A. Schmidt

发表

Evidence for epidermal growth factor (EGF)-induced intermolecular autophosphorylation of the EGF receptors in living cells

A. Ullrich, J. Schlessinger, A. Honegger, 1990, Molecular and cellular biology.

Nuclear factors interacting with the mitogen-responsive regulatory region of the interleukin-2 gene.

A. Schmidt, U. Siebenlist, M. Brunvand, 1988, The Journal of biological chemistry.

Inducible nuclear factor binding to the kappa B elements of the human immunodeficiency virus enhancer in T cells can be blocked by cyclosporin A in a signal-dependent manner

L. Hennighausen, A. Schmidt, U. Siebenlist, 1990, Journal of virology.

A mutant epidermal growth factor receptor with defective protein tyrosine kinase is unable to stimulate proto-oncogene expression and DNA synthesis

A. Ullrich, E. Van Obberghen, J. Schlessinger, 1987, Molecular and cellular biology.

Separate endocytic pathways of kinase-defective and -active EGF receptor mutants expressed in same cells

A. Ullrich, J. Schlessinger, A. Honegger, 1990, The Journal of cell biology.

Point mutation at the ATP binding site of EGF receptor abolishes protein-tyrosine kinase activity and alters cellular routing

A. Ullrich, J. Schlessinger, A. Honegger, 1987, Cell.

Cyclic AMP-induced Chromatin Changes Support the NFATc-mediated Recruitment of GATA-3 to the Interleukin 5 Promoter*

A. Schmidt, E. Serfling, E. Schmitt, 2008, Journal of Biological Chemistry.

An angiogenic growth factor is expressed in human glioma cells.

T. Libermann, M. Jaye, J. Schlessinger, 1987, The EMBO journal.

Two FK506 resistance-conferring genes in Saccharomyces cerevisiae, TAT1 and TAT2, encode amino acid permeases mediating tyrosine and tryptophan uptake

A. Schmidt, M. Hall, A. Koller, 1994 .

Biological activities of EGF‐receptor mutants with individually altered autophosphorylation sites.

A. Ullrich, E. Van Obberghen, J. Schlessinger, 1988, The EMBO journal.

Ligand-induced stimulation of epidermal growth factor receptor mutants with altered transmembrane regions.

A. Ullrich, J. Schlessinger, A. Schmidt, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Domain deletion in the extracellular portion of the EGF-receptor reduces ligand binding and impairs cell surface expression.

A. Ullrich, I. Lax, J. Schlessinger, 1990, Cell regulation.