V. Davé

发表

Pathological unfoldomics of uncontrolled chaos: intrinsically disordered proteins and human diseases.

Vladimir N Uversky, Lilia M Iakoucheva, Ravi Ramesh Pathak, 2014, Chemical reviews.

PTEN proteoforms in biology and disease

V. Uversky, V. Davé, Prerna Malaney, 2017, Cellular and Molecular Life Sciences.

A Unified Nomenclature and Amino Acid Numbering for Human PTEN

P. Pandolfi, F. Furnari, M. Serrano, 2014, Science Signaling.

Galectin-1 inhibition attenuates profibrotic signaling in hypoxia-induced pulmonary fibrosis

V. Davé, Jaymin J. Kathiriya, Niyati Nakra, 2017, Cell Death Discovery.

Presence and utility of intrinsically disordered regions in kinases.

V. Uversky, B. Xue, V. Davé, 2014, Molecular bioSystems.

THE LUNG Gene Expression : FEED-FORWARD ( PTEN ) Induces Leptin-mediated Leptin Loss of Phosphatase and Tensin Homolog Gene Regulation :

V. Davé, Prerna Malaney, R. Pathak, 2013 .

Intrinsic Disorder in PTEN and its Interactome Confers Structural Plasticity and Functional Versatility

V. Uversky, B. Xue, V. Davé, 2013, Scientific Reports.

The activity of the Drosophila morphogenetic protein Bicoid is inhibited by a domain located outside its homeodomain.

David J. Forsthoefel, Jun Ma, S. Small, 2002, Development.

Signaling pathways in the epithelial origins of pulmonary fibrosis

J. Whitsett, V. Davé, J. Hagood, 2010, Cell cycle.

Integrating Omics Technologies to Study Pulmonary Physiology and Pathology at the Systems Level

V. Davé, R. Pathak, 2014, Cellular Physiology and Biochemistry.

Identification of intrinsically disordered regions in PTEN and delineation of its function via a network approach.

V. Uversky, V. Davé, Prerna Malaney, 2015, Methods.

Structural pliability adjacent to the kinase domain highlights contribution of FAK1 IDRs to cytoskeletal remodeling.

A. Bezginov, E. Tillier, V. Uversky, 2017, Biochimica et biophysica acta. Proteins and proteomics.

Data on evolution of intrinsically disordered regions of the human kinome and contribution of FAK1 IDRs to cytoskeletal remodeling

A. Bezginov, E. Tillier, V. Uversky, 2016, Data in brief.

The PTEN Long N-tail is intrinsically disordered: increased viability for PTEN therapy.

V. Uversky, V. Davé, Prerna Malaney, 2013, Molecular bioSystems.

Analyzing aggregation propensities of clinically relevant PTEN mutants: a new culprit in pathogenesis of cancer and other PTENopathies

V. Uversky, B. Xue, V. Davé, 2020, Journal of biomolecular structure & dynamics.

Multiple Sp1 Binding Sites in the Cardiac/Slow Twitch Muscle Sarcoplamsic Reticulum Ca-ATPase Gene Promoter Are Required for Expression in Sol8 Muscle Cells (*)

V. Davé, M. Periasamy, T. Reed, 1996, The Journal of Biological Chemistry.

PTEN Physically Interacts with and Regulates E2F1-mediated Transcription in Lung Cancer

V. Davé, Z. Tian, Diane S. Allen-Gipson, 2018, Cell cycle.

Target Selectivity of Bicoid Is Dependent on Nonconsensus Site Recognition and Protein-Protein Interaction

Chen Zhao, Jun Ma, V. Davé, 2000, Molecular and Cellular Biology.

Reprogrammable Recognition Codes in Bicoid Homeodomain-DNA Interaction

Chen Zhao, Jun Ma, C. Tung, 2000, Molecular and Cellular Biology.

Loss of Phosphatase and Tensin Homolog (PTEN) Induces Leptin-mediated Leptin Gene Expression

V. Davé, R. Pathak, Diane S. Allen-Gipson, 2013, The Journal of Biological Chemistry.

Conditional deletion of Pten causes bronchiolar hyperplasia.

A. R. Thitoff, J. Whitsett, V. Davé, 2008, American journal of respiratory cell and molecular biology.

Genome-wide responses to mitochondrial dysfunction.

Varshal K. Davé, H R Garner, J. Thornton, 2001, Molecular biology of the cell.

One mouse, one patient paradigm: New avatars of personalized cancer therapy.

S. Nicosia, V. Davé, P. Malaney, 2014, Cancer letters.

Transcriptional control of lung morphogenesis.

Jeffrey A Whitsett, J. Whitsett, V. Davé, 2007, Physiological reviews.

The solution structure of the native K50 Bicoid homeodomain bound to the consensus TAATCC DNA-binding site.

J. M. Baird-Titus, Jun Ma, V. Davé, 2006, Journal of molecular biology.

MEF2B Is a Component of a Smooth Muscle-specific Complex That Binds an A/T-rich Element Important for Smooth Muscle Myosin Heavy Chain Gene Expression*

E. Olson, V. Davé, J. Molkentin, 1998, The Journal of Biological Chemistry.

A Composite Motif of the Drosophila Morphogenetic Protein Bicoid Critical to Transcription Control*

Jun Ma, V. Davé, Chen Zhao, 2003, Journal of Biological Chemistry.

A systems approach to mapping transcriptional networks controlling surfactant homeostasis

Minlu Zhang, Pooja Kadambi, Jeffrey A Whitsett, 2010, BMC Genomics.

Calcineurin/Nfat signaling is required for perinatal lung maturation and function.

J. Neilson, G. Crabtree, J. Whitsett, 2006, The Journal of clinical investigation.

PARP-2 Interacts with TTF-1 and Regulates Expression of Surfactant Protein-B*

J. Whitsett, V. Davé, V. Schreiber, 2006, Journal of Biological Chemistry.

Nuclear Factor of Activated T Cells Regulates Transcription of the Surfactant Protein D Gene (Sftpd) via Direct Interaction with Thyroid Transcription Factor-1 in Lung Epithelial Cells*

Jeffrey A. Whitsett, J. Whitsett, V. Davé, 2004, Journal of Biological Chemistry.

Evolutionarily conserved promoter region containing CArG*-like elements is crucial for smooth muscle myosin heavy chain gene expression.

E. Olson, V. Davé, J. Miano, 1998, Circulation research.

Solution structure of the K50 class homeodomain PITX2 bound to DNA and implications for mutations that cause Rieger syndrome.

Beth A. Chaney, Jun Ma, Mark Rance, 2005, Biochemistry.

A novel E box/AT-rich element is required for muscle-specific expression of the sarcoplasmic reticulum Ca2+-ATPase (SERCA2) gene.

V. Davé, M. Periasamy, T. Reed, 1998, Nucleic acids research.

Effects of thyroid hormone on left ventricular performance and regulation of contractile and Ca(2+)-cycling proteins in the baboon. Implications for the force-frequency and relaxation-frequency relationships.

R. Walsh, B. Hoit, V. Davé, 1996, Circulation research.

Regulation of Sarcoplasmic Reticulum Gene Expression during Cardiac Hypertrophy and Heart Failure

R. Walsh, V. Davé, M. Arai, 1995 .

Pathological unfoldomics of uncontrolled chaos: intrinsically disordered proteins and human diseases.

L. Iakoucheva, V. Uversky, S. Metallo, 2014, Chemical reviews.