M. Tilli

发表

Functional mimicry of the acetylated C‐terminal tail of p53 by a SUMO‐1 acetylated domain, SAD

S. Dakshanamurthy, Ricardo E. Perez, C. Knights, 2010, Journal of cellular physiology.

Explant-cell culture of primary mammary tumors from MMTV-c-Myc transgenic mice

R. Dickson, M. Tilli, P. Furth, 2004, In Vitro Cellular & Developmental Biology - Animal.

Reflectance confocal microscopy for characterization of mammary ductal structures and development of neoplasia in genetically engineered mouse models of breast cancer.

M. Freedman, M. Tilli, P. Furth, 2005, Journal of biomedical optics.

Real-time imaging and characterization of human breast tissue by reflectance confocal microscopy.

Minetta C. Liu, E. Makariou, M. C. Cabrera, 2007, Journal of biomedical optics.

Deregulated estrogen receptor alpha expression in mammary epithelial cells of transgenic mice results in the development of ductal carcinoma in situ.

L. Chodosh, Baljit Singh, M. Tilli, 2005, Cancer research.

Loss of protein phosphatase 2A expression correlates with phosphorylation of DP-1 and reversal of dysplasia through differentiation in a conditional mouse model of cancer progression.

J. Renou, M. Tilli, P. Furth, 2003, Cancer research.

Overexpression of an N-terminally truncated isoform of the nuclear receptor coactivator amplified in breast cancer 1 leads to altered proliferation of mammary epithelial cells in transgenic mice.

M. Tilli, P. Furth, A. Riegel, 2005, Molecular endocrinology.

The nuclear receptor coactivator amplified in breast cancer-1 is required for Neu (ErbB2/HER2) activation, signaling, and mammary tumorigenesis in mice.

A. Wellstein, B. O’Malley, Jianming Xu, 2008, Cancer research.

Short Communication Introduction of Estrogen Receptor- (cid:1) into the tTA/TAg Conditional Mouse Model Precipitates the Development of Estrogen-Responsive Mammary Adenocarcinoma

M. Tilli, P. Furth, J. Flaws, 2003 .

Comparison of mouse mammary gland imaging techniques and applications: Reflectance confocal microscopy, GFP Imaging, and ultrasound

M. Tilli, P. Furth, Michael D. Johnson, 2008, BMC Cancer.

Activation of estrogen signaling pathways collaborates with loss of Brca1 to promote development of ERα-negative and ERα-positive mammary preneoplasia and cancer

E. Rosen, M. Tilli, P. Furth, 2008, Oncogene.

Introduction of estrogen receptor-alpha into the tTA/TAg conditional mouse model precipitates the development of estrogen-responsive mammary adenocarcinoma.

M. Tilli, P. Furth, J. Flaws, 2003, The American journal of pathology.

Conditional mouse models demonstrate oncogene-dependent differences in tumor maintenance and recurrence

M. Tilli, P. Furth, 2003, Breast Cancer Research.

Conditional Over-Expression of Estrogen Receptor Alpha in a Transgenic Mouse Model

M. Tilli, P. Furth, J. Flaws, 2002, Transgenic Research.

Chemoprevention of mammary carcinogenesis in a transgenic mouse model by α-difluoromethylornithine (DFMO) in the diet is associated with decreased cyclin D1 activity

M. Tilli, P. Furth, J. Flaws, 2003, Oncogene.

Functional mimicry of the acetylated C‐terminal tail of p53 by a SUMO‐1 acetylated domain, SAD

S. Dakshanamurthy, Ricardo E. Perez, C. Knights, 2010, Journal of cellular physiology.

The nuclear receptor coactivator amplified in breast cancer-1 is required for Neu (ErbB2/HER2) activation, signaling, and mammary tumorigenesis in mice.

A. Wellstein, B. O’Malley, Jianming Xu, 2008, Cancer research.