X. Gu

G. Haddad, X. Gu, L. Buck, 2012, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

DIDS (4,4-Diisothiocyanatostilbenedisulphonic Acid) Induces Apoptotic Cell Death in a Hippocampal Neuronal Cell Line and Is Not Neuroprotective against Ischemic Stress

Mark Ellisman, G. Perkins, G. Haddad, 2013, PloS one.

The role of hco3 −-dependent mechanisms in phi regulation during o2 deprivation

G. Haddad, X. Gu, H. Yao, 2003, Neuroscience.

Ischemic stroke leads to a complex cascade of cellular events

R. Douglas, X. Gu, H. Yao, 2009 .

The sodium-activated potassium channel Slack is modulated by hypercapnia and acidosis

R. Douglas, C. Trouth, G. Haddad, 2008, Neuroscience.

Role of Na(+)/H(+) exchanger during O(2) deprivation in mouse CA1 neurons.

R. Douglas, G. Haddad, X. Gu, 2001, American journal of physiology. Cell physiology.

Action potential-like responses in B 104 cells with low Na+ channel densities

S. Waxman, X. Gu, 1996, Brain Research.

Altered iPSC-derived neurons’ sodium channel properties in subjects with Monge’s disease

A. Muotri, G. Haddad, X. Gu, 2015, Neuroscience.

Drosophila neurons respond differently to hypoxia and cyanide than rat neurons

G. Haddad, X. Gu, 1999, Brain Research.

Mutation in pre-mRNA adenosine deaminase markedly attenuates neuronal tolerance to O2 deprivation in Drosophila melanogaster.

E. Ma, G. Haddad, X. Gu, 2001, The Journal of clinical investigation.

Mitochondrial but not plasmalemmal BK channels are hypoxia‐sensitive in human glioma

X. Gu, D. Siemen, M. Pamenter, 2014, Glia.

Effect of chronically elevated CO2 on CA1 neuronal excitability.

G. Haddad, X. Gu, Jin Xue, 2004, American journal of physiology. Cell physiology.

Na+ channel expression and neuronal function in the Na+/H+ exchanger 1 null mutant mouse.

G. Haddad, X. Gu, H. Yao, 2003, Journal of neurophysiology.

Increased neuronal excitability and seizures in the Na(+)/H(+) exchanger null mutant mouse.

G. Haddad, X. Gu, H. Yao, 2001, American journal of physiology. Cell physiology.

Mutation in pre-mRNA adenosine deaminase markedly attenuates neuronal tolerance to O2 deprivation in Drosophila melanogaster.

X. Gu, Gabriel G. Haddad, Enbo Ma, 2001, The Journal of clinical investigation.

Intermittent hypoxia modulates Na+ channel expression in developing mouse brain

G. Haddad, X. Gu, Ying Xia, 2005, International Journal of Developmental Neuroscience.

Hypoxia Increases Activity of the BK-Channel in the Inner Mitochondrial Membrane and Reduces Activity of the Permeability Transition Pore

G. Haddad, X. Gu, P. Bednarczyk, 2008, Cellular Physiology and Biochemistry.

Effect of extracellular HCO(3)(-) on Na(+) channel characteristics in hippocampal CA1 neurons.

X. Gu, H. Yao, Gabriel G. Haddad, 2000, Journal of neurophysiology.

Isolated CA1 Neurons of the Rat Voltage Dependence of Sodium Currents in Freshly Relation Between Bicarbonate Concentration and

Nace L. Golding, M. Jubeau, S. Perrey, 2015 .

Altered iPSC-derived neurons’ sodium channel properties in subjects with Monge’s disease

G. Perkins, A. Muotri, X. Gu, 2015, Neuroscience.