D. Pim

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Two Polymorphic Variants of Wild-Type p53 Differ Biochemically and Biologically

Miranda Thomas, A. Kalita, S. Labrecque, 1999, Molecular and Cellular Biology.

The function of the human papillomavirus oncogenes

Miranda Thomas, D. Pim, L. Banks, 2001 .

Detection of a common feature in several human tumor cell lines--a 53,000-dalton protein.

P. Goodfellow, E. Gurney, J. Taylor‐Papadimitriou, 1981, Proceedings of the National Academy of Sciences of the United States of America.

The role of the E6-p53 interaction in the molecular pathogenesis of HPV

Miranda Thomas, D. Pim, L. Banks, 1999, Oncogene.

The cellular protein p53 in human tumours.

D. Pim, L. Crawford, P. Lamb, 1984, Molecular biology & medicine.

Detection of antibodies against the cellular protein p53 in sera from patients with breast cancer

D. Pim, L. Crawford, R. Bulbrook, 1982, International journal of cancer.

Expression of human papillomavirus type 6 and type 16 capsid proteins in bacteria and their antigenic characterization.

D. Pim, L. Banks, G. Matlashewski, 1987, The Journal of general virology.

The expression of human papillomavirus type 18 E6 protein in bacteria and the production of anti-E6 antibodies.

D. Pim, L. Banks, G. Matlashewski, 1986, The Journal of general virology.

Primary structure polymorphism at amino acid residue 72 of human p53

D. Pim, G. Matlashewski, L. Crawford, 1987, Molecular and cellular biology.

Choosing the right path: membrane trafficking and infectious entry of small DNA tumor viruses.

D. Pim, L. Banks, Marina Bugnon Valdano, 2019, Current opinion in cell biology.

Identiﬁcation of Human Papillomavirus Type 58 Lineages and the Distribution Worldwide Variant Lineage

Alison, A. Moscicki, J. Palefsky, 2011 .

Regulation of the human papillomavirus oncoproteins by differential phosphorylation

D. Pim, L. Banks, P. Massimi, 2004, Molecular and Cellular Biochemistry.

Regulation of the human papillomavirus oncoproteins by differential phosphorylation.

D. Pim, L. Banks, P. Massimi, 2001 .

Interactions between E6AP and E6 proteins from alpha and beta HPV types.

Miranda Thomas, D. Pim, L. Banks, 2013, Virology.

Analysis of human p53 proteins and mRNA levels in normal and transformed cells.

D. Pim, L. Banks, G. Matlashewski, 1986, European journal of biochemistry.

p53 polymorphic variants at codon 72 exert different effects on cell cycle progression

D. Pim, L. Banks, 2004, International journal of cancer.

Human papillomavirus type 16 E7 binds to the conserved carboxy-terminal region of the TATA box binding protein and this contributes to E7 transforming activity.

D. Pim, L. Banks, P. Massimi, 1997, The Journal of general virology.

Human papillomavirus type 16 E 7 binds to the conserved carboxy-terminal region of the TATA box binding protein and this contributes to E 7 transforming activity

D. Pim, L. Banks, P. Massimi, 1997 .

A Novel PDZ Domain Interaction Mediates the Binding between Human Papillomavirus 16 L2 and Sorting Nexin 27 and Modulates Virion Trafficking

D. Pim, L. Banks, M. Playford, 2015, Journal of Virology.

Regulation of the Human Papillomavirus Type 18 E6/E6AP Ubiquitin Ligase Complex by the HECT Domain-Containing Protein EDD

Miranda Thomas, D. Pim, L. Banks, 2011, Journal of Virology.

The stability of the human papillomavirus E6 oncoprotein is E6AP dependent.

D. Pim, L. Banks, V. Tomaić, 2009, Virology.

Interaction between the HPV-16 E2 transcriptional activator and p53

D. Pim, L. Banks, V. Bouvard, 1999, Oncogene.

Complex of simian virus 40 large-T antigen and host 53,000-molecular-weight protein in monkey cells

D. Pim, E. Harlow, L. Crawford, 1981, Journal of virology.

HPV-16 E7 and adenovirus E1a complex formation with TATA box binding protein is enhanced by casein kinase II phosphorylation.

D. Pim, L. Banks, A. Storey, 1996, Oncogene.

HPV-16 E7 Interacts with the Endocytic Machinery via the AP2 Adaptor μ2 Subunit

Miranda Thomas, D. Pim, L. Banks, 2022, mBio.

An immunochemical investigation of SV40 T-antigens. 2. Quantitation of antigens and antibody activities.

D. Lane, D. Pim, L. Crawford, 1980, Virology.

The Human Papillomavirus (HPV) E6* Proteins from High-Risk, Mucosal HPVs Can Direct Degradation of Cellular Proteins in the Absence of Full-Length E6 Protein

D. Pim, L. Banks, V. Tomaić, 2009, Journal of Virology.

HPV‐18 E6*I modulates HPV‐18 full‐length E6 functions in a cell cycle dependent manner

E. Guccione, D. Pim, L. Banks, 2004, International journal of cancer.

Characterization of the complex between SV40 large T antigen and the 53K host protein in transformed mouse cells.

D. Lane, D. Pim, E. Harlow, 1980, Cold Spring Harbor symposia on quantitative biology.

Lack of immortalizing activity of a human papillomavirus type 16 variant DNA with a mutation in the E2 gene isolated from normal human cervical keratinocytes.

M. Stanley, T. Crook, D. Pim, 1992, Oncogene.

Monoclonal antibodies specific for simian virus 40 tumor antigens

D. Pim, E. Harlow, L. Crawford, 1981, Journal of virology.

Characterizing the spatio-temporal role of sorting nexin 17 in human papillomavirus trafficking.

D. Pim, L. Banks, Špela Peternel, 2017, The Journal of general virology.

HPV-16 E7 Interacts with the Endocytic Machinery via the AP2 Adaptor μ2 Subunit

Miranda Thomas, D. Pim, L. Banks, 2022, mBio.

HPV‐16 E2 contributes to induction of HPV‐16 late gene expression by inhibiting early polyadenylation

D. Pim, L. Banks, C. Johansson, 2012, The EMBO journal.

The Human Papillomavirus E6 PDZ Binding Motif: From Life Cycle to Malignancy

Miranda Thomas, D. Pim, L. Banks, 2015, Viruses.

Human papillomavirus type 16 E5 gene stimulates the transforming activity of the epidermal growth factor receptor.

D. Pim, L. Banks, M. Collins, 1992, Oncogene.

Comparison of the in vitro transforming activities of human papillomavirus types.

D. Pim, L. Banks, A. Storey, 1988, The EMBO journal.

Chimaeric HPV E6 proteins allow dissection of the proteolytic pathways regulating different E6 cellular target proteins

Miranda Thomas, D. Pim, L. Banks, 2002, Oncogene.

Human Papillomavirus Infection Requires the CCT Chaperonin Complex

D. Pim, L. Banks, D. Gardiol, 2021, Journal of Virology.

Human Papillomavirus 16 L2 Recruits both Retromer and Retriever Complexes during Retrograde Trafficking of the Viral Genome to the Cell Nucleus

D. Pim, L. Banks, P. Massimi, 2020, Journal of Virology.

Diverse Papillomavirus Types Induce Endosomal Tubulation

D. Pim, L. Banks, P. Massimi, 2019, Front. Cell. Infect. Microbiol..

Phosphorylation of Human Papillomavirus Type 16 L2 Contributes to Efficient Virus Infectious Entry

R. Garcea, D. Pim, L. Banks, 2019, Journal of Virology.

Human Papillomavirus 16 Infection Induces VAP-Dependent Endosomal Tubulation

D. Pim, L. Banks, P. Massimi, 2018, Journal of Virology.

The VPS4 component of the ESCRT machinery plays an essential role in HPV infectious entry and capsid disassembly

D. Pim, L. Banks, C. Crump, 2017, Scientific Reports.

Radioimmunoassay of the cellular protein p53 in mouse and human cell lines.

D. Pim, S. Benchimol, L. Crawford, 1982, The EMBO journal.

Retriever is a multiprotein complex for retromer-independent endosomal cargo recycling

R. Sessions, D. Pim, L. Banks, 2017, Nature Cell Biology.

HPV-18 E6*I protein modulates the E6-directed degradation of p53 by binding to full-length HPV-18 E6

D. Pim, L. Banks, 1999, Oncogene.

Human papillomavirus E6 protein interactions

Miranda Thomas, D. Pim, L. Banks, 2002 .

Human tumour viruses and the deregulation of cell polarity in cancer

Miranda Thomas, D. Pim, L. Banks, 2012, Nature Reviews Cancer.

The cell polarity regulator hScrib controls ERK activation through a KIM site-dependent interaction

Y. Taketani, Miranda Thomas, D. Pim, 2010, Oncogene.

Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A

D. Pim, L. Banks, S. Dilworth, 2005, Oncogene.

Interaction of viral oncoproteins with cellular target molecules: infection with high‐risk vs low‐risk human papillomaviruses

D. Pim, L. Banks, 2010, APMIS : acta pathologica, microbiologica, et immunologica Scandinavica.

Characterization of the human papillomavirus E2 protein: evidence of trans‐activation and trans‐repression in cervical keratinocytes.

D. Pim, L. Banks, V. Bouvard, 1994, The EMBO journal.

Allosteric Activation of Acid α-Glucosidase by the Human Papillomavirus E7 Protein*

W. Nickel, D. Pim, L. Banks, 2000, The Journal of Biological Chemistry.

Human papillomaviruses, cervical cancer and cell polarity

Miranda Thomas, D. Pim, L. Banks, 2008, Oncogene.

Human papillomaviruses and the specificity of PDZ domain targeting

Miranda Thomas, D. Pim, L. Banks, 2012, The FEBS journal.

The mechanisms and implications of hScrib regulation of ERK

Y. Taketani, D. Pim, L. Banks, 2010, Small GTPases.

Identification of human papillomavirus type 58 lineages and the distribution worldwide.

A. Moscicki, J. Palefsky, A. Ferrera, 2011, The Journal of infectious diseases.

Alternatively spliced HPV-18 E6* protein inhibits E6 mediated degradation of p53 and suppresses transformed cell growth

D. Pim, L. Banks, P. Massimi, 1997, Oncogene.

Isolation and characterization of a human p53 cDNA clone: expression of the human p53 gene.

D. Pim, G. Matlashewski, S. Benchimol, 1984, The EMBO journal.

Induction of apoptosis by p53 is independent of its oligomeric state and can be abolished by HPV-18 E6 through ubiquitin mediated degradation.

Miranda Thomas, D. Pim, L. Banks, 1996, Oncogene.

DNA damage induced p53 mediated transcription is inhibited by human papillomavirus type 18 E6.

S. Labrecque, D. Pim, L. Banks, 1994, Oncogene.

Interaction of HPV E6 oncoproteins with specific proteasomal subunits.

D. Pim, L. Banks, C. Blattner, 2013, Virology.

HPV E6 targeted degradation of the discs large protein: evidence for the involvement of a novel ubiquitin ligase

Miranda Thomas, D. Pim, L. Banks, 2000, Oncogene.

Detection of HPV-16 E2 Protein in Cervical Keratinocytes

D. Pim, L. Banks, V. Bouvard, 1994 .

Viruses and the 26S proteasome: hacking into destruction.

Miranda Thomas, D. Pim, L. Banks, 2003, Trends in biochemical sciences.

HPV E6 and MAGUK protein interactions: determination of the molecular basis for specific protein recognition and degradation

Miranda Thomas, D. Pim, L. Banks, 2001, Oncogene.

Geographical distribution and oncogenic risk association of human papillomavirus type 58 E6 and E7 sequence variations

A. Moscicki, J. Palefsky, A. Ferrera, 2013, International journal of cancer.

Oncogenic comparison of human papillomavirus type 58 E7 variants

Zigui Chen, Miranda Thomas, D. Pim, 2018, Journal of cellular and molecular medicine.

Alternative splicing of human p53 transcripts.

D. Pim, L. Banks, G. Matlashewski, 1987, Oncogene research.

Mutational analysis of HPV-18 E6 identifies domains required for p53 degradation in vitro, abolition of p53 transactivation in vivo and immortalisation of primary BMK cells.

Miranda Thomas, D. Pim, L. Banks, 1994, Oncogene.

Interaction of the Human Papillomavirus E6 Oncoprotein with Sorting Nexin 27 Modulates Endocytic Cargo Transport Pathways

D. Pim, L. Banks, J. Manzo-Merino, 2016, PLoS pathogens.

Retriever , a multiprotein complex for retromer-independent 1 endosomal cargo recycling 2

R. Sessions, D. Pim, L. Banks, 2017 .

Retinoblastoma-independent antiproliferative activity of novel intracellular antibodies against the E7 oncoprotein in HPV 16-positive cells

D. Pim, L. Banks, M. G. Paggi, 2011, BMC Cancer.

Expression of the human papillomavirus E7 oncogene during cell transformation is sufficient to induce susceptibility to lysis by activated macrophages.

K. Vousden, D. Pim, L. Banks, 1991, Journal of immunology.

Allosteric activation of acid alpha-glucosidase by the human papillomavirus E7 protein.

W. Nickel, D. Pim, L. Banks, 2000, Journal of Biological Chemistry.

Interactions between E6AP and E6 proteins from alpha and beta HPV types.

Miranda Thomas, D. Pim, L. Banks, 2013, Virology.