K. Dittmann

发表

The presence of wild-type TP53 is necessary for the radioprotective effect of the Bowman-Birk proteinase inhibitor in normal fibroblasts.

A. Begg, K. Dittmann, N. Gueven, 1998, Radiation research.

The radioprotective effect of BBI is associated with the activation of DNA repair-relevant genes.

K. Dittmann, N. Gueven, C. Mayer, 1998, International journal of radiation biology.

Radiation-induced Epidermal Growth Factor Receptor Nuclear Import Is Linked to Activation of DNA-dependent Protein Kinase*

David J. Chen, M. Schaller, L. Milas, 2005, Journal of Biological Chemistry.

Nuclear EGFR shuttling induced by ionizing radiation is regulated by phosphorylation at residue Thr654

M. Schaller, K. Dittmann, C. Mayer, 2010, FEBS letters.

Inhibition of radiation-induced EGFR nuclear import by C225 (Cetuximab) suppresses DNA-PK activity.

K. Dittmann, C. Mayer, H. Rodemann, 2005, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Radioresistance of K-Ras mutated human tumor cells is mediated through EGFR-dependent activation of PI3K-AKT pathway.

M. Baumann, K. Dittmann, H. Rodemann, 2005, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Nuclear EGFR renders cells radio-resistant by binding mRNA species and triggering a metabolic switch to increase lactate production.

M. Schaller, K. Dittmann, C. Mayer, 2015, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

The radioprotector O-phospho-tyrosine stimulates DNA-repair via epidermal growth factor receptor- and DNA-dependent kinase phosphorylation.

K. Dittmann, C. Mayer, H. Rodemann, 2007, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Radiation-induced survivin nuclear accumulation is linked to DNA damage repair.

K. Dittmann, C. Rödel, F. Rödel, 2010, International journal of radiation oncology, biology, physics.

Radiation-induced caveolin-1 associated EGFR internalization is linked with nuclear EGFR transport and activation of DNA-PK

K. Dittmann, C. Mayer, H. Rodemann, 2008, Molecular Cancer.

Leukemic cells (HL-60) produce a novel extracellular matrix-degrading proteinase that is not inhibited by tissue inhibitors of matrix metalloproteinases (TIMPs).

K. Dittmann, F. Lottspeich, P. Petrides, 1995, Experimental hematology.

EGFR-mediated stimulation of sodium/glucose cotransport promotes survival of irradiated human A549 lung adenocarcinoma cells.

K. Dittmann, C. Mayer, H. Rodemann, 2012, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Bowman-Birk protease inhibitor reduces the radiation-induced activation of the EGF receptor and induces tyrosine phosphatase activity.

K. Dittmann, N. Gueven, C. Mayer, 2009, International journal of radiation biology.

The radioprotector Bowman-Birk proteinase inhibitor stimulates DNA repair via epidermal growth factor receptor phosphorylation and nuclear transport.

K. Dittmann, C. Mayer, H. Rodemann, 2008, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Irradiated homozygous TGF- β 1 knockout fibroblasts show enhanced clonogenic survival as compared with TGF- β 1 wild-type fibroblasts

K. Dittmann, H. Rodemann, C. Herskind, 2002, International journal of radiation biology.

Exposure of primary osteoblasts to combined magnetic and electric fields induced spatiotemporal endochondral ossification characteristic gene- and protein expression profiles

K. Dittmann, C. Mayer, H. Rodemann, 2022, Journal of Experimental Orthopaedics.

Selective enrichment and biochemical characterization of seven human skin fibroblasts cell types in vitro.

K. Dittmann, H. Rodemann, P. I. Francz, 1989, Experimental cell research.

Reduced exosomal L-Plastin is responsible for radiation-induced bystander effect.

K. Dittmann, C. Mayer, H. Rodemann, 2019, Experimental cell research.

Selective Radioprotection of Normal Tissues by Bowman-Birk Proteinase Inhibitor (BBI) in Mice

L. Milas, K. Dittmann, H. Rodemann, 2005, Strahlentherapie und Onkologie.

How do normal and leukemic white blood cells egress from the bone marrow?

K. Dittmann, P. Petrides, 1990, Blut.

MK-886, a leukotriene biosynthesis inhibitor, induces antiproliferative effects and apoptosis in HL-60 cells.

K. Dittmann, C. Mayer, H. Rodemann, 1998, Leukemia research.

Bowman–Birk protease inhibitor activates DNA‐dependent protein kinase and reduces formation of radiation‐induced dicentric chromosomes

K. Dittmann, C. Mayer, H. Rodemann, 2003, International journal of radiation biology.

CHLAMYDIA INFECTION

A. L. Wong, J. Belmont, F. Lammert, 2001 .

EGFR-targeted anti-cancer drugs in radiotherapy: preclinical evaluation of mechanisms.

Mechthild Krause, Michael Baumann, Ulla Kasten-Pisula, 2007, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Human skin fibroblasts in vitro differentiate along a terminal cell lineage.

K. Dittmann, H. Rodemann, R. Hommel, 1988, Proceedings of the National Academy of Sciences of the United States of America.

The Bowman-Birk protease inhibitor enhances clonogenic cell survival of ionizing radiation-treated nucleotide excision repair-competent cells but not of xeroderma pigmentosum cells.

K. Dittmann, C. Mayer, H. Rodemann, 2000, International journal of radiation biology.

Bowman-Birk proteinase inhibitor (BBI) modulates radiosensitivity and radiation-induced differentiation of human fibroblasts in culture.

K. Dittmann, H. Rodemann, M. Bamberg, 1995, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

The radioprotective potential of the Bowman-Birk protease inhibitor is independent of its secondary structure.

K. Dittmann, N. Gueven, C. Mayer, 1998, Cancer letters.

Depletion of Akt1 and Akt2 Impairs the Repair of Radiation-Induced DNA Double Strand Breaks via Homologous Recombination

K. Dittmann, H. Rodemann, Tahereh Mohammadian Gol, 2019, International journal of molecular sciences.

Characterization of the amino acids essential for the photo- and radioprotective effects of a Bowman-Birk protease inhibitor-derived nonapeptide.

K. Dittmann, N. Gueven, C. Mayer, 2001, Protein engineering.

Molecular targeting in radiotherapy of lung cancer.

M. Krause, M. Baumann, D. Zips, 2004, Lung cancer.

O‐phospho‐L‐tyrosine protects TP53 wild‐type cells against ionizing radiation

K. Dittmann, C. Mayer, H. Rodemann, 2001, International journal of cancer.

Radiation-induced lipid peroxidation activates src kinase and triggers nuclear EGFR transport.

K. Dittmann, C. Mayer, R. Kehlbach, 2009, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Radiation-induced EGFR-signaling and control of DNA-damage repair

K. Dittmann, H. Rodemann, M. Toulany, 2007, International journal of radiation biology.

Low-dose total body irradiation facilitates antitumoral Th1 immune responses

K. Dittmann, B. Pichler, M. Röcken, 2021, Theranostics.

Ca2+-Activated IK K+ Channel Blockade Radiosensitizes Glioblastoma Cells

D. Zips, K. Dittmann, P. Ruth, 2015, Molecular Cancer Research.

Blockage of Epidermal Growth Factor Receptor-Phosphatidylinositol 3-Kinase-AKT Signaling Increases Radiosensitivity of K-RAS Mutated Human Tumor Cells In vitro by Affecting DNA Repair

Shaomeng Wang, M. Baumann, K. Dittmann, 2006, Clinical Cancer Research.

Blockage of Epidermal Growth Factor Receptor-Phosphatidylinositol 3-Kinase-AKT Signaling Increases Radiosensitivity of KRAS Mutated HumanTumor Cells In vitro byAffecting

M. Baumann, K. Dittmann, H. Rodemann, 2006 .

Inhibition of DNA repair as a mechanism of enhanced radioresponse of head and neck carcinoma cells by a selective cyclooxygenase-2 inhibitor, celecoxib.

K. Ang, L. Milas, K. Dittmann, 2005, International Journal of Radiation Oncology, Biology, Physics.

Ionizing radiation induces migration of glioblastoma cells by activating BK K(+) channels.

K. Dittmann, P. Ruth, S. Huber, 2011, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

A 41 kDa transferrin related molecule acts as an autocrine growth factor for HL-60 cells.

K. Dittmann, P. Petrides, 1991, Biochemical and biophysical research communications.

Radiosensitization of Ras-mutated human tumor cells in vitro by the specific EGF receptor antagonist BIBX1382BS.

M. Baumann, K. Dittmann, H. Rodemann, 2005, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Epidermal growth factor receptor as a target to improve treatment of lung cancer.

K. Ang, R. Komaki, Z. Liao, 2004, Clinical lung cancer.

The nuclear aryl hydocarbon receptor is involved in regulation of DNA repair and cell survival following treatment with ionizing radiation.

M. Schaller, K. Dittmann, C. Mayer, 2016, Toxicology letters.

Inhibition of cyclooxygenase-2 activity by celecoxib does not lead to radiosensitization of human prostate cancer cells in vitro.

K. Dittmann, P. Ohneseit, H. Rodemann, 2007, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Activation of protein kinase Cepsilon stimulates DNA-repair via epidermal growth factor receptor nuclear accumulation.

K. Dittmann, C. Mayer, H. Rodemann, 2008, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Celecoxib induced tumor cell radiosensitization by inhibiting radiation induced nuclear EGFR transport and DNA-repair: a COX-2 independent mechanism.

L. Milas, K. Dittmann, C. Mayer, 2008, International journal of radiation oncology, biology, physics.

Nuclear epidermal growth factor receptor modulates cellular radio-sensitivity by regulation of chromatin access.

M. Schaller, K. Dittmann, C. Mayer, 2011, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

PI3K-Akt signaling regulates basal, but MAP-kinase signaling regulates radiation-induced XRCC1 expression in human tumor cells in vitro.

M. Baumann, M. Schaller, K. Dittmann, 2008, DNA repair.

Glucose starvation impairs DNA repair in tumour cells selectively by blocking histone acetylation.

K. Dittmann, C. Mayer, H. Rodemann, 2017, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

HL-60 leukemia cells produce an autocatalytically truncated form of matrix metalloproteinase-9 with impaired sensitivity to inhibition by tissue inhibitors of metalloproteinases.

K. Dittmann, F. Lottspeich, P. Petrides, 1996, Leukemia.

Chronic Idiopathic Intestinal Pseudo-obstruction

A. L. Wong, J. Belmont, M. Dichgans, 2011 .

[Cutaneous amyloidoses].

A. L. Wong, J. Belmont, D. Roden, 1990, Der Hautarzt; Zeitschrift fur Dermatologie, Venerologie, und verwandte Gebiete.

The radioprotector ortho-phospho-L-tyrosine (pTyr) attenuates the side effects of fractionated irradiation in retinoblastoma mouse models but also decreases the local tumour control.

K. Dittmann, H. Rodemann, U. Schraermeyer, 2017, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Ca2+-Activated IK K+ Channel Blockade Radiosensitizes Glioblastoma Cells

D. Zips, K. Dittmann, P. Ruth, 2015, Molecular Cancer Research.