C. Scheidereit

发表

A nuclear poly(ADP-ribose)-dependent signalosome confers DNA damage-induced IkappaB kinase activation.

Claus Scheidereit, Anjali D. Zimmer, Michael Stilmann, 2009, Molecular cell.

NF-κB Function in Growth Control: Regulation of Cyclin D1 Expression and G0/G1-to-S-Phase Transition

Claus Scheidereit, A. Eichten, M. Hinz, 1999, Molecular and Cellular Biology.

It takes two to tango: IκBs, the multifunctional partners of NF‐κB

Claus Scheidereit, M. Hinz, Seda Çöl Arslan, 2012, Immunological reviews.

NF-κB transmits Eda A1/EdaR signalling to activate Shh and cyclin D1 expression, and controls post-initiation hair placode down growth

Desmond J. Tobin, Claus Scheidereit, Pascal Schneider, 2006, Development.

The IκB kinase complex in NF‐κB regulation and beyond

Claus Scheidereit, M. Hinz, C. Scheidereit, 2014, EMBO reports.

Luft constitutive apoptosis and abrogates LPS-delayed neutrophil apoptosis binding domain peptide accelerates

M. C. Cardoso, M. Wellner, F. Luft, 2003 .

A pervasive role of ubiquitin conjugation in activation and termination of IκB kinase pathways

C. Scheidereit, D. Krappmann, 2005, EMBO reports.

Opposing roles of NF-κB in anti-cancer treatment outcome unveiled by cross-species investigations.

G. Lenz, Soyoung Lee, B. Dörken, 2011, Genes & development.

cross-species investigations B in anti-cancer treatment outcome unveiled by κ Opposing roles of NF-Material Supplemental

G. Lenz, Soyoung Lee, B. Dörken, 2011 .

The IκB Kinase Complex and NF-κB Actas Master Regulators of Lipopolysaccharide-Induced Gene Expressionand Control Subordinate Activation ofAP-1

A. Thiel, C. Scheidereit, D. Krappmann, 2004, Molecular and Cellular Biology.

Activation of NF‐kappa B in vivo is regulated by multiple phosphorylations.

C. Scheidereit, M. Naumann, 1994, The EMBO journal.

Reoxygenation after severe hypoxia induces cardiomyocyte hypertrophy in vitro: activation of CREB downstream of GSK3β

R. Dietz, C. Scheidereit, M. Bergmann, 2004, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

In vitro susceptibility to TRAIL-induced apoptosis of acute leukemia cells in the context of TRAIL receptor gene expression and constitutive NF-κB activity

B. Dörken, C. Scheidereit, Z. Cai, 2001, Leukemia.

Mixed-Lineage Kinase 3 Delivers CD3/CD28-Derived Signals into the IκB Kinase Complex

T. Hofmann, C. Scheidereit, W. Dröge, 2000, Molecular and Cellular Biology.

Contacts between hormone receptor and DNA double helix within a glucocorticoid regulatory element of mouse mammary tumor virus.

M. Beato, C. Scheidereit, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Requirement of NF-kappaB/Rel for the development of hair follicles and other epidermal appendices.

W. Birchmeier, C. Scheidereit, T. Aebischer, 2001, Development.

The IkappaB kinase complex and NF-kappaB act as master regulators of lipopolysaccharide-induced gene expression and control subordinate activation of AP-1.

A. Thiel, C. Scheidereit, D. Krappmann, 2004, Molecular and cellular biology.

The I j B kinase complex in NF-j B regulation and beyond

M. Hinz, C. Scheidereit, Michael Hinz, 2013 .

IkappaB kinase complexes: gateways to NF-kappaB activation and transcription.

C. Scheidereit, 2006, Oncogene.

Elevated NF-kappaB p50 complex formation and Bcl-3 expression in classical Hodgkin, anaplastic large-cell, and other peripheral T-cell lymphomas.

H. Stein, B. Dörken, P. Lichter, 2005, Blood.

Interaction and functional interference of C / EBPβ with octamer factors in immunoglobulin gene transcription

S. Chen‐Kiang, C. Scheidereit, E. Hatada, 2000, European journal of immunology.

Requirement of Nuclear Factor-&kgr;B in Angiotensin II– and Isoproterenol-Induced Cardiac Hypertrophy In Vivo

R. Dietz, C. Scheidereit, M. Bergmann, 2005, Circulation.

Vascular Endothelial Cell–Specific NF-&kgr;B Suppression Attenuates Hypertension-Induced Renal Damage

R. Dietz, M. Wellner, F. Luft, 2007, Circulation research.

Signal Responsiveness of IκB Kinases Is Determined by Cdc37-assisted Transient Interaction with Hsp90*

M. Hinz, Seda Çöl Arslan, C. Scheidereit, 2007, Journal of Biological Chemistry.

Overexpression of I kappa B alpha without inhibition of NF-kappaB activity and mutations in the I kappa B alpha gene in Reed-Sternberg cells.

H. Stein, B. Dörken, M. Hummel, 1999, Blood.

Serum amyloid A1 mediates myotube atrophy via Toll‐like receptors

S. Felix, F. Luft, C. Scheidereit, 2019, Journal of cachexia, sarcopenia and muscle.

Serum amyloid A1 mediates myotube atrophy via Toll‐like receptors

Katrin, S. Felix, F. Luft, 2019, Journal of cachexia, sarcopenia and muscle.

RC 3 H 1 post-transcriptionally regulates A 20 mRNA and modulates the activity of the IKK / NFk B pathway

M. Landthaler, R. Backofen, J. Wolf, 2015 .

Aspirin inhibits NF‐κB and protects from angiotensin II‐induced organ damage

D. Ganten, R. Dietz, F. Luft, 2001 .

Protein-DNA interactions at steroid hormone regulated genes.

D. von der Ahe, M. Beato, C. Scheidereit, 1989, Endocrine research.

Mechanism of gene regulation by steroid hormones.

D. von der Ahe, M. Beato, C. Scheidereit, 1986, Journal of steroid biochemistry.

The NF-kappa B/Rel and I kappa B gene families: mediators of immune response and inflammation.

C. Scheidereit, D. Krappmann, F. Wulczyn, 1996, Journal of molecular medicine.

Human endogenous retrovirus HERV-K(HML-2) RNA causes neurodegeneration through Toll-like receptors.

V. Tarabykin, F. Heppner, Z. Izsvák, 2020, JCI insight.

Tubular Epithelial NF-κB Activity Regulates Ischemic AKI.

F. Luft, C. Scheidereit, I. Blasig, 2016, Journal of the American Society of Nephrology : JASN.

Regulation of NF-κB activity by IκBα and IκBβ Stability

C. Scheidereit, D. Krappmann, 1997 .

Signal-dependent degradation of IκBα is mediated by an inducible destruction box that can be transferred to NF-κB, Bcl-3 or p53

C. Scheidereit, D. Krappmann, F. Wulczyn, 1998 .

Alternative splicing variants of IkappaB beta establish differential NF-kappaB signal responsiveness in human cells.

D. Moore, C. Scheidereit, H. Tanaka, 1998, Molecular and cellular biology.

Maintenance of NF-kappa B activity is dependent on protein synthesis and the continuous presence of external stimuli

C. Scheidereit, H. Hohmann, A. M. V. Loon, 1991, Molecular and cellular biology.

Aspirin inhibits NF-kappaB and protects from angiotensin II-induced organ damage.

D. Ganten, R. Dietz, F. Luft, 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Inhibition of the NLRP3/IL‐1β axis protects against sepsis‐induced cardiomyopathy

F. Luft, C. Scheidereit, O. Otto, 2021, Journal of cachexia, sarcopenia and muscle.

Human Cells B Signal Responsiveness in κ Differential NF- Establish β B κ Alternative Splicing Variants of I

D. Moore, C. Scheidereit, I. Makino, 2013 .

NF-κ B p 105 is a target of I κ B kinases and controls signal induction of Bcl-3 – p 50 complexes

C. Scheidereit, D. Krappmann, F. Wulczyn, 2013 .

Striking Back at the Activator: How IκB Kinase Terminates Antigen Receptor Responses

M. Hinz, C. Scheidereit, 2007, Science's STKE.

Endothelial NF-κB Blockade Abrogates ANCA-Induced GN.

C. Scheidereit, J. Kamps, R. Kettritz, 2017, Journal of the American Society of Nephrology : JASN.

Tissue-specific expression of a splicing mutation in the IKBKAP gene causes familial dysautonomia.

C Maayan, J. Gusella, C. Robbins, 2001, American journal of human genetics.

Tubular epithelial NF-{ kappa } B activity regulates ischemic

F. Luft, C. Scheidereit, I. Blasig, 2017 .

Inhibition of NF- (cid:1) B essentially contributes to arsenic-induced apoptosis

M. Hinz, C. Scheidereit, K. Bommert, 2003 .

Elevated NF- (cid:1) B p50 complex formation and Bcl-3 expression in classical Hodgkin, anaplastic large-cell, and other peripheral T-cell lymphomas

H. Stein, P. Lichter, S. Joos, 2005 .

Cloning of the DNA-binding subunit of human nuclear factor kappa B: the level of its mRNA is strongly regulated by phorbol ester or tumor necrosis factor alpha.

C. Scheidereit, C. Bartsch, H. Hohmann, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Inhibition of NF-kappaB essentially contributes to arsenic-induced apoptosis.

M. Hinz, C. Scheidereit, K. Bommert, 2003, Blood.

Cloning of the DNA-binding subunit of human nuclear factor cB : The level of its mRNA is strongly regulated by phorbol ester or tumor necrosis factor a

C. Scheidereit, C. Bartsch, E. Hatada, 2022 .

Signal transduction: Docking IκB kinases

C. Scheidereit, 1998, Nature.

Autophagy limits inflammatory gene expression through targeting of nuclear p65/RelA by LC3 and p62 for lysosomal degradation

C. Scheidereit, E. Kaergel, Cristina Brischetto, 2022, bioRxiv.

Cutaneous Inflammation and Proliferation in vitro: Differential Effects and Mode of Action of Topical Glucocorticoids

C. Scheidereit, H. Korting, M. Bader, 2000, Skin Pharmacology and Physiology.

Functional interference of Sp1 and NF-kappaB through the same DNA binding site.

H. Handa, C. Scheidereit, H. Tanaka, 1998, Molecular and cellular biology.

Purified octamer binding transcription factors stimulate RNA polymerase III-mediated transcription of the 7SK RNA gene

A. Pierani, R. Roeder, C. Scheidereit, 1989, Cell.

Identification of novel ubiquitous and cell type-specific factors that specifically recognize immunoglobulin heavy chain and kappa light chain promoters.

C. Scheidereit, G. Scholz, S. Franke, 1994, The Journal of biological chemistry.

Requirement of Hsp90 activity for IkappaB kinase (IKK) biosynthesis and for constitutive and inducible IKK and NF-kappaB activation.

C. Scheidereit, D. Krappmann, Meike Broemer, 2004, Oncogene.

Regulation of NF-kappa B activity by I kappa B alpha and I kappa B beta stability.

C. Scheidereit, D. Krappmann, 1997, Immunobiology.

TSG 101 associates with PARP 1 and is essential for PARylation and DNA damage-induced NF- j B activation

A. Sporbert, C. Scheidereit, Marina Kolesnichenko, 2022 .

Tetrameric Oligomerization of IκB Kinase γ (IKKγ) Is Obligatory for IKK Complex Activity and NF-κB Activation

C. Scheidereit, J. Behlke, Sebastian Tegethoff, 2003, Molecular and Cellular Biology.

Aberrantly expressed c‐Jun and JunB are a hallmark of Hodgkin lymphoma cells, stimulate proliferation and synergize with NF‐κB

H. Stein, B. Dörken, M. Hinz, 2002, The EMBO journal.

The IκB kinase complex is a regulator of mRNA stability

G. Dittmar, P. Mertins, A. Akalin, 2018, The EMBO journal.

The role of oxygen metabolism for the pathological phenotype of Fanconi anemia

H. Klocker, C. Scheidereit, W. Ruppitsch, 1997, Human Genetics.

IκB kinase complexes: gateways to NF-κB activation and transcription

Claus Scheidereit, C. Scheidereit, 2006, Oncogene.

The glucocorticoid receptor binds to defined nucleotide sequences near the promoter of mouse mammary tumour virus

M. Beato, C. Scheidereit, S. Geisse, 1983, Nature.

A cytoplasmic ATM-TRAF6-cIAP1 module links nuclear DNA damage signaling to ubiquitin-mediated NF-κB activation.

G. Dittmar, K. Khanna, Michael Stilmann, 2010, Molecular cell.

RC3H1 post-transcriptionally regulates A20 mRNA and modulates the activity of the IKK/NF-κB pathway

Nils Blüthgen, Rolf Backofen, Caroline C. Friedel, 2015, Nature Communications.

Glucocorticoid receptors recognize DNA sequences in and around murine mammary tumour virus DNA.

B. Groner, M. Beato, N. Hynes, 1982, The EMBO journal.

Candidate proto-oncogene bcl-3 encodes a subunit-specific inhibitor of transcription factor NF-κB

C. Scheidereit, M. Naumann, F. Wulczyn, 1992, Nature.

LHX 2 is a direct NF-{ kappa } B target gene that promotes primary hair follicle placode down-growth

S. Millar, R. Paus, C. Scheidereit, 2017 .

Lhx2 is a direct NF-κB target gene that promotes primary hair follicle placode down-growth

S. Millar, R. Paus, C. Scheidereit, 2016, Development.

Transcription factor NF-κB is constitutively activated in acute lymphoblastic leukemia cells

C. Scheidereit, D. Krappmann, U. Kordes, 2000, Leukemia.

NF-κB determines Paneth versus goblet cell fate decision in the small intestine

C. Scheidereit, C. Klotz, Julian Heuberger, 2021, Development.

Hodgkin/Reed-Sternberg cells network, including CD40, CD86, and a set of antiapoptotic genes in B maintains high expression of a characteristic gene κ Constitutive NF-

B. Dörken, M. Hinz, C. Scheidereit, 2014 .

Malt 1 ubiquitination triggers NF-j B signaling upon T-cell activation

C. Scheidereit, J. Ruland, D. Krappmann, 2013 .

Malt1 ubiquitination triggers NF‐κB signaling upon T‐cell activation

Seda Çöl Arslan, C. Scheidereit, J. Ruland, 2007 .

Nuclear Factor κB–dependent Gene Expression Profiling of Hodgkin's Disease Tumor Cells, Pathogenetic Significance, and Link to Constitutive Signal Transducer and Activator of Transcription 5a Activity

Martin Zenke, Claus Scheidereit, H. Stein, 2002, The Journal of experimental medicine.

Nuclear Factor (cid:2) B–dependent Gene Expression Proﬁling of Hodgkin’s Disease Tumor Cells, Pathogenetic Signiﬁcance, and Link to Constitutive Signal Transducer and Activator of Transcription 5a Activity

H. Stein, B. Dörken, M. Zenke, 2002 .

Constitutive NF-kappaB maintains high expression of a characteristic gene network, including CD40, CD86, and a set of antiapoptotic genes in Hodgkin/Reed-Sternberg cells.

B. Dörken, M. Hinz, C. Scheidereit, 2001, Blood.

Constitutive NFk B maintains high expression of a characteristic gene network , including CD 40 , CD 86 , and a set of antiapoptotic genes in Hodgkin / Reed-Sternberg cells

B. Dörken, M. Hinz, C. Scheidereit, 2001 .

A Dual Role for Ikkα in Tooth Development

M. Karin, C. Scheidereit, P. Sharpe, 2004 .

The AP-1-BATF and -BATF3 module is essential for growth, survival and TH17/ILC3 skewing of anaplastic large cell lymphoma

G. Lenz, B. Dörken, P. Griffin, 2018, Leukemia.

B-Cell Receptor- and Phorbol Ester-Induced NF-κB and c-Jun N-Terminal Kinase Activation in B Cells Requires Novel Protein Kinase C's

C. Scheidereit, D. Krappmann, V. Heissmeyer, 2001, Molecular and Cellular Biology.

Shared Pathways of IκB Kinase-Induced SCFβTrCP-Mediated Ubiquitination and Degradation for the NF-κB Precursor p105 and IκBα

C. Scheidereit, D. Krappmann, V. Heissmeyer, 2001, Molecular and Cellular Biology.

Shared Pathways of I k B Kinase-Induced SCF b TrCP -Mediated Ubiquitination and Degradation for the NF- k B Precursor p105 and I k B a

C. Scheidereit, D. Krappmann, V. Heissmeyer, 2001 .

The IκB Kinase (IKK) Complex Is Tripartite and Contains IKKγ but Not IKAP as a Regular Component*

A. Klippel, C. Scheidereit, K. Giese, 2000, The Journal of Biological Chemistry.

NF-kappaB and the innate immune response.

C. Scheidereit, D. Krappmann, E N Hatada, 2000, Current opinion in immunology.

NF‐κB p105 is a target of IκB kinases and controls signal induction of Bcl‐3–p50 complexes

C. Scheidereit, D. Krappmann, F. Wulczyn, 1999 .

Molecular mechanisms of constitutive NF-κB/Rel activation in Hodgkin/Reed-Sternberg cells

B. Dörken, C. Scheidereit, D. Krappmann, 1999, Oncogene.

The NF‐kappa B precursor p105 and the proto‐oncogene product Bcl‐3 are I kappa B molecules and control nuclear translocation of NF‐kappa B.

C. Scheidereit, M. Naumann, F. Wulczyn, 1993, The EMBO journal.

A human lymphoid- specific transcription factor that activates immunoglobulin genes is a homoeobox protein

R. Roeder, C. Scheidereit, J. A. Cromlish, 1988, Nature.

A roadmap of constitutive NF-κB activity in Hodgkin lymphoma: Dominant roles of p50 and p52 revealed by genome-wide analyses

N. Hübner, Kivia A. P. de Oliveira, Jean-Fred Fontaine, 2016, Genome Medicine.

Identification and purification of a human lymphoid-specific octamer-binding protein (OTF-2) that activates transcription of an immunoglobulin promoter in vitro

A. Heguy, R. Roeder, C. Scheidereit, 1987, Cell.

Regulatory Elements in Steroid Hormone Inducible Genes: Structure and Evolution of DNA Sequences Recognized by Steroid Hormone Receptors

M. Beato, C. Scheidereit, G. Suske, 1985 .

Human endogenous retrovirus HERV-K(HML-2) RNA causes neurodegeneration through Toll-like receptors

V. Tarabykin, F. Heppner, Z. Izsvák, 2019, bioRxiv.

Constitutive nuclear factor-kappaB-RelA activation is required for proliferation and survival of Hodgkin's disease tumor cells.

B. Dörken, C. Scheidereit, E. Grinstein, 1997, The Journal of clinical investigation.

The NF-κB/Rel and IκB gene families: mediators of immune response and inflammation

C. Scheidereit, D. Krappmann, F. Wulczyn, 1996, Journal of Molecular Medicine.

Different mechanisms control signal‐induced degradation and basal turnover of the NF‐kappaB inhibitor IkappaB alpha in vivo.

C. Scheidereit, D. Krappmann, F. Wulczyn, 1996, The EMBO journal.

Reciprocal requirements for EDA/EDAR/NF-kappaB and Wnt/beta-catenin signaling pathways in hair follicle induction.

Boris Jerchow, Walter Birchmeier, Claus Scheidereit, 2009, Developmental cell.

Molecular model of the interaction between the glucocorticoid receptor and the regulatory elements of inducible genes.

M. Beato, C. Scheidereit, H. Westphal, 1986, DNA.

Functional Interference of Sp1 and NF-κB through the Same DNA Binding Site

H. Handa, C. Scheidereit, I. Makino, 1998, Molecular and Cellular Biology.

Purification and Characterization of the Human Elongator Complex*

H. Erdjument-Bromage, P. Tempst, C. Scheidereit, 2002, The Journal of Biological Chemistry.

Ubiquitous transcription factor OTF-1 mediates induction of the MMTV promoter through synergistic interaction with hormone receptors

M. Beato, C. Scheidereit, M. Kalff, 1991, Cell.

Requirement of Hsp90 activity for IκB kinase (IKK) biosynthesis and for constitutive and inducible IKK and NF-κB activation

C. Scheidereit, D. Krappmann, Meike Broemer, 2004, Oncogene.

IκB-ζ controls the constitutive NF-κB target gene network and survival of ABC DLBCL.

G. Lenz, B. Dörken, M. Hummel, 2013, Blood.

of ABC DLBCL B target gene network and survival κ controls the constitutive NF- ζ B- κ I

B. Dörken, M. Hummel, C. Scheidereit, 2013 .

The ankyrin repeat domains of the NF-kappa B precursor p105 and the protooncogene bcl-3 act as specific inhibitors of NF-kappa B DNA binding.

C. Scheidereit, M. Naumann, G. Nucifora, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Inhibition of NF- (cid:1) B essentially contributes to arsenic-induced apoptosis

B. Dörken, M. Hinz, C. Scheidereit, 2003 .

Transcriptional repression of NFKBIA triggers constitutive IKK‐ and proteasome‐independent p65/RelA activation in senescence

Soyoung Lee, C. Schmitt, A. Akalin, 2021, The EMBO journal.

A novel IKK- and proteasome-independent mechanism of RelA activation triggers senescence associated secretome via transcriptional repression of NFKBIA

Soyoung Lee, C. Schmitt, A. Akalin, 2019, bioRxiv.

The Bcl-3 oncoprotein acts as a bridging factor between NF-κB/Rel and nuclear co-regulators

C. Scheidereit, A. Leutz, S. Ansieau, 1999, Oncogene.

TSG101 associates with PARP1 and is essential for PARylation and DNA damage‐induced NF‐κB activation

A. Sporbert, C. Scheidereit, J. V. von Kries, 2021, bioRxiv.

Identification of a chemokine receptor profile characteristic for mediastinal large B‐cell lymphoma

H. Stein, B. Dörken, R. Hetzer, 2009, International journal of cancer.

Mapping of transcription factor motifs in active chromatin identifies IRF5 as key regulator in classical Hodgkin lymphoma

R. Siebert, G. Lenz, B. Dörken, 2014, Proceedings of the National Academy of Sciences.

Loss of PU.1 expression is associated with defective immunoglobulin transcription in Hodgkin and Reed-Sternberg cells of classical Hodgkin disease.

H. Stein, B. Dörken, C. Scheidereit, 2002, Blood.

A roadmap of constitutive NF-κB activity in Hodgkin lymphoma: Dominant roles of p50 and p52 revealed by genome-wide analyses

N. Hübner, Kivia A. P. de Oliveira, Jean-Fred Fontaine, 2016, Genome Medicine.

Chlamydia pneumoniae infection of vascular smooth muscle and endothelial cells activates NF-kappaB and induces tissue factor and PAI-1 expression: a potential link to accelerated arteriosclerosis.

R. Dietz, C. Scheidereit, A. Leutz, 1999, Circulation.

Lymphotoxin and lipopolysaccharide induce NF‐κB‐p52 generation by a co‐translational mechanism

B. Mordmüller, C. Scheidereit, D. Krappmann, 2003, EMBO reports.

Quantitative dissection and modeling of the NF-κB p100-p105 module reveals interdependent precursor proteolysis.

Falk Weih, Inbal Ipenberg, Claus Scheidereit, 2014, Cell reports.

Regulation of Vascular Smooth Muscle Cell Proliferation: Role of NF-&kgr;B Revisited

R. Dietz, C. Scheidereit, F. Mehrhof, 2005, Circulation research.

Elevated NF-κB p50 complex formation and Bcl-3 expression in classical Hodgkin, anaplastic large-cell, and other peripheral T-cell lymphomas

H. Stein, B. Dörken, P. Lichter, 2005 .

The Prevalence of TNFα-Induced Necrosis over Apoptosis Is Determined by TAK1-RIP1 Interplay

C. Scheidereit, Seda Çöl Arslan, 2011, PloS one.

Shared pathways of IkappaB kinase-induced SCF(betaTrCP)-mediated ubiquitination and degradation for the NF-kappaB precursor p105 and IkappaBalpha.

C. Scheidereit, D. Krappmann, V. Heissmeyer, 2001, Molecular and cellular biology.

Autocrine LTA signaling drives NF-κB and JAK-STAT activity and myeloid gene expression in Hodgkin lymphoma.

M. Selbach, N. Hübner, A. Akalin, 2019, Blood.

Inhibition of NF-kappaB by a TAT-NEMO-binding domain peptide accelerates constitutive apoptosis and abrogates LPS-delayed neutrophil apoptosis.

M. C. Cardoso, M. Wellner, F. Luft, 2003, Blood.

Neither the endogenous nor a functional steroid hormone receptor binding site transactivate the ribosomal RNA gene promoter in vitro

M. Beato, C. Scheidereit, A. Misseyanni, 1991, The Journal of Steroid Biochemistry and Molecular Biology.

Deficiency in IκBα in the intestinal epithelium leads to spontaneous inflammation and mediates apoptosis in the gut

J. Wolf, A. Kühl, C. Scheidereit, 2020, The Journal of pathology.

I{kappa}B-{zeta} controls the constitutive NF-{kappa}B target gene network and survival of ABC DLBCL

G. Lenz, M. Hummel, C. Scheidereit, 2013 .

NF-kappa B precursor p100 inhibits nuclear translocation and DNA binding of NF-kappa B/rel-factors.

C. Scheidereit, M. Naumann, A. Nieters, 1993, Oncogene.

High-level nuclear NF-kappa B and Oct-2 is a common feature of cultured Hodgkin/Reed-Sternberg cells.

B. Dörken, C. Scheidereit, D. Krappmann, 1996, Blood.

Identification and purification of a human immunoglobulin-enhancer-binding protein (NF-kappa B) that activates transcription from a human immunodeficiency virus type 1 promoter in vitro.

R. Roeder, C. Scheidereit, K. Kawakami, 1988, Proceedings of the National Academy of Sciences of the United States of America.

The effect of fever‐like temperatures on neutrophil signaling

M. Wellner, F. Luft, C. Scheidereit, 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Transcriptional regulation of the HIV-1 promoter by NF-kappa B in vitro.

R. Roeder, G. Li, M. Kretzschmar, 1992, Genes & development.

NF-κB pathway

C. Scheidereit, D. Krappmann, 2006 .

NF-kappaB p105 is a target of IkappaB kinases and controls signal induction of Bcl-3-p50 complexes.

C. Scheidereit, D. Krappmann, F. Wulczyn, 1999, The EMBO journal.

Common structural constituents confer I kappa B activity to NF‐kappa B p105 and I kappa B/MAD‐3.

C. Scheidereit, M. Naumann, E. Hatada, 1993, The EMBO journal.

Efficient transcription of an immunoglobulin kappa promoter requires specific sequence elements overlapping with and downstream of the transcriptional start site.

C. Scheidereit, G. Scholz, M. Pelletier, 1997, Nucleic acids research.

Alternative Splicing Variants of IκBβ Establish Differential NF-κB Signal Responsiveness in Human Cells

D. Moore, C. Scheidereit, I. Makino, 1998, Molecular and Cellular Biology.

Glucocorticoid and progesterone receptors bind to the same sites in two hormonally regulated promoters

M. Beato, C. Scheidereit, G. Schütz, 1985, Nature.

NF-κB Participates in Mouse Hair Cycle Control and Plays Distinct Roles in the Various Pelage Hair Follicle Types.

S. Millar, R. Paus, C. Scheidereit, 2017, The Journal of investigative dermatology.

High expression of RelA/p65 is associated with activation of nuclear factor-κB-dependent signaling in pancreatic cancer and marks a patient population with poor prognosis

W. Weichert, G. Kristiansen, C. Denkert, 2007, British Journal of Cancer.

Malt1 ubiquitination triggers NF-kappaB signaling upon T-cell activation.

Seda Çöl Arslan, C. Scheidereit, J. Ruland, 2007, The EMBO journal.

Tetrameric oligomerization of IkappaB kinase gamma (IKKgamma) is obligatory for IKK complex activity and NF-kappaB activation.

C. Scheidereit, J. Behlke, Sebastian Tegethoff, 2003, Molecular and cellular biology.

Signal-dependent degradation of IkappaBalpha is mediated by an inducible destruction box that can be transferred to NF-kappaB, bcl-3 or p53.

C. Scheidereit, D. Krappmann, F. Wulczyn, 1998, Nucleic Acids Research.

A computational model of the DNA damage-induced IKK/ NF-κB pathway reveals a critical dependence on irradiation dose and PARP-1

Dorothea Busse, J. Wolf, C. Scheidereit, 2023, bioRxiv.

Transcription factors: important tools and targets for molecular medicine

C. Scheidereit, 1996, Journal of Molecular Medicine.

CLK2 and CLK4 are regulators of DNA damage-induced NF-κB targeted by novel small molecule inhibitors.

P. Lindemann, C. Scheidereit, M. Nazaré, 2023, Cell chemical biology.

constitutive apoptosis and abrogates LPS-delayed neutrophil apoptosis binding domain peptide accelerates - B by a TAT-NEMO κ

M. C. Cardoso, M. Wellner, F. Luft, 2013 .

605 Nuclear Factor (cid:2) B–dependent Gene Expression Proﬁling of Hodgkin’s Disease Tumor Cells, Pathogenetic Signiﬁcance, and Link to Constitutive Signal Transducer and Activator of Transcription 5a

M. Zenke, M. Hinz, C. Scheidereit, 2002 .

A Quantitative Modular Modeling Approach Reveals the Effects of Different A20 Feedback Implementations for the NF-kB Signaling Dynamics

Janina Mothes, Inbal Ipenberg, Seda Çöl Arslan, 2020, Frontiers in Physiology.

A new type of transcriptional reprogramming by an IRF4 mutation in lymphoma

Salam A. Assi, U. Pannicke, K. Schwarz, 2022, bioRxiv.

disease transcription in Hodgkin and Reed-Sternberg cells of classical Hodgkin Loss of PU.1 expression is associated with defective immunoglobulin

B. Dörken, C. Scheidereit, T. Wirth, 2013 .

Inhibition of NF-κB essentially contributes to arsenic-induced apoptosis

M. Hinz, C. Scheidereit, K. Bommert, 2003 .

Identification of a chemokine receptor profile characteristic for mediastinal large B‐cell lymphoma

H. Stein, B. Dörken, R. Hetzer, 2009, International journal of cancer.

A dual role for Ikk alpha in tooth development.

M. Karin, C. Scheidereit, P. Sharpe, 2004, Developmental cell.