C. Coffinier

P. Angrand, C. Coffinier, M. Weiss, 1994, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Neuralin-1 is a novel Chordin-related molecule expressed in the mouse neural plate

C. Coffinier, E. Robertis, U. Tran, 2001, Mechanisms of Development.

Crossveinless-2 Is a BMP feedback inhibitor that binds Chordin/BMP to regulate Xenopus embryonic patterning.

C. Coffinier, E. D. De Robertis, A. Ambrosio, 2008, Developmental cell.

Mouse Crossveinless-2 is the vertebrate homolog of a Drosophila extracellular regulator of BMP signaling.

C. Coffinier, E. D. De Robertis, U. Tran, 2002, Mechanisms of development.

Are B-type lamins essential in all mammalian cells?

S. Young, C. Coffinier, L. Fong, 2011, Nucleus.

Selective deletion of the Hnf1beta (MODY5) gene in beta-cells leads to altered gene expression and defective insulin release.

L. Gresh, M. Yaniv, C. Coffinier, 2004, Endocrinology.

Reciprocal knock-in mice to investigate the functional redundancy of lamin B1 and lamin B2

P. D. de Jong, Y. Yoshinaga, S. Young, 2014, Molecular biology of the cell.

Farnesylation of lamin B1 is important for retention of nuclear chromatin during neuronal migration

P. D. de Jong, Y. Yoshinaga, S. Young, 2013, Proceedings of the National Academy of Sciences.

A farnesyltransferase inhibitor improves disease phenotypes in mice with a Hutchinson-Gilford progeria syndrome mutation.

M. Bergo, S. Young, C. Coffinier, 2006, The Journal of clinical investigation.

A Protein Farnesyltransferase Inhibitor Ameliorates Disease in a Mouse Model of Progeria

Stephen G Young, S. Young, C. Coffinier, 2006, Science.

Regulation of prelamin A but not lamin C by miR-9, a brain-specific microRNA

S. Young, T. Sun, C. Coffinier, 2012, Proceedings of the National Academy of Sciences.

Selective Deletion of the Hnf1β (MODY5) Gene in β-Cells Leads to Altered Gene Expression and Defective Insulin Release

L. Gresh, M. Yaniv, C. Coffinier, 2004 .

Understanding the Roles of Nuclear A- and B-type Lamins in Brain Development*

S. Young, C. Coffinier, L. Fong, 2012, The Journal of Biological Chemistry.

Bile system morphogenesis defects and liver dysfunction upon targeted deletion of HNF1beta.

L. Gresh, F. Tronche, M. Yaniv, 2002, Development.

Essential role for the homeoprotein vHNF1/HNF1beta in visceral endoderm differentiation.

M. Yaniv, C. Coffinier, J. Barra, 1999, Development.

Nuclear lamins and brain development*

C. Coffinier, L. Fong, Hea-Jin Jung, 2012 .

Chordin-like CR domains and the regulation of evolutionarily conserved extracellular signaling systems.

C. Coffinier, E. D. De Robertis, M. Oelgeschläger, 2002, Gene.

HIV protease inhibitors block the zinc metalloproteinase ZMPSTE24 and lead to an accumulation of prelamin A in cells

S. Young, C. Coffinier, Sandy Chang, 2007, Proceedings of the National Academy of Sciences.

Direct Synthesis of Lamin A, Bypassing Prelamin A Processing, Causes Misshapen Nuclei in Fibroblasts but No Detectable Pathology in Mice*

S. Young, J. Lammerding, C. Coffinier, 2010, The Journal of Biological Chemistry.

The posttranslational processing of prelamin A and disease.

S. Young, C. Coffinier, Brandon S. J. Davies, 2009, Annual review of genomics and human genetics.

HIV-protease inhibitors block the enzymatic activity of purified Ste24p.

S. Young, C. Coffinier, L. Fong, 2008, Biochemical and biophysical research communications.

Abnormal development of the cerebral cortex and cerebellum in the setting of lamin B2 deficiency

S. Young, C. Coffinier, J. Toth, 2010, Proceedings of the National Academy of Sciences.

Activating the synthesis of progerin, the mutant prelamin A in Hutchinson–Gilford progeria syndrome, with antisense oligonucleotides

S. Young, C. Coffinier, C. F. Bennett, 2009, Human molecular genetics.

Development of the vertebral morphogenetic field in the mouse: interactions between Crossveinless-2 and Twisted Gastrulation.

C. Coffinier, E. D. De Robertis, L. Zakin, 2008, Developmental biology.

Munster, a novel Paired-class homeobox gene specifically expressed in the Drosophila larval eye

A. Goriely, C. Coffinier, C. Desplan, 1999, Mechanisms of Development.

Mouse Crossveinless-2 is the vertebrate homolog of a Drosophila extracellular regulator of BMP signaling

C. Coffinier, E. Robertis, U. Tran, 2002, Mechanisms of Development.

Selective Deletion of the Hnf1 (MODY5) Gene in -Cells Leads to Altered Gene Expression and Defective Insulin Release

L. Gresh, M. Yaniv, C. Coffinier, 2004 .

The posttranslational processing of prelamin A and disease.

S. Young, C. Coffinier, Brandon S. J. Davies, 2009, Annual review of genomics and human genetics.

Investigating the purpose of prelamin A processing.

S. Young, J. Lammerding, C. Coffinier, 2010, Nucleus.

Expression of the vHNF1/HNF1β homeoprotein gene during mouse organogenesis

M. Yaniv, C. Coffinier, J. Barra, 1999, Mechanisms of Development.