D. C. Cabelof

发表

Transcriptional profiling of the age-related response to genotoxic stress points to differential DNA damage response with age

K. Simon, A. Mukundan, S. Dewundara, 2009, Mechanisms of Ageing and Development.

Aging and base excision repair: in need of a comprehensive approach.

D. C. Cabelof, 2007, DNA repair.

Haploinsufficiency in mouse models of DNA repair deficiency: modifiers of penetrance

D. C. Cabelof, 2011, Cellular and Molecular Life Sciences.

Increased Mutational Response to Carcinogens Haploinsufficiency : Accelerated DNA Damage and β Base Excision Repair Deficiency Caused by Polymerase Updated

Samuel H. Wilson, D. C. Cabelof, A. Richardson, 2003 .

Rodent intestinal folate transporters (SLC46A1): secondary structure, functional properties, and response to dietary folate restriction.

M. Akabas, D. C. Cabelof, L. Matherly, 2007, American journal of physiology. Cell physiology.

D. C. Cabelof, L. Matherly, H. Van Remmen, 2006, The journals of gerontology. Series A, Biological sciences and medical sciences.

The Timing and Duration of Folate Restriction Differentially Impacts Colon Carcinogenesis

D. C. Cabelof, A. Unnikrishnan, Hongzhi Ma, 2021, Nutrients.

Folate deficiency regulates expression of DNA polymerase β in response to oxidative stress.

D. C. Cabelof, A. Unnikrishnan, T. Prychitko, 2011, Free radical biology & medicine.

Caloric restriction promotes genomic stability by induction of base excision repair and reversal of its age-related decline.

D. C. Cabelof, Sunitha Yanamadala, Zhongmao Guo, 2003, DNA repair.

A Screen for Suppressors of Gross Chromosomal Rearrangements Identifies a Conserved Role for PLP in Preventing DNA Lesions

D. Durocher, D. C. Cabelof, Frédéric D. Sweeney, 2007, PLoS genetics.

Apurinic/Apyrimidinic Endonuclease (APE/REF-1) Haploinsufficient Mice Display Tissue-specific Differences in DNA Polymerase β-Dependent Base Excision Repair*

E. Friedberg, L. Meira, D. C. Cabelof, 2004, Journal of Biological Chemistry.

Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice.

E. Friedberg, L. Meira, D. C. Cabelof, 2009, Free radical biology & medicine.

Structure and Regulation of the Murine Reduced Folate Carrier Gene

R. Mohammad, D. C. Cabelof, L. Matherly, 2005, Journal of Biological Chemistry.

Haploinsufficiency in mouse models of DNA repair deficiency: modifiers of penetrance

D. C. Cabelof, 2012, Cellular and Molecular Life Sciences.

Folate Deficiency Provides Protection against Colon Carcinogenesis in DNA Polymerase β Haploinsufficient Mice*

A. Dombkowski, D. C. Cabelof, A. Unnikrishnan, 2010, The Journal of Biological Chemistry.

A sensitive biochemical assay for the detection of uracil

D. C. Cabelof, Jun Nakamura, A. Heydari, 2006, Environmental and molecular mutagenesis.

Imbalanced Base Excision Repair in Response to Folate Deficiency Is Accelerated by Polymerase β Haploinsufficiency*

D. C. Cabelof, Jun Nakamura, A. Heydari, 2004, Journal of Biological Chemistry.

Preventing infection from foodborne pathogens in liver transplant patients.

D. C. Cabelof, 1994, Journal of the American Dietetic Association.

Succinylsulfathiazole modulates the mTOR signaling pathway in the liver of c57BL/6 mice via a folate independent mechanism

D. C. Cabelof, A. Unnikrishnan, Hongzhi Ma, 2021, Experimental Gerontology.

Down syndrome as a model of DNA polymerase beta haploinsufficiency and accelerated aging

D. C. Cabelof, D. Patterson, 2012, Mechanisms of Ageing and Development.

Mutational spectrum at GATA1 provides insights into mutagenesis and leukemogenesis in Down syndrome.

D. C. Cabelof, J. Taub, L. Matherly, 2009, Blood.

Attenuation of DNA polymerase beta-dependent base excision repair and increased DMS-induced mutagenicity in aged mice.

D. C. Cabelof, Sunitha Yanamadala, Zhongmao Guo, 2002, Mutation research.

Folate and Colorectal Cancer in Rodents: A Model of DNA Repair Deficiency

D. C. Cabelof, Hongzhi Ma, R. Rosati, 2012, Journal of oncology.

Base excision repair deficiency caused by polymerase beta haploinsufficiency: accelerated DNA damage and increased mutational response to carcinogens.

Samuel H. Wilson, D. C. Cabelof, A. Richardson, 2003, Cancer research.

Induction of DNA polymerase beta-dependent base excision repair in response to oxidative stress in vivo.

D. C. Cabelof, Sunitha Yanamadala, Zhongmao Guo, 2002, Carcinogenesis.

Excessive Folic Acid Mimics Folate Deficiency in Human Lymphocytes

D. C. Cabelof, A. Heydari, Ali M. Fardous, 2022, Current issues in molecular biology.

Haploinsufficiency in DNA polymerase beta increases cancer risk with age and alters mortality rate.

Samuel H. Wilson, D. C. Cabelof, R. Busuttil, 2006, Cancer research.

Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice.

E. Friedberg, L. Meira, D. C. Cabelof, 2009, Free radical biology & medicine.