with acute immunosuppression followed by syngeneic bone marrow transplantation.

A. Ben-nun, D. Karussis, D. Lehmann, 1992, Journal of immunology.

Chronic-relapsing experimental autoimmune encephalomyelitis (CR-EAE): treatment and induction of tolerance, with high dose cyclophosphamide followed by syngeneic bone marrow transplantation

A. Ben-nun, D. Karussis, D. Lehmann, 1992, Journal of Neuroimmunology.

‘Multi-Epitope-Targeted’ Immune-Specific Therapy for a Multiple Sclerosis-Like Disease via Engineered Multi-Epitope Protein Is Superior to Peptides

A. Ben-nun, R. Zilkha-Falb, N. Kerlero de Rosbo, 2011, PloS one.

Protection against autoimmune disease by bacterial agents. II. PPD and pertussis toxin as proteins active in protecting mice against experimental autoimmune encephalomyelitis

A. Ben-nun, D. Lehmann, S. Yossefi, 1993, European journal of immunology.

Pathogenic and non-pathogenic T lymphocytes specific for the encephalitogenic epitope of myelin basic protein: Functional characteristics and vaccination properties

A. Ben-nun, E. Béraud, A. J. Zamora, 1993, Journal of Neuroimmunology.

DQB1*06:02-Associated Pathogenic Anti-Myelin Autoimmunity in Multiple Sclerosis-Like Disease: Potential Function of DQB1*06:02 as a Disease-Predisposing Allele

A. Ben-nun, N. Kaushansky, 2014, Front. Oncol..

Activation and control of pathogenic T cells in OSP/claudin-11-induced EAE in SJL/J mice are dominated by their focused recognition of a single epitopic residue (OSP58M).

M. Eisenstein, A. Ben-nun, N. Kaushansky, 2008, International immunology.

Pathogenic T cells in MOBP‐induced murine EAE are predominantly focused to recognition of MOBP21F and MOBP27P epitopic residues

M. Eisenstein, A. Ben-nun, R. Zilkha-Falb, 2007, European journal of immunology.

Thyroglobulin-induced murine thyroiditis assessed by intrathyroidal T cell receptor sequencing.

P. Unger, A. Ben-nun, T. Davies, 1994, Journal of immunology.

Differential recognition of MBP epitopes in BALB/c mice determines the site of inflammatory disease induction

R. Bronson, A. Ben-nun, M. Dorf, 1998, Journal of Neuroimmunology.

An Ia-positive mouse T-cell clone is functional in presenting antigen to other T cells

J. Seidman, L. Glimcher, C. Murre, 2004, Immunogenetics.

Dichotomy between the T and the B cell epitopes of the synthetic polypeptide (T,G)‐A–L

M. Sela, A. Ben-nun, E. Mozes, 1994, European journal of immunology.

Generation of HumanThyroid Peroxidase-reactive T Cells and their T cell Receptor Repertoire

Ronald, Terry, David, 2013 .

Generation of Human Thyroid Peroxidase-reactive T Cells and their T cell Receptor Repertoire

A. Ben-nun, T. Davies, R. Magnusson, 2022 .

T cell receptor repertoire in the peripheral blood and intestinal mucosa of coeliac patients

A. Ben-nun, A. Lerner, N. Lahat, 1995, Clinical and experimental immunology.

Autoimmune T-lymphocytes with specificity for pancreatic islet antigens.

H. Kolb, I. Cohen, A. Ben-nun, 1985, Immunology letters.

The human CMV-UL86 peptide 981–1003 shares a crossreactive T-cell epitope with the encephalitogenic MOG peptide 34–56, but lacks the capacity to induce EAE in rhesus monkeys

A. Ben-nun, R. Hintzen, J. Laman, 2007, Journal of Neuroimmunology.

The myelin-associated oligodendrocytic basic protein (MOBP) as a relevant primary target autoantigen in multiple sclerosis.

M. Eisenstein, A. Ben-nun, R. Zilkha-Falb, 2010, Autoimmunity reviews.

EBI 2 Is Highly Expressed i n Multiple Sclerosis Lesions and Promotes Early CNS Migration of Encephalitogenic CD 4 T Cells

K. Rajewsky, A. Waisman, T. Kuhlmann, 2022 .

CCR2 Regulates the Immune Response by Modulating the Interconversion and Function of Effector and Regulatory T Cells

A. Ben-nun, M. Kramer, C. Thaiss, 2017, The Journal of Immunology.

Endogenous antigen presentation by autoantigen-transfected Epstein-Barr virus-lymphoblastoid cells. I. Generation of human thyroid peroxidase-reactive T cells and their T cell receptor repertoire.

A. Ben-nun, T. Davies, E. Concepcion, 1993, The Journal of clinical investigation.

Stable activity of diabetogenic cells with age in NOD mice: dynamics of reconstitution and adoptive diabetes transfer in immunocompromised mice

A. Ben-nun, S. Ash, K. Mizrahi, 2014, Immunology.

Polymorphism and complexity of the human DC and murine I‐A alpha chain genes.

C. Auffray, J. Seidman, R. Germain, 1983, The EMBO journal.

Cells in C57BL/6 Mice T Self-Antigens Is a Common Feature of CD4 Bispecificity for Myelin and Neuronal

M. Eisenstein, A. Sette, A. Ben-nun, 2014 .

Myelin-specific T cells also recognize neuronal autoantigen in a transgenic mouse model of multiple sclerosis

G. Krishnamoorthy, H. Lassmann, A. Ben-nun, 2009, Nature Medicine.

Aberrant T‐Cell Receptor Signalling of Interferon‐γ‐ and Tumour Necrosis Factor‐α‐Producing Cytotoxic CD8+ Vδ1/Vβ16 T Cells in a Patient with Chronic Neutropenia

A. Ben-nun, I. Bank, A. Kneller, 2003 .

Functional expression of a cloned I-A beta k gene in B-lymphoma cells.

J. Seidman, L. Glimcher, A. Ben-nun, 1984, Science.

Selection of human TcR V gene families in autologous mixed lymphocyte reactions: relevance to autoimmune immunopathology.

A. Ben-nun, T. Davies, N. Lahat, 1994, Autoimmunity.

T-cell receptor V region beta-chain gene expression in the autoimmune thyroiditis of non-obese diabetic mice.

A. Ben-nun, T. Davies, E. Concepcion, 1993, Journal of immunology.

T-cell receptor V gene use in autoimmune thyroid disease: direct assessment by thyroid aspiration.

A. Ben-nun, T. Davies, E. Concepcion, 1993, The Journal of clinical endocrinology and metabolism.

Evidence for selective accumulation of intrathyroidal T lymphocytes in human autoimmune thyroid disease based on T cell receptor V gene usage.

W. Cohen, A. Ben-nun, T. Davies, 1992, The Journal of clinical investigation.

Evidence of limited variability of antigen receptors on intrathyroidal T cells in autoimmune thyroid disease.

A. Ben-nun, T. Davies, E. Concepcion, 1991, The New England journal of medicine.

Oligodendrocyte‐specific protein is encephalitogenic in rhesus macaques and induces specific demyelination of the optic nerve

A. Ben-nun, S. Amor, B. Hart, 2008, European journal of immunology.

The central nervous sytem-specific myelin oligodendrocytic basic protein (MOBP) is encephalitogenic and a potential target antigen in multiple sclerosis (MS)

A. Ben-nun, S. Flechter, M. Hoffman, 2000, Journal of Neuroimmunology.

Epitope Specificity of Autoreactive T and B Cells Associated with Experimental Autoimmune Encephalomyelitis and Optic Neuritis Induced by Oligodendrocyte-Specific Protein in SJL/J Mice1

H. Lassmann, A. Ben-nun, N. Kerlero de Rosbo, 2006, The Journal of Immunology.

Myelin Oligodendrocyte Glycoprotein (MOG): An Archetypal Target for Demyelinating Autoantibodies in the Central Nervous System

A. Ben-nun, C. Linington, N. Kaushansky, 2014 .

Anatomy of T cell autoimmunity to myelin oligodendrocyte glycoprotein (MOG): Prime role of MOG44F in selection and control of MOG‐reactive T cells in H‐2b mice

M. Eisenstein, A. Ben-nun, N. Kaushansky, 2006, European journal of immunology.

T-cells specific for soluble recombinant oligodendrocyte-specific protein induce severe clinical experimental autoimmune encephalomyelitis in H-2b and H-2s mice

A. Meshorer, A. Ben-nun, N. Rosbo, 2000, Journal of Neuroimmunology.

Detection of autoimmune cells proliferating to myelin basic protein and selection of T cell lines that mediate experimental autoimmune encephalomyelitis (EAE) in mice.

A. Ben-nun, Z. Lando, 1983, Journal of immunology.

Multiantigen/multiepitope-directed immune-specific suppression of "complex autoimmune encephalomyelitis" by a novel protein product of a synthetic gene.

A. Ben-nun, N. Kerlero de Rosbo, Ming-Chao Zhong, 2002, The Journal of clinical investigation.

OSP/claudin‐11‐induced EAE in mice is mediated by pathogenic T cells primarily governed by OSP192Y residue of major encephalitogenic region OSP179–207

M. Eisenstein, A. Ben-nun, N. Kaushansky, 2007, European journal of immunology.

Rhesus monkeys are highly susceptible to experimental autoimmune encephalomyelitis induced by myelin oligodendrocyte glycoprotein: characterisation of immunodominant T- and B-cell epitopes

A. Ben-nun, J. Bauer, H. Brok, 2000, Journal of Neuroimmunology.

Myelin/Oligodendrocyte Glycoprotein-Induced Autoimmune Encephalomyelitis in Common Marmosets: The Encephalitogenic T Cell Epitope pMOG24–36 Is Presented by a Monomorphic MHC Class II Molecule

K. Nicolay, L. Roccatagliata, R. Bontrop, 2000 .

Class I, II and III major histocompatibility complex gene polymorphisms in BB rats

K. A. Klein, J. Seidman, A. Ben-nun, 1984, Diabetologia.

A 12-kDa protein of Mycobacterium tuberculosis protects mice against experimental autoimmune encephalomyelitis. Protection in the absence of shared T cell epitopes with encephalitogenic proteins.

A. Ben-nun, N. Kerlero de Rosbo, I. Mendel, 1995, Journal of immunology.

The autoimmune reactivity to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis is potentially pathogenic: Effect of copolymer 1 on MOG-induced disease

M. Sela, A. Ben-nun, R. Arnon, 1996, Journal of Neurology.

T lymphocyte lines induce autoimmune encephalomyelitis, delayed hypersensitivity and bystander encephalitis or arthritis

I. Cohen, A. Ben-nun, J. Holoshitz, 1984, European journal of immunology.

Neuroprotection induced by mucosal tolerance is epitope-dependent: Conflicting effects in different strains

M. Schwartz, J. Kipnis, A. Ben-nun, 2006, Journal of Neuroimmunology.

Autoimmune encephalomyelitis (EAE) mediated or prevented by T lymphocyte lines directed against diverse antigenic determinants of myelin basic protein. Vaccination is determinant specific.

I. Cohen, A. Ben-nun, J. Holoshitz, 1983, Journal of immunology.

γδ T cell subsets in patients with arthritis and chronic neutropenia

A. Ben-nun, Z. Farfel, M. Mouallem, 2002, Annals of the rheumatic diseases.

Immunomodulation of autoimmunity in MRL/lpr mice with syngeneic bone marrow transplantation (SBMT)

A. Ben-nun, D. Karussis, D. Lehmann, 1995, Clinical and experimental immunology.

Interleukin‐6 functions in autoimmune encephalomyelitis: a study in gene‐targeted mice

A. Ben-nun, M. Revel, I. Mendel, 1998 .

H‐2 gene products influence susceptibility of target thyroid gland to damage in experimental autoimmune thyroiditis

I. Cohen, Y. Ron, A. Ben-nun, 1980, European journal of immunology.

The mesenchyme expresses T cell receptor mRNAs: relevance to cell growth control

Y. Shav-Tal, A. Ben-nun, A. Zauberman, 2002, Oncogene.

Interleukin-6 functions in autoimmune encephalomyelitis: a study in gene-targeted mice.

A. Ben-nun, M. Revel, I. Mendel, 1998, European journal of immunology.

Inhibition of acute, experimental autoimmune encephalomyelitis by the synthetic immunomodulator linomide

A. Ben-nun, D. Karussis, D. Lehmann, 1993, Annals of neurology.

Prevention and reversal of adoptively transferred, chronic relapsing experimental autoimmune encephalomyelitis with a single high dose cytoreductive treatment followed by syngeneic bone marrow transplantation.

A. Ben-nun, D. Karussis, D. Lehmann, 1993, The Journal of clinical investigation.

Restricted T-cell receptor V beta gene usage by myelin basic protein-specific T-cell clones in multiple sclerosis: predominant genes vary in individuals.

A. Ben-nun, R. Liblau, A. Rosenzweig, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Host stem cells can selectively reconstitute missing lymphoid lineages in irradiation bone marrow chimeras.

Y. Ron, J. Dougherty, A. Ben-nun, 2003, Blood.

Staphylococcal enterotoxin B as a potent suppressant of T lymphocytes: trace levelssuppress T lymphocyte proliferative responses

A. Ben-nun, S. Yossefi, 1992, European journal of immunology.

Staphylococcal enterotoxin B as a potent suppressant of T cell proliferative responses in rats

A. Ben-nun, 1991, European journal of immunology.

Killer-cell lines derived from mouse thymus, resembling large granular lymphocytes and expressing natural killer-like cytotoxicity.

A. Ben-nun, V. Speth, W. Born, 1983, Immunobiology.

A highly conserved sequence associated with the HIV gp41 loop region is an immunomodulator of antigen-specific T cells in mice.

A. Ben-nun, Y. Shai, A. Ashkenazi, 2013, Blood.

is an immunomodulator of antigen-specific T cells in mice A highly conserved sequence associated with the HIV gp41 loop region

A. Ben-nun, Y. Shai, A. Ashkenazi, 2013 .

Modulation of MS-like disease by a multi epitope protein is mediated by induction of CD11c+CD11b+Gr1+ myeloid-derived dendritic cells

A. Ben-nun, H. Allouche-arnon, N. Kaushansky, 2019, Journal of Neuroimmunology.

Genetic control of autoimmune encephalomyelitis and recognition of the critical nonapeptide moiety of myelin basic protein in guinea pigs are exerted through interaction of lymphocytes and macrophages

I. Cohen, A. Ben-nun, H. Otmy, 1981, European journal of immunology.

Self-determinants in autoimmune demyelinating disease: changes in T- cell response specificity.

V. Tuohy, A. Ben-nun, R. Fritz, 1994, Current opinion in immunology.

Delineation of the minimal encephalitogenic epitope within the immunodominant region of myelin oligodendrocyte glycoprotein: diverse Vβ gene usage by T cells recognizing the core epitope encephalitogenic for T cell receptor Vβb and T cell receptor Vβa H‐2b mice

A. Ben-nun, I. Mendel, N. D. de Rosbo, 1996 .

Myelin/Oligodendrocyte Glycoprotein-Induced Autoimmune Encephalomyelitis in Common Marmosets: The Encephalitogenic T Cell Epitope pMOG24–36 Is Presented by a Monomorphic MHC Class II Molecule1

K. Nicolay, L. Roccatagliata, R. Bontrop, 2000, The Journal of Immunology.

Circulating Picomolar Levels of CCL2 Downregulate Ongoing Chronic Experimental Autoimmune Encephalomyelitis by Induction of Regulatory Mechanisms

A. Ben-nun, S. Becker-Herman, N. Kaushansky, 2019, The Journal of Immunology.

Experimental autoimmune encephalomyelitis (EAE) in genetically resistant rats: PVG rats resist active induction of EAE but are susceptible to and can generate EAE effector T cell lines.

I. Cohen, A. Ben-nun, S. Eisenstein, 1982, Journal of immunology.

T cell receptor expression and differential proliferative responses by T cells specific to a myasthenogenic peptide.

A. Waisman, A. Ben-nun, E. Mozes, 1997, Cellular immunology.

Regulation of in-vivo differentiation of myeloid leukemic cells by antigen-specific helper T lymphocytes.

A. Ben-nun, J. Lotem, L. Sachs, 1986, Leukemia research.

Analysis of cross-reactive antigen-specific T cell clones. Specific recognition of two major histocompatibility complex (MHC) and two non- MHC antigens by a single clone

A. Ben-nun, M. Dorf, S. Burakoff, 1983, The Journal of experimental medicine.

RENAL CELLS IN CULTURE AS A MODEL FOR CYSTINOSIS

A. Ben-nun, N. Bashan, R. Potashnik, 1990, Journal of basic and clinical physiology and pharmacology.

Chronic relapsing experimental autoimmune encephalomyelitis with a delayed onset and an atypical clinical course, induced in PL/J mice by myelin oligodendrocyte glycoprotein (MOG)‐derived peptide: Preliminary analysis of MOG T cell epitopes

A. Ben-nun, I. Mendel, N. D. de Rosbo, 1995, European journal of immunology.

Permanent Lines of T Lymphocytes Specific for Acetylcholine Receptors: A Clonal Approach to Study the Pathogenesis of Myasthenia Gravis

I. Cohen, R. Hohlfeld, A. Ben-nun, 1984 .

Delineation of tissue damage mechanisms in experimental autoimmune encephalomyelitis (EAE). I. Cell detachment and lysis induced by encephalitogenic CD4+ T lymphocytes.

A. Ben-nun, H. Wekerle, D. Karussis, 1992, Journal of autoimmunity.

A myelin oligodendrocyte glycoprotein peptide induces typical chronic experimental autoimmune encephalomyelitis in H‐2b mice: Fine specificity and T cell receptor Vβ expression of encephalitogenic T cells

A. Ben-nun, Itzhack Mendel, Nicole Kerlero de Rosbo, 1995, European journal of immunology.

Role of a Novel Human Leukocyte Antigen-DQA1*01:02;DRB1*15:01 Mixed Isotype Heterodimer in the Pathogenesis of “Humanized” Multiple Sclerosis-like Disease*

M. Eisenstein, H. Lassmann, G. Zeilig, 2015, The Journal of Biological Chemistry.

Themed Issue: Cannabinoids in Biology and Medicine, Part I

Z. Vogel, A. Ben-nun, R. Eilam, 2011 .

Vaccination against autoimmune encephalomyelitis (EAE): Attenuated autoimmune T lymphocytes confer resistance to induction of active EAE but not to EAE mediated by the intact T lymphocyte line

I. Cohen, A. Ben-nun, 1981, European journal of immunology.

Genes of the major histocompatibility complex control transmission of information between initiator and recruited T lymphocyte.

I. Cohen, A. Ben-nun, 1982, Advances in experimental medicine and biology.

The interaction between initiator and recruited T lymphocytes

I. Cohen, A. Ben-nun, 1979 .

Anti-idiotypic network induced by T cell vaccination against experimental autoimmune encephalomyelitis.

I. Cohen, A. Ben-nun, E. Béraud, 1988, Science.

Experimental autoimmune encephalomyelitis mediated by T-cell line. II. Specific requirements and the role of pertussis vaccine for the in vitro activation of the cells and induction of disease.

A. Ben-nun, Z. Lando, 1984, Clinical immunology and immunopathology.

From classic to spontaneous and humanized models of multiple sclerosis: impact on understanding pathogenesis and drug development.

Hartmut Wekerle, Reinhard Hohlfeld, Nathali Kaushansky, 2014, Journal of autoimmunity.

HLA-DQB1*0602 Determines Disease Susceptibility in a New “Humanized” Multiple Sclerosis Model in HLA-DR15 (DRB1*1501;DQB1*0602) Transgenic Mice1

H. Lassmann, A. Ben-nun, D. Altmann, 2009, The Journal of Immunology.

HLA-DQB 1 * 0602 Determines Disease Susceptibility in a New “ Humanized ” Multiple Sclerosis Model in HLA-DR 15 ( DRB 1 * 1501 ; DQB 1 * 0602 ) Transgenic Mice 1

H. Lassmann, A. Ben-nun, D. Altmann, 2009 .

mixed-isotype heterodimer in the pathogenesis of ‘ humanized ’ multiple sclerosis-like disease Running Title : Potential effect on MS from DQA 1 * 01 : 02 ; DRB 1 * 15 : 01

M. Eisenstein, H. Lassmann, G. Zeilig, 2015 .

Cannabidiol inhibits pathogenic T cells, decreases spinal microglial activation and ameliorates multiple sclerosis‐like disease in C57BL/6 mice

Z. Vogel, A. Ben-nun, R. Eilam, 2011, British journal of pharmacology.

Mice overexpressing Bcl-2 in their neurons are resistant to myelin oligodendrocyte glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE)

E. Melamed, A. Ben-nun, O. Bernard, 2000, Journal of Molecular Neuroscience.

The Median Eminence, A New Oligodendrogenic Niche in the Adult Mouse Brain

A. Ben-nun, R. Zilkha-Falb, N. Kaushansky, 2020, Stem cell reports.

Post-CNS-inflammation expression of CXCL12 promotes the endogenous myelin/neuronal repair capacity following spontaneous recovery from multiple sclerosis-like disease

A. Ben-nun, N. Kawakami, R. Zilkha-Falb, 2016, Journal of Neuroinflammation.

Cannabinoids Decrease the Th17 Inflammatory Autoimmune Phenotype

Z. Vogel, A. Ben-nun, E. Kozela, 2013, Journal of Neuroimmune Pharmacology.

Regulatory circuits in autoimmunity: recruitment of counter‐regulatory CD8+ T cells by encephalitogenic CD4+ T line cells

A. Ben-nun, H. Wekerle, Deming Sun, 1988, European journal of immunology.

Vaccination against autoimmune encephalomyelitis with T-lymphocite line cells reactive against myelin basic protein

I. Cohen, A. Ben-nun, H. Wekerle, 1981, Nature.

Immunospecific inhibition of nerve conduction by T lymphocytes reactive to basic protein of myelin

V. Lev-Ram, Y. Yarom, I. Cohen, 1983, Nature.

Effector T lymphocyte line cells migrate to the thymus and persist there

I. Cohen, A. Ben-nun, Y. Naparstek, 1982, Nature.

Spontaneous remission and acquired resistance to autoimmune encephalomyelitis (EAE) are associated with suppression of T cell reactivity: suppressed EAE effector T cells recovered as T cell lines.

I. Cohen, A. Ben-nun, 1982, Journal of immunology.

Immune response genes have a variable influence on the selection of antigenic foreign and self determinants of insulin.

V. Lev-Ram, I. Cohen, A. Ben-nun, 1979, Proceedings of the National Academy of Sciences of the United States of America.

Expansion of a unique subpopulation of cytotoxic T cells that express a C alpha V delta 1 T-cell receptor gene in a patient with severe persistent neutropenia.

A. Ben-nun, M. Pras, I. Bank, 1992, Blood.

Reactivity to myelin antigens in multiple sclerosis. Peripheral blood lymphocytes respond predominantly to myelin oligodendrocyte glycoprotein.

R. Milo, A. Ben-nun, C. Bernard, 1993, The Journal of clinical investigation.

Bispecificity for Myelin and Neuronal Self-Antigens Is a Common Feature of CD4 T Cells in C57BL/6 Mice

M. Eisenstein, A. Sette, A. Ben-nun, 2014, The Journal of Immunology.

Erratum to “Myelin specific Th1 cells are necessary for post-traumatic protective autoimmunity” [J. Neuroimmunol. 130 (2002) 78–85]

M. Schwartz, J. Kipnis, E. Yoles, 2002, Journal of Neuroimmunology.

CD151 Regulates T-Cell Migration in Health and Inflammatory Bowel Disease

A. Sonnenberg, R. Alon, A. Ben-nun, 2016, Inflammatory bowel diseases.

Cannabidiol, a non-psychoactive cannabinoid, leads to EGR2-dependent anergy in activated encephalitogenic T cells

G. Coppola, Z. Vogel, A. Ben-nun, 2015, Journal of Neuroinflammation.

Recruitment of effector T lymphocytes by initiator T lymphocytes: application for specific immunosuppression.

I. Cohen, A. Ben-nun, R. Hazan, 1981, Transplantation proceedings.

Delineation of tissue damage mechanisms in experimental autoimmune encephalomyelitis (EAE). II. Characteristics of astrocyte detachment mediated by myelin basic protein (MBP) specific CD4+ T lymphocytes.

S. Miron, A. Ben-nun, R. Pinkus, 1992, Journal of autoimmunity.

Vaccination for Neuroprotection in the Mouse Optic Nerve: Implications for Optic Neuropathies

Hadas Schori, Eti Yoles, E. Yoles, 2001, The Journal of Neuroscience.

Experimental autoimmune encephalomyelitis induced in B6.C-H-2bm12 mice by myelin oligodendrocyte glycoprotein: effect of MHC class II mutation on immunodominant epitope selection and fine epitope specificity of encephalitogenic T cells

A. Ben-nun, H. Gur, N. Rosbo, 1999, Journal of Neuroimmunology.

Experimental Autoimmune Encephalomyelitis Induced by Various Antigens of the Central Nervous System: Overview and Relevance to Multiple Sclerosis

A. Ben-nun, N. Rosbo, 1999 .

DQB1*0602 rather than DRB1*1501 confers susceptibility to multiple sclerosis-like disease induced by proteolipid protein (PLP)

H. Lassmann, A. Ben-nun, D. Altmann, 2012, Journal of Neuroinflammation.

A novel protective model against experimental allergic encephalomyelitis in mice expressing a transgenic TCR-specific for myelin oligodendrocyte glycoprotein

E. Shevach, A. Ben-nun, K. Natarajan, 2004, Journal of Neuroimmunology.

Identification of functional regions on the I-Ab molecule by site-directed mutagenesis.

J. Seidman, L. Glimcher, J. Smith, 1986, Proceedings of the National Academy of Sciences of the United States of America.

EBI2 Is Highly Expressed in Multiple Sclerosis Lesions and Promotes Early CNS Migration of Encephalitogenic CD4 T Cells.

K. Rajewsky, A. Waisman, T. Kuhlmann, 2017, Cell reports.

Cystine loading induces Fanconi's syndrome in rats: in vivo and vesicle studies.

A. Ben-nun, N. Bashan, R. Potashnik, 1993, The American journal of physiology.

Cystine dimethyl ester reduces the forces driving sodium-dependent transport in LLC-PK1 cells.

A. Ben-nun, N. Bashan, R. Potashnik, 1992, The American journal of physiology.

Involvement of phosphodiesterases in autoimmune diseases

A. Ben-nun, Ariel Miller, S. Fuchs, 2010, Journal of Neuroimmunology.

Regulation of experimental autoimmune encephalomyelitis by CD4+, CD25+ and CD8+ T cells: analysis using depleting antibodies.

I. Cohen, A. Ben-nun, E. Montero, 2004, Journal of autoimmunity.

The Myelin-Associated Oligodendrocytic Basic Protein Region MOBP15–36 Encompasses the Immunodominant Major Encephalitogenic Epitope(s) for SJL/J Mice and Predicted Epitope(s) for Multiple Sclerosis-Associated HLA-DRB1*15011

M. Eisenstein, H. Lassmann, A. Ben-nun, 2004, The Journal of Immunology.

Predominance of the autoimmune response to myelin oligodendrocyte glycoprotein (MOG) in multiple sclerosis: Reactivity to the extracellular domain of MOG is directed against three main regions

A. Ben-nun, O. Abramsky, S. Flechter, 1997, European journal of immunology.

T-cell responses to myelin antigens in multiple sclerosis; relevance of the predominant autoimmune reactivity to myelin oligodendrocyte glycoprotein.

A. Ben-nun, N. Rosbo, N. D. de Rosbo, 1998, Journal of autoimmunity.

Elevated levels of antibody to myelin oligodendrocyte glycoprotein is not specific for patients with multiple sclerosis.

A. Ben-nun, O. Abramsky, R. Bakimer-Kleiner, 1999, Archives of neurology.

Cannabinoids Decrease the Th17 Inflammatory Autoimmune Phenotype

Z. Vogel, A. Ben-nun, E. Kozela, 2013, Journal of Neuroimmune Pharmacology.

T lymphocyte lines producing or vaccinating against autoimmune encephalomyelitis (EAE). Functional activation induces peanut agglutinin receptors and accumulation in the brain and thymus of line cells

I. Cohen, A. Ben-nun, Y. Naparstek, 1983, European journal of immunology.

Autoimmune spread to myelin is associated with experimental autoimmune encephalomyelitis induced by a neuronal protein, β-Synuclein

A. Ben-nun, F. Mor, N. Rosbo, 2009, Journal of Neuroimmunology.

Extensive diversity of TCR V-genes expressed by T cell clones specific for the immunodominant encephalitogenic epitope of myelin oligodendrocyte glycoprotein in H-2b mice

A. Ben-nun, N. Rosbo, I. Mendel, 1998, Journal of Neuroimmunology.

Restricted immune responses lead to CNS demyelination and axonal damage

L. Roccatagliata, A. Ben-nun, M. Dono, 2000, Journal of Neuroimmunology.

An Immunomodulating Motif of the HIV-1 Fusion Protein Is Chirality-independent

A. Ben-nun, Y. Shai, A. Ashkenazi, 2013, The Journal of Biological Chemistry.

Specific Class II Histocompatibility Gene Polymorphism in BB Rats

K. A. Klein, J. Seidman, A. Ben-nun, 1984, Diabetes.

Cystine Accumulation Impairs Glucose Transport in LLC-PK1 Cells

A. Ben-nun, N. Bashan, R. Potashnik, 1991 .

Post-CNS-inflammation expression of CXCL12 promotes the endogenous myelin/neuronal repair capacity following spontaneous recovery from multiple sclerosis-like disease

A. Ben-nun, N. Kawakami, R. Zilkha-Falb, 2016, Journal of Neuroinflammation.

Immunomodulation of murine experimental autoimmune encephalomyelitis by pertussis toxin: the protective activity, but not the disease-enhancing activity, can be attributed to the nontoxic B-oligomer.

A. Ben-nun, N. Kerlero de Rosbo, I. Mendel, 1997, Proceedings of the Association of American Physicians.

Interleukin‐6 functions in autoimmune encephalomyelitis: a study in gene‐targeted mice

A. Ben-nun, M. Revel, A. Katz, 1998, European journal of immunology.

DNA-mediated transfer of major histocompatibility class II I-Ab and I-Abm12 genes into B lymphoma cells: molecular and functional analysis of introduced antigens.

J. Seidman, L. Glimcher, A. Ben-nun, 1985, Journal of immunology.

Minor lymphocyte stimulating (Mls) gene products in mice influence their genetic resistance or susceptibility to induction of autoimmune encephalomyelitis

A. Ben-nun, David Soffer, 1990, European journal of immunology.