J. Tinsley

发表

Protein Kinase C Activation Contributes to Microvascular Barrier Dysfunction in the Heart at Early Stages of Diabetes

S. Yuan, E. Ustinova, M. Wu, 2000, Circulation research.

Efficient protein transfection of cultured coronary venular endothelial cells.

J. Tinsley, Yuan Yuan, J. Hawker, 1998, American journal of physiology. Heart and circulatory physiology.

Role of phospholipase C, protein kinase C, and calcium in VEGF-induced venular hyperpermeability.

H. Granger, D. Zawieja, J. Tinsley, 1999, The American journal of physiology.

PKC-dependent, burn-induced adherens junction reorganization and barrier dysfunction in pulmonary microvascular endothelial cells.

S. Yuan, J. Tinsley, J. Breslin, 2005, American journal of physiology. Lung cellular and molecular physiology.

Myosin light chain phosphorylation and pulmonary endothelial cell hyperpermeability in burns.

S. Yuan, J. Tinsley, Nicole R Teasdale, 2004, American journal of physiology. Lung cellular and molecular physiology.

Myosin light chain kinase transference induces myosin light chain activation and endothelial hyperpermeability.

S. Yuan, J. Tinsley, P. de Lanerolle, 2000, American journal of physiology. Cell physiology.

Activated Neutrophils Induce Hyperpermeability and Phosphorylation of Adherens Junction Proteins in Coronary Venular Endothelial Cells*

S. Yuan, J. Tinsley, A. C. Taulman, 1999, The Journal of Biological Chemistry.

In vivo stability and kinetics of absorption and disposition of 3' phosphopropyl amine oligonucleotides.

J. Tinsley, M. Hogan, J. Zendegui, 1992, Nucleic acids research.

Analysis of actin and actin-related protein 3 (ARP3) gene expression following induction of hyphal tip formation and apolar growth in Neurospora

J. Tinsley, M. Plamann, P. Minke, 1998, Molecular and General Genetics MGG.

Apoptotic signaling induces hyperpermeability following hemorrhagic shock.

J. Tinsley, B. Tharakan, Xiaobo X. Cao, 2007, American journal of physiology. Heart and circulatory physiology.

Toll-like receptor 3 activation during pregnancy elicits preeclampsia-like symptoms in rats.

J. Tinsley, B. Mitchell, V. Chiasson, 2009, American journal of hypertension.

Immunosuppression improves blood pressure and endothelial function in a rat model of pregnancy-induced hypertension.

J. Tinsley, B. Mitchell, V. Chiasson, 2009, American journal of hypertension.

Genetic interactions among cytoplasmic dynein, dynactin, and nuclear distribution mutants of Neurospora crassa.

J. Tinsley, M. Plamann, K. Bruno, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Interleukin-10 reduces inflammation, endothelial dysfunction, and blood pressure in hypertensive pregnant rats.

J. Tinsley, B. Mitchell, V. Chiasson, 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

Angiopoietin-1 inhibits intrinsic apoptotic signaling and vascular hyperpermeability following hemorrhagic shock.

J. Tinsley, B. Tharakan, W. Smythe, 2008, American journal of physiology. Heart and circulatory physiology.

&bgr;-Catenin Dynamics in the Regulation of Microvascular Endothelial Cell Hyperpermeability

J. Tinsley, B. Tharakan, W. Smythe, 2012, Shock.

Cytoplasmic dynein and actin-related protein Arp1 are required for normal nuclear distribution in filamentous fungi

J. Tinsley, M. Plamann, K. Bruno, 1994, The Journal of cell biology.

Neurospora crassa ro‐10 and ro‐11 genes encode novel proteins required for nuclear distribution

J. Tinsley, M. Plamann, K. Bruno, 1999, Molecular microbiology.

p150Glued, the largest subunit of the dynactin complex, is nonessential in Neurospora but required for nuclear distribution.

J. Tinsley, M. Plamann, K. Bruno, 1996, Molecular biology of the cell.

Binding of T and T analogs to CG base pairs in antiparallel triplexes.

T. S. Rao, K. Jayaraman, J. Tinsley, 1994, Nucleic acids research.

Involvement of PKCdelta and PKD in pulmonary microvascular endothelial cell hyperpermeability.

S. Yuan, J. Tinsley, Nicole R Teasdale, 2004, American journal of physiology. Cell physiology.

PKC and MLCK-dependent, cytokine-induced rat coronary endothelial dysfunction.

J. Tinsley, E. Childs, F. Hunter, 2009, The Journal of surgical research.

Isoform-specific knockout of endothelial myosin light chain kinase: closing the gap on inflammatory lung disease.

S. Yuan, J. Tinsley, E. Wilson, 2004, Trends in pharmacological sciences.

Src-dependent, neutrophil-mediated vascular hyperpermeability and beta-catenin modification.

S. Yuan, E. Ustinova, J. Tinsley, 2002, American journal of physiology. Cell physiology.

Myosin Light Chain Phosphorylation in Neutrophil-Stimulated Coronary Microvascular Leakage

S. Yuan, E. Ustinova, J. Tinsley, 2002, Circulation research.

Protein Transfection of Intact Microvessels Specifically Modulates Vasoreactivity and Permeability

D. Zawieja, S. Yuan, E. Ustinova, 2001, Journal of Vascular Research.

Efficient protein transfection of cultured coronary venular endothelial cells.

J. Tinsley, Y. Yuan, J. Hawker, 1998, The American journal of physiology.