Martin L. Smith

发表

Phosphorylation of Human p53 at Serine 46 Determines Promoter Selection and whether Apoptosis Is Attenuated or Amplified*

L. Mayo, Y. Seo, M. Jackson, 2005, Journal of Biological Chemistry.

Selenium protection from DNA damage involves a Ref1/p53/Brca1 protein complex.

Martin L. Smith, Joshua L. Fischer, J. Lancia, 2006, Anticancer research.

p53 regulation of DNA excision repair pathways.

Y. Seo, Martin L. Smith, 2002, Mutagenesis.

Dependence of induction of osteocalcin gene expression on the presence of wild‐type p53 in a murine osteosarcoma cell line

Martin L. Smith, N. Chandar, P. Campbell, 1993, Molecular carcinogenesis.

The Xpc gene markedly affects cell survival in mouse bone marrow

K. Pollok, Martin L. Smith, Joshua L. Fischer, 2009, Mutagenesis.

RB stabilizes XPC and promotes cellular NER.

Martin L. Smith, M. Kumar, T. Hardy, 2010, Anticancer research.

RB Stabilizes XPC and Promotes Cellular

Martin L. Smith, Manish Kumar, 2010 .

Implication of p53 in base excision DNA repair: in vivo evidence

S. Amundson, Y. Seo, Martin L. Smith, 2002, Oncogene.

p 53-Mediated DNA Repair Responses to UV Radiation : Studies of Mouse Cells Lacking p 53 , p 21 , and / or gadd 45 Genes

P. Hanawalt, C. Deng, A. Fornace, 2000 .

Manipulation of Base Excision Repair to Sensitize Ovarian Cancer Cells to Alkylating Agent Temozolomide

Martin L. Smith, M. Kelley, M. Fishel, 2007, Clinical Cancer Research.

Cells lacking CIP1/WAF1 genes exhibit preferential sensitivity to cisplatin and nitrogen mustard

P. O'Connor, A. Fornace, Martin L. Smith, 1997, Oncogene.

Mdm2 sensitizes MCF7 breast cancer cells to cisplatin or carboplatin

Martin L. Smith, 1999, Breast Cancer Research and Treatment.

Role of p21Waf1/Cip1/Sdi1 in cell death and DNA repair as studied using a tetracycline-inducible system in p53-deficient cells

A. Fornace, Martin L. Smith, M. Sheikh, 1997, Oncogene.

Chemotherapeutic Targeting of p53

Martin L. Smith, Albert J. Fornace, Jr., 2002, Cancer biology & therapy.

Chemotherapeutic selectivity conferred by selenium: a role for p53-dependent DNA repair

K. Pollok, Martin L. Smith, Joshua L. Fischer, 2007, Molecular Cancer Therapeutics.

Sensitivity of p53-Deficient Cells to Oxaliplatin and Thio-TEPA (N, N′, N″ Triethylenethiophosphoramide)

Y. Seo, Martin L. Smith, E. I. Chen, 2002, Breast Cancer Research and Treatment.

p53-Mediated DNA Repair Responses to UV Radiation: Studies of Mouse Cells Lacking p53, p21, and/orgadd45 Genes

P. Hanawalt, C. Deng, A. Fornace, 2000, Molecular and Cellular Biology.

The "Two faces" of Tumor Suppressor p53-revisited.

Martin L. Smith, M. S. Kumar, 2010, Molecular and cellular pharmacology.

Seleno-L-Methionine Modulation of Nucleotide Excision DNA Repair Relevant to Cancer Prevention and Chemotherapy.

Martin L. Smith, M. S. Kumar, 2009, Molecular and cellular pharmacology.

Selenomethionine induction of DNA repair response in human fibroblasts

Y. Seo, Martin L. Smith, C. Sweeney, 2002, Oncogene.

Lack of mutations of the p53 tumor suppressor gene in hepatocellular carcinomas induced in rats by a peroxisome proliferator

Martin L. Smith, H. Shinozuka, L. Yeleswarapu, 1993, Molecular carcinogenesis.

Response: Does the p53 Up-Regulated Gadd45 Protein Have a Role in Excision Repair?

A. Fornace, Martin L. Smith, I. Chen, 1995, Science.

Abnormalities in the p53 gene in tumors and cell lines of human squamous‐cell carcinomas of the head and neck

T. Whiteside, J. Locker, Martin L. Smith, 1993, International journal of cancer.

A Radiation-Induced Acute Apoptosis Involving TP53 and BAX Precedes the Delayed Apoptosis and Neoplastic Transformation of CGL1 Human Hybrid Cells

Martin L. Smith, D. Lewis, D. Spandau, 2005, Radiation research.

Association with Cdc2 and inhibition of Cdc2/Cyclin B1 kinase activity by the p53-regulated protein Gadd45

X. Wang, C. Harris, A. Fornace, 1999, Oncogene.

Selenomethionine regulation of p53 by a ref1-dependent redox mechanism

Y. Seo, Martin L. Smith, M. Kelley, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Tumorigenesis, protooncogene activation, and other gene abnormalities in methyl deficiency

Martin L. Smith, B. Lombardi, 1994 .

Regulation of the c-met Proto-oncogene Promoter by p53*

Martin L. Smith, D. Seol, Q. Chen, 1999, The Journal of Biological Chemistry.

Imbalancing the DNA base excision repair pathway in the mitochondria; targeting and overexpressing N-methylpurine DNA glycosylase in mitochondria leads to enhanced cell killing.

Y. Seo, Martin L. Smith, M. Kelley, 2003, Cancer research.

Nuclear Factor-κB Is Constitutively Activated in Prostate Cancer In vitro and Is Overexpressed in Prostatic Intraepithelial Neoplasia and Adenocarcinoma of the Prostate

Liang Cheng, Lang Li, Harikrishna Nakshatri, 2004, Clinical Cancer Research.

Selenomethionine or methylseleninic acid inhibits mutagenesis of a reporter gene in mouse bone marrow.

K. Pollok, Martin L. Smith, M. Kumar, 2010, Anticancer research.

Low frequency of retinoblastoma gene alterations in rat hepatocellular carcinomas

Martin L. Smith, N. Chandar, B. Lombardi, 1993, Molecular carcinogenesis.

Lack of Prognostic Importance of Reverse-Transcriptase Polymerase Chain Reaction Detection of Circulating Messenger RNA in Patients with Melanoma

J. J. Coleman, Martin L. Smith, J. Daggy, 2005, Plastic and reconstructive surgery.

Analysis oftheMouseDhfrlRep-3 MajorPromoter Regionby UsingLinker-Scanning andInternal Deletion Mutations andDNaseIFootprinting

P. Mitchell, Martin L. Smith, G. Crouse, 1990 .

Selenium compounds regulate p53 by common and distinctive mechanisms.

Martin L. Smith, J. Lancia, C. Ip, 2004, Anticancer research.

Enhanced Cell Killing-Methylpurine DNA Glycosylase in Mitochondria Leads to N Mitochondria ; Targeting and Overexpressing Imbalancing the DNA Base Excision Repair Pathway in the Updated Version

Y. Seo, Martin L. Smith, M. Kelley, 2003 .

Interaction of the p53-regulated protein Gadd45 with proliferating cell nuclear antigen.

P. O'Connor, A. Fornace, Q. Zhan, 1994, Science.

Antisense GADD45 expression results in decreased DNA repair and sensitizes cells to u.v.-irradiation or cisplatin.

P. O'Connor, A. Fornace, Q. Zhan, 1996, Oncogene.

Involvement of the p53 tumor suppressor in repair of u.v.-type DNA damage.

P. O'Connor, A. Fornace, Q. Zhan, 1995, Oncogene.