D. Baruch

发表

A genetically-engineered von Willebrand disease type 2B mouse model displays defects in hemostasis and inflammation

M. Bryckaert, F. Adam, C. Casari, 2016, Scientific Reports.

Microfluidic model of the platelet-generating organ: beyond bone marrow biomimetics

D. Baruch, A. Blin, M. Reyssat, 2015, Scientific Reports.

Inhibition of αIIbβ3 Ligand Binding by an αIIb Peptide that Clasps the Hybrid Domain to the βI Domain of β3

D. Baruch, N. Kieffer, V. Moussis, 2015, PloS one.

Increased metastatic potential of tumor cells in von Willebrand factor‐deficient mice

D Meyer, D. Baruch, P. Lenting, 2006, Journal of thrombosis and haemostasis : JTH.

Effect of recombinant von Willebrand factor reproducing type 2B or type 2M mutations on shear-induced platelet aggregation.

D. Baruch, D. Meyer, A. Ribba, 2000, Blood.

von Willebrand factor-mediated platelet adhesion to collagen involves platelet membrane glycoprotein IIb-IIIa as well as glycoprotein Ib.

D. Baruch, É. Fressinaud, K. Sakariassen, 1988, The Journal of laboratory and clinical medicine.

Role of von Willebrand factor associated to extracellular matrices in platelet adhesion.

D. Baruch, C. Denis, L. Coulombel, 1991 .

Localization of von Willebrand factor binding domains to endothelial extracellular matrix and to type VI collagen.

D. Baruch, O. Christophe, C. Denis, 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.

Modulation by heparin of the interaction of the A1 domain of Von Willebrand factor with glycoprotein Ib.

P. Legendre, D. Baruch, D. Meyer, 1999, Blood.

The Cleavage of Neutrophil Leukosialin (CD43) by Cathepsin G Releases Its Extracellular Domain and Triggers Its Intramembrane Proteolysis by Presenilin/γ-Secretase*

D. Baruch, M. Fukuda, Misa Suzuki, 2008, Journal of Biological Chemistry.

Relative importance of the glycoprotein Ib-binding domain and the RGD sequence of von Willebrand factor for its interaction with endothelial cells.

J. Sixma, D. Baruch, D. Meyer, 1997, Blood.

Solid-phase von Willebrand factor contains a conformationally active RGD motif that mediates endothelial cell adhesion through the alpha v beta 3 receptor.

J. Williams, D. Baruch, C. Denis, 1993, Blood.

Kinetics of thrombin-induced release and activation of platelet factor V.

D. Baruch, H. Hemker, T. Lindhout, 1986, European journal of biochemistry.

Subacute Stent Thrombosis: A Case-Control Study Enhanced Shear-Induced Platelet Aggregation in Patients Who Experience

D. Baruch, L. Feldman, A. Cachier, 2011 .

Enhanced shear-induced platelet aggregation in patients who experience subacute stent thrombosis: a case-control study.

D. Baruch, L. Feldman, A. Cachier, 2005, Journal of the American College of Cardiology.

von Willebrand factor stimulates thrombin‐induced exposure of procoagulant phospholipids on the surface of fibrin‐adherent platelets

D. Baruch, H. Hemker, J. Heemskerk, 2003, Journal of thrombosis and haemostasis : JTH.

von Willebrand factor is a major determinant of ADAMTS‐13 decrease during mouse sepsis induced by cecum ligation and puncture

D. Baruch, C. Denis, D. Motto, 2009, Journal of thrombosis and haemostasis : JTH.

Role of αv integrins in mesangial cell adhesion to vitronectin and von Willebrand factor

D. Baruch, L. Halbwachs‐Mecarelli, P. Lesavre, 1997 .

von Willebrand factor C1C2 domain is involved in platelet adhesion to polymerized fibrin at high shear rate.

P. Legendre, D. Baruch, P. Lenting, 2004, Blood.

Characterization of Murine Anti-Glycoprotein Ib Monoclonal Antibodies That Differentiate between Shear-Induced and Ristocetin/Botrocetin-Induced Glycoprotein Ib-von Willebrand Factor Interaction

D. Baruch, P. Declerck, M. Hoylaerts, 2000, Pathophysiology of Haemostasis and Thrombosis.

CHO cells expressing the high affinity αIIbβ3T562N integrin demonstrate enhanced adhesion under shear

P. Legendre, D. Baruch, N. Kieffer, 2006 .

Activation of integrin αIIbβ3 expressed in Chinese hamster ovary cells is required for interaction with solid‐phase von Willebrand factor

P. Legendre, D. Baruch, N. Kieffer, 2002 .

Activation of recombinant αIIbβ3 expressed in Chinese hamster ovary cells exposes different binding sites for fibrinogen or von Willebrand factor: evidence using monoclonal antibodies to αIIbβ3

D. Baruch, N. Kieffer, M. Mekrache, 2002 .

A comparison of haematopoietic stem cells from umbilical cord blood and peripheral blood for platelet production in a microfluidic device

D. Baruch, G. Sicot, V. Compernolle, 2019, Vox sanguinis.

Inhibition of thrombin-catalyzed reactions in blood coagulation and platelet activation by heparin fractions in the absence of antithrombin III.

D. Baruch, H. Hemker, R. Wagenvoord, 1986, Haemostasis.

Effect of heparin and low molecular weight heparins on thrombin-induced blood platelet activation in the absence of antithrombin III.

D. Baruch, H. Hemker, T. Lindhout, 1985, Thrombosis research.

Platelet shape change and subsequent glycoprotein redistribution in human stenosed arteries

D. Baruch, H. Kobeiter, E. Cramer, 2005, Platelets.

Role of von Willebrand factor associated to extracellular matrices in platelet adhesion.

D. Baruch, C. Denis, L. Coulombel, 1991, Blood.

Production in Escherichia coli of a biologically active subfragment of von Willebrand factor corresponding to the platelet glycoprotein Ib, collagen and heparin binding domains.

D. Baruch, D. Meyer, P. Meulien, 1989, Biochemical and biophysical research communications.

von Willebrand factor and platelet function.

D. Baruch, B. Bahnak, D. Meyer, 1989, Bailliere's clinical haematology.

[In vitro platelet production].

D. Baruch, C. Dunois‐Lardé, 2011, Transfusion clinique et biologique : journal de la Societe francaise de transfusion sanguine.

Plasma Renin Activity in the Renal Veins of Three Patients with Juxtaglomerular Cell Renin Tumors Treatment Case 1 Sodium depletion + beta-blockers Case 2 Sodium depletion Sodium depletion Captopril Captopril Case 3 Normal sodium intake Captopril Right renal vein

J. Ménard, J. Gaux, D. Baruch, 2005 .

Diagnosis and Treatment of Renin-Secreting T \ imors Report of Three Cases

Brisset, J. Ménard, J. Gaux, 2005 .

Platelet-Vessel Wall Interactions

D. Baruch, 1988, The Bloomsbury Series in Clinical Science.

Activation of pp125FAK by type 2B recombinant von Willebrand factor binding to platelet GPIb at a high shear rate occurs independently of (cid:1) IIb (cid:2) 3 engagement

P. Legendre, D. Baruch, A. Saci, 2003 .

6 Von Willebrand factor and platelet function

D. Baruch, B. Bahnak, D. Meyer, 1989 .

Binding of Heparin Fractions to von Willebrand Factor: Effect of Molecular Weight and Affinity for Antithrombin III

P. Legendre, D. Baruch, C. Denis, 1994, Thrombosis and Haemostasis.

Platelet aggregation induced by a monoclonal antibody to the A1 domain of von Willebrand factor.

D. Baruch, C. Lacombe, H. Deckmyn, 1998, Blood.

Influence of mutations and size of multimers in type II von Willebrand disease upon the function of von Willebrand factor.

D. Baruch, O. Christophe, K. Niinomi, 1994, Blood.

Activation of pp125FAK by type 2B recombinant von Willebrand factor binding to platelet GPIb at a high shear rate occurs independently of αIIbβ3 engagement

P. Legendre, D. Baruch, A. Saci, 2003 .

A monoclonal antibody directed against human von Willebrand factor induces type 2B‐like alterations

D. Baruch, H. Deckmyn, J. Vermylen, 2004, Journal of thrombosis and haemostasis : JTH.

von Willebrand factor binding to heparin in various types of von Willebrand disease.

P. Legendre, D. Baruch, D. Meyer, 2000, The hematology journal : the official journal of the European Haematology Association.

Inhibition of cell adhesion by antibodies to Arg-Gly-Asp (RGD) in normal immunoglobulin for therapeutic use (intravenous immunoglobulin, IVIg).

S. Kaveri, M. Kazatchkine, D. Baruch, 1999, Blood.

Exposure of human megakaryocytes to high shear rates accelerates platelet production.

D. Baruch, S. Fichelson, E. Cramer-Bordé, 2009, Blood.

Decreased ADAMTS-13 (A disintegrin-like and metalloprotease with thrombospondin type 1 repeats) is associated with a poor prognosis in sepsis-induced organ failure*

L. Trinquart, P. Legendre, D. Baruch, 2007, Critical care medicine.

The Integrin α2β1 (GPIa/IIa)-I-Domain Inhibits Platelet-Collagen Interaction

D. Baruch, P. Stanssens, H. Deckmyn, 1997, Thrombosis and Haemostasis.

Thrombin generation and inactivation in the presence of antithrombin III and heparin.

D. Baruch, H. Hemker, P. Schoen, 1986, Biochemistry.

Terminal Platelet Production is Regulated by Von Willebrand Factor

C. Casari, D. Baruch, C. Denis, 2013, PloS one.

Thrombin-Induced Platelet Factor Va Formation in Patients with a Gray Platelet Syndrome

D. Baruch, E. Dupuy, J. Caen, 1987, Thrombosis and Haemostasis.

Human neutrophil integrin α9β1: up‐regulation by cell activation and synergy with β2 integrins during adhesion to endothelium under flow

D. Baruch, L. Halbwachs‐Mecarelli, P. Lesavre, 2010, Journal of leukocyte biology.

Activation of pp125FAK by type 2B recombinant von Willebrand factor binding to platelet GPIb at a high shear rate occurs independently of alpha IIb beta 3 engagement.

P. Legendre, D. Baruch, A. Saci, 2003, Blood.

Role of oculocerebrorenal syndrome of Lowe (OCRL) protein in megakaryocyte maturation, platelet production and functions: a study in patients with Lowe syndrome

C. Elie, R. Salomon, D. Baruch, 2021, British journal of haematology.

The integrin alpha 2 beta 1 (GPIa/IIa)-I-domain inhibits platelet-collagen interaction.

D. Baruch, P. Stanssens, H. Deckmyn, 1997, Thrombosis and haemostasis.

Two clusters of charged residues located in the electropositive face of the von Willebrand factor A1 domain are essential for heparin binding.

B. Villoutreix, P. Legendre, D. Baruch, 2002, Biochemistry.

Three-Dimensional Environment Sustains Hematopoietic Stem Cell Differentiation into Platelet-Producing Megakaryocytes

A. Kauskot, D. Baruch, D. Letourneur, 2015, PloS one.

Defect of Heparin Binding in Plasma and Recombinant von Willebrand Factor with Type 2 von Willebrand Disease Mutations

B. Villoutreix, P. Legendre, D. Baruch, 2001, Thrombosis and Haemostasis.

Distinct sequences of the glycoprotein Ib-binding domain of von Willebrand factor involved in shear induced platelet aggregation.

D. Baruch, C. Lacombe, H. Deckmyn, 1998, Platelets.

Platelet von Willebrand factor: evidence for its involvement in platelet adhesion to collagen.

D. Baruch, C. Rothschild, É. Fressinaud, 1987, Blood.

Ser752 mutation to Pro or Ala in the beta3 integrin subunit differentially affects the kinetics of cell spreading to von Willebrand factor and fibrinogen.

D. Baruch, N. Kieffer, D. Schoevaert, 1998, Cell adhesion and communication.

Mitochondrial dynamics and reactive oxygen species initiate thrombopoiesis from mature megakaryocytes.

A. Kauskot, D. Baruch, I. Dusanter-Fourt, 2021, Blood advances.

LIM kinase/cofilin dysregulation promotes macrothrombocytopenia in severe von Willebrand disease-type 2B.

M. Bryckaert, F. Adam, C. Casari, 2016, JCI insight.

Clinical researchInterventional cardiologyEnhanced Shear-Induced Platelet Aggregation in Patients Who Experience Subacute Stent Thrombosis: A Case-Control Study

D. Baruch, L. Feldman, A. Cachier, 2005 .

Clinical researchInterventional cardiologyEnhanced Shear-Induced Platelet Aggregation in Patients Who Experience Subacute Stent Thrombosis: A Case-Control Study

D. Baruch, L. Feldman, A. Cachier, 2005 .

Complete Defect in vWF-cleaving Protease Activity Associated with Increased Shear-induced Platelet Aggregation in Thrombotic Microangiopathy

D. Baruch, C. Denis, D. Meyer, 2002, Thrombosis and Haemostasis.

Platelet von Willebrand factor: evidence for its involvement in platelet adhesion to collagen.

D. Baruch, É. Fressinaud, H. Baumgartner, 1987, Blood.

Thrombin generation and inactivation in the presence of antithrombin III and heparin.

D. Baruch, H. Hemker, P. Schoen, 1986, Biochemistry.