S. Caniglia

发表

Targeting Wnt signaling at the neuroimmune interface for dopaminergic neuroprotection/repair in Parkinson's disease.

S. Pluchino, F. L'Episcopo, C. Tirolo, 2014, Journal of molecular cell biology.

Glucocorticoid receptor–nitric oxide crosstalk and vulnerability to experimental parkinsonism: pivotal role for glia–neuron interactions

Pier Andrea Serra, Bianca Marchetti, Egidio Miele, 2005, Brain Research Reviews.

Targeting Wnt signaling at the neuroimmune interfacefordopaminergicneuroprotection/repairin

S. Pluchino, C. Tirolo, S. Caniglia, 2014 .

Reactive astrocytes are key players in nigrostriatal dopaminergic neurorepair in the MPTP mouse model of Parkinson's disease: focus on endogenous neurorestoration.

C. Tirolo, S. Caniglia, N. Testa, 2013, Current aging science.

Exposure to a Dysfunctional Glucocorticoid Receptor from Early Embryonic Life Programs the Resistance to Experimental Autoimmune Encephalomyelitis Via Nitric Oxide-Induced Immunosuppression1

C. Tirolo, S. Caniglia, N. Testa, 2002, The Journal of Immunology.

Uncovering novel actors in astrocyte–neuron crosstalk in Parkinson's disease: the Wnt/β‐catenin signaling cascade as the common final pathway for neuroprotection and self‐repair

S. Pluchino, C. Tirolo, S. Caniglia, 2013, The European journal of neuroscience.

Glucocorticoid receptor deficiency increases vulnerability of the nigrostriatal dopaminergic system: critical role of glial nitric oxide

Rossana Migheli, Maria Speranza Desole, Pier Andrea Serra, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

microRNAs in Parkinson’s Disease: From Pathogenesis to Novel Diagnostic and Therapeutic Approaches

C. Tirolo, S. Caniglia, B. Marchetti, 2017, International journal of molecular sciences.

Neuroendocrine–immune (NEI) circuitry from neuron–glial interactions to function: Focus on gender and HPA–HPG interactions on early programming of the NEI system

C. Tirolo, S. Caniglia, N. Testa, 2001, Immunology and cell biology.

Corrigendum to “Reactive astrocytes and Wnt/β-catenin signaling link nigrostriatal injury to repair in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease” [Neurobiol. Dis. 41/2 (2011) 508–527]

G. Martino, S. Pluchino, C. Tirolo, 2011, Neurobiology of Disease.

Reactive astrocytes and Wnt/β-catenin signaling link nigrostriatal injury to repair in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease

G. Martino, S. Pluchino, C. Tirolo, 2011, Neurobiology of Disease.

Glia as a turning point in the therapeutic strategy of Parkinson's disease.

C. Tirolo, S. Caniglia, N. Testa, 2010, CNS & neurological disorders drug targets.

Gender, Neuroendocrine‐Immune Interactions and Neuron‐Glial Plasticity: Role of Luteinizing Hormone‐Releasing Hormone (LHRH)

C. Tirolo, S. Caniglia, N. Testa, 2000, Annals of the New York Academy of Sciences.

Bilirubin protects astrocytes from its own toxicity by inducing up-regulation and translocation of multidrug resistance-associated protein 1 (Mrp1).

C. Tirolo, S. Caniglia, N. Testa, 2004, Proceedings of the National Academy of Sciences of the United States of America.

The reproductive system at the neuroendocrine-immune interface: focus on LHRH, estrogens and growth factors in LHRH neuron-glial interactions.

C. Tirolo, S. Caniglia, M. C. Morale, 2003, Domestic animal endocrinology.

Oxide-Induced Immunosuppression Autoimmune Encephalomyelitis Via Nitric Programs the Resistance to Experimental Receptor from Early Embryonic Life Exposure to a Dysfunctional Glucocorticoid

C. Tirolo, S. Caniglia, N. Testa, 2002 .

Hormones Are Key Actors in Gene X Environment Interactions Programming the Vulnerability to Parkinson's Disease: Glia as a Common Final Pathway

Maria Speranza Desole, Pier Andrea Serra, Bianca Marchetti, 2005, Annals of the New York Academy of Sciences.

GSK-3β-induced Tau pathology drives hippocampal neuronal cell death in Huntington's disease: involvement of astrocyte–neuron interactions

R. Barker, R. Giugno, A. Pulvirenti, 2016, Cell Death and Disease.

Loss of aromatase cytochrome P450 function as a risk factor for Parkinson's disease?

N. Harada, C. Tirolo, S. Caniglia, 2008, Brain Research Reviews.

Vulnerability to Parkinson's Disease: Towards an Unifying Theory of Disease Etiology

C. Tirolo, S. Caniglia, N. Testa, 2011 .

Reactive astrocytes and Wnt/β-catenin signaling link nigrostriatal injury to repair in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease

G. Martino, S. Pluchino, C. Tirolo, 2011, Neurobiology of Disease.